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1 g adjunct to inhaled NO for the treatment of persistent pulmonary hypertension.
2 e decreases severe hypoxemia in infants with persistent pulmonary hypertension.
3 ate or severe right ventricular hypokinesis, persistent pulmonary hypertension, a patent foramen oval
4 oembolic pulmonary hypertension (CTEPH), but persistent pulmonary hypertension after PTE, as a result
5 mproves systemic oxygenation in infants with persistent pulmonary hypertension and may reduce the nee
6 ysis may identify CTEPH patients at risk for persistent pulmonary hypertension and poor outcome after
7 ed to the underlying disease associated with persistent pulmonary hypertension and that lung recruitm
8 borns with hypoxemic respiratory failure and persistent pulmonary hypertension, and is now a standard
9 ates with hypoxaemic respiratory failure and persistent pulmonary hypertension, but potential adverse
10 neonates transferred for rescue therapy for persistent pulmonary hypertension during the study perio
12 rginine would correlate with the presence of persistent pulmonary hypertension in newborns and that t
13 psis, septicemia, septic shock, endotoxemia, persistent pulmonary hypertension, nitric oxide, and ext
14 psis, septicemia, septic shock, endotoxemia, persistent pulmonary hypertension, nitric oxide, extraco
16 idepressant use during pregnancy and risk of persistent pulmonary hypertension of the newborn (PPHN)
21 nary vascular resistance after birth, and in persistent pulmonary hypertension of the newborn (PPHN),
22 e serotonin reuptake inhibitors (SSRIs) with persistent pulmonary hypertension of the newborn (PPHN),
23 nation and clinical outcomes in infants with persistent pulmonary hypertension of the newborn and ass
24 ness of inhaled NO treatment in infants with persistent pulmonary hypertension of the newborn and ass
27 ression may increase levels of superoxide in persistent pulmonary hypertension of the newborn lung ti
29 ation (ECMO) use or death among infants with persistent pulmonary hypertension of the newborn who rec
33 reliable in identifying patients at risk for persistent pulmonary hypertension or predicting postoper
34 ation fetal lambs, and in newborn lambs with persistent pulmonary hypertension (PPHN) after prenatal
35 led nitric oxide (iNO) in newborn lambs with persistent pulmonary hypertension (PPHN) following prena
38 full-term infants with severe hypoxemia and persistent pulmonary hypertension were randomly assigned
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