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1 thesized in the blade and transported to the petiole.
2 ical meristem, leaf primordium, and emerging petiole.
3 in the major venation, and 14% and 4% in the petiole.
4 mbium tissue present in roots, stem and leaf petiole.
5 AB and KNOX1 gene activity in the developing petiole.
6 s were only slightly increased in blades and petioles.
7 observed in all organs except hypocotyls and petioles.
8 , and the heads of trichomes on the stem and petioles.
9 green leaves, and short stems, pedicels, and petioles.
10 d in elongating seedlings and senescing leaf petioles.
11 1 causes more extensive maceration of celery petioles.
12  of roots and nodules and in the pulvinus of petioles.
13  confined to the guard cells, trichomes, and petioles.
14 nchyma, and in the exuding phloem sap of cut petioles.
15 id growth" was likewise reduced in xxt1/xxt2 petioles.
16 sal region of shoot organs, such as BLADE ON PETIOLE 2 and the GROWTH REGULATORY FACTOR pathway.
17 nd to a necrotic response along the stem and petioles accompanied by ROS production.
18 nsin mRNA accumulated are also seen: wounded petiole accumulating extensin message to a level higher
19 ins, shortened petioles, increased rachises, petioles acquiring motor organ characteristics, and ecto
20  wounded leaf is seen after 36 h, in wounded petioles after 11 h and in wounded stem after 17 h.
21 hylphthalamic acid (1% [w/w] in lanolin), to petioles also inhibited long-term leaf growth.
22 n it was inoculated directly onto cut tomato petioles, an inoculation method that did not require bac
23 polarized structure consisting of a proximal petiole and a distal blade, but the molecular mechanisms
24 arenchyma, and maceration and rotting of the petiole and central bud.
25 mutants, which have a constitutive elongated-petiole and early-flowering pheno-type, do not display a
26                                      In leaf petiole and flower pedicel zones this activity was enhan
27 sion at the lower (abaxial) side of the leaf petiole and involves the volatile phytohormone ethylene
28 erences explaining variation in the ratio of petiole and leaf length could be identified.
29 x vulnerability was strongly correlated with petiole and midrib conduit dimensions.
30 pressed in the root, expression in the leaf, petiole and stem being absent.
31 s typified by increased elongation growth of petioles and accelerated flowering and can be fully indu
32 n the absence of FB1, most notably elongated petioles and enhanced leaf margin serration.
33 movement of RNA originates in leaf veins and petioles and is induced by a short-day photoperiod, regu
34 ines had smaller rosettes because of shorter petioles and leaf blades and often acquired a twisted le
35 dration was driven by embolism initiating in petioles and midribs across all species, and Kx vulnerab
36 ypocotyls, more expanded cotyledons, shorter petioles and modestly higher levels of CAB gene expressi
37 ession of ELP1 resulted in dwarf plants with petioles and rachises reduced in length, and the epiderm
38 roponic medium through both Arabidopsis leaf petioles and roots, without apparent aggregation, and sh
39             Tissue-print immunoblots of rape petioles and stems showed that the rape ptGRP1 homologue
40 and in association with phloem cells in both petioles and stems.
41 so resists infection by H. parasitica in its petioles and this phenotype is complemented by transform
42 ipening fruit, abscission zones of senescent petioles and unfertilized flowers, and at wound sites.
43 d blue light, plants exhibited elongation of petioles and upward leaf reorientation, symptoms consist
44  investigate how embolisms spread throughout petioles and vein orders during leaf dehydration in rela
45                   Expression of SoGA20ox1 in petioles and young leaves was strongly up-regulated by a
46 pes in the hypocotyl, cotyledon, stem, leaf, petiole, and root.
47 imarily in young leaves, PPO2 in flowers and petioles, and PPO3 in leaves and possibly flowers.
48 rning the growth of cotyledons, true leaves, petioles, and primary and secondary roots and root hairs
49 lls and a predominantly aligned array in the petioles, and provide an excellent system for determinin
50 g metaxylem elements in young internodes and petioles, and stylar transmitting tissue cells.
51 x3 were higher in shoot tips than in blades, petioles, and young leaves.
52      Cucurbits exude profusely when stems or petioles are cut.
53  vascular tissues of a S. pinnata young leaf petiole as well as in guttation fluid.
54 sed elongation of the hypocotyl and the leaf petioles as well as with an acceleration of flowering ti
55 otyledon, lateral organ boundaries, blade-on-petiole, asymmetric leaves, and lateral organ fusion.
56 during which plants elongate their stems and petioles at the expense of leaf development.
57 es upward leaf movement (hyponasty) from the petiole base.
58 aptive advantage over local signaling in the petiole, because it optimizes the timing of leaf movemen
59  Here, we present evidence that the BLADE-ON-PETIOLE (BOP) genes, which have previously been shown to
60 er with homologs of the Arabidopsis BLADE-ON-PETIOLE (BOP) transcriptional cofactors, defined by the
61 odimers, mediate R sensitivity in leaves and petioles but not hypocotyls.
62 in tomato leaf abscission zones and adjacent petioles but not in ethylene-treated stem tissue or frui
63 increased lignin syringyl monomer content in petioles, but had no detectable effect on lignification
64 rinsic ET-SHYG-ACO5 activator loop for rapid petiole cell expansion upon waterlogging.
65  thaliana) wall, we compared the behavior of petiole cell walls from xxt1/xxt2 and wild-type plants u
66 mbrane of cotyledon epidermal, mesophyll and petiole cells during blade expansion.
67                                              Petiole cells from antisense plants were smaller than co
68                                              Petiole cells in the cotyledon epidermis exhibit well-al
69 fects on the nuclear distribution of phyB in petiole cells of light-grown plants.
70                 In contrast, in the adjacent petiole cells, SPR2 is constantly moving along the micro
71 cular bundles and scattered through stem and petiole cortex tissues [extrafascicular phloem (EFP)].
72 rgans, e.g. junctions between stems and leaf petioles, cotyledons and hypocotyls, roots and hypocotyl
73 sponse phenotypes including long and bending petioles, curly leaves, accelerated senescence, and cons
74           Our analyses show that the altered petiole development requires ectopic expression of ELONG
75 and FCL1 act additively and are required for petiole development.
76 ion, in adult plants both the leaves and the petioles display epinastic curvature and there is consti
77 ypes similar to those of axr1, namely, short petioles, downwardly curling leaves, short inflorescence
78 ientation defects, reduction of rosette leaf petioles, dramatically misshapen rosette leaves, one to
79                                              Petiole elongation also was inhibited by nuclear, but no
80 nsitivity, including increased hypocotyl and petiole elongation and increased numbers of lateral root
81 in leaf area, with reduced low R:FR-mediated petiole elongation and leaf hyponasty responses.
82 o be mediated by phyB, such as inhibition of petiole elongation and the shade avoidance response.
83 early-flowering pheno-type, do not display a petiole elongation growth response to EOD FR, but they d
84 ations at MAX2 cause increased hypocotyl and petiole elongation in light-grown seedlings.
85 ld-type or monogenic phyA or phyB seedlings, petiole elongation in phyA phyB seedlings is reduced in
86 A genes are essential for hypocotyl and leaf petiole elongation in response to low R:FR, in a fashion
87                            This reduction in petiole elongation is accompanied by the appearance of e
88 eals signaling bifurcation in the control of petiole elongation versus hyponasty.
89  light response, inhibits leaf expansion and petiole elongation, and attenuates the expression of CAB
90                                Internode and petiole elongation, and changes in overall leaf area and
91 t other phyB-controlled responses, including petiole elongation, are not sensitive to the same temper
92 that discrete pathways control flowering and petiole elongation, components of the shade-avoidance re
93 erexpression of HBI1 increased hypocotyl and petiole elongation, whereas dominant inactivation of HBI
94 ude increased elongation growth of stems and petioles, enabling plants to overtop competing vegetatio
95 s longitudinal cell expansion in the abaxial petiole epidermis to induce hyponasty and simultaneously
96 ht-grown mutant plants are dwarfs with short petioles, epinastic leaves, short inflorescence stems, a
97 duced inhibition of hypocotyl elongation and petiole epinasty are normal in Gr and Nr-2, suggesting t
98   sku6 roots, etiolated hypocotyls, and leaf petioles exhibit right-handed axial twisting, and root g
99                                              Petiole exudate experiments indicate that dir1-1 is defe
100 wever, compared with the phloem-sap enriched petiole exudate from the WT plant, mpl1 petiole exudate
101 ed accumulation of an antibiotic activity in petiole exudate of the Arabidopsis ssi2 mutant, which ex
102 ched petiole exudate from the WT plant, mpl1 petiole exudate was deficient in an activity that restri
103                     We demonstrate here that petiole exudates (PeXs) collected from Arabidopsis leave
104 e of inoculation, and, most specifically, in petiole exudates from inoculated leaves.
105 -derived oxylipins increased in roots and in petiole exudates of GPA-colonized plants.
106 ts accumulate reduced levels of G3P in their petiole exudates, suggest that the cooperative interacti
107 n the weakening abscission zones of the leaf petiole, flower and fruit pedicel, flower corolla, and f
108 , elf3 mutants have elongated hypocotyls and petioles, flower early, and have defects in the red ligh
109 ht regimes showed signs of impaired stem and petiole function which was not observed in wild-type tob
110  cellular and histological features of these petiole galls have been preserved in exquisite detail, i
111 scriptional core unit underlying directional petiole growth in Arabidopsis thaliana, governed by the
112            In V. vinifera, both the stem and petiole had similar sigmoidal vulnerability curves but d
113 s are imposed primarily by the leaves, whose petioles had unlignified, thin-walled xylem fibers with
114 G QDs moved faster than PEI QDs through leaf petioles; however, 8-fold more cadmium accumulated in PE
115 ncert with stomatal conductance and stem and petiole hydraulic measurements.
116    pPLAIIIbeta-KO plants have longer leaves, petioles, hypocotyls, primary roots, and root hairs than
117 nd 4HBA are synthesized de novo in stems and petioles in response to a mobile signal from the inocula
118 nd deep serration of leaf margins, shortened petioles, increased rachises, petioles acquiring motor o
119 d directly into the plant stem through a cut petiole, indicating that taxis makes its contribution to
120 of ectopic blade tissue along bop1 bop2 leaf petioles is strongly suppressed in a dosage-dependant ma
121                 These plants also had longer petioles, larger leaf area, increased specific leaf area
122             Sense plants had slightly longer petioles, larger leaf blades, and larger cells than cont
123 owed by auxin long-distance transport to the petiole leads to proliferation of J0121-marked xylem-ass
124   During axillary bud development in a model petiole-leaf cutting system, the levels of POTM1-1 trans
125 itro develop disorganized tumorous tissue in petioles, leaves and stems.
126 re characterized by elongated hypocotyls and petioles, leaves that are narrow and somewhat epinastic
127 is species, including roots, nodules, stems, petioles, leaves, flowers, pods and seeds.
128 for traits related to flowering time and for petiole length and successfully mapped QTL controlling e
129 In the Ws genetic background, an increase in petiole length, a reduction in cotyledon area and in ant
130 cts on hypocotyl elongation, leaf shape, and petiole length, as well as on gene expression.
131 ly, myr1 myr2 mutants exhibited increases in petiole length, leaf angle and apical dominance.
132 directed mutant have increased hypocotyl and petiole lengths, relative to wild-type BRI1-Flag (both i
133 at overexpress a closely related gene, LEAFY PETIOLE (LEP).
134 he leaf develops only the basal part of leaf petioles, main vascular tissues, and leaf veins (not bla
135 istance within the leaf is distributed among petiole, major veins, minor veins, and the pathways down
136 Y1) caused a shortened hypocotyl and shorter petioles, most dramatically under low-intensity red ligh
137 utant, was found to cosegregate with a short petiole mutant phenotype, and thus may serve as an examp
138 reduction in the import of auxin through the petioles of abcb19 cotyledons during the period of maxim
139               Cells associated with veins of petioles of C(3) tobacco possess high activities of the
140 va, a heat-girdling treatment was applied to petioles of cassava leaves at the end of the light cycle
141 ted from the leaves of Tabebuia argentea and petioles of Catalpa bignonioides.
142                                In young leaf petioles of clmp1, where clustering is prevalent, cells
143 urified indol-3-ylmethylglucosinolate to the petioles of cyp79B2 cyp79B3 mutant leaves, which do not
144 d in petioles of wild-type plants but not in petioles of dde2 plants, indicating that fungal compound
145 lalanine ammonia-lyase (PAL) activity in the petioles of inoculated leaves and in stems above inocula
146 uch as the style of elongating siliques, the petioles of maturing leaves, and most of the root.
147                                      Indeed, petioles of plants under -DIF had reduced ACC content, a
148 l phloem-associated cells in major veins and petioles of the inoculated leaf and stems below the inoc
149  DNA accumulated to almost similar levels in petioles of wild-type and coi1 plants at 10 d post infec
150  JA/ethylene defense pathway were induced in petioles of wild-type plants but not in petioles of dde2
151  roots and etiolated hypocotyls, whereas the petioles of WVD2-overexpressing rosette leaves exhibit l
152 cells in stems above the inoculated leaf and petioles or major veins of sink leaves.
153 sion PG mRNAs are expressed in fruit, stems, petioles, or anthers of fully open flowers.
154 T and COCH are Arabidopsis thaliana BLADE-ON-PETIOLE orthologs, and we have shown that their function
155 rounded, lobed leaves with shorter and wider petioles, overexpression of either RS2 or AS1 results in
156       We investigated the structure of their petioles, petiolules, leaflets, and tendrils through his
157 s were slightly pale green and had elongated petioles, phenotypes that are observed in mutants altere
158                   We identify both blade and petiole positioning as important components of leaf move
159 egulated processes in Arabidopsis, including petiole positioning, leaf expansion, stomatal opening an
160 CUC3::P1-GFP partially or fully complemented petiole positioning, leaf flattening and chloroplast acc
161 2 activity is specifically suppressed in the petiole region under -DIF conditions.
162 cell expansion in abaxial cells of the basal petiole region, while both responses are largely diminis
163 ion rate at the proximal abaxial side of the petiole relative to the adaxial side was implemented.
164 ongation at the proximal abaxial side of the petiole relative to the adaxial side).
165 c conductivity (-1.7 and -1 MPa for stem and petiole, respectively).
166 d ACC content, and application of ACC to the petiole restored leaf growth patterns.
167                                      Stem or petiole segments were tested for cavitation resistance b
168 lar and nonvascular tissues of mature celery petioles showed a strong anti-MTD sera cross-reactive ba
169 hus annuus stems, and Aesculus hippocastanum petioles) showed considerable reduction in cavitation re
170 ple, grape, corn, and tomato and leaf blade, petiole, stem, and pod tissues from soybean plants.
171 indicated that the protein occurs in leaves, petioles, stems, and cotyledons of seedlings but not in
172 9 promoter activity is robust in leaf veins, petioles, stems, and vascular tissues and induced by sho
173 9 promoter activity is robust in leaf veins, petioles, stems, and vascular tissues and induced by sho
174 ts in longer and narrower leaves with longer petioles than wild type.
175 s of C4 photosynthesis in cells of stems and petioles that surround the xylem and phloem, and that th
176                                       In the petiole, the initial flame-wound-evoked transient increa
177 growth inhibition and weakening of stems and petioles, the severity of which positively correlated wi
178 ted with the shoot apex and the base of leaf petioles throughout the vegetative phase.
179 d this enhanced resistance response protects petiole tissue alone.
180 pontaneous lesion formation, all confined to petiole tissue.
181  in the axillary bud, or in adjacent stem or petiole tissue.
182                                        Also, petiole treatment of Arabidopsis with 1-N-naphthylphthal
183 titutive luciferase activity specifically in petioles, was chosen for further analysis.
184  at the base of pedicels, and at the base of petioles where leaves attach to the stem.
185 he adaxial-abaxial polarity axis in the leaf petiole, where they regulate PHB and FIL expression and
186 ective of equilibrium leaf water potentials, petioles, whose vessels extend into the major veins, sho
187 e and in excised leaves supplied through cut petioles with peptides derived from the C terminus of ea
188 sayed by feeding leaves, via freshly excised petioles, with 1% (weight in volume, w/v) neutral red (N

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