ライフサイエンスコーパス: phagocytic

1 crete proinflammatory cytokines but are less phagocytic.

2 an brain slices shows DAM with intracellular/phagocytic Abeta particles.

3 ue and showed they had amoeboid movement and phagocytic abilities.

4 i.v.) 1 h after surgery increased neutrophil phagocytic ability and Fcgamma receptor I (CD64) express

5 4 directly augments CD64-mediated neutrophil phagocytic ability but does not directly increase neutro

6 ddition of LXA4 to CLP neutrophils increased phagocytic ability but not CD64 expression.

7 face expression of immunological markers and phagocytic ability were assessed by flow cytometry.

8 PE dysfunction, especially impairment of its phagocytic ability, plays an essential role in the patho

9 -forward manner that results in an increased phagocytic ability.

10 ar macrophages with low pro-inflammatory and phagocytic ability.

11 Small alveolar macrophages had the highest phagocytic ability.

12 IFN-gamma was a critical regulator of phagocytic/activating receptors on CNS APCs.

13 f these immune cells in the period following phagocytic activation, and provides a marker of neutroph

14 e that stimulated macrophages possess higher phagocytic activities and that classically activated (M1

15 antibody levels, and blood bactericidal and phagocytic activities.

16 20 targets (p = 0.026) and with Ab-dependent phagocytic activity (p = 0.010).

17 against scaffolded V1V2, antibody-dependent phagocytic activity against VLP-coated beads, and antibo

18 rophages were collected for determination of phagocytic activity and classically activated macrophage

19 egulatory protein alpha (SIRPalpha) inhibits phagocytic activity and protects red blood cells (RBCs)

20 ctionality was determined by analyzing their phagocytic activity and reactive oxygen species (ROS) ge

21 d neutrophils were found to exhibit a higher phagocytic activity as compared with the subsequently re

22 on lipid mediator, could increase neutrophil phagocytic activity as well as reduce bacterial virulenc

23 anoparticles with CD47 preferentially lowers phagocytic activity by the M1 phenotype.

24 ted by both bacterial species, they lack the phagocytic activity demonstrated by neutrophils.

25 ults suggested that astrocytes may engage in phagocytic activity during extended wake, but direct evi

26 urthermore, resident renal MPCs had impaired phagocytic activity in the absence of THP.

27 gentamicin-induced injury and had conserved phagocytic activity in zebrafish.

28 ity of myeloid cells is increased, and their phagocytic activity is promoted.

29 unication, leading to impaired migratory and phagocytic activity of CD11c cells.Collectively, our stu

30 MSCs to prepare native MSCs, so that (i) the phagocytic activity of cultured MSCs can be recovered an

31 peripheral lymphocyte apoptosis but promotes phagocytic activity of macrophages and neutrophils.

32 nm)- and resolvin D1 (0.01-10 nm)-stimulated phagocytic activity of macrophages.

33 ouse orthologs of many AD risk genes and the phagocytic activity of mouse microglial cells.

34 this issue, O'Connor et al. demonstrate that phagocytic activity of systemic immune cells is compromi

35 e chemerin, fMLF, and MCP-1; and doubled the phagocytic activity of these macrophages toward microbia

36 at phagocytose nanoparticles in vivo and how phagocytic activity relates to disease severity is not w

37 we investigated whether increased microglial phagocytic activity that clears amyloid can also cause p

38 Neutrophil phagocytic activity was decreased, and spontaneous oxida

39 an inflammatory response and a reduction in phagocytic activity, and connects RIPK1-mediated transcr

40 eep loss activates microglia, promotes their phagocytic activity, and does so in the absence of overt

41 nce, including low cytokine production, high phagocytic activity, and impaired Ag presentation.

42 Apoptotic indices, phagocytic activity, and phenotypic characterization of

43 idence that, in spite of an apparent lack of phagocytic activity, basophils can kill bacteria through

44 duction, phagocytic receptor expression, and phagocytic activity, enhancing parasite clearance by mac

45 e CD14(++) CD16(-) subset and exhibited high phagocytic activity, whereas RT monocytes originated fro

46 knockout reduced macrophage infiltration and phagocytic activity, which is consistent with TSP-1-enha

47 antly, they also displayed clear evidence of phagocytic activity.

48 ecretion of IL-10, and TGFbeta and increased phagocytic activity.

49 s a significant role in signaling late-stage phagocytic activity.

50 ng and these migratory cells displayed clear phagocytic activity.

51 autocrine mechanism to stimulate or maintain phagocytic activity.

52 ifest elevated M1 polarization with enhanced phagocytic activity.

53 o exhibited higher levels of active RhoA and phagocytic activity.

54 tokines with LPS stimulation and showed more phagocytic activity.

55 lting in killing of encapsulated bacteria by phagocytic activity.

56 lopment, phagocytic receptor expression, and phagocytic activity.

57 xposure to a MFGE8-blocking antibody reduces phagocytic activity.

58 ononuclear phagocyte number, plasticity, and phagocytic activity.

59 Altogether, both the phagocytic and immunomodulatory functions of Mer regulat

60 tem cell type that produces APOE, are highly phagocytic and participate in normal synapse pruning and

61 he recruitment and differentiation of highly phagocytic and stimulatory macrophages within tumours.

62 Six subsets of phagocytic APC (HLA-DR(+)) were consistently observed.

63 gen's response is mainly directed to prevent phagocytic attacks.

64 Conventional studies of dynamic phagocytic behavior have been limited in terms of spatia

65 iod, altering their morphology, motility and phagocytic behaviour as well as interactions with synaps

66 The phagocytic blockade resulted from reduced microglial sur

67 released were temporally correlated with OS phagocytic burst after light onset.

68 Marrow-derived macrophages are highly phagocytic, but whether they can also traffic into solid

69 ges showed significant reduction (46-72%) in phagocytic capabilities.

70 ly undergo phenotypic modulation and develop phagocytic capabilities.

71 ha, IL1-beta) and exhibit lower motility and phagocytic capacities.

72 Importantly, neutrophils exhibit enhanced phagocytic capacity after activation by conditioned medi

73 demonstrate a dramatic and lasting change in phagocytic capacity after serum exposure.

74 astrocytes are accompanied by alterations in phagocytic capacity and effects on neuronal calcium sign

75 ort a novel role for APOE in controlling the phagocytic capacity of astrocytes that is highly depende

76 f APOE4 may originate in part from defective phagocytic capacity of astrocytes which accelerates the

77 macrophage marker expression, and augmented phagocytic capacity of human MDMs stimulated with LPS or

78 ains of F. nucleatum significantly increased phagocytic capacity of neutrophils.

79 globulin levels and a modest decrease in the phagocytic capacity of pulmonary macrophage populations

80 y activated (M1) macrophages exhibit greater phagocytic capacity than alternatively activated (M2) ma

81 a non-contractile, migratory phenotype with phagocytic capacity that may act as a macrophage-like ce

82 This neutrophil phagocytic capacity was further enhanced after TI antibo

83 reductions in Deltapsim and ATP impeded the phagocytic capacity whereas ROS interfered with a later

84 These G-Neutrophils have high phagocytic capacity, high peroxide production, low myelo

85 The combination of neutrophil phagocytic capacity, plasma cfDNA levels, and IG count a

86 , increased cytokine production, and reduced phagocytic capacity.

87 ith MMP-9 or a CD36-blocking peptide reduced phagocytic capacity.

88 vealed impeded M2 polarization and decreased phagocytic capacity.

89 Phagocytic cargo, upon internalization, follows a define

90 We conclude that highly phagocytic, CD138(+) SPM-like cells with an anti-inflamm

91 at the ability of S. aureus strains to evade phagocytic cell killing and to survive temporarily withi

92 eased bacterial clearance, and iv) increased phagocytic cell killing of bacteria compared with tail t

93 ammatory cells to the peritoneum, or improve phagocytic cell killing of pathogens.

94 GS-9620 induced phagocytic cell maturation and improved effector-mediate

95 s replace CCR2(+) macrophages as the primary phagocytic cell.

96 is their rapid recognition and engulfment by phagocytic cells (a process called efferocytosis).

97 However, many phagocytic cells also act as antigen-presenting cells an

98 nsmission of pathogenic mycobacteria between phagocytic cells also depends on nonlytic ejection throu

99 with IL-6 that can bind Fcgamma receptors on phagocytic cells and are rapidly internalized.

100 important virulence factors that lyse human phagocytic cells and contribute to immune evasion.

101 Macrophages are myeloid-derived phagocytic cells and one of the first immune cell types

102 plicate in professional and non-professional phagocytic cells and subvert immune responses for chroni

103 Depletion of phagocytic cells by clodronate liposomes in wild-type mi

104 The phagocytic cells from MGL1(-/-) mice did not exhibit any

105 hese results highlight an unexpected role of phagocytic cells in processing T. gondii oocysts, in lin

106 Because microglia, which are phagocytic cells in the mammalian brain, are bone marrow

107 Professional phagocytic cells ingest microbial intruders by engulfing

108 Macrophages are specialized phagocytic cells involved in clearing invading pathogens

109 Clodronate-mediated depletion of phagocytic cells markedly prolonged the serum half-life

110 requirements for survival and replication in phagocytic cells of organisms from different kingdoms.

111 LRRK2 protein is highly expressed in phagocytic cells of the innate immune system, most notab

112 s, allowing them to survive and replicate in phagocytic cells of vertebrate hosts.

113 opsonophagocytic killing of staphylococci by phagocytic cells offers opportunities to establish such

114 totic tissue is recognized for engulfment by phagocytic cells such as macrophages.

115 ive production of reactive oxygen species in phagocytic cells that results in life-threatening infect

116 ental processes or injury must be cleared by phagocytic cells to maintain and repair tissues.

117 Thus, MSCs are novel phagocytic cells with a potential for immunotherapy in t

118 As the first-responder phagocytic cells, neutrophils are recruited in large num

119 icroglia (MG), a heterogeneous population of phagocytic cells, play important roles in central nervou

120 Macrophages are specialized phagocytic cells, present in all tissues, which engulf a

121 the capacity to concentrate drug delivery to phagocytic cells, significantly reducing off-target toxi

122 ogen, Francisella survives and replicates in phagocytic cells, such as macrophages.

123 , is involved in the handling of pristane by phagocytic cells, which is required to trigger disease i

124 o the malaria parasite from clearance by the phagocytic cells, which may be an immune escape mechanis

125 icity mediated by complement-fixing DSAs and phagocytic cells.

126 layer, particles of which can be ingested by phagocytic cells.

127 nd a specific viability deficit inside human phagocytic cells.

128 diabetic mice transfer secretory vesicles to phagocytic cells.

129 the interactions between A. pittii and human phagocytic cells.

130 everal cell types, including mitotic and non-phagocytic cells.

131 amentous actin and altered actin dynamics in phagocytic cells.

132 lammasome, Th17 signaling and recruitment of phagocytic cells.

133 by liver-, spleen-, and bone marrow-resident phagocytic cells.

134 ction of Yersinia outer proteins (Yops) into phagocytic cells.

135 e protein carriers, potential mechanisms for phagocytic chloride preservation and acquisition, intrac

136 a act as guardians of the brain by promoting phagocytic clearance and providing trophic support to en

137 We sought to test the significance of phagocytic clearance by resident and recruited phagocyte

138 ction produces elongated hyphae resistant to phagocytic clearance compelling alternative neutrophil e

139 her, these findings suggest that the myeloid phagocytic clearance of apoptotic cancer cells accelerat

140 Phagocytic clearance of apoptotic germ cells by Sertoli

141 rm cells by Sertoli cells using LAP.Although phagocytic clearance of apoptotic germ cells by Sertoli

142 and the discovery of cellular immunity, the phagocytic clearance of cellular debris has been conside

143 ses from glia, including glial migration and phagocytic clearance of damaged neurons.

144 We now know that the phagocytic clearance of dying cells (efferocytosis), par

145 The phagocytic clearance of dying cells in a tissue is a hig

146 ating parasite sequestration to host organs, phagocytic clearance of parasites, and regulation of imm

147 These changes were associated with increased phagocytic clearance of the platelets by macrophages.

148 ins to constitute a functional LC3-dependent phagocytic complex.

149 omly, they were displaced from the advancing phagocytic cup by an expanding diffusional barrier.

150 a delay in the transition to PI(3,4,5)P3 and phagocytic cup closure.

151 dominant negative mutant of EhRab35 reduced phagocytic cup formation and thereby reduced RBC interna

152 filopodia-guided actin anterograde flow with phagocytic cup formation, and (iii) the rapid growth of

153 Cdc42, had normal morphology, motility, and phagocytic cup formation, but displayed markedly reduced

154 Cdc42 is not critical for filopodia or phagocytic cup formation, but plays a key role in drivin

155 Cdc42-deficient macrophages exhibited rapid phagocytic cup kinetics, but reduced particle clearance,

156 the progression and ultimate closure of the phagocytic cup.

157 filamentous bacteria occurs through tubular phagocytic cups (tPCs) and takes many minutes to engulf

158 structures based on PIP3 patches, including phagocytic cups and basal waves.

159 ith RBCs EhRab35 is recruited to the site of phagocytic cups as well as to the nascent phagosomes tha

160 At phagocytic cups, Rab31 is first recruited during the pho

161 f fungal stress responses in countering host phagocytic defenses, we also report that C. albicans pho

162 cells, as well as rescue the cells from the phagocytic dysfunction induced by cholesterol crystals a

163 15 genes whose expression was upregulated in phagocytic E. histolytica trophozoites to determine whet

164 very of internalized yeasts, with comparable phagocytic efficacy.

165 orted to different strategies to boost their phagocytic efficiency and compensate for the increased n

166 and urge to routinely assess the microglial phagocytic efficiency in neurodegenerative disorders.

167 Akita/Ncf1 mice had normal degranulation and phagocytic efficiency when compared with wild-type mice.

168 for trafficking soluble proteins within the phagocytic/endocytic network to exosomes.

169 ial scavenger receptor Draper and downstream phagocytic engulfment machinery.

170 d a prorepair phenotype, and increase of the phagocytic engulfment of neutrophils by macrophages.

171 the human tumors, while TAMs were minimally phagocytic, even toward CD47-knockdown tumors.

172 tracellular replication rather than multiple phagocytic events or fusion of macrophages.

173 th LPS-induced signals to mediate macrophage phagocytic function and lung injury resolution.

174 rt that high dose atorvastatin increased the phagocytic function of ARPE-19 cells, as well as rescue

175 ity complex (MHC) class I in controlling the phagocytic function of macrophages.

176 e serious adverse effects, help preserve the phagocytic function of the RPE while also exhibiting ant

177 Phagocytic function requires cytoskeletal rearrangements

178 r macrophages with more pro-inflammatory and phagocytic function respectively, and large alveolar mac

179 more, that defective MCI macrophages recover phagocytic function via omega-3.

180 ucin domain markedly impaired KIM-1-mediated phagocytic function, resulting in increased proinflammat

181 l WD and reduced PI3K/Draper-dependent glial phagocytic function.

182 sults suggest a potential mechanism by which phagocytic glia contribute to both protein aggregate-rel

183 Drosophila model to investigate the role of phagocytic glia in clearance of neuronal mutant huntingt

184 previously that introduction of trpml(+) in phagocytic glia rescued the locomotor deficit by removin

185 acrophages quickly differentiated toward non-phagocytic, high-SIRPalpha TAMs.

186 ole for Fmr1 in regulating the activation of phagocytic immune cells both in the body and the brain.

187 , cell death, estrogen receptor pathways and phagocytic immune responses.

188 8CPT-2Me-cAMP reversed phagocytic impairment induced by Rho kinase inhibition b

189 In healthy neutrophils phagocytic impairment was induced experimentally by usin

190 sting that cAMP signals through PKA to drive phagocytic impairment.

191 pression in M-MPhi is correlated with higher phagocytic indices in post-ischemic brain immune cells.

192 Macrophages are phagocytic innate immune cells that are important mediat

193 sed survival in human whole blood and during phagocytic interaction with polymorphonuclear leukocytes

194 Here we show that glia-axon phagocytic interactions change dramatically in the aged

195 easing complement-mediated attack, improving phagocytic killing activity of neutrophils, and preventi

196 utrophils, but does not affect intracellular phagocytic killing by neutrophils.

197 on of C3b on their surface and in diminished phagocytic killing in a whole-blood assay.

198 AZM is more effective than AMX at enhancing phagocytic killing of A. actinomycetemcomitans by neutro

199 activity of AB307.30 but failed to increase phagocytic killing of capsule-positive strains.

200 Moreover, COMP inhibits phagocytic killing of M. catarrhalis by human neutrophil

201 Furthermore, PRELP enhances phagocytic killing of serum-opsonized M. catarrhalis by

202 ed with R-specific antibodies, which trigger phagocytic killing of staphylococci and protect mice aga

203 eumococcal nasopharyngeal carriage, reducing phagocytic killing, and resulting in increased inflammat

204 ich is recruited to the GAS surface to block phagocytic killing, interacts with a remarkably large nu

205 ophagosomes, the bacteria are protected from phagocytic killing, thus providing an intracellular surv

206 d phenotype, and corresponding resistance to phagocytic killing.

207 AMP, reduce antibiotic efficacy and enhance phagocytic killing.

208 , rendering this species still vulnerable to phagocytic killing.

209 cape of the microorganism from SP-A-mediated phagocytic killing.

210 recognize the microbial surface and promote phagocytic killing.

211 Binding of SP-A to OprH promoted phagocytic killing; thus, late CF isolates were at least

212 Thanks to phagocytic leucocytes and other host defenses, the vast

213 Additionally, phagocytic leukocytes are involved in fibrinolysis and t

214 surrounded by the PV membrane, suggesting a phagocytic-like process for vesicle engulfment.

215 The phagocytic machinery of the follicle cells, including Dr

216 e these focal myelin dystrophies using known phagocytic machinery, including the opsonin milk fat glo

217 MSCs promote an antiinflammatory and highly phagocytic macrophage phenotype through EV-mediated mito

218 d-derived immune cell populations, including phagocytic macrophages and microglia.

219 Phagocytic macrophages expressed CD206, displayed blunte

220 hemotin mutants accumulate undigested phagocytic material inside enlarged endo-lysosomes and a

221 silico 3D modeling revealed activated Abeta phagocytic microglia in APP/PS1(+)Il10(-/-) mice that re

222 impaired spatial learning and persistence of phagocytic microglia without loss of hippocampal neurons

223 omplement receptor CR3 reduces the number of phagocytic microglia, as well as the extent of early syn

224 glial scar-like formation and recruitment of phagocytic microglia.

225 olar epithelia to switch from secretion to a phagocytic mode and rapidly remove dying neighbors.

226 ed predominantly of a novel subset of highly phagocytic MPhi resembling small peritoneal MPhi (SPM) t

227 e acetylcholinesterase(+) megakaryocytes and phagocytic myeloid cells in vitro and can also engraft i

228 e (PI3K), which is required to stimulate the phagocytic NADPH-oxidase that generates reactive oxygen

229 en, thus promoting their eradication by live phagocytic neighbors while within the epithelium.

230 Phagocytic neutrophils generate reactive oxygen species

231 ione adducts were detected in the cytosol of phagocytic neutrophils.

232 Defects in phagocytic nicotinamide adenine dinucleotide phosphate o

233 Contravening the canonical phagocytic pathway, tPCs mature by fusing with endosomes

234 s of the intimately connected autophagic and phagocytic pathways that are responsible for degradation

235 hancers of genes in the lipid metabolism and phagocytic pathways.

236 tor and its ligand MFG-E8, thus generating a phagocytic peak.

237 M PD-1 expression correlates negatively with phagocytic potency against tumour cells, and blockade of

238 IIA expression by alveolar macrophages after phagocytic process via NOD2-NF-kappaB-dependent mechanis

239 gated by shed POS at different stages of the phagocytic process.

240 whether these genes actually function in the phagocytic process.

241 brates, showing that this novel regulator of phagocytic processing is widely conserved, emphasizing t

242 c responses involve the coordination of both phagocytic programs and the "arming" of cytotoxic cells.

243 orphological alterations and increased their phagocytic rate, without affecting matrix metalloproteas

244 adenine dinucleotide phosphate/NADPH levels, phagocytic reactive oxygen species production, neutrophi

245 n model, we noted that the expression of the phagocytic receptor Bai1 was progressively downmodulated

246 Here we report that the phagocytic receptor CED-1 mEGF10 is required for the for

247 his study, we report that the absence of the phagocytic receptor Draper in glia leads to a pronounced

248 quently, reduced expression of the conserved phagocytic receptor Draper/MEGF10.

249 regulated B1 cell expansion, IgM production, phagocytic receptor expression, and phagocytic activity,

250 s and natural killer cells, Th1 development, phagocytic receptor expression, and phagocytic activity.

251 ssed, or excess neurons and synapses via the phagocytic receptor Mer tyrosine kinase (MerTK).

252 The tyrosine kinase phagocytic receptor MerTK was one of the most upregulate

253 However, the pro-phagocytic receptor(s) responsible for tumour cell phago

254 ney injury molecule 1 (KIM-1), an epithelial phagocytic receptor, is markedly upregulated in the prox

255 s and neutrophils expressed higher levels of phagocytic receptors and showed enhanced phagocytosis of

256 ly, these experiments establish that not all phagocytic receptors are functionally equal, and that co

257 ro and rapidly induced the expression of the phagocytic receptors Axl and MerTK.

258 Blocking phagocytosis or specific phagocytic receptors may alleviate synapse loss and axon

259 ever, the contribution of precursor monocyte phagocytic receptors, which are the first to interact wi

260 partments associated with both endocytic and phagocytic recycling functions, confirming evolutionary

261 agocytic regulators, HIF-2alpha can act as a phagocytic repressor.

262 to be a slow process mediated exclusively by phagocytic resident macrophages, Kupffer cells (KC).

263 In addition, the microglial phagocytic response and elevation of Trem2, both necessa

264 ity in aged glia significantly reverses slow phagocytic responses.

265 of the Brucella-containing vacuole from the phagocytic route in professional phagocytes.

266 cell surface protein that transmits an anti-phagocytic signal, known as the "don't-eat-me" signal, t

267 on with gadolinium, fluorescein and "eat-me" phagocytic signals (Gd-FITC-LiLa) a) demonstrates high r

268 Collectively, our results suggest that phagocytic signals can preferentially target inflammator

269 pha receptor which typically transduces anti-phagocytic signals from the 'don't eat me' CD47 ligand t

270 rid lipid-latex (LiLa) nanoparticles bearing phagocytic signals.

271 ytosis assays to quantitatively characterize phagocytic spreading dynamics.

272 Our investigation reveals that frustrated phagocytic spreading occurs in phases and is punctuated

273 To get insight into the role of phagocytic superoxide in the onset of diabetic complicat

274 se rewire the CD47-SIRPalpha axis to the pro-phagocytic Syk kinase activation.

275 which locally overwhelms self-signaling at a phagocytic synapse.

276 specific cell corpse/phagocyte interactions (phagocytic synapses) that impinge on host immunity, with

277 lternative approach to evade the mononuclear phagocytic system and facilitate transport across the en

278 hils are polymorphonuclear leukocytes of the phagocytic system that act as first line of host defense

279 the microvasculature and hepatic mononuclear phagocytic system.

280 Physical properties of phagocytic targets thus regulate self signaling, as is r

281 helia (enterocytes) are considered to be non-phagocytic towards bacteria with invasive pathogenic str

282 HIV-1 were unable to respond efficiently to phagocytic triggers and to clear bacteria.

283 o these key pathways that underpin the daily phagocytic turnover of photoreceptor outer segments (POS

284 The role of Golgi apparatus during phagocytic uptake by macrophages has been ruled out in t

285 BLCLs, thereby enhancing their Fc-dependent phagocytic uptake by macrophages.

286 microbial pathogens and dead cells and their phagocytic uptake by specialized immune cells are essent

287 rface of apoptotic cells, and promotes their phagocytic uptake by TIM1-expressing cells.

288 ication of bacterial peptidoglycans restored phagocytic uptake in the lysosomal degradation-defective

289 Filopodia supported the phagocytic uptake of bacterial (Escherichia coli) partic

290 ent inhibited mannosidase-II recruitment and phagocytic uptake of serum-coated or -uncoated latex bea

291 outer surface and a significant increase in phagocytic uptake of the NPs; ii) nanoparticle-containin

292 phage-mediated bacterial clearance relies on phagocytic uptake of the pathogen, subsequent phagolysos

293 ecruitment of mannosidase-II vesicles during phagocytic uptake required Ca(2+) from both extra- and i

294 role of Golgi-derived secretory vesicles in phagocytic uptake, the key innate defense function.

295 t lysosomal degradation of bacteria sustains phagocytic uptake.

296 pha, HIF-2alpha was not required for hypoxic phagocytic uptake.

297 uce permeabilization of Escherichia coli and phagocytic uptake.

298 ygen species suppressed Marco expression and phagocytic uptake.

299 asm, enabling Salmonella to replicate in the phagocytic vacuole.

300 Endocytic, autophagic, and phagocytic vesicles move on microtubule tracks to fuse w