ライフサイエンスコーパス: phalloidin

1 C (calphostin C), and actin destabilization (phalloidin).

2 ), microglia (CD11b), and filamentous actin (phalloidin).

3 sing extracellular applications of rhodamine-phalloidin.

4 cofilin-induced partial release of rhodamine phalloidin.

5 bbit psoas fiber were labeled with rhodamine-phalloidin.

6 x at branching sites that were stabilized by phalloidin.

7 polymerization in the presence of MgCl2 and phalloidin.

8 ilamentous actin with fluorescein-conjugated phalloidin.

9 on was studied after staining with rhodamine phalloidin.

10 toskeletal actin when stained with rhodamine-phalloidin.

11 s jasplakinolide and 4-fold more active than phalloidin.

12 observe polymerization in real time, without phalloidin.

13 probing new actin assembly with fluorescent phalloidin.

14 yofibrillar proteins, and FITC- or rhodamine phalloidin.

15 hate (PtdIns(3,4,5)P3) was also prevented by phalloidin.

16 nce microscopy after labeling with rhodamine-phalloidin.

17 of changes in actin bundles were blocked by phalloidin.

18 chalasin B with or without pretreatment with phalloidin.

19 alogues, KCl concentration, and the level of phalloidin.

20 tin in the CL was labeled with conjugates of phalloidin.

21 mentous actin organization was visualized by phalloidin.

22 sensitized emission by tetramethyl-rhodamine phalloidin.

23 by calcineurin inhibitors, staurosporine, or phalloidin.

24 cytochalasin D, and by the inability to bind phalloidin.

25 xtension when stained with rhodamine-labeled phalloidin.

26 ected by the microtubule polymerizing agent, phalloidin.

27 ared thicker after staining with fluorescent phalloidin.

28 ere visualized with AlexaFluor488-conjugated phalloidin.

29 determined using Alexa Fluor 488-conjugated phalloidin.

30 F-actin cytoskeleton was visualized with phalloidin.

31 half-sarcomere was labeled with fluorescent phalloidin.

32 this mutant was sensitive to nocodazole and phalloidin.

33 Actin filaments were detected with phalloidin.

34 winding in both the presence and absence of phalloidin.

35 on was visualized by staining with Texas-red phalloidin.

36 n was quantified using fluorescently labeled phalloidin.

37 alpha on the actin cytoskeleton by rhodamine-phalloidin.

38 ls treated with the microfilament stabilizer phalloidin.

39 t a solid-phase synthetic approach to [Ala7]-phalloidin (1).

40 dialysis with the actin filament stabiliser phalloidin (10 microM) prevented KATP channel activation

41 Microsomes prepared in the presence of phalloidin (a toxin that binds to F actin and stabilizes

42 peptide (pHLIP)-facilitated translocation of phalloidin, a cell-impermeable polar toxin, inhibits the

43 he intensity of staining with Alexa-Fluor488-phalloidin, a compound that permits visualization and qu

44 Staining with BODIPY phalloidin, a fluorescent dye selective for F-actin, sho

45 protein that binds and severs filaments, and phalloidin, a fungal toxin that binds and stabilizes F-a

46 Phalloidin, a fungal toxin which stabilizes F-actin and

47 oduce the cell-impermeable bi-cyclic peptide phalloidin, a specific marker for actin filaments, into

48 nt acid phosphatase (TRAP), Oregon Green 488-phalloidin, a stain for cytoskeletal proteins, and count

49 The anisotropy of phalloidin-actin changed rapidly at first and was follow

50 luorescent nucleotide was similar to that of phalloidin-actin.

51 mbly and MMP secretion and pretreatment with phalloidin again retarded actin disassembly and MMP secr

52 n was investigated by visualizing actin with phalloidin-Alexa 488 after infection or transfection of

53 Dialysis with phalloidin also prevented hypo-osmotically activated Cl-

54 inject nanomolar concentrations of rhodamine phalloidin (an impermeable dye molecule for staining fil

55 ostsynaptic role for actin, latrunculin B or phalloidin, an actin filament stabilizer, was perfused i

56 s stained with fluorescent rhodamine-labeled phalloidin, an actin stain, that the transient viscoelas

57 In contrast, phalloidin, an agent that prevents depolymerization of a

58 ation by confocal fluorescein isothiocyanate-phalloidin analysis.

59 ed by the findings that the actin stabilizer phalloidin and a cofilin inhibitory peptide each blocked

60 1, 3, or 7 days, matrices were labeled with phalloidin and a nucleic acid dye, and were imaged using

61 The actions of phalloidin and actin were reversed by coapplication of g

62 actin cytoskeleton with rhodamine-conjugated phalloidin and analysis by confocal fluorescence microsc

63 body and anti-CSP24 antibody, or fluorescent phalloidin and anti-CSP24 antibody showed that CSP24 is

64 ons was determined by labeling cultures with phalloidin and anti-talin or ILK antibodies, respectivel

65 tics (anti-Ki-67, anti-p16), stratification (phalloidin and anti-ZO-1), and differentiation (anti-K3,

66 ECs were subjected to immunostaining to FITC-phalloidin and antibodies to different junction componen

67 ed cells by using fluorescein isothiocyanate-phalloidin and antibodies to phosphotyrosine and cortact

68 Buttons were single and double labeled using phalloidin and antibodies to ZO-1, Ki67, fibronectin, al

69 te here that together, but not individually, phalloidin and cofilin co-rescue the polymerization of c

70 The NRG 1beta effect was also inhibited by phalloidin and cytochalasin D.

71 ities were noticed, including attenuation of phalloidin and cytoplasmic active beta-catenin staining,

72 Significantly, the unique actin inhibitors, phalloidin and DNase I, also inhibit synthesis of poly P

73 ocked by concurrent whole-cell dialysis with phalloidin and DNase, respectively.

74 or 15 to 120 min were stained with rhodamine phalloidin and examined with a confocal microscope.

75 actin) content by using rhodamine-conjugated phalloidin and flow cytometry showed an elevated F-actin

76 Rhodamine-phalloidin and flow cytometry were used to measure chang

77 vitro cultures were fixed, stained with FITC-phalloidin and hair cells were counted.

78 uld be restored to wild type actin levels by phalloidin and improved greatly through copolymerization

79 h this hypothesis, the actin modifying drugs phalloidin and jasplakinolide enhanced secretion, while

80 erates nuclear actin filaments-detectable by phalloidin and live-cell actin probes-with three charact

81 distribution were assessed using fluorescent phalloidin and LSCM.

82 pletely resistant to hepatoxicity induced by phalloidin and microcystin-LR.

83 We found that a combination of phalloidin and NSF/GluR2 inhibitor reduced the response

84 he association rate constant is low for both phalloidin and rhodamine phalloidin because the filament

85 ease is prevented by the F-actin stabilizers phalloidin and to a lesser extent beryllium fluoride.

86 urons with fluorescently labeled tubulin and phalloidin and used fluorescence time-lapse imaging to a

87 After 24 hours, constructs were labeled with phalloidin and were imaged using fluorescent and reflect

88 ssed by confocal microscopy with fluorescein phalloidin and were not prevented by staurosporine or ca

89 ing peptide nucleic acids, a cyclic peptide (phalloidin), and organic compounds.

90 compared (-)-doliculide with jasplakinolide, phalloidin, and chondramide C to gain insight into a pos

91 ained with antibodies or fluorescent-labeled phalloidin, and viewed with a confocal microscope.

92 us were evaluated by staining with rhodamine-phalloidin, anti-paxillin, and anti-phosphotyrosine anti

93 e branches in samples treated with rhodamine-phalloidin arises from multiple influences of the peptid

94 Using rhodamine-phalloidin as a probe, the individualization complex is

95 concentrations of microinjected fluorescent phalloidin as a tracer for actin filaments, we found tha

96 er donor and anti-actin tetramethylrhodamine phalloidin as an acceptor.

97 High-affinity fluorescent phalloidin as well as immunocytochemistry using anti-act

98 gelsolin and stains intensely with rhodamine-phalloidin, as does the zebrafish extraocular muscle.

99 We observe differences in the phalloidin association kinetics between muscle alpha- an

100 s of wild-type actin, beryllium fluoride, or phalloidin at room temperature, although at 4 degrees C

101 ant is low for both phalloidin and rhodamine phalloidin because the filaments must undergo conformati

102 The rates of rhodamine phalloidin binding are independent of the pH, ionic stre

103 At equilibrium, rhodamine phalloidin binding generates a positive entropy change (

104 ces in actin filaments, we have examined the phalloidin binding kinetics and the bulk rheologic prope

105 mpens these conformational fluctuations, and phalloidin binding kinetics are inhibited.

106 ional changes (i.e. "breathe") to expose the phalloidin binding site.

107 ctin) in pollen were quantified by measuring phalloidin binding sites, a sensitive assay that had not

108 the kinetics and thermodynamics of rhodamine phalloidin binding to actin purified from rabbit skeleta

109 The cyclic peptide phalloidin binds and stabilizes actin filaments.

110 Here we show that rhodamine-phalloidin binds both Arp2/3 complex and the VCA domain

111 Mg(2+)-containing filaments with phalloidin bound also displayed increased rotational mot

112 tetramethylrhodamine isothiocyanate-labeled phalloidin bound to F-actin and N-(1-pyrenyl)iodoacetami

113 escued by filament seeds added together with phalloidin but not with cofilin.

114 ngly and reversibly the release of rhodamine phalloidin by cofilin.

115 Fluorescein-labeled phalloidin confirmed marked disruption of filamentous ac

116 d that fibroblasts stained for f-actin using phalloidin conjugated with common fluorophores display d

117 All three mutants display abnormal phalloidin cytoskeletal staining.

118 FRET) between GFP-tagged Hsp27 and rhodamine phalloidin-decorated actin, minimal interaction was foun

119 flexibility of the filament, the binding of phalloidin decreased the torsional flexibility of all fi

120 1(-/-);Rac2(-/-) RBCs stained with rhodamine-phalloidin demonstrated F-actin meshwork gaps and aggreg

121 g of Kv1.5-GFP and retrospective labeling of phalloidin demonstrated motility of Kv1.5 vesicles on ac

122 stained with saturating levels of rhodamine-phalloidin demonstrated that changes in the level of F-a

123 es also increased KATP channel activity in a phalloidin-dependent manner.

124 plakinolide, readily displaced a fluorescent phalloidin derivative from actin polymer.

125 ctin polymer nor was it able to displace the phalloidin derivative from polymer.

126 did not inhibit the binding of a fluorescent phalloidin derivative to actin polymer nor was it able t

127 ulin (Tmod) or probes such as phallacidin (a phalloidin derivative).

128 Addition of phalloidin did not alter cooperative effects between bou

129 Phalloidin dissociates from all mammalian actin isoforms

130 heir binding sites are distinct, cofilin and phalloidin do not bind simultaneously to F-actin.

131 es not lock actin in filaments (as rhodamine-phalloidin does), possibly allowing for its use in inves

132 However, in contrast to jasplakinolide and phalloidin, dolastatin 11 did not inhibit the binding of

133 In neurons coinjected with rhodamine-phalloidin, F-actin was observed in dynamic cortical pat

134 ANP quenching of rhodamine phalloidin fluorescence showed that neither Ca(2+) nor S

135 We show that SIF causes the intensity of phalloidin fluorescence to increase 4-5 fold and its flu

136 This increase in cortical phalloidin fluorescence was inhibited by latrunculin B a

137 The distribution of the increasing phalloidin fluorescence was uniform with respect to the

138 during recovery; but when adjusted for total phalloidin fluorescence, FRET between Hsp27 and F-actin

139 ation of permeabilized strips with 50 microM phalloidin for 1 h, the increases in isometric force and

140 creases the association rate and affinity of phalloidin for actin.

141 version method based in the high affinity of phalloidin for filamentous (F)-actin.

142 tant and lower binding affinity of rhodamine phalloidin for S. cerevisiae actin filaments provide a q

143 -actin yet synergize in promoting release of phalloidin from filaments, suggesting that Crn1/Cof1 co-

144 tabilization of F-actin by microinjection of phalloidin had no effect on GVBD.

145 Phalloidin had no effect on Vmax during steady-state iso

146 horylated GAP-43 inhibit binding of actin to phalloidin, implying a lateral interaction with filament

147 Stabilizing filaments with phalloidin in most experiments eliminated any contributi

148 xpressed GFP-mTn co-localized with rhodamine-phalloidin in permeabilized tobacco BY-2 suspension cell

149 angement of actin filaments (Alexa Fluor 568-phalloidin) in primary cultures of IMCD cells.

150 Phalloidin increased both filamentous actin (F-actin) an

151 F-actin, as measured by binding of rhodamine-phalloidin, increased transiently during phagocytosis, a

152 the hypothesis that F-actin stabilization by phalloidin increases tension cost (i.e. ATP hydrolysis r

153 reasing the rigidity of actin filaments with phalloidin increases the extent of depletion, whereas sh

154 epithelium stains very weakly with rhodamine-phalloidin, indicating little F-actin in the cytoplasm.

155 these cells coincided with that of rhodamine-phalloidin, indicating that GFP-ABD specifically binds f

156 ifferential salt precipitation or binding to phalloidin-induced actin filaments, had no effect on ves

157 otein kinase and allows entry of fluorescent phalloidin into the cytoplasm of epithelial cells.

158 n the cell after introduction of fluorescent phalloidin into the medium, and the cytokinetic ring was

159 his indicates that the stabilizing effect of phalloidin is achieved mainly through constraining struc

160 or association and dissociation of rhodamine phalloidin is dominated by entropic changes (delta S++).

161 om temperature, although at 4 degrees C only phalloidin is effective.

162 The affinity of NFA filaments for rhodamine phalloidin is lower than that of native actin filaments,

163 for actin marked by microinjected rhodamine phalloidin is very similar, 0.033 +/- 0.013 s(-1), sugge

164 Like phalloidin, jasplakinolide stabilizes F-actin and promot

165 proximately 1-micrometer stripe in rhodamine phalloidin-labeled actin appears stable up to at least 3

166 ectrodes elevated above a surface, rhodamine-phalloidin-labeled actin filaments were attracted to the

167 Rhodamine-phalloidin-labeled actin filaments were visualized glidi

168 velocity matches measurements with rhodamine-phalloidin-labeled actin.

169 the fluorescence lifetime (tau) of Alexa488-phalloidin-labeled actin.

170 fluorescence microscopy was used to identify phalloidin-labeled CLANs and to ascertain the presence o

171 scopy (FPM) has been used to study rhodamine phalloidin-labeled red cell ghosts.

172 Three days after laser injury, phalloidin-labeled RPE cells and isolectin-labeled endot

173 rdiac troponin and tropomyosin and rhodamine-phalloidin-labeled skeletal actin and skeletal heavy mer

174 e stretching stiffness of a single rhodamine-phalloidin-labeled, 1-microm-long F-actin is 34.5 +/- 3.

175 unts of conjunctiva prepared using rhodamine-phalloidin labeling followed by confocal microscopy.

176 Phalloidin labeling intensified in the ciliary stalk wit

177 while the loss of: 1) membrane integrity; 2) phalloidin labeling of F-actin; and 3) TO-PRO-1 labeling

178 BDNF caused no changes in phalloidin labeling of filamentous actin (F-actin) when

179 6 d, because calmodulin immunoreactivity and phalloidin labeling of filamentous actin are retained.

180 Phalloidin labeling of filamentous actin revealed profou

181 ions of spines in these areas indicated that phalloidin labeling was restricted to the largest and mo

182 n-activating mAb AP-5 and were identified by phalloidin labeling.

183 Rhodamine-phalloidin labelling was used wherever possible, and rev

184 The delivery construct carries phalloidin linked to its inserting C terminus via a disu

185 Yeast actin filaments bind rhodamine phalloidin more rapidly, suggesting that perhaps they ar

186 aments of S. cerevisiae actin bind rhodamine phalloidin more weakly than Acanthamoeba and rabbit skel

187 conjugate quantum dot (Qdot) binding biotin-phalloidin on actin.

188 at 25 degrees C but will in the presence of phalloidin or beryllium fluoride.

189 Interestingly, phalloidin or the presence of wild type actin reversed t

190 Pretreatment with phalloidin or treatment with MMP inhibitors partly preve

191 onent was found by using an actin inhibitor (phalloidin) or an inhibitor of NSF (N-ethylmaleimide-sen

192 ropelled actin filaments is similar (without phalloidin) or slightly lower (with phalloidin) than tha

193 eys were fluorescently labeled with lectins, phalloidin, or antibody.

194 LS-WH2 can bind to but fails to depolymerize phalloidin- or jasplakinolide-bound actin filaments.

195 Stabilization of actin filaments with phalloidin partially prevents the rundown, whereas colla

196 omic force microscopy, F-actin staining with phalloidin, passage of FITC-conjugated dextran through a

197 cybe albipes that cleaves a synthetic 22-mer phalloidin peptide to release the mature toxin peptide (

198 ofibrils were labeled lightly with rhodamine-phalloidin, placed on coverslips coated with SIF, illumi

199 t the plasmalemma, actin polymerization into phalloidin-positive stress fibers, and finally CAM endoc

200 160- and 130-kDa proteins was attenuated by phalloidin preloading the cells, a condition which also

201 Both were retarded by phalloidin pretreatment.

202 Phalloidin prevented pressure-stimulated adhesion.

203 Stabilization of F-actin with phalloidin prevented the Ca(2+) response in AF and TZ ce

204 tabilizing actin filaments with jaspamide or phalloidin prevented vesicle release induced by ischaemi

205 binding to regulated alpha-actin affects the phalloidin-probe distance.

206 We purified an enzyme from the phalloidin-producing mushroom Conocybe albipes that clea

207 Not only does phalloidin promote nucleation of pure actin monomers but

208 Conversely, phalloidin protects the L267C and L269C filaments and in

209 Although the numbers of synapsin and phalloidin puncta do not differ from WT, preCGG neurons

210 ucleus and actin distribution using DAPI and phalloidin respectively.

211 actin were identified by DNase and rhodamine phalloidin, respectively.

212 ls, the induction of actin polymerization by phalloidin resulted in the incorporation of both IQGAP a

213 CCV nucleocapsid (N) monoclonal antibody and phalloidin revealed a colocalization of the BCCV N prote

214 analysis of filaments rescued by cofilin and phalloidin revealed a dense contact between opposite str

215 Quantitative immunostaining with phalloidin revealed a significant increase in axonal F-a

216 ng of 3T3 fibroblasts with anti-vinculin and phalloidin revealed clear cytoskeletal filaments and foc

217 HT29 cells stained with fluorescein-labeled phalloidin revealed contraction of the cytoskeleton and

218 ining of the cells with rhodamine-conjugated phalloidin revealed rapid disassembly of actin filaments

219 Coprecipitation using phalloidin revealed that tau interacts with the most pre

220 T-treated monolayers (stained with rhodamine-phalloidin) revealed diminished and flocculated staining

221 stigated using vital staining with rhodamine phalloidin (RP).

222 s countered by filament stabilizing factors, phalloidin, S1, and tropomyosin.

223 ctron microscopy and staining with rhodamine-phalloidin showed that these lamellipodia displayed ruff

224 orce microscopy (cryo-AFM) was used to image phalloidin-stabilized actin filaments adsorbed to mica.

225 Phalloidin-stabilized actin filaments were used in order

226 e range 0.4-1.2 s-1, and subsequently severs phalloidin-stabilized F-actin with a first-order rate co

227 sin inhibited the disulfide cross-linking of phalloidin-stabilized F-actin.

228 Phalloidin stabilizes actin polymers and DNase inhibits

229 s B and D, but was not affected by 10 microM phalloidin (stabilizes actin filaments) or 50 microM col

230 Whereas both anti-MYC and Rho-phalloidin stained intra-Z-band F-alpha-actin oligomers,

231 bution, quantitative fluorescence imaging of phalloidin-stained cells, and ultrastructural studies in

232 ted thick actin assemblies seen in rhodamine phalloidin-stained GG cells.

233 Confocal microscopy of rhodamine phalloidin-stained infected Vero cells revealed the typh

234 ments of F-actin by immunoblot and rhodamine phalloidin staining after ultracentrifugation.

235 ular cytoskeleton, visualized by fluorescein phalloidin staining and confocal microscopy.

236 Cholestatic hepatocytes were analyzed by phalloidin staining and electron microscopy.

237 n reorganization was examined by fluorescent-phalloidin staining and Rac activity assay.

238 Furthermore, phalloidin staining and reactive oxygen species estimati

239 With fluorescent phalloidin staining and serial thin sections, we show th

240 ng and reduces actin filaments determined by phalloidin staining and Western blotting of Triton X-100

241 ent by fluorescein isothiocyanate-conjugated phalloidin staining as well as by indirect immunolabelin

242 al-stromal communications was evaluated with phalloidin staining as well as electron microscopy.

243 Immunofluorescence combined with phalloidin staining demonstrated the colocalization of t

244 g 3T3L1 adipocyte differentiation, rhodamine-phalloidin staining demonstrated the formation of a cort

245 Fluorescent phalloidin staining indicated that a well defined fringe

246 Rhodamine phalloidin staining indicates that ATPgammaS treatment i

247 was accompanied by a reduction of rhodamine-phalloidin staining most prominent in the growth cone pe

248 site of attachment, as visualized by either phalloidin staining of fixed cells or the active recruit

249 BODIPY-phalloidin staining of four different stably transfected

250 Phalloidin staining of hepatocytes showed a prominent sh

251 In contrast, phalloidin staining of infected cells showed that the vi

252 Rhodamine phalloidin staining of karst mutant ovaries similarly re

253 Rhodamine-labeled phalloidin staining of morphologically differentiated 3T

254 Phalloidin staining revealed dispersed, shorter and diso

255 Rhodamine-phalloidin staining revealed increase stress fiber forma

256 Rhodamine phalloidin staining reveals that the actin-based hexagon

257 Phalloidin staining showed that FTI treatment did not re

258 morphologic studies, AlexaFluor 488-labeled phalloidin staining was used to examine actin filament,

259 Annexin-V and phalloidin staining were used to detect apoptosis and th

260 cts of lovastatin on F-actin reorganization (phalloidin staining), focal adhesion formation (paxillin

261 By phalloidin staining, a colocalization of pyrin with acti

262 ured and analyzed by histology, immunoblots, phalloidin staining, immunohistochemistry, electron micr

263 hanges in actin cytoskeletal organization by phalloidin staining, MMP-2 activation by gelatin zymogra

264 at this later stage as well and, based upon phalloidin staining, we propose that the second half of

265 Phalloidin staining, which labels actin filaments of ste

266 leton (thickening of fibrils) as assessed by phalloidin staining, with more pronounced effects at 20

267 Actin structures were visualized by phalloidin staining.

268 n) was confirmed by the absence of rhodamine-phalloidin staining.

269 h an anti-acetylated tubulin antibody and by phalloidin staining.

270 sualized by light microscopy and fluorescent-phalloidin staining.

271 actin cytoskeletal network was visualized by phalloidin staining.

272 Actin was visualized by phalloidin staining.

273 ve-muscle cultures, as revealed by rhodamine-phalloidin staining.

274 migration using inserts, wound healing, and phalloidin staining; and cell synthesis using ELISA and

275 n fibers within 30 minutes, as determined by phalloidin stainings of the polymerized actin and tropon

276 Computational modeling reveals how bound phalloidin stiffens actin filaments and inhibits the rel

277 In contrast, although phalloidin strongly stabilizes F-actin, it causes little

278 n the rat central nervous system (CNS) using phalloidin tagged with the fluorophore eosin followed by

279 thalene-1-sulfonic acid at Cys-190 of Tm and phalloidin-tetramethylrhodamine B isothiocyanate bound t

280 (without phalloidin) or slightly lower (with phalloidin) than that of HMM-free filaments observed in

281 With beryllium fluoride, but not phalloidin, this polymerization rescue is cold-sensitive

282 actin and/or competition between cofilin and phalloidin to alter F-actin symmetry.

283 r and extracellular application of rhodamine-phalloidin to conventional hippocampal slices to test wh

284 ion, consistent with the expected binding of phalloidin to F actin, stabilizing the filaments against

285 n gelation activity, even in the presence of phalloidin, to stabilize actin filaments against debranc

286 n, to assist with actin nucleation, and with phalloidin, to stabilize the elongating filament segment

287 Phalloidin treatment altered actin polymerization, as de

288 erization defect can be partially rescued by phalloidin treatment, which stabilizes F-actin.

289 immunoreactivity with F-actin (labeled with phalloidin) was observed at the apices and bases of RPE

290 ystrophin antibody and tetramethyl-rhodamine phalloidin were present.

291 incubating the cells overnight in 10 microM phalloidin, which binds to actin filaments (n = 5).

292 Branches were stabilized by phalloidin, which inhibits phosphate dissociation from A

293 Phalloidin, which is known to stabilize actin filaments,

294 dual filaments after treatment with the drug phalloidin, which perturbs filament dynamics.

295 Rhodamine-phalloidin, which selectively binds to polymerized actin

296 Direct application of phalloidin, which stabilizes actin, preserved low PO and

297 However, preapplication of phalloidin, which stabilizes the actin polymerization, s