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1 There was no evidence of a pharmacokinetic interaction.
2 itaxel dose is reduced to compensate for the pharmacokinetic interaction.
3 st likely related to P450, contribute to the pharmacokinetic interaction.
4 ersal in cancer treatment and drug-flavonoid pharmacokinetic interactions.
5 ose of the combination regimen and to assess pharmacokinetic interactions.
6 rotein cholesterol-occurred independently of pharmacokinetic interactions.
7 There were no pharmacokinetic interactions.
9 ated treatment regimens designed to minimize pharmacokinetic interactions and to potentiate immunosup
15 conducted a phase I/IIa study to investigate pharmacokinetic interaction between IVC and gemcitabine.
18 ESLD) (substudy 1) and to assess the lack of pharmacokinetic interaction between RAL and the immunosu
22 coinfection; however, little is known about pharmacokinetic interactions between boceprevir and anti
23 A randomized, open-label study to assess the pharmacokinetic interactions between boceprevir and rito
25 nsplant recipients, the authors describe the pharmacokinetic interactions between cyclosporine and th
30 of data from these trials is complicated by pharmacokinetic interactions between the target cytotoxi
31 suggested that pharmacological, rather than pharmacokinetic, interactions between the parent drugs w
33 Detailed mechanistic interrogation of this pharmacokinetic interaction demonstrated that lenalidomi
35 rsers have in many instances been limited by pharmacokinetic interactions exacerbating the clinical t
39 dose reductions were planned because of the pharmacokinetic interactions known to occur with PSC 833
40 mbination with cyclosporine (CsA) produces a pharmacokinetic interaction, namely increases in the who
41 n of renal dysfunction seemed to be due to a pharmacokinetic interaction of RAPA to greatly increase
44 udy investigated the efficacy, toxicity, and pharmacokinetic interactions resulting from simultaneous
46 toxicity profile without clinically relevant pharmacokinetic interactions, support the performance of
47 red administration of RAD+Neoral avoided the pharmacokinetic interactions that caused the elevated dr
56 tes CsA-induced renal dysfunction owing to a pharmacokinetic interaction, whereas CsA produces a phar
59 efficacious modulator that apparently lacks pharmacokinetic interactions with coadministered antican
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