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1 , 3-butenyl, 4-(methylthio)butyl, benzyl and phenethyl isothiocyanates.
2 mice in each group were fed diets containing phenethyl isothiocyanate (3 and 1.5 mmol/kg diet), sulfo
5 is for future evaluation of the potential of phenethyl isothiocyanate and sulforaphane and their conj
6 this study, the chemopreventive activity of phenethyl isothiocyanate and sulforaphane and their N-ac
7 toward tubulin among benzyl isothiocyanate, phenethyl isothiocyanate, and sulforaphane correlate wel
8 hrough the addition of glutathione mainly to phenethyl isothiocyanate, but alternatively and in a com
9 ence of adenocarcinoma in the 3 mmol/kg diet phenethyl isothiocyanate group and 8 mmol/kg diet phenet
11 t, inhibition of aldehyde dehydrogenase with phenethyl isothiocyanate increased the inhibitory effect
12 iet), sulforaphane (3 and 1.5 mmol/kg diet), phenethyl isothiocyanate-N-acetylcysteine (8 and 4 mmol/
13 thyl isothiocyanate group and 8 mmol/kg diet phenethyl isothiocyanate-N-acetylcysteine group was redu
14 Knockdown of IQGAP1 significantly attenuated phenethyl isothiocyanate- or MEK-mediated activation of
15 with human plasma over a range of 49-3003 nM phenethyl isothiocyanate (PEITC) (r(2) = 0.996 +/- 0.003
16 prostate cancer cells in culture showed that phenethyl isothiocyanate (PEITC) and curcumin have signi
18 a mechanism for inhibiting tumorigenesis by phenethyl isothiocyanate (PEITC) and sulforaphane (SFN).
19 cluding the cruciferous vegetable derivative phenethyl isothiocyanate (PEITC) can reduce risk of huma
24 thiocyanates, such as sulforaphane (SFN) and phenethyl isothiocyanate (PEITC) possess strong antitumo
26 re more sensitive to cytotoxicity induced by phenethyl isothiocyanate (PEITC) than those with the wil
29 hose from our laboratory, have revealed that phenethyl isothiocyanate (PEITC), a constituent of many
31 is report, we examined the effect of dietary phenethyl isothiocyanate (PEITC), an inhibitor of lung t
32 ounds, 6-phenylhexyl isothiocyanate (PHITC), phenethyl isothiocyanate (PEITC), and N-acetylcysteine (
34 t p53 is required for apoptosis induction by phenethyl isothiocyanate (PEITC), which is a highly prom
36 tment with the N-acetylcysteine conjugate of phenethyl isothiocyanate (PEITC-NAC) inhibited benzo(a)p
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