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1  directly measured positions of alaninol and phenethylamine.
2 e slowly and bound 75-fold more tightly than phenethylamine.
3 lass of 5-HT(2A) receptor agonists, N-benzyl phenethylamines.
4 iety affords pharmaceutically important beta-phenethylamines.
5 rimethamine with substituted benzylamines or phenethylamines.
6 y restricted analogues of the hallucinogenic phenethylamine 1 (2,5-dimethoxy-4-bromophenethylamine, 2
7 , and a series of substituted tryptamine and phenethylamine 5-HT(2A) receptor agonists, we found that
8 g affinities of a series of para-substituted phenethylamine analogues to MAO A show an increase in af
9 nd isotopic characterization of alaninol and phenethylamine, analogues of alanine and phenylalanine,
10 ding site for the aryl ring is identical for phenethylamine and for benzylamine analogues and that st
11                                              Phenethylamine and tyramine prepared from a mixture of 1
12 nd further allows the simulation analysis of phenethylamine and tyramine, which are major monoamine c
13 each catalyze conversion of phenylalanine to phenethylamine and tyrosine to tyramine.
14 a-selective C-H arylation of nosyl-protected phenethylamines and benzylamines is disclosed using a co
15                    A common binding mode for phenethylamines and imidazoles with alpha 2-adrenoceptor
16 an with conformational analysis of classical phenethylamines and medetomidine analogs.
17 strated on amides derived from benzylamines, phenethylamines and phenylpropylamines; amine-containing
18 rototypical hallucinogenic drug classes, the phenethylamines and the tryptamines/ergolines.
19 benzyl analogs, whereas most N-unsubstituted phenethylamines and traditional agonists were only weakl
20 zamide, homologation of the benzylamine to a phenethylamine, and incorporation of a methyl group at t
21 ic study of the nitrosation of ethylbenzene, phenethylamine, and tyramine was carried out, using UV-v
22 mides prepared from readily available chiral phenethylamines, and allows easy variation of the stereo
23 ctional assays confirmed that these N-benzyl phenethylamines are potent and highly efficacious agonis
24  of piperidinone- and piperidine-constrained phenethylamines as novel DPP4 inhibitors.
25   P450 2D6 discriminated between the various phenethylamines, as evidenced by binding and steady-stat
26                                              Phenethylamine-betaxanthin was the most potent in the in
27 t catalyzes the oxidation of methylamine and phenethylamine, but not that of benzylamine.
28 hrine, and epinephrine, as well as for other phenethylamines, but not for imidazolines, a class of st
29 phenylalanine were converted to tyramine and phenethylamine by tyrosine and phenylalanine decarboxyla
30  implies, therefore, that the hallucinogenic phenethylamines cannot be directly superimposed on LSD i
31                            A wide variety of phenethylamines containing a potentially free primary am
32        Although fenethylline shares a common phenethylamine core with other amphetamine-type stimulan
33 eral approach for (13)C(2)- and (2)H-labeled phenethylamine derivatives has been developed, based on
34 ence, we prepared nearly two dozen different phenethylamine derivatives, attached them to the C termi
35 be used to easily access biologically active phenethylamine derivatives.
36                    The NBOMe derivatives are phenethylamines derived from the 2C class of hallucinoge
37                              Tryptamine- and phenethylamine-derived imides were selectively monotrifl
38 tes, such as tyrosine, tryptamine, tyramine, phenethylamine, dopamine, 3-methoxytyramine, serotonin,
39 thousand for alaninol, 6.54 per thousand for phenethylamine) due to propagation of errors.
40 e a higher potency and intrinsic activity of phenethylamines for polyphosphoinositide turnover but no
41                                 Finally, the phenethylamine fragment incorporated into these molecule
42                                 Pyrolysis of phenethylamine generated benzene and toluene fragments.
43                            Unexpectedly, the phenethylamine hallucinogen, DOI, a partial agonist at 5
44 phetamine (DOM; 0.6 mg/kg, i.p.), which is a phenethylamine hallucinogen, increased glutamate to 206%
45 pulation of 5-HT cells in the DRN induced by phenethylamine hallucinogens in vivo.
46 nt with the proposed mechanism of action for phenethylamine hallucinogens, that such compounds must b
47 terium exchange (HDX) in aqueous droplets of phenethylamine has been determined with submillisecond t
48                Benzyl cyanide was reduced to phenethylamine in 83% yield.
49 it enhanced inhibitory activity (relative to phenethylamine), including para-substituted sulfonamide
50  We selected naphthylpropane-2-amines of the phenethylamine library (PAL) including the partial subst
51 ses concerning the bioactive conformation of phenethylamine ligands upon binding to the 5-HT(2A) rece
52  reproducibility of delta15N of tyramine and phenethylamine measured by GCC-IRMS averaged SD(delta15N
53 sitized, and internalized on exposure to the phenethylamines norepinephrine (NE), epinephrine, or phe
54                                              Phenethylamine oxidation by MAO A can be described as th
55 e-3-acetic acid (5-HIAA), tryptophane, and 2-phenethylamine (PEA) in rat brain using liquid chromatog
56 urthermore, Mg2(olz) was used to encapsulate phenethylamine (PEA), a model drug for a broad class of
57 ynthetic cannabinoids, synthetic cathinones, phenethylamines, piperazines, ketamine and phencyclidine
58 interacting with Phe339((6.51)), whereas the phenethylamine portion was likely to be interacting with
59  of medetomidine conformations with those of phenethylamines provided a tentative explanation for the
60 ion of peptide-extended glycine residues and phenethylamines, respectively.
61       Examination of side chain analogues of phenethylamine show 3-phenylpropylamine to be oxidized 2
62 monoamine oxidase A (MAO A) with a series of phenethylamine substrate analogues has been investigated
63 is, we analyzed the binding and oxidation of phenethylamine substrates.
64                              A comparison of phenethylamine systems bearing different leaving groups
65  effects similar to DisA overexpression, and phenethylamine, the product of phenylalanine decarboxyla
66 exposed to the trace amines p-tyramine, beta-phenethylamine, tryptamine, and octopamine.
67 ve binding conformation of the more flexible phenethylamine type hallucinogens.
68  this class of ligand binds differently than phenethylamine-type agonists and may be more antagonist-
69 hylurapidil, with no changes in affinity for phenethylamine-type agonists such as epinephrine, methox
70                     In studies of the SAR of phenethylamine-type serotonin 5-HT(2A) receptor agonists
71  of sophorolipid ethyl ester in dry THF with phenethylamine, tyramine, p-methoxyphenethylamine, 2-(p-
72  analogues, such as ascorbic acid, catechol, phenethylamine, tyrosine, epinephrine, and norepinephrin
73 ffects for para-substituted benzylamines and phenethylamines, unlike native AADH for which a poor cor
74                                              Phenethylamine (varphiCH2CH2NH2) was chosen to study bec
75 is method delivers valuable beta-substituted phenethylamines via a challenging reductive elimination
76 incorporation of two carbon-13 isotopes into phenethylamines was accomplished using a palladium-catal
77                The putative product of DisA, phenethylamine, was able to inhibit disA expression, ind
78           The configurations of these chloro-phenethylamines were determined by 1D and 2D NMR analysi
79 In contrast to the 2,5-dimethoxy-substituted phenethylamines, where rigidification of the methoxy gro
80                   Acylation of N-(benzoyloxy)phenethylamine with the acid chloride of N(alpha)-Fmoc-L

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