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1 etaldehyde, 4-hydroxyphenylacetaldehyde, and phenylethylamine.
2 and identified it to be a biogenic amine, 2-phenylethylamine.
3 ive sensory neurons that also responded to 2-phenylethylamine.
4 s the anti-Markovnikov addition product beta-phenylethylamine.
5 monoamines such as serotonin, tyramine, and phenylethylamine.
6 of serotonin, norepinephrine, dopamine, and phenylethylamine.
7 ffects for para-substituted benzylamines and phenylethylamines.
8 (*) from three chiral benzylic substrates (1-phenylethylamine, 1-phenylethanol, and alpha-vinylbenzyl
9 t study, this is exemplified by subjecting 1-phenylethylamine (1a) and analogous benzylic amines to D
11 6 receptors a series of compounds, including phenylethylamines 6, pyrroloethylamine 7, and phenylpipe
13 nd rTAAR1, respectively) insensitive to beta-phenylethylamine, amphetamine (AMPH), and methamphetamin
15 methyl N-Boc-phosphoramidates derived from 1-phenylethylamine and 1,2,3,4-tetrahydronaphthalen-1-ylam
19 hanism because the Ea of formation of both 2-phenylethylamine and phenylacetaldehyde remained unchang
20 e that the benzenesulfonamide portion of the phenylethylamine and phenylpiperazine analogues can be "
21 revalence of tyramine followed by histamine, phenylethylamine and putrescine; and total amine levels
22 lative affinity for indoleethylamines versus phenylethylamines and temperature-activity relationships
23 indole, the neurotransmitter precursor beta-phenylethylamine, and a variety of peroxidase and laccas
25 adienyl)ethanol, and the aromatized product, phenylethylamine, and that the two pathways do not cross
26 ytryptamine, a decreased preference for beta-phenylethylamine, and, similar to MAO A, was more sensit
29 not a requirement for the binding of certain phenylethylamines at 5-HT(2A) receptors, the presence of
31 ation of biogenic and dietary amines such as phenylethylamine, benzylamine, dopamine, and tyramine.
32 lycine and agmatine and neuromodulators beta-phenylethylamine (beta-PEA) and taurine are important bi
34 -aryl[or (1S,2S)-1-alkyl]-2-(phenylseleno)-2-phenylethylamines by reaction with t-BuLi and subsequent
35 spermidine, putrescine, histamine, tyramine, phenylethylamine, cadaverine and serotonin) were determi
37 reactions of QHNDH with chiral deuterated 2-phenylethylamines demonstrated that the enzyme abstracts
41 n studies involving enzymatic depletion of 2-phenylethylamine from a carnivore odor indicate it to be
42 ptamine), norepinephrine, dopamine, and beta-phenylethylamine; in humans and mice, these neurochemica
43 of serotonin, norepinephrine, dopamine, and phenylethylamine increased, and serotonin metabolite 5-h
47 Two prey species, rat and mouse, avoid a 2-phenylethylamine odor source, and loss-of-function studi
48 without significant changes of the K(m) for phenylethylamine or dopamine or the IC(50) for deprenyl
51 , spermine, spermidine), aromatic (tyramine, phenylethylamine) or heterocyclic (histamine, tryptamine
52 ependent biosynthesis of the TPQ cofactor in phenylethylamine oxidase (PEAO) and histamine oxidase fr
53 ling of the C2, C4, and C5 oxygens of TPQ in phenylethylamine oxidase (PEAO) from Arthrobacter globif
54 y of the topa quinone biogenesis reaction in phenylethylamine oxidase from Arthrobacter globiformis (
56 ect, the periplasmic compartmentalization of phenylethylamine oxidase serves the same purpose as the
57 atic that E. coli can synthesize a catabolic phenylethylamine oxidase that stoichiometrically generat
58 thway that is required for the catabolism of phenylethylamine (PEA) and its hydroxylated derivatives
59 ntinuous intravenous infusion over 30 min of phenylethylamine (PEA), an endogenous substrate of MAOB,
61 inatorial library of 10 structurally related phenylethylamines (PEAs) is investigated for chiral sepa
62 ss 38 mammalian species indicates enriched 2-phenylethylamine production by numerous carnivores, with
64 The quantity of biogenic amines (tryptamine, phenylethylamine, putrescine, cadaverine, histamine, ser
65 ight biogenic amines (cadaverine, histamine, phenylethylamine, putrescine, spermine, spermidine, tyra
66 apparent affinities for a preferred ligand, phenylethylamine, rivaling those seen with mammalian phe
67 amine dehydrogenase-catalyzed reaction with phenylethylamine shows complex pressure and temperature
69 Putrescine, cadaverine, tryptamine, beta-phenylethylamine spermidine, spermine were analysed by U
71 of the achiral parent ligand with a chiral 1-phenylethylamine substituent on the open face of the com
72 to perpendicular to the aromatic ring of the phenylethylamine substrate or cyclohexadiene ring of CHD
74 which exhibits a higher preference for beta-phenylethylamine than for the MAO A preferred substrate
75 roles in the neurotoxicity produced by this phenylethylamine, the primary source of DA responsible f
77 s was prepared from N-(o-nitrophenylsulfenyl)phenylethylamines through BINOL-phosphoric acid [(R)-TRI
78 tivity (from 0.02mgkg(-1) for spermidine and phenylethylamine to 0.2mgkg(-1) for spermine) when compa
79 imethyl N-Boc-phosphoramidate derived from 1-phenylethylamine to the N-Boc alpha-aminophosphonate pre
80 , in enclosed laboratory cultures, growth on phenylethylamine triggered induction of the OxyR H2O2 st
82 of six biogenic amines (histamine, tyramine, phenylethylamine, tryptamine, putrescine, and cadaverine
83 cted during substrate (either benzylamine or phenylethylamine) turnover, by anaerobic incubation with
86 model compound, first (S)- and/or (RS)-alpha-phenylethylamine was used, and their diastereomeric salt
91 es, tyramine, histamine, tryptamine and beta-phenylethylamine, were detected in practically all ferme
92 ion of histamine, tyramine, tryptamine and 2-phenylethylamine, which eliminates the purification step
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