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1 n with the first use of a product containing phenylpropanolamine.
2 the use of cough or cold remedies containing phenylpropanolamine.
3  use of appetite suppressants that contained phenylpropanolamine.
4                                              Phenylpropanolamine, 12.5 to 25 mg PO, increased systoli
5 (3, the active metabolite of midodrine), and phenylpropanolamine (4).
6                                              Phenylpropanolamine, a well-known over-the-counter appet
7 dence interval: 2.5, 25.3; 4 exposed cases), phenylpropanolamine and ear defects (odds ratio = 7.8; 9
8 e interval: 2.2, 27.2; 4 exposed cases), and phenylpropanolamine and pyloric stenosis (odds ratio = 3
9  the use of appetite suppressants containing phenylpropanolamine and the risk of a hemorrhagic stroke
10 tion between the use of a product containing phenylpropanolamine and the risk of a hemorrhagic stroke
11 use of cough or cold remedies that contained phenylpropanolamine, and 15.92 (95 percent confidence in
12 ential association with hemorrhagic strokes, phenylpropanolamine has been largely substituted for by
13                     The results suggest that phenylpropanolamine in appetite suppressants, and possib
14 ed with 16 oz of room temperature tap water, phenylpropanolamine increased SBP by 82+/-2 mm Hg.
15                            All first uses of phenylpropanolamine involved cough or cold remedies.
16                                              Phenylpropanolamine is commonly found in appetite suppre
17                The cardiovascular effects of phenylpropanolamine or pseudoephedrine, alone and in com
18  MAP than the nonselective alpha(1)-agonists phenylpropanolamine (PPA) (1) and ST-1059 (2, the active
19 age patterns of the nonprescription products phenylpropanolamine (PPA) and ephedra are of particular
20 s have linked the use of products containing phenylpropanolamine to hemorrhagic stroke, often after t

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