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1 bolism, as demonstrated with exogenously fed phenylurea.
2 bolized herbicides belonging to the class of phenylurea.
3 targeting 52 carbamtes, thiocarbamates, and phenylureas.
4 rolidin-2-yl]methyl}amino)hexyl]-N-methyl-N'-phenylurea (1396-12), but not a structurally related con
5 of phenyl hydroxycarbamate 1 and 1-hydroxy-3-phenylurea 2 using CuCl2 and 2-ethyl-2-oxazoline in meth
7 eido dihomooxacalix[4]arene anion receptors (phenylurea 4a, phenylthiourea 4b, and tert-butylurea 4c)
9 plied to tumor cell lines in culture, active phenylurea-based compounds induced apoptosis in a rapid,
11 nt with these findings, apoptosis induced by phenylurea-based compounds was not altered by genetic al
12 Altogether, these findings indicate that phenylurea-based XIAP antagonists block interaction of d
14 een of a compound library, we identified the phenylurea BX430 (1-(2,6-dibromo-4-isopropyl-phenyl)-3-(
15 nt, the discovery of a 2-(phenoxypyridine)-3-phenylurea chemotype that inhibited ADP-mediated platele
20 yzes rapid oxidative dealkylation of various phenylurea herbicides to yield nonphytotoxic metabolites
21 10) specifically catalyzed the metabolism of phenylurea herbicides, converting four herbicides of thi
23 tuents at the meta- or para-positions of the phenylurea, increases ligand affinity and beta1-selectiv
24 le following modification of the substituted phenylurea moiety appended off the lysine revealed that
27 For example, neutral and basic pesticides (phenylureas, triazines) are more sensitive using APCI (e
29 lphenoxy)pyridin-3-yl)-3-4-(trifluoromethoxy)phenylurea, which demonstrated a 68 +/- 7% thrombus weig
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