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1 SC block, verifying strict use dependence of philanthotoxin.
2 yl)-spermine (PPS), and virtually absent for philanthotoxin 343 (PhTX 343), suggesting that permeatio
3                                The action of philanthotoxin 343 (PhTX) on rat homomeric GluR6(Q) reco
4 nule cells loaded with fluo-3, ArgTX-636 and philanthotoxin-343 (PhTX-343) antagonised increases of i
5  receptor (nAChR) in BC(3)H1 muscle cells by philanthotoxin-343 (PhTX-343), a synthetic analogue of p
6                                              Philanthotoxin-433 (PhTX-433) is an active component of
7             The GluA2-lacking AMPAR blocker, philanthotoxin-433 (PhTx-433), was used as an alternativ
8 oxin-343 (PhTX-343), a synthetic analogue of philanthotoxin-433, a wasp toxin, was investigated using
9 ndent antagonist of GluA2-containing AMPARs, philanthotoxin-74 (PhTx-74), on recombinant AMPARs and o
10 auditory nerve input alone were inhibited by philanthotoxin, a blocker of calcium-permeable AMPA rece
11 s use, recent findings have indicated that a philanthotoxin analog, PhTX-74, can distinguish among Gl
12 ess the potential neuroprotective effects of philanthotoxins and argiotoxin-636 (ArgTX-636).
13 6), polycationic compounds (spermine, Naspm, philanthotoxin) and polyanionic compounds (Evans Blue, s
14 e of the naturally occurring polyamine toxin philanthotoxin), and Mg(2+) each blocked in a dose- and
15                        In contrast, the same philanthotoxin application at rest decreased the subsequ
16  neurons obtained from GluR2-deficient mice, philanthotoxin application decreased AMPA-receptor-media
17 fit, assuming that spermine, spermidine, and philanthotoxin are weakly permeable open-channel blocker
18                   A 10-min-long perfusion of philanthotoxin further decreased AMPA-eEPSC amplitudes t
19 -dependent glutamate receptor (GluR) blocker philanthotoxin, indicated that spontaneous and evoked tr
20 R synapses in young PICK1 knock-out mice are philanthotoxin insensitive with linear I-V relationships
21 tinic acetylcholine receptors (nAChR) by the philanthotoxins, PhTX-343 and PhTX-(12).
22 Polyamine toxins such as joro spider toxins, philanthotoxins (PhTXs), and argiotoxins are use-depende
23 ing activity and can be interrupted by NBQX, philanthotoxin, postsynaptic BAPTA, or external sequestr
24        Finally, stimulation after removal of philanthotoxin resulted in reversal of AMPA-eEPSC block,
25 ns occurs by afferent-specific activation of philanthotoxin-sensitive and -insensitive AMPA receptors
26 ate SE animals were inwardly rectifying, and philanthotoxin-sensitive; similar changes were observed
27 ent-voltage (I-V) relationships, and partial philanthotoxin sensitivity of synaptic events.
28 ndencies of the two modes of action of these philanthotoxins suggest two binding sites, one deep in t
29 eurotransmission, we used a polyamine agent, philanthotoxin, that selectively blocks AMPA receptors l
30                    Using the polyamine toxin philanthotoxin, which selectively blocks calcium-permeab

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