ライフサイエンスコーパス: phosphatidylethanolamine

1 f the membrane caused by the presence of PE (phosphatidylethanolamine).

2 te through cellular membranes via binding to phosphatidylethanolamine.

3 e biosynthetic pathways for the synthesis of phosphatidylethanolamine.

4 interaction prevents PfAtg8 lipidation with phosphatidylethanolamine.

5 by PtdSer decarboxylase 2 (Psd2p) to produce phosphatidylethanolamine.

6 omes containing only phosphatidylcholine and phosphatidylethanolamine.

7 utral phospholipids, phosphatidylcholine, or phosphatidylethanolamine.

8 specially dilinoleoyl phosphatidylcholine or phosphatidylethanolamine.

9 osphatidylcholine, phosphatidylinositol, and phosphatidylethanolamine.

10 (CL), mono-lyso CL, phosphatidylcholine, and phosphatidylethanolamine.

11 and as a mixture of phosphatidylglycerol and phosphatidylethanolamine.

12 mbrane phospholipids phosphatidylserine, and phosphatidylethanolamine.

13 formation involves the membrane phospholipid phosphatidylethanolamine.

14 n unknown phospholipid determined to be lyso-phosphatidylethanolamine.

15 was correlated with phosphatidylcholine and phosphatidylethanolamine.

16 s involved in the synthesis of mitochondrial phosphatidylethanolamine.

17 otoisomerization of N-ret-PE (N-retinylidene-phosphatidylethanolamine.

18 chidonate into liver phosphatidylcholine and phosphatidylethanolamine.

19 ctive, causing increased inward transport of phosphatidylethanolamine.

20 ygenase-derived hydroxyeicosatetraenoic acid-phosphatidylethanolamines.

21 r both treatment groups (q<0.00005), whereas phosphatidylethanolamine (36:3e) contributed most to dis

22 tions, but showed an almost complete loss of phosphatidylethanolamine(42:4), phosphatidylserine(42:4)

23 At intermediate levels of phosphatidylethanolamine a mixture of native and topolog

24 bisretinoids A2E, A2-GPE, A2-dihydropyridine-phosphatidylethanolamine (A2-DHP-PE), and all-trans-reti

25 P4-ATPase that flips phosphatidylserine and phosphatidylethanolamine across cell membranes.

26 phosphatidylserine and, to a lesser extent, phosphatidylethanolamine across the lipid bilayers of in

27 epare blank oil (O), glucose added oil (OG), phosphatidylethanolamine added oil (OP), and both phosph

28 ial for synthesizing the ATG12-ATG5 and ATG8-phosphatidylethanolamine adducts that are central to aut

29 Synthesis of the ATG12-ATG5 and ATG8-phosphatidylethanolamine adducts, which are essential fo

30 Depletion of intermediate levels of phosphatidylethanolamine after final protein assembly re

31 ine (A2-DHP-PE), and all-trans-retinal dimer-phosphatidylethanolamine (all-trans-retinal dimer-PE) al

32 With dioleoyl phosphatidylethanolamine alone, the proteins had no effe

33 , and Arg69, ensures stable insertion of the phosphatidylethanolamine anchor into membranes.

34 hosphate-limited plants, phosphatidylcholine/phosphatidylethanolamine and associated transcript level

35 n not only locally curved membranes but also phosphatidylethanolamine and cardiolipin, lipids with hi

36 at high levels of Mdm33 affect the levels of phosphatidylethanolamine and cardiolipin, the two key in

37 osynthesis of the non-bilayer-forming lipids phosphatidylethanolamine and cardiolipin.

38 Conical lipids, phosphatidylethanolamine and diacylglycerol, enhanced tr

39 hatidylethanolamine added oil (OP), and both phosphatidylethanolamine and glucose added oil (OPG).

40 Phosphatidylethanolamine and glucose were added to the d

41 tion of synthesis involves the conversion of phosphatidylethanolamine and glycerol into PG and is cat

42 By targeting phosphatidylethanolamine and lysylphosphatidylglycerol,

43 lycerophospholipid in both seed oils whereas phosphatidylethanolamine and phosphatidic acid were less

44 The major phospholipids, phosphatidylethanolamine and phosphatidylcholine (PtdCho

45 These comprise eicosanoids attached to phosphatidylethanolamine and phosphatidylcholine and for

46 headgroups found in mitochondrial membranes, phosphatidylethanolamine and phosphatidylcholine.

47 maintain membrane asymmetry by translocating phosphatidylethanolamine and phosphatidylserine from the

48 tion required to induce PFO binding, whereas phosphatidylethanolamine and phosphatidylserine in the i

49 s of host-encoded cofactor molecules such as phosphatidylethanolamine and RNA molecules are required

50 in aqueous extracts and phosphatidylcholine, phosphatidylethanolamine and sphingomyelin in lipid extr

51 s were observed by reduced ratios between PC:phosphatidylethanolamine and sphingomyelin:cholesterol,

52 host yeast has revealed essential roles for phosphatidylethanolamine and sterols in viral replicatio

53 osphocoenzyme A, spermidine, putrescine, and phosphatidylethanolamines and elevated alanine, leucine,

54 The virion envelope contained twofold more phosphatidylethanolamines and threefold less phosphatidy

55 re made for neutral (phosphatidylcholine and phosphatidylethanolamine) and anionic (phosphatidylserin

56 ted by zwitterionic (phosphatidylcholine and phosphatidylethanolamine) and cationic (sphinganine) lip

57 authentic standards (phosphatidylcholine and phosphatidylethanolamine) and two purified PL from marin

58 phosphatidic acid, a phosphatidylcholine, a phosphatidylethanolamine, and a phosphatidylglycerol wer

59 arrhalis membranes are phosphatidylglycerol, phosphatidylethanolamine, and cardiolipin (CL).

60 major types of lipids (phosphatidylcholine, phosphatidylethanolamine, and cardiolipin) in a roughly

61 sphatidic acid together with sphingomyelins, phosphatidylethanolamine, and cholesterol) gives rise to

62 ly found in F1, with higher triglyceride and phosphatidylethanolamine, and lower sphingomyelin concen

63 lactosyldiacylglycerol, whereas cardiolipin, phosphatidylethanolamine, and phosphatidic acid are pres

64 Defective cardiolipin (CL), phosphatidylethanolamine, and phosphatidylglycerol remod

65 ain-containing lipase 1) that hydrolyzes CL, phosphatidylethanolamine, and phosphatidylglycerol.

66 cids, diacylglycerides, phosphatidylcholine, phosphatidylethanolamine, and phosphatidylinositol in th

67 id (phosphatidylcholine, phosphatidylserine, phosphatidylethanolamine, and phosphatidylinositol) prof

68 r phosphatidylcholine, phosphatidylinositol, phosphatidylethanolamine, and sphingomyelin between phos

69 SG phosphatidylserine, phosphatidylinositol, phosphatidylethanolamine, and sphingomyelin, fatty acids

70 lipin, decreases in phosphatidylglycerol and phosphatidylethanolamine, and the dramatic appearance of

71 of lipid mixtures that includes cholesterol, phosphatidylethanolamine, and varied PS content.

72 for the synthesis of phosphatidylcholine and phosphatidylethanolamine are attractive drug targets to

73 sported to mitochondria, but cardiolipin and phosphatidylethanolamine are produced in mitochondria.

74 ilayer-forming phospholipids cardiolipin and phosphatidylethanolamine are required for the activity o

75 eivably uses heparin and phosphatidylcholine/phosphatidylethanolamine as anchors to increase protein

76 s a potential molecular link between loss of phosphatidylethanolamine asymmetry and the dynamic buddi

77 re that with physiological concentrations of phosphatidylethanolamine, Atg8 does not act as a fusogen

78 h as FLOWERING LOCUS T (FT), a member of the phosphatidylethanolamine-binding protein (PEBP) family.

79 To study the evolution of phosphatidylethanolamine-binding protein (PEBP) gene fam

80 ether 15-lipoxygenase-1 (15LO1) binding with phosphatidylethanolamine-binding protein 1 (PEBP1) contr

81 n is regulated by interactions of Raf-1 with phosphatidylethanolamine-binding protein 1 (PEBP1).

82 nases, and selected gene family members with phosphatidylethanolamine-binding protein domains.

83 nd assisted by two proteins with homology to phosphatidylethanolamine-binding proteins.

84 ation exposure, using (99m)Tc-duramycin as a phosphatidylethanolamine-binding radiopharmaceutical.

85 We previously demonstrated increased phosphatidylethanolamine biosynthesis subsequent to elev

86 thanolamine branch of the Kennedy pathway of phosphatidylethanolamine biosynthesis.

87 trol blood samples as an indication of liver phosphatidylethanolamine biosynthesis.

88 und to interact with phosphatidylcholine and phosphatidylethanolamine but not with phosphatidylserine

89 egulatory enzyme for de novo biosynthesis of phosphatidylethanolamine by the CDP-ethanolamine pathway

90 ng cell damage or activation, exposed PS and phosphatidylethanolamine collaborate to bind GLA domains

91 From day 100 postpartum on, phosphatidylethanolamine concentration was constant in t

92 Atg8, a phosphatidylethanolamine-conjugated protein, was previou

93 Atg7 and other autophagy factors and undergo phosphatidylethanolamine conjugation to preautophagic me

94 mine, derivatives of phosphatidylcholine and phosphatidylethanolamine containing a 7-nitro-2-1,3-benz

95 id bilayer triggered by a change in membrane phosphatidylethanolamine content, both in vivo and in vi

96 We now show that carboxyalkylpyrrole-phosphatidylethanolamine derivatives (CAP-PEs) are prese

97 o phosphatidylserine, phosphatidylcholine or phosphatidylethanolamine destabilizes the Na,K-ATPase.

98 There was no threshold level of phosphatidylethanolamine determining a sharp transition

99 ps that tend to form non-bilayer structures (phosphatidylethanolamine, diacylglycerol, and ergosterol

100 The polar lipid profile consisted of phosphatidylethanolamine, diphosphatidylglycerol, phosph

101 leaflet of vesicles composed of 1,2-dioleoyl-phosphatidylethanolamine (DOPE) and 1-palmitoyl-2-oleoyl

102 dylglycerol (DOPG), and zwitterionic dioleyl phosphatidylethanolamine (DOPE), with the addition of mo

103 anolamine (PE), DC-cholesterol, and dioleoyl-phosphatidylethanolamine (DOPE).

104 nolamine (DPoPE), or monomethylated dioleoyl phosphatidylethanolamine (DOPE-Me), at lipid/peptide mol

105 Synthetic mouse prions formed with phosphatidylethanolamine exhibit levels of specific infe

106 the plasma membrane and its loss results in phosphatidylethanolamine exposure on cell surfaces.

107 is revealed alteration to levels of specific phosphatidylethanolamine fatty acyl species in patients,

108 or the final step in Kennedy pathway forming phosphatidylethanolamine from CDP-ethanolamine.

109 te that AtMic60 contributes to the export of phosphatidylethanolamine from mitochondria and the impor

110 d by EASI(+)-MS, whereas EASI(-)-MS provided phosphatidylethanolamines, glycophospholipids and free f

111 in the following order: phosphatidylserine > phosphatidylethanolamine > phosphatidylcholine.

112 the abundances of endogenous cardiolipin and phosphatidylethanolamine halve during elongation of the

113 through binding to phospholipids containing phosphatidylethanolamine headgroups.

114 Duramycin, a peptide that binds phosphatidylethanolamine in enveloped virions and preclu

115 and offer insight into the critical role of phosphatidylethanolamine in fusion.

116 SDs) play a central role in the synthesis of phosphatidylethanolamine in numerous species of prokaryo

117 t enzyme that converts phosphatidylserine to phosphatidylethanolamine in the inner mitochondrial memb

118 namics of Atto646N-labeled sphingomyelin and phosphatidylethanolamine in the plasma membrane.

119 In contrast, ABCA4 transported phosphatidylethanolamine in the reverse direction.

120 tidylserine and other phospholipids, such as phosphatidylethanolamine, in ISG could play important ro

121 -associated protein 1 light chain 3 (LC3) to phosphatidylethanolamine, including Atg7, Atg3, and the

122 opagation with only one functional cofactor (phosphatidylethanolamine) induced the conversion of thre

123 ryl hemisuccinate, linoleamide/oleamide, and phosphatidylethanolamine inhibit and phosphatidic acid a

124 Phosphatidylethanolamine is a glycerophospholipid that,

125 The presence of phosphatidylethanolamine is however required for correct

126 Phosphatidylethanolamine is proposed to regulate mitocho

127 romyces cerevisiae, the majority of cellular phosphatidylethanolamine is synthesized by the mitochond

128 Phosphatidylserine and N,N-dimethyl phosphatidylethanolamine isomers in a bovine brain total

129 formulation contained N-lactobionyl-dioleoyl phosphatidylethanolamine (Lac-DOPE), a ligand for the as

130 accessibility we undertook quantification of phosphatidylethanolamine levels and species in patient a

131 The regulation of phosphatidylethanolamine levels by the MSF1 domain is an

132 mine fatty acyl species in patients, overall phosphatidylethanolamine levels were broadly unaffected

133 kinase, leading to a severe decrease in the phosphatidylethanolamine levels within P. falciparum, wh

134 e reaction products such as Amadori product, phosphatidylethanolamine-linked pyrrolecarbaldehyde and

135 avorable for the postulated binding of E7 to phosphatidylethanolamine lipid headgroups.

136 ESCs were evident in phosphatidylcholine and phosphatidylethanolamine lipid structures, essential and

137 single compound level, 168 sphingolipids, 36 phosphatidylethanolamine lipids, and 5 tobacco-related c

138 in the bilayer, through addition of lamellar phosphatidylethanolamine lipids, lowers lactose permease

139 old by liver PC or 1,2-dioleoyl-sn-glycero-3-phosphatidylethanolamine lipids.

140 perties, with particularly high affinity for phosphatidylethanolamine lipids.

141 K contained 4-10-fold higher amounts of lyso phosphatidylethanolamine (LPE) and 2-4-fold higher amoun

142 essential for maintaining adequate levels of phosphatidylethanolamine, LPE, and LPC in the cells.

143 Escherichia coli model cell system in which phosphatidylethanolamine membrane content can be systema

144 sphatidylcholine, phosphatidylserine, and/or phosphatidylethanolamine metabolism had a major effect o

145 n, respiratory competency, and mitochondrial phosphatidylethanolamine metabolism.

146 e-derived methyl groups for PC synthesis via phosphatidylethanolamine N-methyltransferase (PEMT) as i

147 Biosynthesis of hepatic choline via phosphatidylethanolamine N-methyltransferase (PEMT) play

148 pathway for PC biosynthesis is catalyzed by phosphatidylethanolamine N-methyltransferase (PEMT) that

149 by endogenous synthesis catalyzed by hepatic phosphatidylethanolamine N-methyltransferase (PEMT).

150 en choline-derived methyl groups are used by phosphatidylethanolamine N-methyltransferase (PEMT)] was

151 king key enzymes in PC biosynthesis, namely, phosphatidylethanolamine N-methyltransferase and hepatic

152 ulation of N-(lissamine rhodamine B sulfonyl)phosphatidylethanolamine (N-Rh-PE), a lipid marker of ex

153 rofolate reductase 1 (MTHFD1) rs2236225, and phosphatidylethanolamine-N-methyltransferase (PEMT) rs12

154 tion, possibly via impaired folate-dependent phosphatidylethanolamine-N-methyltransferase (PEMT)-PC s

155 ficantly more likely to have the G allele of phosphatidylethanolamine-N-methyltransferase (PEMT; V175

156 ors oleoylethanolamide (OEA) and N-palmitoyl-phosphatidylethanolamine (NPPE), which reduce food intak

157 directly uncouple Atg8 proteins attached to phosphatidylethanolamine on autophagosome membranes.

158 y native Tar contained as the majority lipid phosphatidylethanolamine or a related zwitterionic lipid

159 ed at opposite voltage polarities, either in phosphatidylethanolamine or phosphatidylcholine membrane

160 eicosatetraenoic acid (15HETE) conjugated to phosphatidylethanolamine (PE) (15HETE-PE).

161 nhibited by cholesterol (CHOL) as well as by phosphatidylethanolamine (PE) and -glycerol (PG) lipids.

162 The non-bilayer-forming phospholipids phosphatidylethanolamine (PE) and cardiolipin (CL) are r

163 cells or reconstituted in liposomes lacking phosphatidylethanolamine (PE) and containing only anioni

164 M is enriched in phosphatidylserine (PS) and phosphatidylethanolamine (PE) and contains minor populat

165 hingomyelin (dhSM) levels, and a decrease in phosphatidylethanolamine (PE) and ether phosphatidylchol

166 hosphatidylserine as well as 1-alkyl, 2-acyl phosphatidylethanolamine (PE) and phosphatidycholine wer

167 we show that neutral phospholipids, namely, phosphatidylethanolamine (PE) and phosphatidylcholine (P

168 chromatography (TLC) analysis indicated that phosphatidylethanolamine (PE) and phosphatidylcholine (P

169 constituents of cellular membranes, such as phosphatidylethanolamine (PE) and phosphatidylcholine.

170 chia coli with the remainder being primarily phosphatidylethanolamine (PE) and phosphatidylglycerol (

171 ISA, and reporter cell assays, we identified phosphatidylethanolamine (PE) and phosphatidylserine (PS

172 Phosphatidylethanolamine (PE) and phosphatidylserine (PS

173 inding of CD300a and CD300c to their ligands phosphatidylethanolamine (PE) and phosphatidylserine (PS

174 he inner mitochondrial membrane, synthesizes phosphatidylethanolamine (PE) and, in some cells, synthe

175 such as cardiolipin, phosphatidic acid, and phosphatidylethanolamine (PE) are proposed to generate n

176 membranes containing phosphatidylcholine and phosphatidylethanolamine (PE) bind factor Va with high a

177 reased expression (2- to 3-fold) of genes in phosphatidylethanolamine (PE) de novo biosynthesis resul

178 content and decreased ratio of hepatic PC to phosphatidylethanolamine (PE) have previously been linke

179 We found that PgpB was inhibited by phosphatidylethanolamine (PE) in a competitive mode in v

180 ymes in phospholipid metabolism that produce phosphatidylethanolamine (PE) in bacteria, protists, pla

181 the greatest decrease found in the level of phosphatidylethanolamine (PE) in root hairs and stripped

182 aazacyclododecane tetraacetic acid (Gd-DOTA) phosphatidylethanolamine (PE) into the surfactant were m

183 Phosphatidylethanolamine (PE) is a major cellular phosph

184 Phosphatidylethanolamine (PE) is a major phospholipid sp

185 Here we demonstrate that phosphatidylethanolamine (PE) is also a ligand for PS re

186 gy and then to completely native topology as phosphatidylethanolamine (PE) is increased from 0% to 70

187 and cerebellar phosphatidylcholine (PC) and phosphatidylethanolamine (PE) levels, while hippocampal

188 e TMD is predominantly alpha-helical, but in phosphatidylethanolamine (PE) membranes, the TMD changes

189 genetic inactivation of de novo synthesis of phosphatidylethanolamine (PE) mitigates OPA cytotoxicity

190 Phosphatidylethanolamine (PE) N-methyltransferase (PEMT)

191 phatidylcholine (PC) synthesis by way of the phosphatidylethanolamine (PE) N-methyltransferase (PEMT)

192 duced via the S-adenosylmethionine-dependent phosphatidylethanolamine (PE) N-methyltransferase (PEMT)

193 ubiquitin-like protein LC3 is conjugated to phosphatidylethanolamine (PE) on the inner and outer mem

194 ere demethylated either by substitution with phosphatidylethanolamine (PE) or chemically by using mon

195 lipid composition where the primary lipid is phosphatidylethanolamine (PE) or its methyl derivatives.

196 chondrial phospholipids cardiolipin (CL) and phosphatidylethanolamine (PE) play crucial roles in main

197 Phosphatidylethanolamine (PE) plays important roles for

198 r structure with phosphatidylserine (PS) and phosphatidylethanolamine (PE) restricted to the cytosoli

199 rophospholipids phosphatidylcholine (PC) and phosphatidylethanolamine (PE) show distinct reactivities

200 d (eas) mutants that harbor a disturbance in phosphatidylethanolamine (PE) synthesis display tachycar

201 o bushy stunt virus (TBSV) are enriched with phosphatidylethanolamine (PE) through a previously unkno

202 acyl chain from acyl-CoA to the headgroup of phosphatidylethanolamine (PE) to form N-acylphosphatidyl

203 rved in phospholipid content as the ratio of phosphatidylethanolamine (PE) to phosphatidylcholine (PC

204 rict most of the phosphatidylserine (PS) and phosphatidylethanolamine (PE) to the inner cytosolic lea

205 ious lipids, we have developed an artificial phosphatidylethanolamine (PE) vesicle-based Tomato bushy

206 Recently, higher quantities of phosphatidylethanolamine (PE) were also found at these c

207 e enrichments of phosphatidylserine (PS) and phosphatidylethanolamine (PE) were characterized with UP

208 phospholipids (phosphatidylcholine (PC) and phosphatidylethanolamine (PE)) and enhance the signals f

209 of phosphatidylinositol (PI), 12 species of phosphatidylethanolamine (PE), 18 species of phosphatidy

210 Phosphatidylethanolamine (PE), a cone-shaped phospholipi

211 ing head groups of phosphatidylcholine (PC), phosphatidylethanolamine (PE), and cholesterol were crea

212 phatidylcholine (PC) and non-bilayer-forming phosphatidylethanolamine (PE), are not clearly defined.

213 composed of polyethylene glycol (PEG-2000), phosphatidylethanolamine (PE), DC-cholesterol, and diole

214 e of the major components of the inner leaf, phosphatidylethanolamine (PE), displays a strong tendenc

215 s, particularly the nonbilayer-forming lipid phosphatidylethanolamine (PE), in mitochondrial function

216 we show that methylation of a phospholipid, phosphatidylethanolamine (PE), is a major consumer of SA

217 polyunsaturated phosphatidylcholine (PC) or phosphatidylethanolamine (PE), known to stimulate Na,K-A

218 Phosphatidylcholine (PC), phosphatidylethanolamine (PE), phosphatidylinositol (PI)

219 e ion/ion reactions of (13)C-TrEnDi-modified phosphatidylethanolamine (PE), phosphatidylserine (PS),

220 (13)C-TrEnDi-modified phosphatidylethanolamine (PE), phosphatidylserine (PS),

221 idylinositol (PI), and C43H80O8PN (C38:3), a phosphatidylethanolamine (PE), reached the Bonferroni co

222 at ABCA4 can transport N-11-cis-retinylidene-phosphatidylethanolamine (PE), the Schiff-base conjugate

223 demonstrate that Cu(2+) binds bivalently to phosphatidylethanolamine (PE), the second most abundant

224 re, we report that mitochondria deficient in phosphatidylethanolamine (PE), the second non-bilayer-fo

225 that this may result from trimethylation of phosphatidylethanolamine (PE), which partakes in biliary

226 ntly reduced levels of PS and its derivative phosphatidylethanolamine (PE).

227 ylserine decarboxylase Psd1, which generates phosphatidylethanolamine (PE).

228 he phospholipids phosphatidylserine (PS) and phosphatidylethanolamine (PE).

229 so be applied for cross-linking proteins and phosphatidylethanolamine (PE).

230 ant cofactor activity in brain is endogenous phosphatidylethanolamine (PE).

231 inverted when assembled in membranes lacking phosphatidylethanolamine (PE).

232 , that readily modify the amine headgroup of phosphatidylethanolamine (PE).

233 Phosphatidylethanolamines (PE) and phosphatidylcholines

234 executed via oxygenation of polyunsaturated phosphatidylethanolamines (PE) by 15-lipoxygenases (15-L

235 spholipid classes, phosphatidylcholine (PC), phosphatidylethanolamines (PE), and phosphatidylinositol

236 rdiolipins (CL), phosphatidylglycerols (PG), phosphatidylethanolamines (PE), phosphatidic acids (PA),

237 , we synthesized a novel polyethylene glycol-phosphatidylethanolamine (PEG-PE) conjugate with the TPP

238 Herein, we show that distearyl-phosphatidylethanolamine-PEG (DSPE-PEG), which forms 12-

239 living systems: phosphatidylcholines (PCs), phosphatidylethanolamine (PEs), and phosphatidylserines

240 Phosphatidylcholines (PCs) and phosphatidylethanolamines (PEs) are usually the most abu

241 f a series of phosphatidylcholines (PCs) and phosphatidylethanolamines (PEs) in nitrogen using a drif

242 ophosphatidylcholines, phosphatidylcholines, phosphatidylethanolamines (PEs), sphingomyelins, and tri

243 alize two phosphatidylserines (PSs) and five phosphatidylethanolamines (PEs).

244 icosatetraenoic acid (KETE) attached to four phosphatidylethanolamines (PEs).

245 Other lipids were imaged, including phosphatidylethanolamines, phosphatidic acids, sterols,

246 aflet of cell membranes (phosphatidylserine, phosphatidylethanolamine, phosphatidylcholine, and chole

247 pholipids commonly found in lipid membranes, phosphatidylethanolamine, phosphatidylcholine, and phosp

248 and phosphatidic acid, phosphatidylinositol, phosphatidylethanolamine, phosphatidylcholine, lyso-phos

249 ds in phosphatidylserine increased; and most phosphatidylethanolamines, phosphatidylcholines, sphingo

250 ed molecular species of phosphatidylcholine, phosphatidylethanolamine, phosphatidylglycerol, digalact

251 ix major lipid classes: phosphatidylcholine, phosphatidylethanolamine, phosphatidylglycerol, phosphat

252 choline/phosphatidylglycerol (PC/PG) and 1:1 phosphatidylethanolamine/phosphatidylglycerol (PE/PG) bi

253 ing phosphatidylcholine, phosphatidylserine, phosphatidylethanolamine, phosphatidylinositol, and the

254 dated at the N-terminus by transfer from PC, phosphatidylethanolamine, phosphatidylserine or phosphat

255 sphatidylcholine (PC) to mixtures of PC with phosphatidylethanolamine, phosphatidylserine or phosphat

256 rs2, and Neo1) are implicated in flipping of phosphatidylethanolamine, phosphatidylserine, and phosph

257 Similarly, phosphatidylethanolamine, phosphatidylserine, and sphing

258 osphatidylcholine, lyso-phosphatidylcholine, phosphatidylethanolamine, phosphatidylserine, triacylgly

259 different lipid species (phosphatidic acids, phosphatidylethanolamines, phosphatidylserines, phosphat

260 N-Arachidonoyl phosphatidylethanolamine-phospholipase D (NAPE-PLD), gly

261 itterionic 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphatidylethanolamine (POPE) and anionic 1-palmitoyl-

262 tidylcholine (POPC); 2) 1-palmitoyl 2-oleoyl-phosphatidylethanolamine (POPE); and 3) a mixture of 75%

263 me required for the mitochondrial pathway of phosphatidylethanolamine production, is closely monitore

264 species in the CL, phosphatidylcholine, and phosphatidylethanolamine profiles, was reduced in PGC-1a

265 be selective, since phosphatidylcholine and phosphatidylethanolamine promoted ISVP* formation, where

266 Phosphatidylethanolamine (PtdEtn) is the second major ph

267 ow the expected decreased methyl-pool and PC/phosphatidylethanolamine ratio and are resistant to the

268 sults in a deleterious decrease in the PC-to-phosphatidylethanolamine ratio.

269 Decreasing phosphatidylethanolamine reduces the rate of lipid mixin

270 Maillard type reaction products of phosphatidylethanolamine showed potent antioxidant activ

271 s) and depletion of phosphatidylinositol and phosphatidylethanolamine species containing unsaturated

272 presumed to include phosphatidylcholine and phosphatidylethanolamine species with two intact acyl ch

273 such as diacylglycerol lipase alpha, N-acyl-phosphatidylethanolamine-specific phospholipase D, and 1

274 l-3-sn-phosphatidylcholine/1,2-dioleoyl-3-sn-phosphatidylethanolamine/ Sphingomyelin/Cholesterol (35:

275 id entities (including phosphatidylcholines, phosphatidylethanolamines, sphingomyelins, phosphatidyls

276 ed Pcyt2alpha phosphorylation, activity, and phosphatidylethanolamine synthesis by 50-90%.

277 netic studies have validated the pathway for phosphatidylethanolamine synthesis from phosphatidylseri

278 sd2Deltadpl1Delta yeast strain, defective in phosphatidylethanolamine synthesis, with the P. knowlesi

279 of EPT1, thereby hindering the final step in phosphatidylethanolamine synthesis.

280 ner membrane-localized metabolic enzymes for phosphatidylethanolamine synthesis.

281 r binding affinity to phosphatidylserine and phosphatidylethanolamine than to phosphatidylcholine.

282 using mixtures of phosphatidic acid (PA) and phosphatidylethanolamine that reflects its physiological

283 racellular ceramide, phosphatidylserine, and phosphatidylethanolamine, that are exposed on the outer

284 eves its antibacterial activity by degrading phosphatidylethanolamine, the major component of bacteri

285 leoyl phosphatidylglycerol added to dioleoyl phosphatidylethanolamine, the proteins induced a dose-re

286 Phosphatidylethanolamine, the second most abundant eukar

287 ine N-methyltransferase (PEMT) that converts phosphatidylethanolamine to PC.

288 associated with an increase in the ratio of phosphatidylethanolamine to phosphatidylcholine in host

289 ps2p antagonistically regulate conversion of phosphatidylethanolamine to phosphatidylcholine.

290 o2, which is required for the methylation of phosphatidylethanolamine to produce phosphatidylcholine,

291 mbrane and exposure of phosphatidylserine or phosphatidylethanolamine to the radiotracer, which binds

292 lyzes the transfer of the sn-1-acyl chain of phosphatidylethanolamine to this N-terminal cysteine, ge

293 hat CD300a recognizes phosphatidylserine and phosphatidylethanolamine, two aminophospholipids exposed

294 brane insertion of LC3 protein modified with phosphatidylethanolamine using multiple coarse-grain sim

295 ing of human phosphatidylserine to bacterial phosphatidylethanolamine was identified as CT699.

296 plant tissues, phosphatidylcholine (PC) and phosphatidylethanolamine were major phospholipids, but g

297 led that cord-blood phosphatidylcholines and phosphatidylethanolamines were significantly decreased i

298 osed conserved glycine forms conjugates with phosphatidylethanolamine, which associate with membranes

299 en prion conversion and lipids, specifically phosphatidylethanolamine, which is a critical cofactor i

300 y that attaches AUTOPHAGY-RELATED8 (ATG8) to phosphatidylethanolamine, which then coats emerging auto