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2 rinary phosphate excretion suggests that the phosphaturic action of MEPE correlates with a significan
4 tions of bone, such as those mediated by the phosphaturic and 1,25(OH)2D-regulating hormone FGF23, an
5 lcium and by acting as a co-receptor for the phosphaturic and vitamin D-regulating hormone fibroblast
9 senger, the plasma levels of the circulating phosphaturic factor FGF-23 were measured using a c-termi
11 ms, and there is evidence that a circulating phosphaturic factor plays a role in the bone disease.
12 y expressed in such tumors, is a circulating phosphaturic factor that antagonizes renal Wnt-signaling
13 Fibroblast growth factor 23 (FGF23) is a phosphaturic factor that suppresses both sodium-dependen
18 factor-23 (FGF-23) is one of the circulating phosphaturic factors associated with renal phosphate was
19 te that elevated levels of novel circulating phosphaturic factors such as fibroblast growth factor 23
21 correlated with reduced serum levels of the phosphaturic hormone FGF23 and increased protein express
22 thyroidism (HPT) and serum elevations in the phosphaturic hormone FGF23, which may be maladaptive and
23 gical mechanism, potentially mediated by the phosphaturic hormone fibroblast growth factor-23 (FGF-23
24 ine and serum phosphorus levels, circulating phosphaturic hormone fibroblast growth factor-23 levels,
25 high or low circulating levels of the novel phosphaturic hormone fibroblastic growth factor 23 (FGF2
26 on of fibroblast growth factor 23 (FGF23), a phosphaturic hormone related to tumor necrosis factor al
27 roblast growth factor-23 (FGF-23) is a novel phosphaturic hormone that also inhibits renal 1alpha-hyd
29 f FGF23 may be to act as a counterregulatory phosphaturic hormone to maintain phosphate homeostasis i
31 vels of fibroblast growth factor (FGF)-23, a phosphaturic hormone, increase from the early stages of
33 measured concentrations of calciotropic and phosphaturic hormones, and urine and serum calcium and i
37 To determine whether serotonin modulates the phosphaturic response to PTH during conditions of dietar
38 n phosphate-replete rats, and attenuates the phosphaturic response to PTH in phosphate-deprived rats.
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