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1 ceptor complex may cause a resistance to the phosphaturic action of FGF23.
2 rinary phosphate excretion suggests that the phosphaturic action of MEPE correlates with a significan
3                                          The phosphaturic activity of FGF23 is abrogated by proteolyt
4 tions of bone, such as those mediated by the phosphaturic and 1,25(OH)2D-regulating hormone FGF23, an
5 lcium and by acting as a co-receptor for the phosphaturic and vitamin D-regulating hormone fibroblast
6 we found no evidence for intestinal-specific phosphaturic control mechanisms in humans.
7                                          The phosphaturic effect of intestinal phosphate is specific
8                                          The phosphaturic effect was present in FGF23-null mice, indi
9 senger, the plasma levels of the circulating phosphaturic factor FGF-23 were measured using a c-termi
10                        The regulation of the phosphaturic factor fibroblast growth factor 23 (FGF23)
11 ms, and there is evidence that a circulating phosphaturic factor plays a role in the bone disease.
12 y expressed in such tumors, is a circulating phosphaturic factor that antagonizes renal Wnt-signaling
13     Fibroblast growth factor 23 (FGF23) is a phosphaturic factor that suppresses both sodium-dependen
14 increased expression of the MEPE and another phosphaturic factor, Fgf23.
15  (XLH) by increasing levels of a circulating phosphaturic factor.
16                FGF23 is a candidate for this phosphaturic factor.
17 e that is a candidate for the tumor-secreted phosphaturic factor.
18 factor-23 (FGF-23) is one of the circulating phosphaturic factors associated with renal phosphate was
19 te that elevated levels of novel circulating phosphaturic factors such as fibroblast growth factor 23
20 hich results in the accumulation of putative phosphaturic factors, called phosphatonins.
21  correlated with reduced serum levels of the phosphaturic hormone FGF23 and increased protein express
22 thyroidism (HPT) and serum elevations in the phosphaturic hormone FGF23, which may be maladaptive and
23 gical mechanism, potentially mediated by the phosphaturic hormone fibroblast growth factor-23 (FGF-23
24 ine and serum phosphorus levels, circulating phosphaturic hormone fibroblast growth factor-23 levels,
25  high or low circulating levels of the novel phosphaturic hormone fibroblastic growth factor 23 (FGF2
26 on of fibroblast growth factor 23 (FGF23), a phosphaturic hormone related to tumor necrosis factor al
27 roblast growth factor-23 (FGF-23) is a novel phosphaturic hormone that also inhibits renal 1alpha-hyd
28       Fibroblast growth factor (FGF) 23 is a phosphaturic hormone that directly targets cardiac myocy
29 f FGF23 may be to act as a counterregulatory phosphaturic hormone to maintain phosphate homeostasis i
30                     Serum levels of FGF23, a phosphaturic hormone, increase at the earliest stages of
31 vels of fibroblast growth factor (FGF)-23, a phosphaturic hormone, increase from the early stages of
32 ic growth factor 23 (FGF23) is a circulating phosphaturic hormone.
33  measured concentrations of calciotropic and phosphaturic hormones, and urine and serum calcium and i
34                          Thus, the two major phosphaturic hormones, PTH and FGF23, have opposing effe
35                            Recently, several phosphaturic peptides such as fibroblast growth factor-2
36                                 The earliest phosphaturic response occurred after plasma PO4 and para
37 To determine whether serotonin modulates the phosphaturic response to PTH during conditions of dietar
38 n phosphate-replete rats, and attenuates the phosphaturic response to PTH in phosphate-deprived rats.
39                            Klotho is a novel phosphaturic substance that acts as an enzyme in the pro

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