ライフサイエンスコーパス: phosphoethanolamine

1 gmentation that yields hexadecanaldehyde and phosphoethanolamine.

2 out 2% of the total polar lipid, of ceramide phosphoethanolamine.

3 enosyl-L-methionine-dependent methylation of phosphoethanolamine.

4 showed that Pfpmt has strong specificity for phosphoethanolamine.

5 the parasite, is subsequently converted into phosphoethanolamine.

6 ification of lipid A with aminoarabinose and phosphoethanolamine.

7 phosphocholine and 1,2-dioleoyl-sn-glycero-3-phosphoethanolamine.

8 -acetyl)-1-palmitoyl-2-hydroxy-sn-gly cero-3-phosphoethanolamine.

9 ace by attaching it to the headgroup of lyso-phosphoethanolamine.

10 e, GABA) and decreased that of aspartate and phosphoethanolamine.

11 tamate, gamma-aminobutyric acid, taurine and phosphoethanolamine.

12 efflux of aspartate, glutamate, taurine and phosphoethanolamine.

13 s of aspartate, glutamate, GABA, taurine and phosphoethanolamine.

14 eleases of aspartate, glutamate, taurine and phosphoethanolamine.

15 P lyase (S1PL) yielding (2E)-hexadecenal and phosphoethanolamine.

16 egion by addition of 4-aminoarabinose and/or phosphoethanolamine.

17 lymyxin B was consistent with the absence of phosphoethanolamine.

18 ine, 8-epi-legionaminic acid, phosphate, and phosphoethanolamine.

19 rs derived from 1,2-dimyristoyl-sn-glycero-3-phosphoethanolamine, 1,2-distearoyl-sn-glycero-3-phospho

20 10-30mol% DOTAP or 1,2-dioleoyl-sn-glycero-3-phosphoethanolamine, 1-20mol% DOPE or 1,2-dioleoyl-3-tri

21 all cases), and 1,2-dimyristoyl-sn-glycero-3-phosphoethanolamine/1, 2-dimyristoyl-sn-glycero-3-phosph

22 (mol/mol), and 1,2-dipalmitoyl-sn-glycero-3-phosphoethanolamine/1, 2-dipalmitoyl-sn-glycero-3-phosph

23 tures with 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphoethanolamine (16:0-18:1PE, POPE) or 1-palmitoyl-2

24 1-[(2)H(31)]palmitoyl-2-oleoyl-sn-glycero-3-phosphoethanolamine (16:0-18:1PE-d(31)) with equimolar C

25 1-[(2)H(31)]palmitoyl-2-oleoyl-sn-glycero-3-phosphoethanolamine (16:0-18:1PE-d(31))/SM (1:1) and the

26 r 1-palmitoyl-2-docosahexaenoyl-sn-glycero-3-phosphoethanolamine (16:0-22:6PE, PDPE) and cholesterol.

27 31)]palmitoyl-2-docosahexaenoyl-sn-glycero-3-phosphoethanolamine (16:0-22:6PE-d(31)) or 1-[(2)H(31)]p

28 31)]palmitoyl-2-docosahexaenoyl-sn-glycero-3-phosphoethanolamine (16:0-22:6PE-d(31))/SM (1:1) in the

29 evels of the bisretinoids A2E and A2-glycero-phosphoethanolamine (A2-GPE).

30 Lipid A modification by the addition of phosphoethanolamine accounted for colistin resistance.

31 c proteins play an essential role in lipid A phosphoethanolamine addition and affect lipid A palmitat

32 but no palmitoylated lipid A, suggests that phosphoethanolamine addition is sufficient to confer EGT

33 racenylmethyl)-1, 2-dihexadecyl-sn-glycero-3-phosphoethanolamine (ADHP), synthesized from anthracenal

34 lutamate, GABA, glycine, taurine, glutamine, phosphoethanolamine, alanine, serine and the free fatty

35 Position 4 of Kdo is substituted by phosphoethanolamine, also present in position 6 of the b

36 was found to be extensively derivatized with phosphoethanolamine, aminoarabinose, 2-hydroxymyristate

37 and lipid A can be chemically modified with phosphoethanolamine, aminoarabinose, or glycine residues

38 izine leads to a sharp elevation of cellular phosphoethanolamine, an intermediate in the ethanolamine

39 s, derived from 1,2-dipalmitoyl-sn-glycero-3-phosphoethanolamine and 1,2-distearoyl-sn-glycero-3-phos

40 he two phosphates in the lipid A region with phosphoethanolamine and 4-aminoarabinose, which has been

41 e pyridoxal-phosphate-dependent breakdown of phosphoethanolamine and 5-phosphohydroxy-L-lysine.

42 hingosine-1-phosphate (S1P) lyase to produce phosphoethanolamine and a fatty aldehyde is the final de

43 Reperfusion caused a further increase in phosphoethanolamine and arachidonic acid levels and tran

44 n the CDP-ethanolamine pathway intermediates phosphoethanolamine and CDP-ethanolamine, and an increas

45 ld-type CRP, mutant CRP bound more avidly to phosphoethanolamine and could be purified by affinity ch

46 ic insult as evidenced by its attenuation of phosphoethanolamine and free fatty acid efflux.

47 phospholipid metabolism in gliomas involving phosphoethanolamine and glycerophosphocholine.

48 tumors, characterized by decreased levels of phosphoethanolamine and increased levels of glycerophosp

49 urring Escherichia coli lipids, we show that phosphoethanolamine and phosphoglycerol head groups impo

50 oligosaccharide of the LOS, the presence of phosphoethanolamine and sialic acid substituents can be

51 There were fewer O-acetyl groups and more phosphoethanolamine and sialic acid substitutions on the

52 ibly degrades sphingoid base-1-phosphates to phosphoethanolamine and the corresponding fatty aldehyde

53 -phosphocholine, 1,2-dihexanoyl-sn-glycero-3-phosphoethanolamine, and 1,2-dihexanoyl-sn-glycero-3-pho

54 phoethanolamine, 1,2-distearoyl-sn-glycero-3-phosphoethanolamine, and cholesterol, have provided stro

55 of phosphatidylcholine, phosphatidylserine, phosphoethanolamine, and phosphatidylinositol between co

56 The significance of increased asparagine, phosphoethanolamine, and taurine in the asthmatic patien

57 Asparagine, phosphoethanolamine, and taurine were significantly incr

58 n of the cationic sugar 4-aminoarabinose and phosphoethanolamine, and the LpxO-catalyzed addition of

59 ted singly or in combination with palmitoyl, phosphoethanolamine, and/or aminodeoxypentose residues.

60 3-deoxy-D-manno-octulosonic acid, and PEA is phosphoethanolamine] and four, three, or two hexoses, re

61 ine (POPC):1-palmitoyl-2-oleoyl-sn-glycero-3-phosphoethanolamine approximately 6:4 POPC:cholesterol<P

62 penta-acylated lipid A with an alpha-linked phosphoethanolamine attached to C-1 of GlcN (I) in the h

63 evious reports with N. meningitidis, loss of phosphoethanolamine attached to lipid A rendered strain

64 us free and the GPI with its nonreducing end phosphoethanolamine bearing a free amino group were synt

65 Mutation of residues implicated in zinc or phosphoethanolamine binding, or catalytic activity, rest

66 % biotinylated-cap-1,2-dioleoyl-sn-glycero-3-phosphoethanolamine (biotin-cap-DOPE).

67 atalyzes not only the first N-methylation of phosphoethanolamine but also the two subsequent N-methyl

68 ins, have increased amounts of palmitate and phosphoethanolamine but no aminoarabinose addition, sugg

69 nt msbB pmrA(Con) pagP::Tn10, which contains phosphoethanolamine but no palmitoylated lipid A, sugges

70 lta mutant relies on specific methylation of phosphoethanolamine but not phosphatidylethanolamine.

71 microM) significantly inhibited effluxes of phosphoethanolamine, but had no effect on glutamate, asp

72 ntenance of physiological levels of ceramide phosphoethanolamine by CERT in vivo is required to preve

73 choline (C6PC), 1, 2-dihexanoyl-sn-glycero-3-phosphoethanolamine (C6PE), and 1, 2-dihexanoyl-sn-glyce

74 Sphingomyelin (SM) and ceramide-phosphoethanolamines (cer-PEs) are related lipids presen

75 l phosphorus) that contained high amounts of phosphoethanolamine (compared to those of phosphocholine

76 h-, pneumococcal C-polysaccharide (PnC)-, or phosphoethanolamine-conjugated agarose columns.

77 be purified by affinity chromatography using phosphoethanolamine-conjugated Sepharose.

78 tants) to dodecylphosphocholine micelles and phosphoethanolamine-containing lipid bilayers.

79 AGXT2L1 catalyzed a similar reaction on phosphoethanolamine, converting it to ammonia, inorganic

80 instead synthesizes the SM analogue ceramide phosphoethanolamine (CPE) as the principal membrane sphi

81 Phosphoethanolamine cytidylyltransferase (ECT) catalyzes

82 CTP:phosphoethanolamine cytidylyltransferase (Pcyt2) is the

83 irect inhibition of the cytosolic enzyme CTP:phosphoethanolamine cytidylyltransferase (PCYT2).

84 GTA resistance and polymyxin resistance with phosphoethanolamine-decorated lipid A and demonstrate th

85 serum and polymyxin B resistance as well as phosphoethanolamine decoration of lipid A were restored

86 dependent inhibition of LpxT is required for phosphoethanolamine decoration of lipid A, which is show

87 cine, glycerol, phenylalanine, tyrosine, and phosphoethanolamine; decreases in choline-containing com

88 The phosphoethanolamine derivatives 1b and 2b readily form e

89 The aminolipid 1,2-dihexadecyl-sn-glycero-3-phosphoethanolamine (DHPE), which has been used extensiv

90 olding into mixed 1,2-dilauroyl-sn-glycero-3-phosphoethanolamine:diC(12:0)PC liposomes resulted in a

91 The chelate, 1,2-dimyristoyl-sn-glycero-3-phosphoethanolamine diethylenetriaminepentaacetic acid,

92 find that these lipids, and particularly the phosphoethanolamine dihydroceramide (PE DHC) fraction, s

93 ctions, phosphoglycerol dihydroceramides and phosphoethanolamine dihydroceramides, were prepared free

94 ds derived from 1,2-dimyristoyl-sn-glycero-3-phosphoethanolamine (DMPE) and 1,2-distearoyl-sn-glycero

95 lipid mixtures, 1,2-dimyristoyl-sn-glycero-3-phosphoethanolamine (DMPE):DMPC (7:3) and 1,2-dilauroyl-

96 d with two lipids, 1,2-dioleoyl-sn-glycero-3-phosphoethanolamine (DOPE) and 1,2-dioleoyl-3-trimethyla

97 c properties such as 2-dioleoyl-sn-glycero-3-phosphoethanolamine (DOPE) are integrated into the nanoc

98 Immobilization of 1,2-dioleoyl-sn-glycero-3-phosphoethanolamine (DOPE) liposome-gold nano-particle (

99 (PEI)(1.8 kDa), and 1,2-dioleyl-sn-glycero-3-phosphoethanolamine (DOPE) units (the nanocarrier is ref

100 tterionic liposome 1,2-Dioleoyl-sn-Glycero-3-Phosphoethanolamine (DOPE) were tethered on thiol monola

101 elike helper lipid 1,2-dioleoyl-sn-glycero-3-phosphoethanolamine (DOPE).

102 al (HII) phases of 1,2-dioleoyl-sn-glycero-3-phosphoethanolamine (DOPE).

103 yer composed of 1,2-dipalmitoyl-sn-glycero-3-phosphoethanolamine (DPPE) and subsequently 1,2-dipalmit

104 line (DPPC) and 1,2-dipalmitoyl-sn-glycero-3-phosphoethanolamine (DPPE) bilayers at 0, 25, 50, 75, an

105 choline (DPPC), 1,2-dipalmitoyl-sn-glycero-3-phosphoethanolamine (DPPE), cholesterol, and bradykinin

106 palmitoyl phosphocholine (DPPC), dipalmitoyl phosphoethanolamine (DPPE), dipalmitoyl phosphate (DPPA)

107 line (DSPC) and 1,2-dipalmitoyl-sn-glycero-3-phosphoethanolamine (DPPE), were used to study Gb3 packi

108 oline (DSPC) and 1,2-distearoyl-sn-glycero-3-phosphoethanolamine (DSPE) as a function of both pressur

109 gh proportion of 1,2 distearoyl-sn-glycero-3-phosphoethanolamine (DSPE) released up to 30% of payload

110 amine (DMPE) and 1,2-distearoyl-sn-glycero-3-phosphoethanolamine (DSPE), in the presence of nonexchan

111 ce of either anionic lipids, cholesterol, or phosphoethanolamine eliminates membrane binding at neutr

112 have a conserved trimannosyl core bearing a phosphoethanolamine (EthN-P) moiety on the third mannose

113 ed Man on the alpha1,2-Man that receives the phosphoethanolamine (EthN-P) moiety through which GPIs b

114 estigation of the biological significance of phosphoethanolamine extensions from lipooligosaccharide,

115 n significantly reduced levels of glutamate, phosphoethanolamine, GABA and arachidonic, myristic, pal

116 sretinoid A2-GPE is detected as sn-glycero-3-phosphoethanolamine (GPE) derivatized by two all-trans-r

117 one or two glycine residues attached to the phosphoethanolamine group at the nonreducing end.

118 osphorylated lipid A (LA) with and without a phosphoethanolamine group, and both hexa- and pentaacyla

119 y strong oriented H-bonds between waters and phosphoethanolamine groups at channel interfaces.

120 5), Phosphoethanolamine groups caused a marked attenuation o

121 active antibodies exist: one is dependent on phosphoethanolamine groups in LPS, and one is not.

122 omonoesters such as beta-glycerophosphate or phosphoethanolamine had no effect.

123 The ceramide phosphoethanolamine has a fatty amide profile with only

124 a, it has now been shown that loss of the 4' phosphoethanolamine has an impact on virulence in an ani

125 :1/OH lyso-PLs bearing the phosphoserine and phosphoethanolamine head groups, presented on albumin, w

126 g by lowering the kinetic barrier imposed by phosphoethanolamine head groups.

127 oinositol) and zwitterionic (phosphocholine, phosphoethanolamine) head groups, doubly mutated V172D/S

128 lyunsaturated lipids with phosphocholine and phosphoethanolamine headgroups.

129 e demonstrating the specificity of Pfpmt for phosphoethanolamine in vivo.

130 rabinose biosynthesis also prevented lipid A phosphoethanolamine incorporation and reduced the levels

131 in that is required for the incorporation of phosphoethanolamine into lipid A and for polymyxin B res

132 hree-step methylation reaction that converts phosphoethanolamine into phosphocholine, a precursor for

133 Phosphoethanolamine is a poor substrate.

134 that in the absence of the lactosyl group, a phosphoethanolamine is added to generate a new antigenic

135 For pathogenic Neisseria, phosphoethanolamine is added to lipid A by the phosphoet

136 modification of GPI anchors with side chain phosphoethanolamine is also discussed.

137 ification of lipid A with aminoarabinose and phosphoethanolamine is responsible for PmrA-regulated po

138 e, the anionic fluorescent lipid fluorescein phosphoethanolamine is seen to rearrange, forming worm-l

139 The N-methylation of phosphoethanolamine is the committing step in choline bi

140 I anchor is a complex structure comprising a phosphoethanolamine linker, glycan core, and phospholipi

141 maintain the balance of 4-aminoarabinose and phosphoethanolamine lipid A modifications.

142 d complex formation, specific binding to the phosphoethanolamine-lipid headgroup is also required, wh

143 to attach a PEG chain to several distearoyl phosphoethanolamine lipids, thereby differing from conve

144 ate exogenous 1-acyl-2-hydroxyl-sn-glycero-3-phosphoethanolamine (lyso-PtdEtn).

145 diseases may involve a perturbation of brain phosphoethanolamine metabolism.

146 Wild-type yeast cells, which inherently lack phosphoethanolamine methylation, acquire this activity a

147 pathway involving serine decarboxylation and phosphoethanolamine methylation.

148 ylcholine via a plant-like pathway involving phosphoethanolamine methylation.

149 of phosphoethanolamine to phosphocholine by phosphoethanolamine methyltransferase (PEAMT).

150 ite Plasmodium falciparum, a multifunctional phosphoethanolamine methyltransferase (PfPMT) catalyzes

151 P. falciparum phosphoethanolamine methyltransferase (Pfpmt) is a monop

152 Phosphoethanolamine methyltransferase (PMT) catalyzes th

153 sor, and the plant-like serine decarboxylase-phosphoethanolamine methyltransferase (SDPM) pathway, wh

154 hat knock-out of the PfPMT gene encoding the phosphoethanolamine methyltransferase enzyme completely

155 phobase methylation pathway catalyzed by the phosphoethanolamine methyltransferase in Plasmodium falc

156 ity and shown that its product is an unusual phosphoethanolamine methyltransferase with no human homo

157 e identification and characterization of the phosphoethanolamine methyltransferase, Pfpmt, of P. falc

158 The monopartite phosphoethanolamine methyltransferase, Pfpmt, plays an i

159 ative pathway named the serine-decarboxylase-phosphoethanolamine-methyltransferase (SDPM) pathway usi

160 two S-adenosylmethionine (AdoMet)-dependent phosphoethanolamine methyltransferases (PMT).

161 Phosphoethanolamine methyltransferases (PMTs) catalyze t

162 lipid synthesis in nematodes and compare the phosphoethanolamine methyltransferases (PMTs) from nemat

163 ber of a large family of known and predicted phosphoethanolamine methyltransferases (PMTs) recently i

164 Phosphoethanolamine methyltransferases add three methyl

165 med that MgrR effectively silences EptB; the phosphoethanolamine modification associated with EptB is

166 Moreover, the role that the phosphoethanolamine modification of lipid A plays in the

167 Phosphoethanolamine modification of lipid A was present

168 ogues with a glycine residue attached to the phosphoethanolamine moiety at the nonreducing end to for

169 alogues contain an aryl group linked to an O-phosphoethanolamine moiety through amide, sulfonamide, o

170 and is catalyzed by S-adenosyl-L-methionine:phosphoethanolamine N-methyltransferase (PEAMT, EC ).

171 and we measure differences among variants of phosphoethanolamine N-methyltransferase and actin-I acro

172 Parasites lacking the phosphoethanolamine N-methyltransferase enzyme, which ca

173 emical screening identified 11 inhibitors of phosphoethanolamine N-methyltransferase that block paras

174 enzyme of the plant Cho-synthesis pathway is phosphoethanolamine N-methyltransferase, which catalyzes

175 rhodamine B 1,2-dihexadecanoyl-sn-glycero-3-phosphoethanolamine (N-Rh-DPPE) were used as fluorescent

176 rhodamine B 1,2-dihexadecanoyl-sn-glycero-3-phosphoethanolamine (N-Rh-DPPE).

177 eight glycoforms, containing the addition of phosphoethanolamine, N-acetylgalactosamine, and N-acetyl

178 phocholine and 1,2-dipalmitoyl- sn-glycero-3-phosphoethanolamine- N-[methoxy(polyethylene glycol-2000

179 the exchange of 1,2-dipalmitoyl-sn-glycero-3-phosphoethanolamine-n,n-Dimethyl-n-(2',2',6', 6'-tetrame

180 for biotin-cap-1,2-dipalmitoyl-sn-glycero-3-phosphoethanolamine-N-(7-nitro-2-1,3-benz oxadiazol-4-yl

181 d lipid, NBD-DOPE [1,2-dioleoyl-sn-glycero-3-phosphoethanolamine-N-(7-nitro-2-1,3-benzoxadiazol-4-y l

182 tidylcholine and 1,2-distearoyl-sn-glycero-3-phosphoethanolamine-N-[amino (polyethylene glycol)-2000]

183 ne and DSPE-PEG [1,2-distearoyl-sn-glycero-3-phosphoethanolamine-N-[amino(polythylene glycol)].

184 ugates (i.e. , 1, 2-dipalmitoyl-sn-glycero-3-phosphoethanolamine-N-[methoxypoly(e thylen e glycol)200

185 arboxamide and 1, 2-dipalmitoyl-sn-glycero-3-phosphoethanolamine-N-[methoxypoly(e thylen e oxide)5000

186 We have previously shown that phosphoethanolamine on the lipid A portion of lipooligos

187 However, the in vivo substrate (phosphoethanolamine or phosphatidylethanolamine) is not

188 Cho), are synthesized by the transfer of the phosphoethanolamine or phosphocholine polar head group,

189 ipids as 1-O-alk-1'-enyl-2-acyl-sn-glycero-3-phosphoethanolamine or plasmenylethanolamine (PME) throu

190 We also show that the phosphoethanolamine (P-EtN) addition on heptose I in E.

191 In particular, it contains a phosphoethanolamine (P-Etn) instead of a free phosphate

192 ng transcriptional activator peptide-PEG1000-phosphoethanolamine (PE) (a cell-penetrating enhancer),

193 Phospholipids containing phosphoethanolamine (PE) headgroups within biological me

194 negative curvature lipids such as those with phosphoethanolamine (PE) headgroups.

195 ifferent minimum threshold concentrations of phosphoethanolamine (PE) lipids to reconstruct the membr

196 varying compositions of phosphocholine (PC), phosphoethanolamine (PE), cholesterol and the photo-poly

197 g lyso-phospholipid, phosphatidic acid (PA), phosphoethanolamine (PE), phosphatidylserine (PS), phosp

198 the native heptameric form at 2.3 A, and the phosphoethanolamine (PE)-bound octameric form at 2.7 A.

199 1,2-O-Bis[11-(Z)-hexadecenoyl]-sn-glycero-3-phosphoethanolamine (PE-16:1 omega 5c/16:1 omega 5c) was

200 ipid signals in the range of m/z 250-350 and phosphoethanolamines (PE) m/z 700-800 observed in negati

201 Phosphoethanolamine (PEA) decoration of lipid A increase

202 Loss of phosphoethanolamine (PEA) from the lipid A of gonococcal

203 Phosphoethanolamine (PEA) residues on the second heptose

204 The gamma-chain Hep II contains two phosphoethanolamine (PEA) substitutions at C3 and C6/7.

205 se (PMT) catalyzes the triple methylation of phosphoethanolamine (pEA) to pCho.

206 sferase (PfPMT) catalyzes the methylation of phosphoethanolamine (pEA) to phosphocholine for membrane

207 PfPMT methylates phosphoethanolamine (pEA) to phosphocholine for use in m

208 Further, a mutation in lptA, the phosphoethanolamine (PEA) transferase responsible for mo

209 Lpt-3, a phosphoethanolamine (PEA) transferase, and LgtG, a gluco

210 ns heptose residues that can be decorated by phosphoethanolamine (PEA).

211 revealed three major components present in a phosphoethanolamine (PEA)0 and a PEA1 series.

212 ingly, blocking of the PCh-binding site with phosphoethanolamine (PEt) dramatically increased the bin

213 d a gene, lpt3, required for the addition of phosphoethanolamine (PEtn) at the 3 position on the beta

214 monoclonal antibody (Mab B5) that recognises phosphoethanolamine (PEtn) attached to the inner core of

215 ain lipopolysaccharide (LPS) modified with a phosphoethanolamine (pEtN) group at position 7 of the ou

216 f lgtG and determines whether a glucose or a phosphoethanolamine (PEtn) is added at a specific positi

217 ossess this epitope are immunotypes in which phosphoethanolamine (PEtn) is linked to the 3-position o

218 The zwitterionic lipids possess phosphoethanolamine (PEtn) linked to one or more GlcNAc

219 Addition of a phosphoethanolamine (pEtN) moiety to the outer 3-deoxy-D

220 ther these effects require its metabolism to phosphoethanolamine (PEtn) or PtdEtn.

221 4-amino-4-deoxy-l-arabinose (l-Ara4N) and/or phosphoethanolamine (pEtN) substituents.

222 as 4-amino-4-deoxy-l-arabinose (l-Ara4N) and phosphoethanolamine (pEtN) to Escherichia coli and Salmo

223 4-aminoarabinose (Ara4N) to the lipid A and phosphoethanolamine (pEtN) to the lipid A and core.

224 nsible for the transfer of the amino-residue phosphoethanolamine (pEtN) to the lipid A of V. cholerae

225 In this work, we identify a gene encoding a phosphoethanolamine (pEtN) transferase (Cj0256) that ser

226 er jejuni identified a gene encoding a novel phosphoethanolamine (pEtN) transferase Cj0256, renamed E

227 C. jejuni identified a gene encoding a novel phosphoethanolamine (pEtN) transferase, EptC (Cj0256), t

228 he lipid A of Helicobacter pylori contains a phosphoethanolamine (pEtN) unit directly linked to the 1

229 Phosphoethanolamine (pEtN)-modified lipid A species are

230 f these data allowed the identification of a phosphoethanolamine (pEtN)-modified variant of the N-gly

231 ng mice with synthetic supplement containing phosphoethanolamine (PHO-S) promoted an accentuated decr

232 i PHOSPHO1 is a dual-specific phosphocholine/phosphoethanolamine phosphatase enriched in mineralizing

233 orphism of 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphoethanolamine (POPE), using differential scanning

234 omposed of 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphoethanolamine (POPE), with or without rough Escher

235 hosphocholine (POPC) or 1-palmitoyl 2-oleoyl phosphoethanolamine (POPE).

236 with amine-containing substituents, such as phosphoethanolamine, reduces the overall net negative ch

237 xEHP (Hp0021), followed by the addition of a phosphoethanolamine residue catalyzed by EptAHP (Hp0022)

238 substituents 4-amino-4-deoxy-L-arabinose and phosphoethanolamine respectively.

239 s of aspartate, glutamate, taurine, GABA and phosphoethanolamine rose during ischemia and then declin

240 addition of monosaccharides, fatty acid, and phosphoethanolamine(s) to phosphatidylinositol (PI).

241 ly mannosylated GPI structure containing one phosphoethanolamine side chain; and (iv) the mitochondri

242 and 1-alk-1-enyl-2-arachidonoyl-sn-glycero-3-phosphoethanolamine species.

243 elated pathways, such as changes in ceramide phosphoethanolamines, sphingomyelin, carnitines, tyrosin

244 olecules was phosphorylated, presumably by a phosphoethanolamine substituent.

245 However, loss of phosphoethanolamine substitution from the lipid A compon

246 respectively, and do not contain additional phosphoethanolamine substitution in their core glycan st

247 ions from lipooligosaccharide, we found that phosphoethanolamine substitutions from the heptose II gr

248 Our results support a role for lipid A phosphoethanolamine substitutions in resistance of this

249 sis and binding to S-adenosyl-methionine and phosphoethanolamine substrates.

250 mplexed with 1-stearoyl-2-palmitoylglycero-3-phosphoethanolamine suggested why these 3-Cl-AHPC groups

251 t releases of aspartate, glutamate, glycine, phosphoethanolamine, taurine and GABA from the rat cereb

252 superfusate levels of aspartate, glutamate, phosphoethanolamine, taurine, gamma-aminobutyric acid (G

253 1-sulfonyl]-1,2-dihexadecanoyl-sn- glycero-3-phosphoethanolamine, that are sensitive to water content

254 They display >70% decrease in ceramide phosphoethanolamine (the sphingomyelin analog in Drosoph

255 PMTs) catalyze the three-step methylation of phosphoethanolamine to form phosphocholine, a critical s

256 ssion in E coli resulting in the addition of phosphoethanolamine to lipid A.

257 osphatidylcholine requires the conversion of phosphoethanolamine to phosphocholine by phosphoethanola

258 hree of the methylations required to convert phosphoethanolamine to phosphocholine.

259 ates that HcPMT1 catalyzes the conversion of phosphoethanolamine to phosphomonomethylethanolamine (pM

260 ipid A modification involves the addition of phosphoethanolamine to the 1 and 4' headgroup positions

261 lpt-3, that is required for the addition of phosphoethanolamine to the 3-position (PEtn-3) on the be

262 1 encodes a membrane-bound enzyme catalysing phosphoethanolamine transfer onto bacterial lipid A.

263 single zinc ion may be sufficient to support phosphoethanolamine transfer.

264 MCR-1 is a member of the phosphoethanolamine transferase enzyme family, with expr

265 e crystal structure of a full-length lipid A phosphoethanolamine transferase from Neisseria meningiti

266 osphoethanolamine is added to lipid A by the phosphoethanolamine transferase specific for lipid A, wh

267 ely to be carried out by three different GPI-phosphoethanolamine transferases (GPI-PETs).

268 amine to the 1 and 4' headgroup positions by phosphoethanolamine transferases.

269 -O-hexadecyl-2-N-methylcarbamyl-sn-glycero-3-phosphoethanolamine was covalently attached to the CH-Se

270 xadecanoyl-2-(9-Z-octadecenoyl)-sn-glycero-3-phosphoethanolamine) was added.

271 iazol-4-yl)-1,2-dihexadecanoyl-sn- glycero-3-phosphoethanolamine, was found to be relatively insensit

272 fragment ions with three phosphates and one phosphoethanolamine were detected in all LOS analyzed.

273 ls of all amino acids, with the exception of phosphoethanolamine, were elevated during reperfusion.

274 in hexagonal phase 1,2-dioleoyl-sn-glycero-3-phosphoethanolamine, which further indicates that FP23 p

275 led to rapid accumulation of its substrate, phosphoethanolamine, which is itself an inhibitor of mit

276 mixtures of DOPE (1,2-dioleoyl-sn-glycero-3-phosphoethanolamine) with phosphatidylcholines (PCs) of

277 ethanolamine and 1,2-distearoyl-sn-glycero-3-phosphoethanolamine, with an exchangeable form of choles