ライフサイエンスコーパス: phosphoramide mustard

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1 ntracellular concentrations of the cytotoxic phosphoramide mustard.

2 resulted in rapid expulsion of the cytotoxic phosphoramide mustard.

3 s treated with melphalan, mechlorethamine or phosphoramide mustard.

4 base-catalyzed beta-elimination of cytotoxic phosphoramide mustard.

5 The more active acyclic 4-nitrobenzyl phosphoramide mustard (7) showed 167 500x selective cyto

6 unds were more potent than the corresponding phosphoramide mustards against V-79 Chinese hamster lung

7 ed to provide a pathway for expulsion of the phosphoramide mustard alkylating agent.

8 Cyclic and acyclic nitroaryl phosphoramide mustard analogues were activated by E. col

9 t is metabolized by cytochrome P450 to yield phosphoramide mustard and acrolein, which alkylate DNA a

10 CPA undergoes metabolic activation to phosphoramide mustard and nornitrogen mustard (NOR) whic

11 y bound intermediates of mechlorethamine and phosphoramide mustard assumed thermodynamically stable c

12 ts in the highest quartile of o-carboxyethyl-phosphoramide mustard exposure had a 5.9-fold higher ris

13 ontrol compounds 5 and 15, which lack in the phosphoramide mustard group.

14 amide and 4-hydroperoxyifosfamide but not to phosphoramide mustard, ifosfamide mustard, melphalan, or

15 ere cross-resistant to other OAPs but not to phosphoramide mustard, ifosfamide mustard, melphalan, or

16 Mechlorethamine and phosphoramide mustard induced intrastrand crosslinks bet

17 4-HC generates two active metabolites, phosphoramide mustard (PM) and acrolein.

18 iques were used to study the partitioning of phosphoramide mustard (PM) and its aziridinium ions amon

19 amide (4-HDC), a progenitor of acrolein, and phosphoramide mustard (PM).

20 icancer prodrugs that liberate the cytotoxic phosphoramide mustards (PM, IPM, and tetrakis-PM) via be

21 ter than 1) and/or excellent cytotoxicity of phosphoramide mustard released.

22 diamidates have been designed as prodrugs of phosphoramide mustard requiring bioreductive activation.

23 eduction by DTD followed by expulsion of the phosphoramide mustard substituent from the hydroquinone.

24 rticularly for the metabolite o-carboxyethyl-phosphoramide mustard, whose area under the curve varied

25 t all these compounds spontaneously liberate phosphoramide mustards with half-lives in the range of 0

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