ライフサイエンスコーパス: photoinactivation

1 indirect mechanism to the overall bacterial photoinactivation.

2 asma membrane, cannot be re-formed after Clc photoinactivation.

3 esicle re-formation does not occur after Clc photoinactivation.

4 nt membrane internalization occurs after Clc photoinactivation.

5 y and/or protection against oxygen-dependent photoinactivation.

6 reased (approximately 2-fold) sensitivity to photoinactivation.

7 ly, 2.7- and 4-fold increased sensitivity to photoinactivation.

8 or in combination with Mg2+, protect against photoinactivation.

9 esence of 12 microM dequalinium led to rapid photoinactivation.

10 exhibiting a 3-fold increase in the rate of photoinactivation.

11 which was directly correlated to luciferase photoinactivation.

12 indicated that the mutant was susceptible to photoinactivation.

13 d because DOM acted as an antioxidant toward photoinactivation, a phenomenon recently established for

14 erial cells, as shown by the higher bacteria photoinactivation activity retained after washing the ba

15 We used acute photoinactivation and found that loss of Dynamin functio

16 The results of photoinactivation and kinetic and bioluminescence studie

17 unteract a lower intrinsic susceptibility to photoinactivation, and C. radiatus thus did not need to

18 The mutants had a high rate of photoinactivation, and many mutants showed an up to 1000

19 e retrieval has also been seen upon Clathrin photoinactivation, and superresolution imaging indicated

20 nthesis, nonphotochemical quenching and PSII photoinactivation arises from changes in the abundances

21 e mutant proved to be extremely sensitive to photoinactivation at high light intensities, exhibiting

22 nd these PSII centers were very sensitive to photoinactivation at high light intensities.

23 Aggregation lowers the efficiency of photoinactivation because of self-quenching of the dye.

24 olution imaging indicated that acute Dynamin photoinactivation blocked Clathrin and alpha-adaptin rel

25 Effective protection against photoinactivation by 150 microM GS-Succ-BP is provided b

26 We first demonstrate that Clc photoinactivation does not impair synaptic-vesicle fusio

27 e exogenous indirect mechanism by conducting photoinactivation experiments with eight health-relevant

28 indirect mechanisms contribute to bacterial photoinactivation in natural surface waters.

29 absorbance and depth, suggesting endogenous photoinactivation is a major pathway for bacterial decay

30 s of tryptophan-like fluorescence paralleled photoinactivation kinetics and because DOM acted as an a

31 cous drag impeding traveling waves; targeted photoinactivation locally interrupts this compensation.

32 gesting that although the exogenous indirect photoinactivation mechanism may be active against Ent. f

33 A time-dependent photoinactivation occurs upon irradiation at long wavele

34 loride-limiting conditions, with a t(1/2) of photoinactivation of 2.6 min under chloride-limiting con

35 olved organic matter in marsh water enhanced photoinactivation of a laboratory strain of Enterococcus

36 ation allowed selective and potent UV-driven photoinactivation of both homomeric (GluA2) and heterome

37 Moreover, photoinactivation of Dynamin in shi(ts1) animals convert

38 ynthetic photosensitizers generally enhanced photoinactivation of Gram-positive facultative anaerobes

39 We used photoinactivation of identified neurons and pharmacologi

40 structure are significantly involved in the photoinactivation of phosphatase because a loss of trypt

41 Thus, photoinactivation of phosphatase can be significantly sl

42 tes but can also significantly quench direct photoinactivation of phosphatase.

43 adiance-sensitive phenotype with significant photoinactivation of photosystem II (PSII), indicated by

44 o HL benefited cells by reducing the rate of photoinactivation of PSII under high light.

45 the triple mutant revealed that the rate of photoinactivation of PSII was the same in wild-type and

46 rain of Enterococcus faecalis, but depressed photoinactivation of sewage-sourced enterococci and E. c

47 In biological media, photoinactivation of Staphylococcus aureus was evaluated

48 e live imaging of the synapto-pHluorins with photoinactivation of Syt I, through fluorescein-assisted

49 ease in neurotransmitter release after acute photoinactivation of the V0 a1-I subunit in neuronal pai

50 for application in cancer therapy and in the photoinactivation of viruses.

51 smaller T. pseudonana, photosystem II (PSII) photoinactivation outran the clearance of PSII protein s

52 Their photoinactivation performance was tested against Escheri

53 ditional investigations on the nature of the photoinactivation process strongly suggested that BPTC c

54 (1) FlAsH-FALI-mediated protein photoinactivation rapidly and specifically disrupts Clc

55 sensitizers either reduced or did not affect photoinactivation rate constants.

56 , a firefly luciferin analogue, was a potent photoinactivation reagent for luciferase.

57 he bc(1) complex by hematoporphyrin-promoted photoinactivation resulted in the complex becoming proto

58 Substrate studies, inhibition kinetics, photoinactivation studies, and photolabeling experiments

59 3) After complete photoinactivation, the level of incorporation of radioac

60 We employ FlAsH-FALI-mediated protein photoinactivation to rapidly (3 min) and specifically di

61 mutant exhibited an enhanced sensitivity to photoinactivation under chloride-limiting conditions, wi

62 onstrate a noninvasive technique for protein photoinactivation using a transgenically encoded tag.

63 and the photoaffinity label was specific: 1) photoinactivation was inhibited in the presence of a non

64 Direct photoinactivation was slowed by more than 50% in the pre

65 ter VL irradiation, Sigma-TiO2 showed higher photoinactivation, whereas S-TiO2 and P-25 showed modera