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1 ith the key signaling protein constitutively photomorphogenic 1 (COP1) and induction of UV-B-protecti
2 is study, we identify mammalian constitutive photomorphogenic 1 (COP1) as a novel E3 ubiquitin ligase
3 n dark-grown wild-type roots by constitutive photomorphogenic 1 (COP1) E3 ligase and 26S proteasome a
4                                 Constitutive photomorphogenic 1 (COP1) is a p53-targeting E3 ubiquiti
5                     Arabidopsis constitutive photomorphogenic 1 (COP1) is an E3 ubiquitin ligase that
6            Here, we show that CONSTITUTIVELY PHOTOMORPHOGENIC 1 (COP1) is required for UV-B-induced n
7                             The constitutive photomorphogenic 1 (COP1) protein of Arabidopsis functio
8  followed by interaction with CONSTITUTIVELY PHOTOMORPHOGENIC 1 (COP1), a major factor in UV-B signal
9                               Constitutively photomorphogenic 1 (COP1), a RING finger ubiquitin ligas
10  by suppressing the activity of CONSTITUTIVE PHOTOMORPHOGENIC 1 (COP1), resulting in activation of th
11 rphogenesis involves UVR8 and CONSTITUTIVELY PHOTOMORPHOGENIC 1 (COP1)-mediated repression of PIF4 tr
12  integrators DE-ETIOLATED 1 and CONSTITUTIVE PHOTOMORPHOGENIC 1 maintain heterochromatin in a deconde
13                         COP1 (constitutively photomorphogenic 1) is a RING-finger-containing protein
14 ulated development, the COP1 (constitutively photomorphogenic 1) protein is characterized by a RING-f
15 or of photomorphogenesis, COP1 (constitutive photomorphogenic 1), is not a chief regulator of the ear
16 attenuating the activity of the CONSTITUTIVE PHOTOMORPHOGENIC 1-SUPPRESSOR OF PHYA-105 1 (COP1-SPA1)
17 ds in etiolated roots of cop (constitutively photomorphogenic)1, cop9, and det (de-etiolated)1 mutant
18 ator of Cullins-1 (ROC1), and constitutively photomorphogenic-1.
19 s the smallest subunit of the constitutively photomorphogenic 9 (COP9) signalosome (CSN), which consi
20 rpins against subunits of the COnstitutively Photomorphogenic-9- (COP9-) signalosome (CSN) in somatic
21 reduced chlorophyll in leaves and additional photomorphogenic abnormalities when the seedlings are gr
22 r AtZFP1 in shoot development, downstream of photomorphogenic activation.
23                     We found that GI acts in photomorphogenic and circadian blue light signaling path
24 cial environmental signal that controls many photomorphogenic and circadian responses in plants.
25 part through downregulation of BNQ-dependent photomorphogenic and developmental signaling pathways.
26  opposing in cis the promoting effect of the photomorphogenic and thermomorphogenic regulator Phytoch
27           Mutants of COP1 are constitutively photomorphogenic, and this has been attributed to their
28 lecular basis of translational regulation in photomorphogenic Arabidopsis thaliana, we adopted a ribo
29 mutant seedlings also display characteristic photomorphogenic cellular differentiation and elevated e
30 iate signal transduction pathways leading to photomorphogenic changes in development.
31         rfd3 and rfd4 belong to the group of photomorphogenic cop/det/fus mutants.
32 alizes in nuclear bodies with CONSTITUTIVELY PHOTOMORPHOGENIC (COP) 1, a RING motif-containing E3 lig
33                            This constitutive photomorphogenic (COP) phenotype was not observed for mu
34                 The pleiotropic CONSTITUTIVE PHOTOMORPHOGENIC (COP), DEETIOLATED (DET), and FUSCA (FU
35 the dark, N282 expression led to pleiotropic photomorphogenic cotyledon development, including cellul
36                 The pleiotropic constitutive photomorphogenic/deetiolated/fusca (cop/det/fus) mutants
37 duce the transition from skotomorphogenic to photomorphogenic development (deetiolation) in dark-germ
38 ance of COP1, a transcriptional repressor of photomorphogenic development [1] [2].
39 e-mediated blue light regulation of seedling photomorphogenic development and genome expression profi
40 ncluding shoot and root growth, vascular and photomorphogenic development and leaf senescence.
41 ithin the nucleus as a repressor of seedling photomorphogenic development and that high inactivation
42 ifically as a light-inactivable repressor of photomorphogenic development and to elucidate the functi
43 omponent mechanism in the broader control of photomorphogenic development by phytochrome and cryptoch
44 downstream regulators, dictate the extent of photomorphogenic development in a quantitative manner.
45 F3, and PIF4/5 as an underlying mechanism of photomorphogenic development in Arabidopsis thaliana.
46 chanism between PIFs and HFR1 that underlies photomorphogenic development in Arabidopsis thaliana.
47  complex initially defined as a repressor of photomorphogenic development in Arabidopsis.
48 gy is the collective repression of premature photomorphogenic development in dark-grown seedlings by
49 t abolish the complex result in constitutive photomorphogenic development in darkness and pleiotropic
50 etic screens, one for mutations resulting in photomorphogenic development in darkness and the other f
51 ys a key role in the repression of the plant photomorphogenic development in darkness.
52 -box protein, positively regulates facets of photomorphogenic development in response to light.
53           Plant photoreceptors that regulate photomorphogenic development include red/far-red-light-a
54       Under conditions which induce or mimic photomorphogenic development including growth in the lig
55 e ability to switch from skotomorphogenic to photomorphogenic development is essential for seedling s
56               PIFs are central regulators of photomorphogenic development that act to promote stem gr
57 icotyledon seedling undergoes organ-specific photomorphogenic development when exposed to light.
58 Y5 is directly correlated with the extent of photomorphogenic development, and that the COP1-HY5 inte
59 y-photoreceptor system to induce appropriate photomorphogenic development, but at excessive levels, s
60 OP1 acts as a light-inactivable repressor of photomorphogenic development, but its molecular mode of
61      COP1 acts within the nucleus to repress photomorphogenic development, but light inactivates COP1
62 emitted by dysfunctional chloroplasts impact photomorphogenic development, but the molecular link bet
63 ce of COP1 for the appropriate regulation of photomorphogenic development.
64 negatively regulate COP1, a key repressor of photomorphogenic development.
65 ucible genes, promoting their expression and photomorphogenic development.
66 th intact WD-40 repeats are able to suppress photomorphogenic development.
67 , a bZIP protein and a positive regulator of photomorphogenic development.
68 anism for its control of gene expression and photomorphogenic development.
69 undance of COP1, an established repressor of photomorphogenic development.
70 s genes that are necessary for repression of photomorphogenic development.
71 sisting in etioplast development, and normal photomorphogenic development.
72  originally identified as a key regulator of photomorphogenic development.
73 nes (BRASSINOSTEROID-6-OXIDASE, CONSTITUTIVE PHOTOMORPHOGENIC DWARF, and DIMINUTO) and one brassinost
74 ar data support that SCF(EBF1/2) function as photomorphogenic E3s during seedling development.
75 yB double mutants were less sensitive to the photomorphogenic effects of UV-B.
76 le to unequivocally associate several of the photomorphogenic effects seen in phyB mutants with phyto
77   The light-induced stabilization of HFR1, a photomorphogenic factor targeted for degradation by COP1
78 ng pathways, also plays an important role in photomorphogenic growth and light-regulated gene express
79 omplex, which targets positive regulators of photomorphogenic growth for degradation by the proteasom
80                     In dark-grown seedlings, photomorphogenic growth is suppressed by the action of t
81 5 work in an antagonistic manner to regulate photomorphogenic growth.
82  triggering the transition from etiolated to photomorphogenic growth.
83 ortant roles than HYH in blue light-mediated photomorphogenic growth.
84 actors (PIF1, 3, 4, and 5) is constitutively photomorphogenic in darkness establishes that these fact
85 d red or white light, phyA mutants are hyper-photomorphogenic in many respects.
86 /far-red photoreceptor phytochrome A and are photomorphogenic in the dark.
87 tubule dynamics for hours without triggering photomorphogenic inhibition of growth, we used Arabidops
88                           The root-perceived photomorphogenic inhibition of shoot greening demonstrat
89 ants that define four additional pleiotropic photomorphogenic loci and a null mutant allele of the pr
90 h arm of chromosome 4, and is not allelic to photomorphogenic loci identified previously.
91  which, cin4, is allelic to the constitutive photomorphogenic mutant fus9/cop10.
92            Through positional cloning of the photomorphogenic mutant hy1, the Arabidopsis HO (designa
93 niscent of the previously characterised dark-photomorphogenic mutant, de-etiolated 3 (det3); conseque
94       We report that a previously identified photomorphogenic mutant, hypersensitive to red and blue
95                        We report here that a photomorphogenic mutant, red and far-red insensitive 2-1
96            We have identified an Arabidopsis photomorphogenic mutant, sub1, which exhibits hypersensi
97 analysis, the data show that red1 is a novel photomorphogenic mutant.
98  the previously characterized constitutively photomorphogenic mutants (cop, det, fus, and shy).
99 tum) yg-2 and Nicotiana plumbaginifolia pew1 photomorphogenic mutants are defective in specific HO ge
100                             The constitutive photomorphogenic mutants cop1 and det1 did not show sign
101 nd the elg mutation acts additively with the photomorphogenic mutants phyB, hy1 and hy2.
102  chromosome 2, distinct from any other known photomorphogenic mutants.
103 toreceptor deficient plants and constitutive photomorphogenic mutants.
104 nd1-1 mutant displays a partial constitutive photomorphogenic phenotype and has defects in HY5 degrad
105  had the same ectopic lignification and dark-photomorphogenic phenotype as that of the det3 mutant.
106  compared to pif1, indicating that the hyper-photomorphogenic phenotype of phyA requires PIF1.
107 ated based on their deetiolated/constitutive photomorphogenic phenotypes in the dark.
108 truncated form of PIF1 causes constitutively photomorphogenic phenotypes in the dark.
109                                          The photomorphogenic phenotypes of ATAF2 loss- and gain-of-f
110 mutant partially suppresses the constitutive photomorphogenic phenotypes of cop1-6 pif1 and of the qu
111 o explain the ectopic lignification and dark-photomorphogenic phenotypes of the det3 mutant.
112                                       Severe photomorphogenic phenotypes, including the defect of phy
113  blue light, and are compromised in multiple photomorphogenic processes, including seed germination,
114 ubiquitination by the E3 ligase constitutive photomorphogenic protein (COP1).
115 ouse double minute 2 (Mdm2) and constitutive photomorphogenic protein 1 (COP1).
116 on with the E3 ubiquitin ligase constitutive photomorphogenic protein 1 (COP1).
117  The subcellular localization of COP1, a key photomorphogenic repressor, is regulated by light in Ara
118  this transition, plants must rapidly remove photomorphogenic repressors accumulated in the dark.
119 gly inhibits hypocotyl elongation during the photomorphogenic response known as de-etiolation, the tr
120 uces the phosphorylation of cry2, triggering photomorphogenic responses and eventually degradation of
121 elatively little is known about the types of photomorphogenic responses and signal transduction pathw
122 notype, elg seedlings retain a full range of photomorphogenic responses and the elg mutation acts add
123                         Here, we examine two photomorphogenic responses in maize: inhibition of mesoc
124 w doses of UV-B light (280 to 315 nm) elicit photomorphogenic responses in plants that modify biochem
125 me blue light photoreceptors mediate various photomorphogenic responses in plants, including hypocoty
126 or ultraviolet-B (UV-B) light that initiates photomorphogenic responses in plants.
127 r specifically for UV-B light that initiates photomorphogenic responses in plants.
128                         UV-B light initiates photomorphogenic responses in plants.
129 but not damage, occurs at low doses of UV-B, photomorphogenic responses of UV-B sensitive mutants wer
130 hotoreception, W233, W285, and W337, impairs photomorphogenic responses to different extents.
131 s a photoreceptor that specifically mediates photomorphogenic responses to ultraviolet (UV)-B in plan
132         UV RESISTANCE LOCUS8 (UVR8) mediates photomorphogenic responses to UV-B by regulating transcr
133 ceptor UV RESISTANCE LOCUS 8 (UVR8) mediates photomorphogenic responses to UV-B in Arabidopsis throug
134 ISTANCE LOCUS 8 (UVR8) specifically mediates photomorphogenic responses to UV-B wavelengths.
135                                              Photomorphogenic responses triggered by low fluence rate
136 UVR8) is a UV-B photoreceptor that initiates photomorphogenic responses underlying acclimation and UV
137                                      Whereas photomorphogenic responses were observed at low doses, h
138 K1 and LNK2 genes control circadian rhythms, photomorphogenic responses, and photoperiodic dependent
139 ceptors in plants and often regulate similar photomorphogenic responses.
140 ate into the nucleus in red light to mediate photomorphogenic responses.
141 C80 motif and signal transduction to trigger photomorphogenic responses.
142 zation of UVR8 are required for UV-B-induced photomorphogenic responses.
143 udies of phytochrome C (phyC) have suggested photomorphogenic roles for this receptor, conclusive evi
144 sting global effects not directly related to photomorphogenic signaling; and 12 (37%) lines displayed
145 henotypes are intimately associated with the photomorphogenic transition in an organ-specific manner.
146 (COP1), which functions with UVR8 to control photomorphogenic UV-B responses.
147 ion; understanding the mechanisms underlying photomorphogenic variation is therefore of significant i

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