ライフサイエンスコーパス: photooxidative

1 uorescence bleaching of bisretinoid involves photooxidative and photodegradative processes.

2 anthin and lutein and undergoes irreversible photooxidative bleaching and cell death at moderate to h

3 damage to intracellular membranes caused by photooxidative chemistries or by phagocytosis of ground

4 but are not the longest lived species under photooxidative conditions, contrary to popular perceptio

5 tic study of each derivative under identical photooxidative conditions.

6 ogenic peptide sequences and exposed them to photooxidative conditions.

7 On the basis of photooxidative cross-linking of the wild type but not K1

8 esence of all-trans-retinaldehyde results in photooxidative cytotoxicity.

9 nstration of protection of RPE cells against photooxidative damage by induction of phase 2 proteins m

10 Targeting of photooxidative damage by triplex formation extends our p

11 ating a role for FeSOD in protection against photooxidative damage during moderate chilling in light.

12 Blue-light photooxidative damage has been implicated in the etiolog

13 n in these strains was sufficient to prevent photooxidative damage in the npq1 lor1 background.

14 A frequently occurring, irreversible photooxidative damage inhibits the PSII charge separatio

15 ere photosynthetic function is optimized and photooxidative damage is minimized in graduated response

16 e observed decreased photosynthesis and that photooxidative damage might be involved in the establish

17 was confirmed by the decreased tolerance to photooxidative damage of jasmonate-treated ch1 plants an

18 There is evidence of photooxidative damage of the photosynthetic apparatus in

19 inhibition of photosystem II without causing photooxidative damage of the plant.

20 re disrupted, the magnitude of resistance to photooxidative damage paralleled the basal levels of glu

21 various stimuli such as lipofuscin-mediated photooxidative damage to lysosomal membranes.

22 rately elevated light intensities eliminated photooxidative damage without suppressing (1)O(2) format

23 hogenesis including lipofuscin accumulation, photooxidative damage, complement activation, and RPE de

24 sponse to the high vulnerability of PS II to photooxidative damage, exacerbated by high-light (HL) st

25 ency, higher relative water content and less photooxidative damage.

26 es in some process relevant to the repair of photooxidative damage.

27 ation by stimuli such as lipofuscin-mediated photooxidative damage.

28 ass of UVA photosensitizers, capable of skin photooxidative damage.

29 absorbed energy in the cell can cause lethal photooxidative damage.

30 ll green and displayed signs of irreversible photooxidative damage.

31 essential in protecting the chloroplast from photooxidative damage.

32 hotosynthesis and protects the plant against photooxidative damage.

33 ]) confer cytoprotection from oxidative- and photooxidative-induced cellular damage and to explore th

34 rging by making tumor cells more tolerant to photooxidative insult.

35 hanced plant survival and reproduction under photooxidative light conditions, evidence that the plast

36 kinase-dependent stress signaling suggest a photooxidative mechanism of skin cell photosensitization

37 photoreductive Fe-N bond breakage as well as photooxidative N-N bond breakage occur on a time scale w

38 fluorescence; this effect is consistent with photooxidative processes known to precede bisretinoid de

39 tissues, light induces primary and secondary photooxidative processes.

40 heptacene derivatives with varying levels of photooxidative resistance (1 < 2 < 3 < 4) have been synt

41 uantitative assessment of HOMO-LUMO gaps and photooxidative resistances for a large series of pentace

42 x1 in retinal tissue and was protective from photooxidative retinal damage.

43 Under photooxidative stress conditions, the gene expression pr

44 from the Q(B) site in photosystem II, under photooxidative stress conditions.

45 photosynthetic organisms, protection against photooxidative stress due to singlet oxygen is provided

46 To examine the long-term effects of acute photooxidative stress in the retina, retinal pigment epi

47 senting a molecular mechanism of UVA-induced photooxidative stress potentially operative in human ski

48 We also found that photooxidative stress signaling pathway is constitutivel

49 hat HL-induced plastid to nucleus retrograde photooxidative stress signaling takes place after loss o

50 hich limits photophosphorylation, leading to photooxidative stress, causing the chlorotic and stunted

51 re resistant to cell damage induced by acute photooxidative stress, progressive loss of cone cells co

52 esults in mutants that are hypersensitive to photooxidative stress, whereas overexpression produces p

53 known for their roles in protecting against photooxidative stress, whereas the photoprotective funct

54 Chloroplast DNA (cpDNA) is under great photooxidative stress, yet its evolution is very conserv

55 The concept of a photooxidative stress-induced vicious cycle of increased

56 In response to HL, H2O2- and photooxidative stress-responsive marker genes were found

57 t, fix CO2 , perform biosynthesis and resist photooxidative stress.

58 lorophyll precursors, which can cause deadly photooxidative stress.

59 he seedlings and adult plants susceptible to photooxidative stress.

60 herols and carotenoids in protection against photooxidative stress.

61 , an overreduced cellular state, and limited photooxidative stress.

62 as associated with an increased tolerance to photooxidative stress.

63 nce and that ClpC2 might act by accelerating photooxidative stress.

64 type, implying that they experienced chronic photooxidative stress.

65 c free radicals as key mediators of cellular photooxidative stress.