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1 and prednisone) alone or in conjunction with photopheresis.
3 titox, mycophenolate mofetil, extracorporeal photopheresis, and several monoclonal antibodies have be
4 tosis in patients clearing on extracorporeal photopheresis but persisted in nonresponsive patients.
5 The safety and efficacy of extracorporeal photopheresis (ECP) for 12 to 24 weeks together with sta
9 gulatory T (Treg) cells after extracorporeal photopheresis (ECP) is thought to contribute to how ECP
16 27), and significantly fewer patients in the photopheresis group had two or more rejection episodes (
18 etected significantly less frequently in the photopheresis group than in the standard-therapy group (
19 bexarotene, interferon alpha, extracorporeal photopheresis, histone deacetylase inhibitors, and antib
20 y study to assess the safety and efficacy of photopheresis in the prevention of acute rejection of ca
23 nti-LFA-3-IgG fusion protein, extracorporeal photopheresis, mesenchymal stem cells and regulatory T c
25 ts for chronic GVHD including extracorporeal photopheresis, rituximab, sirolimus, mycofenolate mofeti
26 loaded dendritic cells as well as the use of photopheresis to generate an anti-idiotype cytotoxic T-c
28 e photopheresis group received a total of 24 photopheresis treatments, each pair of treatments given
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