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1 in mice as a measure of migraine-associated photophobia.
2 ement as well as reduction of both glare and photophobia.
3 decreased visual quality marked by glare and photophobia.
4 n body sensation, conjunctival hyperemia and photophobia.
5 rgins and also moderate dry eyes with severe photophobia.
6 for sleep disorders and migraine-associated photophobia.
7 void confounding effects of illumination and photophobia.
8 provide a neural substrate for migraine-type photophobia.
9 g head pain and associated symptoms, such as photophobia.
10 s for migraine and other disorders involving photophobia.
11 ral visual impairment (CVI) often experience photophobia.
12 olors, impaired visual acuity, nystagmus and photophobia.
13 had ocular involvement, with 65% describing photophobia.
14 we noted transient headaches (11 patients), photophobia (11 patients), reduction in serum HDL concen
15 58%] of 353 vs 126 [37%] of 342 [p<0.0001]), photophobia (180 [51%] of 353 vs 99 [29%] of 342 [p<0.00
16 , with the greatest differences observed for photophobia (70% versus 6%), poor balance (63% versus 4%
17 ache relief 2 hours after dosing, absence of photophobia, absence of phonophobia, and absence of naus
18 ed visual acuity, nystagmus, strabismus, and photophobia, although pigmentation of skin and hair is r
19 ter surgery 12 patients (75.00%) reported no photophobia and 10 patients (62.50%) reported no glare.
20 This behavior appears to be an indicator of photophobia and cannot be fully explained by gross abnor
24 ying protein 1 (RAMP1), can be a modifier of photophobia and, by extension, suggest that genetic or e
25 acterized by the clinical triad of epiphora, photophobia, and blepharospasm; increased intraocular pr
28 ontaneous photopsia, self-light of the eye), photophobia, and nyctalopia (impaired night vision); and
30 changes may result in reduced visual acuity, photophobia, and ocular irritation, though these symptom
31 ce, and migraine symptoms, such as headache, photophobia, and phonophobia, is a requisite diagnostic
34 arring alopecia, slightly runted growth, and photophobia arose at The Jackson Laboratory in 1993 in t
35 and study 2, 57% vs 29%; P<.001), absence of photophobia at 2 hours (58% vs 26%; P<.001 and 50% vs 32
39 atients' experience with colour and migraine photophobia could originate in cone-driven retinal pathw
40 The clinical phenotype was typical CORD with photophobia, decreased central vision, and dyschromatops
44 utosomal recessive disorder characterized by photophobia, low visual acuity, nystagmus and a total in
46 17; 39%), followed by tearing (n = 10; 23%), photophobia (n = 6; 14%), itching (n = 4; 9%), swelling
47 e disorder characterized by color blindness, photophobia, nystagmus and severely reduced visual acuit
48 y alterations in sensory perception, such as photophobia or allodynia, which have in common an uncomf
52 ing, foreign body sensation, eye discomfort, photophobia, pain), ocular discomfort score (ODS), and s
54 to understand better the neural substrate of photophobia paved a way to the development of different
55 s reported so far include: headaches, severe photophobia, persistent foreign body sensation, and migr
56 ere pain freedom, pain relief, or absence of photophobia, phonophobia, or nausea at 2 h after treatme
58 characterized by reduced visual acuity, mild photophobia, reduced amplitude of the cone electroretino
59 with variable degrees of night blindness or photophobia, reduced visual acuity, high myopia, and nys
62 a or vomiting, fever, weight loss, headache, photophobia, seizure, extremity weakness, or sensory dis
63 defects, cleft palate, extradural cysts, and photophobia, suggesting a defect in a gene with pleiotro
64 ary measures include reduction in nausea and photophobia, use of rescue medication, relapse of headac
69 ich was unilateral in 14 (48%); 29 (74%) had photophobia, which was unilateral in 14 (48%); and 27 (6
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