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1 a deficiency in immunochemically detectable phytochrome B.
2 onsistently, BRC1 is negatively regulated by phytochrome B.
3 ponent of SCFTIR1 and with the photoreceptor phytochrome B.
4 in-of-function mutant, sob1-D (suppressor of phytochrome B-4 [phyB-4] dominant), which suppresses the
5 is inhibited in sorghum genotypes that lack phytochrome B (58M, phyB-1) until after floral initiatio
6 e genes (INDOLE-3-ACETIC ACID-INDUCIBLE1 and PHYTOCHROME B ACTIVATION-TAGGED SUPPRESSOR1) were impair
8 monstrate that this response is dominated by phytochrome B and also identify a role for the transcrip
9 specifically requires phytochrome A but not phytochrome B and also requires the cryptochrome1 blue l
10 between the C-terminal domain of Arabidopsis phytochrome B and COP1, suggesting that phytochrome sign
12 toreversible extent of greening) mediated by phytochrome B and other photo-stable phytochromes, both
13 nses to continuous red light are mediated by phytochrome B and other photostable phytochromes, we hav
14 ht and temperature by dual receptors such as phytochrome B and phototropin leads to immediate signall
16 uch more light stable, although among these, phytochromes B and C are reduced 4- to 5-fold in red- or
18 is similar to plants that are known to lack phytochrome B, and ma3 sorghum lacks a 123-KD phytochrom
21 Here we show that full-length photoactive phytochrome B binds PIF3 in vitro only upon light-induce
22 ous light-regulated gene promoters, and that phytochrome B binds reversibly to G-box-bound PIF3 speci
23 n and may be one way by which the absence of phytochrome B causes early flowering in 58M under most p
25 bicolor [L.] Moench) deficient in functional phytochrome B exhibits reduced photoperiodic sensitivity
26 ed and validated signaling-active alleles of phytochrome B (eYHB) as plant-derived selection marker g
27 s in sorghum, one in response to the loss of phytochrome B function and another in response to shadin
28 pocotyl growth induced by light perceived by phytochrome B in deetiolating Arabidopsis thaliana seedl
29 ocot species have defined a central role for phytochrome B in mediating responses to light in the con
31 et al. (2016a) show that red-light-activated phytochrome B interacts with transcriptional regulators
32 We conclude that photosensory signalling by phytochrome B involves light-induced, conformer-specific
38 h cycles in a circadian rhythm; however, the phytochrome B mutant produces ethylene peaks with approx
39 ene production by seedlings of wild-type and phytochrome B-mutant cultivars progresses through cycles
41 in Arabidopsis, the negative effects of the phytochrome B mutation and of low red light:far-red ligh
42 (Ma1Ma1, Ma2Ma2, phyB-1phyB-1, and Ma4Ma4 [a phytochrome B null mutant]); 90M (Ma1Ma1, Ma2Ma2, phyB-2
44 of the level of immunochemically detectable phytochrome B of equivalent wild-type EIN/EIN seedlings.
45 eedlings deficient in both phytochrome A and phytochrome B (phyA phyB), have a greatly reduced statur
46 o-His mutant alleles of Arabidopsis thaliana phytochrome B (PHYB(Y276H)) and Arabidopsis phytochrome
47 GDP-bound form to the photosensory domain of phytochrome B (PhyB) and fused the Cdc42 effector, the W
48 ersensitivity can be overcome by eliminating phytochrome B (phyB) and phyD, indicating that LRB1/2 ac
49 RACK/BROAD (LRB) E3 ubiquitin ligases target phytochrome B (phyB) and PIF3 primarily under high-light
52 show that inactivation of the photoreceptor phytochrome B (phyB) by a low red/far-red ratio (R:FR),
53 antisense plants that have reduced levels of phytochrome B (PHYB) compared with wild-type plants, imp
56 ic Arabidopsis line (ABO) that overexpresses phytochrome B (phyB) display enhanced deetiolation speci
60 the Arabidopsis EARLY FLOWERING 3 (ELF3) and PHYTOCHROME B (PHYB) genes cause early flowering and inf
61 far-red light photoreceptors-in Arabidopsis, phytochrome B (phyB) has the most significant role in sh
62 tors [3-5], with the red-light photoreceptor phytochrome B (phyB) having a dominant role in white lig
65 hat light-activated phytochrome A (phyA) and phytochrome B (phyB) interact with SPA1 and other SPA pr
69 ow that the C-terminal module of Arabidopsis phytochrome B (PHYB) is sufficient to mediate the degrad
75 ibe the steady-state dynamics of Arabidopsis phytochrome B (phyB) localization in response to differe
76 nt, HEMERA (HMR), that is essential for both phytochrome B (phyB) localization to photobodies and PIF
77 pects of plant growth, and the photoreceptor phytochrome B (phyB) mediates many responses to red ligh
80 study, oat phytochrome A (phyA), Arabidopsis phytochrome B (phyB) or Arabidopsis phytochrome C (phyC)
82 growth through the cryptochrome 1 (cry1) and phytochrome B (phyB) photosensory pathways, respectively
87 ith critical roles in photomorphogenesis are phytochrome B (phyB), a red/far-red absorbing photorecep
89 tion of low red to far-red ratios (R:FRs) by phytochrome B (phyB), which leads to the direct activati
98 he red/far red light absorbing photoreceptor phytochrome-B (phyB) cycles between the biologically ina
99 in the phytochrome A (phyA) null mutant, the phytochrome B- (phyB) deficient mutant, and in two trans
100 vegetation shade, which we show to occur via phytochrome B, PHYTOCHROME INTERACTING FACTOR4 (PIF4), a
102 esponse, mediated by either phytochrome A or phytochrome B, represents a prime example of cross-talk
104 ition, we demonstrate that the photoreceptor PHYTOCHROME B restricts ethylene biosynthesis and constr
105 the red light sensing network that modulates phytochrome B signaling input into the circadian system.
107 response is controlled by the photoreceptor, phytochrome B, through the deactivation and proteolytic
109 introduce a constitutively active version of phytochrome B-Y276H (YHB) into both wild-type and phytoc
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