ライフサイエンスコーパス: pigment epithelium

1 retinal, 38% subretinal, and 36% sub-retinal pigment epithelium).

2 lls, and the basolateral side of the retinal pigment epithelium.

3 mophores and recycling in the nearby retinal pigment epithelium.

4 ce ectoderm, lens, neuro-retina, and retinal pigment epithelium.

5 ized dysfunction at the level of the retinal pigment epithelium.

6 n sFlt-1 expression is attenuated in retinal pigment epithelium.

7 iltrating myeloid cells but not from retinal pigment epithelium.

8 larly toxic to photoreceptors and/or retinal pigment epithelium.

9 mum or >/=425 mum, and elevation of retinal pigment epithelium.

10 by macular atrophy and flecks in the retinal pigment epithelium.

11 e carrier between the retina and the retinal pigment epithelium.

12 ntation consistent with transplanted retinal pigment epithelium.

13 s-retinyl esters (11-REs) within the retinal pigment epithelium.

14 led the visual cycle in cells of the retinal pigment epithelium.

15 e re-attachment of the retina to the retinal pigment epithelium.

16 reted in exosomes by polarized human retinal pigment epithelium.

17 al separation of the retina from the retinal pigment epithelium.

18 hyperreflective lesions adherent to retinal pigment epithelium.

19 ppress consumption of glucose by the retinal pigment epithelium.

20 ith PVR, and reactive changes in the retinal pigment epithelium.

21 c nerve head pallor, and mottling of retinal pigment epithelium.

22 e interdigitation zone and an intact retinal pigment epithelium.

23 the cone outer segment closer to the retinal pigment epithelium.

24 ent cells, including melanocytes and retinal pigment epithelium.

25 ina preparation after removal of the retinal pigmented epithelium.

26 cative of pathological events in the retinal pigmented epithelium.

27 omposed of 6 layered components: (1) retinal pigment epithelium, (2) basal laminar deposits, (3) fibr

28 otographs were graded for drusen and retinal pigment epithelium abnormalities and were evaluated for

29 (n = 37) in those 80 years or older, retinal pigment epithelium abnormalities from 4.1% (n = 85) to 7

30 The copresence of medium drusen plus retinal pigment epithelium abnormalities signals a greater risk

31 Retinal pigment epithelium abnormalities, AVLs, neovascularizati

32 injection site demonstrated diffuse retinal pigment epithelium alterations with dense hard exudates.

33 position of the CNV relative to the retinal pigment epithelium and Bruch membrane were described usi

34 sualize CNV location relative to the retinal pigment epithelium and Bruch's layer and classify type I

35 of vitamin A derivatives across the retinal pigment epithelium and Bruch's membrane, 2 tissues with

36 hat toll-like receptor activation of retinal pigment epithelium and cellular metabolic switch upregul

37 The retinal pigment epithelium and choroid are involved in severely

38 n accumulate in the lysosomes of the retinal pigment epithelium and display cytotoxic effects.

39 Quantifying preserved retinal pigment epithelium and EZ areas on FAF and OCT images, r

40 n export the lactate as fuel for the retinal pigment epithelium and for neighboring Muller glial cell

41 ultimodal imaging supports an intact retinal pigment epithelium and inner retina but an abnormal phot

42 cient to alter Fpn levels within the retinal pigment epithelium and Muller cells, but may limit iron

43 e choriocapillaris in the absence of retinal pigment epithelium and outer retinal abnormalities suppo

44 adelta T cells in protection against retinal pigment epithelium and retinal injury.

45 trongly expressed in the presumptive retinal pigment epithelium and RGCs.

46 ssociated with absence of underlying retinal pigment epithelium and were longer (r = -0.62; 95% CI, -

47 nfirmed that these areas had loss of retinal pigmented epithelium and ellipsoids zones, with or witho

48 n involving the neurosensory retina, retinal pigment epithelium, and choroid in 4 eyes (44%).

49 ior ocular segment (neuronal retina, retinal pigment epithelium, and choroid) of wild-type (WT) C57BL

50 rees of atrophy of the outer retina, retinal pigment epithelium, and choroid, with outer retinal tubu

51 of structural alterations of the CC, retinal pigment epithelium, and photoreceptors with multimodal i

52 tive characteristics of the choroid, retinal pigment epithelium, and retina were compared between the

53 ely by promoting inflammation of the retinal pigment epithelium, and validate TLR2 as a novel therape

54 ty and genotoxicity studies in human retinal pigment epithelium (ARPE-19) cells.

55 stimulate the growth of normal human retinal pigment epithelium (ARPE-19) cells.

56 The atrophic area of the retinal pigment epithelium assessed on the basis of FAF increase

57 nd NeuN(-) and possibly arose from embryonic pigment epithelium at the edge of the retinochoroidal co

58 the small irregular elevation of the retinal pigment epithelium at the site of the quiescent CNV visu

59 veral nummular perifoveal islands of retinal pigment epithelium atrophy and adjacent pale deposits in

60 e of the extent of photoreceptor and retinal pigment epithelium atrophy in the macula.

61 44.4% vs 12.5%, P = .03), absence of macular pigment epithelium atrophy on FA (88.9% vs 62.5%, P = .0

62 an irregularly thickened and rippled retinal pigment epithelium band in 2 eyes.

63 as hyperreflective debris above the retinal pigment epithelium band in all 3 eyes, and were associat

64 ghlight an unrecognised link between retinal pigment epithelium bioenergetic status and tissue remode

65 Retinal pigment epithelium-BM thickness, as measured by SD OCT s

66 graphy (OCT) revealed a split in the retinal pigment epithelium-Bruch membrane band.

67 ance between the outer border of the retinal pigment epithelium-Bruch's membrane complex, and the cho

68 mes and condensation products in the retinal pigment epithelium by their characteristic localization,

69 ession of glucose consumption in the retinal pigment epithelium can increase the amount of glucose th

70 analysis of LC/MS data from a human retinal pigment epithelium cell line (ARPE-19) grown on normal a

71 ncluding Bruchs membrane thickening, retinal pigment epithelium cell loss, retinal functional deficit

72 zation and a decrease in mesenchymal retinal pigment epithelium cells in alphaB-crystallin knockout m

73 icroglia, whereas both microglia and retinal pigment epithelium cells produced Ccl2.

74 Retinal pigment epithelium cells were in the centre, photorecept

75 Finally, in primary human fetal retinal pigment epithelium cells, ligand binding to TLR2 induced

76 olar, horizontal, photoreceptor, and retinal pigment epithelium cells, thus exposing the anatomical s

77 n the mammalian cochlea and in human retinal pigment epithelium cells.

78 lial-mesenchymal transition (EMT) of retinal pigment epithelium cells.

79 nelles in the OS region and abnormal retinal pigmented epithelium cells.

80 appearance but later develop mottled retinal pigment epithelium change along the arcades, followed by

81 ations lead to clinically detectable retinal pigment epithelium changes remain unclear.

82 rated, such as macular degeneration, retinal pigment epithelium changes, and glaucoma.

83 o CNV-associated macrophages and the retinal pigment epithelium/choroid complex.

84 nent 3 and factor B in plasma and in retinal pigment epithelium/choroid/sclera, establishing that hum

85 ST2 was highly expressed in retinal pigment epithelium, choroidal mast cells, and choroidal

86 loss of pigment and thinning of the retinal pigment epithelium, choroidal thinning, undifferentiated

87 resembling congenital hypertrophy of retinal pigment epithelium (CHRPE) lesions.

88 combined hamartoma of the retina and retinal pigment epithelium (CHRRPE) involving the macula.

89 sed autofluorescence as a measure of retinal pigment epithelium damage and lipofuscin accumulation, r

90 rentiation of the dorsal and ventral retinal pigment epithelium, defective optic cup periphery, and c

91 ed accumulation of lipofuscin in the retinal pigment epithelium, degeneration of the neuroretina, and

92 hloroquine retinopathy involving the retinal pigment epithelium demonstrated progressive damage on op

93 ker outer nuclear layer and less sub-retinal pigment epithelium deposit accumulation.

94 Pigment Epithelium Derived Factor (PEDF) is a secreted f

95 ransmembrane (IFITM)-like protein (BRIL) and pigment epithelium-derived factor (PEDF) defects cause t

96 tudy, we tested the effect of treatment with pigment epithelium-derived factor (PEDF) in combination

97 a1 transcription factor and are dependent on pigment epithelium-derived factor (PEDF) on the outer su

98 ation of the known neuroprotective molecule, pigment epithelium-derived factor (PEDF) plus docosahexa

99 The cytoprotective effects of pigment epithelium-derived factor (PEDF) require interac

100 vIII induces the expression and secretion of pigment epithelium-derived factor (PEDF) via activation

101 asminogen activator inhibitor-1 (PAI-1), and pigment epithelium-derived factor (PEDF) were measured b

102 Furthermore, pigment epithelium-derived factor (PEDF), a secreted gly

103 ociated transcription factor (MITF) acts via pigment epithelium-derived factor (PEDF), an antiangioge

104 One of these molecules, pigment epithelium-derived factor (PEDF), is a broadly e

105 Null mutations in for pigment epithelium-derived factor (PEDF), the protein pr

106 Loss of pigment epithelium-derived factor (PEDF, SERPINF1) in ca

107 elial growth factor while increasing that of pigment epithelium-derived factor in diabetic retinas.

108 Pigment epithelium-derived factor restoration increases

109 Inhibitor, Clade F) gene which encodes PEDF (pigment epithelium-derived factor), a potent inhibitor o

110 l, revealed that they constitutively secrete pigment epithelium-derived factor, PEDF, which is known

111 ing alpha-1 microglobulin/bikunin precursor, pigment epithelium-derived factor, surfactant protein B,

112 Drusenoid pigment epithelium detachment (DPED) is a known precurso

113 The majority (n = 4) showed a drusenoid pigment epithelium detachment (PED) preceding the lesion

114 h nonneovascular DPED and 2 with neovascular pigment epithelium detachment [PED]) and 49 eyes of 33 c

115 ate hypercyanescence on ICGA (P < .001), and pigment epithelium detachment on SD OCT (P < .001) were

116 worse vision, presence of atrophy/fibrosis, pigment epithelium detachment, and geographic atrophy/fi

117 ad thickening of Bruchs membrane and retinal pigment epithelium dysfunction.

118 led malformations in the choroid and retinal pigmented epithelium, early cone photoreceptor cell deat

119 antitative RT-PCR in the retina, the retinal pigment epithelium, fibroblasts, and whole-blood cells (

120 eye, hemorrhage, and absence of sub-retinal pigment epithelium fluid at baseline were associated wit

121 nal fluid, subretinal fluid, and sub-retinal pigment epithelium fluid.

122 of intraretinal, subretinal, and subretinal pigment epithelium fluid; thickness at the foveal center

123 ulin 2 dramatically increased in the retinal pigment epithelium following retinal detachment, suggest

124 isual cycle proteins or with reduced retinal pigment epithelium function due to aging.

125 o AMD but rather may be a marker for retinal pigment epithelium function.

126 Eyes with elevation of the retinal pigment epithelium had lower risk (aHR, 0.6; CI, 0.5-0.8

127 across the blood-retinal barrier or retinal pigment epithelium have not been identified.

128 human induced pluripotent stem cell-retinal pigment epithelium (hiPSC-RPE) derived from patients wit

129 ages in 2 (17%) and 3 (18%); loss of retinal pigment epithelium in 1 (8%) and 4 (24%); and drusen in

130 and hyperreflectivity underlying the retinal pigment epithelium in 9 eyes (100%), retinal thinning in

131 e that Mfsd2a is highly expressed in retinal pigment epithelium in embryonic eye, before the developm

132 lammasome to cause cell death of the retinal pigment epithelium in geographic atrophy, a type of age-

133 TLR2 was robustly expressed by the retinal pigment epithelium in mouse and human eyes, both normal

134 inal transplantation of hESC-derived retinal pigment epithelium in nine patients with Stargardt's mac

135 incidence of atrophic lesions of the retinal pigment epithelium in patients with Stargardt disease as

136 y abnormalities of photoreceptors or retinal pigment epithelium in the retina leading to progressive

137 l melanosomes forming the remains of retinal pigment epithelium indicates that it is a vertebrate; co

138 uch's membrane, the Bruch's membrane-retinal pigment epithelium interface, or both in the pathogenesi

139 The variable extent of retinal pigment epithelium involvement was reflected in variable

140 hologies arranged in layers, forming retinal pigment epithelium, is a synapomorphy of vertebrates.

141 ssive geographic atrophy (GA) of the retinal pigment epithelium leads to loss of central vision.

142 coherence tomography revealed a mean retinal pigment epithelium lesion area of 2.6 mm(2), preserved f

143 phy parameters (IS/OS alteration and retinal pigment epithelium lesion area) were obtained in only 49

144 The subretinal pigment epithelium lesion underlying PED appears to be t

145 ly coupled, the results suggest that retinal pigment epithelium loss is more extensive than photorece

146 ination, neural retinal attenuation, retinal pigment epithelium loss, or hypertrophy was seen in seve

147 inal vasculature, optic atrophy, and retinal pigment epithelium loss.

148 unt of pigment granules deposited in retinal pigment epithelium microvilli area and an abnormal respo

149 interface between photoreceptors and retinal pigment epithelium microvilli, a region critical for ret

150 antiangiogenic protein, to regulate retinal pigment epithelium migration.

151 tions of photoreceptor cells and the retinal pigmented epithelium necessitate precise gene regulation

152 of either groups was atrophy of the retinal pigment epithelium observed in the area of treatment.

153 TLR2 signaling, was detected in the retinal pigment epithelium of human eyes, particularly in eyes w

154 o subnormal lipofuscin levels in the retinal pigment epithelium or, alternatively, limitations to det

155 luding congenital hypertrophy of the retinal pigment epithelium, ora serrata pearl, TCD, cystic retin

156 erreflective foci represent cells of retinal pigment epithelium origin that are similar to those foun

157 acquired vitelliform lesions are of retinal pigment epithelium origin, and the natural course and fu

158 ischemic infarction of the choroid, retinal pigment epithelium, outer part of the retina and the opt

159 vein, congenital hypertrophy of the retinal pigment epithelium, pars plana, ora serrata pearl, typic

160 notypes, including the occurrence of retinal pigment epithelium (RPE) abnormalities, choroidal neovas

161 hat begins around the age of 30 with retinal pigment epithelium (RPE) and Bruch's membrane changes re

162 hyperreflective deposits between the retinal pigment epithelium (RPE) and Bruch's membrane on SD-OCT,

163 increased deposition of C5b-9 in the retinal pigment epithelium (RPE) and choroid.

164 remains elusive, dysfunction of the retinal pigment epithelium (RPE) and dysregulation of complement

165 ents is an important function of the retinal pigment epithelium (RPE) and it is essential for retinal

166 Histology data show aberrant retinal pigment epithelium (RPE) and late-onset photoreceptor ce

167 ophin1 (BEST1) is expressed in human retinal pigment epithelium (RPE) and mutations in the BEST1 gene

168 tial progenitor cells from which the retinal pigment epithelium (RPE) and neural retina fates segrega

169 n the diseased retina, damage in the retinal pigment epithelium (RPE) and neural retina from individu

170 ng OV specification and formation of retinal pigment epithelium (RPE) and neural retina progenitor ce

171 ces (BMOM, BMOH) and the terminal of retinal pigment epithelium (RPE) and ONH surfaces (RPEM, RPEH) w

172 AMD that manifests with progressive retinal pigment epithelium (RPE) and photoreceptor degeneration

173 by neighboring epithelial cells, the retinal pigment epithelium (RPE) and podocytes, respectively.

174 We semiautomatically delineated the retinal pigment epithelium (RPE) and RPE drusen complex (RPEDC,

175 action between autophagy impaired in retinal pigment epithelium (RPE) and the responses of macrophage

176 n the morphology and function of the retinal pigment epithelium (RPE) are common features shared by m

177 outer boundaries of the choroid and retinal pigment epithelium (RPE) as well as the inner retinal su

178 etinal disruption and atrophy of the retinal pigment epithelium (RPE) associated with ORT on spectral

179 e vitelliform material was above the retinal pigment epithelium (RPE) at any stage of the macular dys

180 toreceptor atrophy can occur without retinal pigment epithelium (RPE) atrophy and that atrophy can un

181 horiocapillaris in correspondence of retinal pigment epithelium (RPE) atrophy in 80% (n = 16) of case

182 ected visual acuity (BCVA), age, and retinal pigment epithelium (RPE) atrophy were recorded and corre

183 se disorders is the formation of sub-retinal pigment epithelium (RPE) basal deposits.

184 s localized to the apical aspects of retinal pigment epithelium (RPE) cells and contributes to a dela

185 lls, mitochondria, Muller cells, and retinal pigment epithelium (RPE) cells and were visualized using

186 Retinal Pigment Epithelium (RPE) cells generated from a patient

187 te the intraretinal migration of the retinal pigment epithelium (RPE) cells in age-related macular de

188 Cholesterol accumulation beneath the retinal pigment epithelium (RPE) cells is supposed to contribute

189 s cadherin-3, a protein expressed in retinal pigment epithelium (RPE) cells that may have a key role

190 trophic and functional support from retinal pigment epithelium (RPE) cells.

191 ucial for volume regulation in human retinal pigment epithelium (RPE) cells.

192 not HSV-1 infection of human primary retinal pigment epithelium (RPE) cells.

193 ed either no abnormalities or foveal retinal pigment epithelium (RPE) changes in 10 and 9 patients, r

194 murine eye, melanin synthesis in the retinal pigment epithelium (RPE) coincides with neurogenesis of

195 V4 expression in the endothelium and retinal pigment epithelium (RPE) components of the BRB, and that

196 ar degeneration and atypical central retinal pigment epithelium (RPE) defects not attributable to geo

197 Adamtsl4(tvrm267) mice exhibit focal retinal pigment epithelium (RPE) defects primarily in the inferi

198 ophy, characterised by extensive sub-retinal pigment epithelium (RPE) deposits, RPE atrophy, choroida

199 RF), subretinal fluid (SRF), and sub-retinal pigment epithelium (RPE) fluid and performed manual meas

200 d (IRF), subretinal fluid (SRF), sub-retinal pigment epithelium (RPE) fluid, and subretinal tissue co

201 d pluripotent stem cells to generate retinal pigment epithelium (RPE) from an individual suffering fr

202 in three-dimensional optic cups and retinal pigment epithelium (RPE) from iPSCs with this common CEP

203 ways necessary for photoreceptor and retinal pigment epithelium (RPE) function is critical to uncover

204 To report on the presence of retinal pigment epithelium (RPE) humps in high myopia, and to de

205 ofiles specifically localized to the retinal pigment epithelium (RPE) in Abca4 (-/-) Stargardt model

206 Daily, the retinal pigment epithelium (RPE) ingests a bolus of lipid and pr

207 While AMD histopathology involves retinal pigment epithelium (RPE) injury associated with immune c

208 The retinal pigment epithelium (RPE) is a key site of injury in inhe

209 The retinal pigment epithelium (RPE) is a monolayer of pigmented cel

210 -containing deposits external to the retinal pigment epithelium (RPE) is common in the aging eye, and

211 ce between the neural retina and the retinal pigment epithelium (RPE) is critical for several process

212 lipofuscin bisretinoids (LBs) in the retinal pigment epithelium (RPE) is the alleged cause of retinal

213 0.03) were: hyperreflective foci and retinal pigment epithelium (RPE) layer atrophy or absence, follo

214 rs (P = 0.024) and diffusely thinned retinal pigment epithelium (RPE) layers (P = 0.009) versus contr

215 ated from the SD-OCT and the area of retinal pigment epithelium (RPE) loss from the FAF.

216 to mitochondrial DNA (mtDNA) in the retinal pigment epithelium (RPE) may play a key role in AMD.

217 ase 1/2 (ERK1/2) is increased in the retinal pigment epithelium (RPE) of age-related macular degenera

218 ical phenotypes were observed in the retinal pigment epithelium (RPE) of individuals with butterfly-s

219 to outer retinal atrophy with viable retinal pigment epithelium (RPE) on spectral-domain OCT and may

220 inflammasome have been implicated in retinal pigment epithelium (RPE) pathology in age-related macula

221 The retinal pigment epithelium (RPE) performs specialized functions

222 D, estimate of coverage by different retinal pigment epithelium (RPE) phenotypes in the DPED surface;

223 licit a Wnt/beta-catenin response in retinal pigment epithelium (RPE) progenitors near the optic cup

224 f autoantibodies against retinal and retinal pigment epithelium (RPE) proteins.

225 height of 45.3 (36.1) mum above the retinal pigment epithelium (RPE) reference plane that was preset

226 e measurements of vitreous (VIT) and retinal pigment epithelium (RPE) signal intensities, which were

227 growth factor (VEGF) inhibitors, (2) retinal pigment epithelium (RPE) tear, (3) subretinal hemorrhage

228 To investigate when retinal pigment epithelium (RPE) tears occur and their associate

229 CT measurement parameters, including retinal pigment epithelium (RPE) thickness, central macular thic

230 transport is a major function of the retinal pigment epithelium (RPE) to support the neural retina.

231 biologically and genetically defined retinal pigment epithelium (RPE) to the diseased human eye.

232 Retinectomies expose the retinal pigment epithelium (RPE) to the vitreous cavity; the dir

233 onditionally inactivate genes in the retinal pigment epithelium (RPE) transgenic mouse strains have b

234 In contrast, the canonical retinal pigment epithelium (RPE) visual cycle produces exclusive

235 The retinal pigment epithelium (RPE) was absent (n = 2), thickened (

236 s in regions with normally appearing retinal pigment epithelium (RPE) were the loss of the POS and el

237 cular degeneration (AMD) affects the retinal pigment epithelium (RPE), a cell monolayer essential for

238 nisms that modulate autophagy in the retinal pigment epithelium (RPE), a key site of insult in macula

239 retinal projection through SHRM onto retinal pigment epithelium (RPE), and (4) masking of choriocapil

240 ies were lower at the photoreceptor, retinal pigment epithelium (RPE), and choroid layers (standardiz

241 ve deposits primarily in the choroid, retina pigment epithelium (RPE), and inner segment and outer se

242 sulting from loss of photoreceptors, retinal pigment epithelium (RPE), and underlying choriocapillari

243 isomerization occurs in the adjacent retinal pigment epithelium (RPE), but because ipRGCs are far fro

244 ound in the photoreceptor-supporting retinal pigment epithelium (RPE), especially in a zone correspon

245 ordinated terminal maturation of the retinal pigment epithelium (RPE), fenestrated choroid endothelia

246 pathy, congenital hypertrophy of the retinal pigment epithelium (RPE), hemorrhagic RPE detachment, ch

247 ontaining lipofuscin pigments in the retinal pigment epithelium (RPE), increased oxidative stress, au

248 nt membrane-associate protein in the retinal pigment epithelium (RPE), is a key retinoid isomerase of

249 y cell types, including those of the retinal pigment epithelium (RPE), is a regulatory protein that i

250 eta signaling in the entire eye, the retinal pigment epithelium (RPE), or the vascular endothelium.

251 ingerprint' lysosomal storage in the retinal pigment epithelium (RPE), replicating the human disease.

252 totoxic bisretinoid formation in the retinal pigment epithelium (RPE), which is associated with the p

253 of ocular and systemic factors with retinal pigment epithelium (RPE)-Bruch's membrane (BM) complex t

254 rfering RNA (siRNA) to transfect the retinal pigment epithelium (RPE)-derived cell line ARPE-19, and

255 The volumes of retinal pigment epithelium (RPE)-drusen complex, RPE-drusen comp

256 P = .001; SE -2.27 D [SD 4.65]), and retinal pigment epithelium (RPE)-related dystrophies (OR low myo

257 n the retinoid cycle in the adjacent retinal pigment epithelium (RPE).

258 model of chronic degeneration of the retinal pigment epithelium (RPE).

259 pigment clumping at the level of the retinal pigment epithelium (RPE).

260 cking caveolin-1 specifically in the retinal pigment epithelium (RPE).

261 sk membranes, is a major role of the retinal pigment epithelium (RPE).

262 e pigment regeneration driven by the retinal pigment epithelium (RPE).

263 e-onset morphological changes in the retinal pigment epithelium (RPE).

264 eceptors, as well as the choroid and retinal pigment epithelium (RPE).

265 is characterized by the loss of the retinal pigment epithelium (RPE).

266 na, the ciliary margin (CM), and the retinal pigment epithelium (RPE).

267 lial mesenchymal transition (EMT) of retinal pigment epithelium (RPE).

268 ulate the phagocytosis of OSs by the retinal pigment epithelium (RPE).

269 s, especially the involvement of the retinal pigment epithelium (RPE).

270 and mild transient thickening of the retinal pigment epithelium (RPE)/Bruch's complex (Bc).

271 y IL-1beta was markedly increased in retinal pigment epithelium (RPE)/choroid and positively correlat

272 egulated manner in chicken embryonic retinal pigment epithelium (RPE)/choroid in the absence of light

273 ns showed 2 distinct profiles of the retinal pigment epithelium (RPE): a slight RPE detachment with s

274 through a series of reactions in the retinal pigmented epithelium (RPE visual cycle).

275 acular degeneration characterized by retinal pigmented epithelium (RPE) death; the RPE also exhibits

276 The retinal pigmented epithelium (RPE) forms the outer blood-retinal

277 Accumulation of bis-retinoids in the retinal pigmented epithelium (RPE) is a hallmark of aging and re

278 he major biological functions of the retinal pigmented epithelium (RPE) is the clearance of shed phot

279 we delivered a wild-type Mfrp to the retinal pigmented epithelium (RPE) of Mfrp (rd6) /Mfrp (rd6) mic

280 he intraretinal visual cycle and the retinal pigmented epithelium (RPE) visual cycle.

281 photoreceptor cells and the adjacent retinal pigmented epithelium (RPE), reportedly display the earli

282 evealed predominant up-regulation of retinal pigmented epithelium (RPE)-specific genes associated wit

283 occur in photoreceptor cells and the retinal pigmented epithelium (RPE).

284 ocessing enzyme DICER1 in the mature retinal pigmented epithelium (RPE).

285 ed phagocytes: Sertoli cells and the retinal pigmented epithelium (RPE).

286 er, photoreceptor outer segments and retinal pigment epithelium show promise for the diagnostic class

287 /=196350 mum2) and depigmentation of retinal pigment epithelium (slope of -19.17 for the NEI-VFQ-25 c

288 d levels of CFH led to increased sub-retinal pigmented epithelium (sub-RPE) deposit formation, specif

289 en cultured with interleukin-33-rich retinal pigment epithelium supernatant.

290 Retinal pigment epithelium tears act differently depending on wh

291 excluded 5 eyes from analysis (4 had retinal pigment epithelium tears, and 1 had a laser scar).

292 Loss of retinal pigment epithelium, the presence of a thin choroid, a pe

293 utant lacks pigmentation also in the retinal pigment epithelium, therefore enabling optical access to

294 e choroidal blood passes through the retinal pigment epithelium to the retina where photoreceptors co

295 cement of the temporal peripapillary retinal pigment epithelium (tRPE) from its position in central g

296 and 16.9% (45/266) were type 1 (sub-retinal pigment epithelium), type 2 (subretinal), type 3 (intrar

297 Damage of the pigment epithelium was also confirmed.

298 al soft drusen and depigmentation of retinal pigment epithelium was associated with substantially low

299 Labeling of retinal pigment epithelium was observed in some cases of advance

300 -activated chloride channel from the retinal pigment epithelium, where mutations are associated with