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1  their ability to stimulate phagocytosis and pinocytosis.
2 isceral yolk sac cells, suggesting uptake by pinocytosis.
3 d-phase pinocytosis or inducible fluid-phase pinocytosis.
4 delivered by DEC205-mediated endocytosis and pinocytosis.
5 tosis and the documented dynamin-independent pinocytosis.
6 or of actin assembly, cell polarization, and pinocytosis.
7 serum treatment appeared to be the result of pinocytosis.
8 hilic leukemia cells and in cells performing pinocytosis.
9 ysiological downregulation of dendritic cell pinocytosis.
10 GF-induced membrane ruffling and fluid phase pinocytosis.
11  unaffected by inhibitors of endocytosis and pinocytosis.
12 timulates cell surface membrane ruffling and pinocytosis.
13  either through these pores or by reparative pinocytosis.
14 dicated by the ability of v-Abl to stimulate pinocytosis.
15 s as they are internalized into endosomes by pinocytosis.
16 omitant reduction in the rate of fluid phase pinocytosis, a significant decrease in the efficiency of
17 rophages that show high rates of fluid-phase pinocytosis also show similar high rates of uptake of na
18 t cross the plasma membrane, utilizing fluid pinocytosis and a specific lysosomal transporter.
19            Together, these data suggest that pinocytosis and adhesion are raft-dependent functions in
20                    In addition to inhibiting pinocytosis and autophagy, VPS34 depletion induced membr
21 vitelline fluid is initially internalized by pinocytosis and degraded by lysosomes; in later stages,
22 ortmannin and LY294002 inhibited fluid-phase pinocytosis and Fc receptor-mediated phagocytosis, but t
23 ke up LDL-sized nanoparticles by fluid-phase pinocytosis and indicate that fluid-phase pinocytosis of
24          IL-4 and IL-13 enhanced fluid phase pinocytosis and mannose receptor-mediated uptake by acti
25 L-10 or IFN-gamma decreased both fluid phase pinocytosis and mannose receptor-mediated uptake.
26                                              Pinocytosis and membrane ruffling are among the earliest
27 ne proteins and that SP-D enhances efficient pinocytosis and phagocytosis of DNA by macrophages and m
28 gregated LDL taken up by macrophages through pinocytosis and phagocytosis, respectively, aggregated L
29  the cell, and it has been shown to regulate pinocytosis and phagocytosis; however, its effects on po
30 as(G12V) separately activates Rab5-dependent pinocytosis and Rac1-dependent membrane ruffling.
31 ed CDG greatly enhances Ag uptake, including pinocytosis and receptor-mediated endocytosis in vivo.
32 al cells and extend the half-life of IgG via pinocytosis and recycling into the extracellular milieu.
33  we measured internalization of gelonin (via pinocytosis) and gelonin-based immunotoxins (via antigen
34 l important virulence functions, fluid-phase pinocytosis, and adhesion to host cell monolayers.
35 ative regulator of cell growth, cytokinesis, pinocytosis, and phagocytosis, as all are enhanced in co
36 e trafficking events including phagocytosis, pinocytosis, and transferrin receptor recycling were als
37  stimulation, was independent of endocytosis/pinocytosis, and was consistent with bidirectional, tran
38 tudy demonstrates that membrane ruffling and pinocytosis are regulated by distinct Ras signal transdu
39 mined that the mutant strain was impaired in pinocytosis but normal in phagocytosis of beads or bacte
40                  The Ha-Ras(G12V)-stimulated pinocytosis but not membrane ruffling was abolished by e
41 cs the stimulatory effect of Ha-Ras(G12V) on pinocytosis but not membrane ruffling.
42              Cytochalasin B, which inhibited pinocytosis by 65% and phagocytosis by 93%, decreased ed
43 than that achieved previously by nonspecific pinocytosis, CBATP mediates time-, temperature- and conc
44 ied LDL by receptor-independent, fluid-phase pinocytosis converting these macrophages into foam cells
45 also taken up by cells in a temperature- and pinocytosis-dependent manner.
46 iameter beads, which can enter cells through pinocytosis, did not stimulate IL-12 expression.
47 ond, we found that CDG selectively activated pinocytosis-efficient-DCs, leading to T(H) polarizing cy
48 tive LDL by receptor-independent fluid-phase pinocytosis, either constitutively or in response to spe
49 -based behaviors that were tested, including pinocytosis, exocytosis, cytokinesis and morphogenesis,
50 eptor-mediated endocytosis and non-saturable pinocytosis for uptake of the conjugate.
51                                  Fluid-phase pinocytosis has been demonstrated by macrophages within
52 serve as models of LDL uptake by fluid-phase pinocytosis in cultured human monocyte-derived macrophag
53 amily of GTPases are involved in and control pinocytosis in dendritic cells.
54  The mechanisms of regulation of fluid-phase pinocytosis in macrophages and, specifically, the role o
55  SH2 domain, inhibited membrane ruffling and pinocytosis induced by GH and PDGF.
56 ed phagocytosis by >90% but had no effect on pinocytosis, inhibited edge cell liposome-DNA complex en
57 eptide transporter, and (iii) was blocked by pinocytosis inhibitors but not by brefeldin A.
58 oes not affect cell growth, phagocytosis, or pinocytosis, inhibits the formation of head-to-tail cell
59  phagocytosis, but not the receptor-mediated pinocytosis, is highly associated with the production of
60 fore sought to determine whether fluid-phase pinocytosis occurs in vivo in macrophages in atheroscler
61 ibited receptor-mediated endocytosis of LDL, pinocytosis of 10-nm microspheres, and phagocytosis of 2
62  overexpression, wortmannin abolished amebic pinocytosis of dextrans but had no inhibitory effects on
63                       Despite ongoing (macro)pinocytosis of extracellular fluid, the volume of the en
64  defective in capping and cytokinesis, while pinocytosis of fluorescent dextrans was not affected.
65                                  Fluid-phase pinocytosis of LDL by macrophages is regarded as a novel
66 se pinocytosis and indicate that fluid-phase pinocytosis of LDL is a mechanism for macrophage foam ce
67 s on an SP-A substrate demonstrated enhanced pinocytosis of mannose BSA and phagocytosis of Mycobacte
68 s not necessary for MR up-regulation, and 3) pinocytosis of mannose BSA via MR recycling was increase
69 , but degradation of internalized ligand and pinocytosis of the fluid-phase marker Lucifer Yellow wer
70 endocytosis, but not by compounds that block pinocytosis or cellular entry via scavenger or mannose r
71  demonstrate either constitutive fluid-phase pinocytosis or inducible fluid-phase pinocytosis.
72 cated by possible uptake through fluid-phase pinocytosis or membranous ducts.
73       HDMEC are similar to HUVEC in rates of pinocytosis or receptor-mediated endocytosis.
74  is internalized by a relatively nonspecific pinocytosis or transcytosis mechanism.
75  monocytes by receptor-mediated endocytosis, pinocytosis, or phagocytosis and measured internalizatio
76 is protein is not essential for cytokinesis, pinocytosis, phagocytosis, or development.
77 eristic signatures of neutrophil activation: pinocytosis, phagosomal alkalinisation, and human neutro
78 of wild-type SH2-Bbeta enhanced ruffling and pinocytosis produced by submaximal GH but not submaximal
79  mediated by phagocytosis and endocytosis or pinocytosis, respectively.
80       Thus, targeting macrophage fluid-phase pinocytosis should be considered when investigating stra
81 and was specific for endocytosis rather than pinocytosis since a TR mutant lacking an internalization
82 readouts: induction of membrane ruffling and pinocytosis, stimulation of cell motility, and Pak bindi
83 d Rac on plasma membrane morphology and bulk pinocytosis, there has been no evidence for their involv
84 to that by which Ags taken up by fluid phase pinocytosis traffick, suggesting that the accelerated BC
85 ve to control Ax2 cells, whereas fluid-phase pinocytosis was reduced threefold, primarily as the resu
86 nd activated Rho has been shown to stimulate pinocytosis when microinjected into Xenopus oocytes.
87 hosphatase inhibitor reduces Cdc42-regulated pinocytosis while stimulating caveolar endocytosis.