ライフサイエンスコーパス: pistil

1 xpanding leaves (about 3.5-fold less than in pistils).

2 ion involves interactions between pollen and pistil.

3 ecifically, in the transmitting tract of the pistil.

4 oral tissues to access the ovules within the pistil.

5 male gametophyte) that is encased within the pistil.

6 xpressing pollen tubes elongating within the pistil.

7 hat express specificities in common with the pistil.

8 tches either S-allele present in the diploid pistil.

9 e-specific rejection of "self" pollen by the pistil.

10 is induced in pollen tubes by growth in the pistil.

11 ing specific interactions between pollen and pistil.

12 promoters from genes expressed primarily in pistils.

13 1, GACO2, GACO3) were isolated from geranium pistils.

14 tically marked Col-0 and RIL pollen on Van-0 pistils.

15 marked Col-0 pollen and Van-0 pollen on RIL pistils.

16 with Col and Landsberg erecta pollen on RIL pistils.

17 pollinations with Col and RIL pollen on Col pistils.

18 loret in each spikelet on the ear includes a pistil abortion process that requires the action of the

19 M1 gene appears to perform a similar role in pistil abortion.

20 Binding between NaPCCP and NaSBP1 and the pistil AGPs may contribute to signaling and trafficking

21 ollen tube gene products that respond to the pistil and are required for reproductive success; moreov

22 In Papaver rhoeas, cognate pistil and pollen S-determinants, PrpS, a pollen-express

23 nflata S-locus F-box (Pi SLF), determine the pistil and pollen specificity, respectively.

24 ering plants, pollen grains germinate on the pistil and send pollen tubes down the transmitting tract

25 tion, corolla tube structure, nectar volume, pistil and stamen length) remains poorly understood.

26 S-RNase is produced in the pistil and taken up by pollen tubes in a non-S-haplotype

27 otein that is expressed predominantly in the pistils and anthers of Brassica flowers late in flower d

28 moderate levels in leaves, pedicels, sepals, pistils and petals, and at very low levels in roots.

29 Message levels of PhETR1 and PhETR2 in pistils and receptacles are unaffected by self-pollinati

30 n, and nitrogen allocation to female whorls (pistils and sepals) decreased under high density, wherea

31 in floral organ size including elongation of pistils and shortened stamen filaments that resulted in

32 ated hypocotyls (about 2.5-fold less than in pistils) and in young expanding leaves (about 3.5-fold l

33 ally similar tissues such as leaves, anther, pistil, and embryo, while orthologs that are highly expr

34 owth, similar to what normally occurs in the pistil, and this ability correlates with the accumulatio

35 ot density, delayed sepal opening, elongated pistils, and reduced fertility in the primary infloresce

36 TTS) and 120-kDa glycoprotein (120K) are two pistil arabinogalactan proteins (AGPs) that share a cons

37 uidance on extracellular matrices within the pistil are essential processes that convey the pollen tu

38 sms of guidance for pollen tubes through the pistil are not known, the female tissues play a critical

39 len tubes are reduced when NaStEP-suppressed pistils are pollinated with either compatible or incompa

40 ecific antibody first detects the protein in pistils at one day prior to flowering, with higher level

41 nd weakened (or absent) interspecific pollen-pistil barriers.

42 control the enhanced style elongation of pro pistils, because its expression was not higher in pro st

43 minish pollen tube length in vitro or in the pistil, but it reduces ovule targeting by twofold.

44 LeSTIG1, a small cysteine-rich protein from pistil, can bind the extracellular domains of both LePRK

45 17; Chi2;1) identified by screening a tomato pistil cDNA library has been found to encode a protein s

46 results suggest that cytokinin can determine pistil cell fate during maize floret development.

47 ncluding deterioration and death in specific pistil cell types.

48 Ill-fated pistil cells undergo a cell death process associated wit

49 d1 and tasselseed2 are required for death of pistil cells.

50 for the accumulation of TASSELSEED2 mRNA in pistil cells.

51 The establishment of pollen-pistil compatibility is strictly regulated by factors de

52 f non-functional S-haplotypes with disrupted pistil component (stylar-S) and/or pollen component (pol

53 t, nonfunctional S-haplotypes with disrupted pistil component (stylar-S) and/or pollen component (pol

54 e with S3-, S5-, S7-, S11-, and S13-carrying pistils, confirming that other SLF proteins are responsi

55 In addition, cbl10 mutant pistils could not sustain the growth of wild-type pollen

56 ollen tube growth assays in vitro and in the pistil demonstrate that hydroxyl free radicals are likel

57 Aniline blue staining of pollinated pistils demonstrated that pollen tube growth was affecte

58 y interactions: the S-RNase gene encodes the pistil determinant and the previously unidentified S-gen

59 The S-RNase gene encodes the pistil determinant, whereas the pollen determinant gene,

60 tains two separate genes encoding pollen and pistil determinants in SI interactions.

61 t positive regulators or effectors of SI and pistil development are regulated by ta-siRNA(s).

62 We show that ARF3, a regulator of pistil development that is expressed in the vascular tis

63 CO1, and leafy, genes regulating anther and pistil development, and stress-related transcription fac

64 ed expression during certain stages of early pistil development, Cel4 mRNA was also detected at high

65 y pathway and the dual role of SRK in SI and pistil development, our study provides a molecular expla

66 for PCP in pollen-pistil interactions or in pistil development.

67 iator of cross-talk between SI signaling and pistil development.

68 hich are normally expressed very late in the pistil developmental pathway and function in the final s

69 efore, it could function in the transport of pistil ECM proteins in the pollen tube endomembrane syst

70 receptor kinase (SRK) gene further enhances pistil elongation and stigma exsertion in this mutant ba

71 are essential for pollen tube elongation in pistil, especially, free Ca(2+) providing a concentratio

72 ection response, the identification of three pistil essential modifier genes unlinked to the S-locus

73 ucuronidase staining occurred throughout the pistil, except in the stigma.

74 ted predominantly in the stigma and style of pistils excised from open flowers; much lower levels of

75 expressed transmembrane protein, and PrsS, a pistil-expressed secreted protein, interact to trigger a

76 TS) protein, 120 kDa glycoprotein (120K) and pistil extensin-like protein III (PELP III) are stylar g

77 ovary, they are in constant contact with the pistil extracellular matrix (ECM).

78 interaction between pollen proteins and the pistil extracellular matrix.

79 We isolated an Arabidopsis pistil extract fraction that stimulates Arabidopsis poll

80 ompound that is specifically present in this pistil extract fraction.

81 ect of pistils on pollen germination and the pistil factors that stimulate pollen germination remain

82 A model is proposed for the control of pistil fate by the action of the ts1-ts2-sk1 pathway.

83 ordia express TASSELSEED2 RNA but functional pistils found in ear spikelets are protected from cell d

84 highest levels of transcript accumulation in pistils from flowers at anthesis.

85 mutations in pollen-S that reduce the set of pistils from which the pollen accepts inhibition and dis

86 wing pollination, the upper and lower floret pistils fuse, producing a connated kernel with two genet

87 Most pro pistils had exserted stigmas, thus preventing self-polli

88 o be correlated with rapid cell expansion in pistils, hypocotyls and leaves.

89 The pistil in each floret was fertile, but the spikelet prod

90 m pollen tubes to penetrate farther into the pistil in HT suppressed plants, but not to reach the ova

91 PRKs) control pollen tube growth through the pistil in response to extracellular signals, and regulat

92 stamen in ear spikelets and the abortion of pistils in both the tassel spikelets and in the secondar

93 in plantacyanin levels in the overexpression pistil, including the transmitting tract.

94 y involves separate genes for the pollen and pistil incompatibility recognition processes.

95 -carboxylic acid (ACC), to the flower or the pistil induced overall deterioration in the entire flowe

96 Pollen and pistil interact to kill invading pollen from an incompat

97 specific gene ontology classes (e.g., pollen-pistil interaction) in apomicts implies that gene enrich

98 nsduction' pathway is enhanced during pollen-pistil interaction.

99 MP) mating systems, and characterized pollen-pistil interactions among S. habrochaites populations an

100 The high degree of specificity in pollen-pistil interactions and the precision of directional pol

101 Pollen-pistil interactions are critical early events regulating

102 Initial pollen-pistil interactions in the Brassicaceae are regulated by

103 genes in compatible and incompatible pollen-pistil interactions is discussed.

104 stent with a possible role for PCP in pollen-pistil interactions or in pistil development.

105 l compatibility results from multiple pollen-pistil interactions with additive effects.

106 ial signal transduction components of pollen-pistil interactions, and isolated two structurally relat

107 evelopment is dependent on successful pollen-pistil interactions.

108 and LePRK2 are involved in mediating pollen-pistil interactions.

109 olarized secretion, in the context of pollen-pistil interactions.

110 Pollen tube elongation in the pistil is a crucial step in the sexual reproduction of p

111 Although the pistil is a great facilitator of pollen function, it can

112 imination of self and non-self pollen by the pistil is controlled by a single polymorphic locus, the

113 ation between self and nonself pollen by the pistil is controlled by the highly polymorphic S-RNase g

114 elf recognition mechanism between pollen and pistil is controlled by two polymorphic genes at the S-l

115 elf-/non-self-recognition between pollen and pistil is regulated by the pistil-specific S-RNase gene

116 combination with either HT-A or HT-B in the pistil is sufficient to cause rejection of pollen from a

117 determinants of S-allele specificity in the pistil, it is not known how allele-specific information

118 expression patterns in subepidermal cells of pistils just before abortion.

119 brary enriched in transcripts present in the pistil late in flower development - potentially encodes

120 the pollen tube nucleus during growth in the pistil leads to pollen tube differentiation required for

121 locus genes, which control anther position, pistil length and pollen size in pin and thrum flowers,

122 long stamen length, short stamen length, and pistil length) in a cosmopolitan sample of 15 ecotypes.

123 of reproductive and immune responses of the pistil makes it a prime system in which to study the con

124 llen rejection; therefore, NaStEP is a novel pistil-modifier gene.

125 To date, two essential pistil-modifier genes, 120K and High Top-Band (HT-B), ha

126 ponents are taken up during growth, and some pistil molecules exert their effect inside the pollen tu

127 subtle pleiotropic effects on both sepal and pistil morphology.

128 The pistil of 28-5 ap2-1 mutant flowers shows a structure si

129 c self-incompatibility mechanism enables the pistil of a plant to reject self-pollen and therefore pr

130 sm between the pollen of one species and the pistil of another.

131 nsmitting tissue extracellular matrix of the pistil of tobacco.

132 d in leaf and flower abscission zones and in pistils of fully open flowers.

133 y expressed in both abscission zones and the pistils of mature flowers.

134 -RNase, the determinant of SI specificity in pistils of Nicotiana alata.

135 ith higher levels of the protein seen in the pistils of open flowers.

136 hat lack S-RNase, a key protein expressed in pistils of SI Solanum species.

137 .1, are required for pollen compatibility on pistils of SI species or hybrids.

138 ction often follows the 'SI x SC' rule, i.e. pistils of SI species reject the pollen of SC (self-comp

139 ible (SC) red-fruited species is rejected on pistils of the predominantly self-incompatible (SI) gree

140 Pistils of the wild tomato relative Solanum lycopersicoi

141 ns during pollination between pollen and the pistil on which it lands.

142 However, in Arabidopsis, the effect of pistils on pollen germination and the pistil factors tha

143 ary analysis of mutations that affect either pistil or pollen specificity indicates that natural sele

144 on typically favored increased allocation to pistils (or stamens) but decreased allocation to other w

145 s exhibit normal growth and guidance in pop2 pistils, perhaps by degrading excess GABA and sharpening

146 CE1 mRNA accumulated specifically within the pistil, petals, and stamen filaments.

147 Wild-type pistils pollinated with a limited number of single and d

148 tic self-incompatibility (GSI) system in the pistil predates speciation.

149 ) has been shown to be identical to a tomato pistil-predominant EGase cDNA, TPP18.

150 All pistil primordia express TASSELSEED2 RNA but functional

151 Abortion of pistil primordia in staminate florets is controlled by a

152 Elimination of pistil primordia occurs in the primary and secondary flo

153 blocks tasselseed-induced cell death in the pistil primordia of primary ear florets.

154 Our results show that Arabidopsis pistils promote germination by producing azadecalin-like

155 rmination by restricting the function of the pistil-protecting factor, silkless1, from the apical inf

156 use specific interactions between pollen and pistil proteins as "self" recognition and/or rejection m

157 The pistil provides everything the pollen requires for succe

158 ences between male pollen release and female pistil receptivity (dichogamy), and self-pollen rejectio

159 nia possesses self-incompatibility, by which pistils reject self-pollen but accept non-self-pollen fo

160 intra-specific reproductive barrier by which pistils reject self-pollen to prevent inbreeding and acc

161 ce interactions in male (pollen) and female (pistil) reproductive tissues.

162 ts and that at least LePRK2 may mediate some pistil response.

163 PrpS is a single-copy gene linked to the pistil S gene (currently called S, but referred to herea

164 patible) pollen by interaction of pollen and pistil S locus components, and is subsequently inhibited

165 In Papaver rhoeas, the pistil S locus product is a small protein that interacts

166 /S-locus Protein11 (SCR/SP11) ligand and the pistil S Receptor Kinase (SRK).

167 multiallelic S locus, comprising pollen and pistil S-determinants.

168 ceae, the S-specific interaction between the pistil S-RNase and the pollen S-Locus F-box protein cont

169 The protein products of S alleles in the pistil, S proteins, were initially identified based on t

170 ms, roots, and cotyledons) and reproductive (pistils, sepals, petals, stamens, and floral buds) organ

171 cific reproductive barriers or loss of known pistil SI factors.

172 ecific and interpopulation compatibility and pistil SI factors.

173 For example, the pistil SI proteins S-RNase and HT protein function in a

174 roteins S-RNase and HT protein function in a pistil-side IRB that causes rejection of pollen from sel

175 Expression of pistil-side UI is weakened in S. lycopersicum x S. lycop

176 When present in the pistil (silk and ovary) a number of maize genes discrimi

177 The class III pistil-specific extensin-like protein (PELPIII) was cons

178 etween pollen and pistil is regulated by the pistil-specific S-RNase gene and by multiple pollen-spec

179 s a gene encoding an S-RNase, which controls pistil specificity, and multiple S-locus F-box (SLF) gen

180 ty in Petunia inflata; the S-RNase regulates pistil specificity, and multiple S-locus F-box (SLF) gen

181 LHS1 expression in pistils, stamens, and lodicules varies among the cereals

182 Expression in the pistil steadily increases during flower development and

183 hat stigma, style, and ovules in Arabidopsis pistils stimulate pollen germination.

184 In many plants, pistils stimulate pollen germination.

185 enhance reproductive defects in lre-5/lre-5 pistils, suggesting that LLG1 function is not redundant

186 cloned from tomato (Lycopersicon esculentum) pistils, than to any other reported EGases.

187 we purified an additional molecule from the pistil that enhances pollen tube adhesion when combined

188 between pollen/pollen tubes and cells of the pistil that line their path.

189 type pollen was used to pollinate the mutant pistil, the pollinated 28-5 silique became >10% longer a

190 As pollen tubes grow through the pistil they are thought to perceive and respond to diver

191 ems and roots) as well as in floral tissues (pistil tips, developing anthers and sepal vasculature).

192 to a ca. 2.4 kb mRNA primarily expressed in pistil tissues.

193 n shown to indeed control the ability of the pistil to recognize and reject self pollen.

194 ompatibility (SI), which allows cells of the pistil to recognize and specifically inhibit "self" poll

195 cialized extracellular matrices (ECM) of the pistil to the ovule.

196 pollen-tube success and its variation among pistils to pollen receipt.

197 This enabled pistils to reject pollen expressing cognate PrpS.

198 a mutation in the S-RNase gene that enables pistils to reject the new pollen type.

199 ch allows the female reproductive organ, the pistil, to distinguish between self pollen and non-self

200 differentially expressed genes (DEGs) in the pistil transcriptomes of Arabidopsis thaliana and Arabid

201 of the angiosperms, pollen tubes grow in the pistil transmitting tract (TT) and are guided to the ovu

202 nting self-pollination, similar to wild-type pistils treated with GA(3) or auxins.

203 howed that LTP5 is present in pollen and the pistil TT in low levels.

204 Phosphorus allocation decreased by half in pistils under drought, while stamen phosphorus was unaff

205 Activity was not apparent in pistils until the flowers had opened and was confined to

206 e and methylsalicylate emission, whereas the pistil was the main source of benzylbenzoate.

207 Ethylene production in geranium pistils was not autocatalytic.

208 mosaic virus 35S, and protein levels in the pistil were examined as well as the pollination process.

209 ntrast to its non-expression in unpollinated pistils, where expression decreased after anthesis.

210 st number of DEGs was identified in infected pistils, where genes encoding regulators of cell divisio

211 ID1: GID1A is expressed throughout the whole pistil, while GID1B is expressed in ovules, and GID1C is

212 c S locus where "self" pollen is rejected on pistils with matching S alleles.

213 significantly higher levels than HLP mRNA in pistils, with the opposite pattern in leaves.