ライフサイエンスコーパス: pituitary

1 thke's pouch - the precursor of the anterior pituitary.

2 spheres, optic tectum/tegmentum, retina, and pituitary.

3 MNCs) and hormone release from the posterior pituitary.

4 cells and their projections in the brain and pituitary.

5 release OT into the blood from the posterior pituitary.

6 t the same relationship likely exists in the pituitary.

7 e and follicle-stimulating hormone) from the pituitary.

8 regulator of TGFbeta/Activin pathways in the pituitary.

9 tegrated development of the hypothalamus and pituitary.

10 nfundibulum - the precursor of the posterior pituitary.

11 tial are drastically increased in the mutant pituitaries.

12 ersible damage, such as neurologic (75%) and pituitary (72.9%) injuries.

13 Pituitary abnormalities were identified in a large propo

14 te matter T2-weighted hyperintense areas and pituitary abnormalities, with a low incidence of microhe

15 Pituitary adenoma (PA) is one of the most common intracr

16 However, the potential role of LDHA in pituitary adenoma (PA) remains unknown.

17 and the most common cause in adulthood is a pituitary adenoma, or treatment with pituitary surgery o

18 /secretion, cell viability and apoptosis) in pituitary adenomas (n = 74), and to compare with the res

19 Non-functioning pituitary adenomas (NFPAs) are the most frequent pituita

20 nfirmed in a validation group of 37 surgical pituitary adenomas and 11 normal pituitary glands.

21 multiple, clinically relevant parameters on pituitary adenomas and may represent a valuable therapeu

22 Pituitary adenomas may hypersecrete hormones or cause ma

23 Observations: Prevalence of pituitary adenomas ranges from 1 in 865 adults to 1 in 2

24 Patients with pituitary adenomas should be identified at an early stag

25 markers to predict the ultimate response of pituitary adenomas to BIM-23A760.

26 For all other pituitary adenomas, initial therapy is generally transsp

27 her conditions such as aseptic meningitis or pituitary adenomas.

28 emerged as promising new approaches to treat pituitary adenomas.

29 Pituitary adenylate cyclase activating polypeptide (PACA

30 stress (CVS) has been shown to increase BNST pituitary adenylate cyclase activating polypeptide (PACA

31 determine the effects of blocking glutamate, pituitary adenylate cyclase activating polypeptide, and

32 Chemicals, such as glutamate and pituitary adenylate cyclase activating polypeptide, whos

33 The activation of pituitary adenylate cyclase-activating peptide (PACAP) s

34 Pituitary adenylate cyclase-activating polypeptide (PACA

35 Recent work indicates that pituitary adenylate cyclase-activating polypeptide (PACA

36 The G protein-coupled pituitary adenylate cyclase-activating polypeptide recep

37 ist verucerfont potently blocks hypothalamic-pituitary adrenal (HPA) axis activation in adrenalectomi

38 and competition may involve the hypothalamic-pituitary-adrenal (HPA) and hypothalamic-pituitary-gonad

39 tream molecular consequences of hypothalamic-pituitary-adrenal (HPA) axis activation by exogenous adr

40 iday" so that the potential for hypothalamic-pituitary-adrenal (HPA) axis activation might be mitigat

41 independent of their effects on hypothalamic pituitary-adrenal (HPA) axis activation, aversive condit

42 promising measure of long-term hypothalamus-pituitary-adrenal (HPA) axis activity.

43 aviors as well as regulation of hypothalamic-pituitary-adrenal (HPA) axis activity.

44 se, including activation of the hypothalamic-pituitary-adrenal (HPA) axis and increases in anxiety be

45 fective disturbance and promote hypothalamic-pituitary-adrenal (HPA) axis dysregulation, a key featur

46 cits, by reestablishment of the hypothalamic-pituitary-adrenal (HPA) axis feedback and corticosterone

47 The hypothalamic-pituitary-adrenal (HPA) axis has been implicated in the

48 Genetic variation within the hypothalamic-pituitary-adrenal (HPA) axis has been linked to risk for

49 The stress-responsive hypothalamo-pituitary-adrenal (HPA) axis plays a central role in pro

50 ute CIRCI: dysregulation of the hypothalamic-pituitary-adrenal (HPA) axis, altered cortisol metabolis

51 The normal function of the hypothalamic-pituitary-adrenal (HPA) axis, and resultant glucocortico

52 nge is through regulation of the hypothalamo-pituitary-adrenal (HPA) axis, the neuroendocrine system

53 s, leading to activation of the hypothalamic-pituitary-adrenal (HPA) axis, the sympathetic nervous sy

54 derived cytokines stimulate the hypothalamic-pituitary-adrenal (HPA) axis, triggering endogenous gluc

55 ion via hormonal priming of the hypothalamic-pituitary-adrenal (HPA) axis.

56 s from adrenergic input and the hypothalamus-pituitary-adrenal (HPA) axis.

57 xis, the fish equivalent of the hypothalamic-pituitary-adrenal (HPA) axis.

58 mmalian stress response via the hypothalamic-pituitary-adrenal (HPA) axis.

59 between the limbic forebrain and hypothalamo-pituitary-adrenal (HPA) effector neurons in the paravent

60 he newborn decreases the limbic-hypothalamic-pituitary-adrenal (LHPA) axis activity in the offspring.

61 promotes the activation of the hypothalamic-pituitary-adrenal and hypothalamic-pituitary-thyroid axe

62 ubpopulations with demonstrable hypothalamic-pituitary-adrenal axis abnormalities.

63 Instead of consecutive hypothalamus-pituitary-adrenal axis activation, we report that acute

64 related to anxiety behavior and hypothalamic-pituitary-adrenal axis activity, likely through modulati

65 (GR) expression, and increased hypothalamic-pituitary-adrenal axis activity.

66 stress-mediated attenuation of hypothalamic-pituitary-adrenal axis activity.

67 ic inflammation, or exacerbated hypothalamic-pituitary-adrenal axis activity.

68 is that PVN Sirt1 activates the hypothalamic-pituitary-adrenal axis and basal GC levels by enhancing

69 kely mediated by inhibiting the hypothalamic-pituitary-adrenal axis and inflammatory responses to str

70 data reveal that impairment of hypothalamic-pituitary-adrenal axis during depression can lead to olf

71 tic actions without influencing hypothalamic-pituitary-adrenal axis function.

72 t in GABAergic, neurons induced hypothalamic-pituitary-adrenal axis hyperactivity and reduced fear- a

73 ion, microglial activation, and hypothalamic-pituitary-adrenal axis hyperactivity in stress vulnerabi

74 The hypothalamic-pituitary-adrenal axis is a dynamic system regulating gl

75 The hypothalamic-pituitary-adrenal axis is a pivotal component of an orga

76 The hypothalamus-pituitary-adrenal axis is sensitive to changes in the ea

77 vation of catecholaminergic and hypothalamic-pituitary-adrenal axis leads to splenic atrophy and cont

78 of depression to study whether hypothalamic-pituitary-adrenal axis perturbation could be sufficient

79 important central component of hypothalamic-pituitary-adrenal axis regulation that prepares the orga

80 nding protein 51 is involved in hypothalamic-pituitary-adrenal axis regulation.

81 Evaluation of the hypothalamic-pituitary-adrenal axis response in these animals reveale

82 ted Glp1r knockdown had reduced hypothalamic-pituitary-adrenal axis responses to both acute and chron

83 adversity group showed altered hypothalamus-pituitary-adrenal axis responses to stress, evidenced by

84 cess glucocorticoid release via hypothalamus-pituitary-adrenal axis stimulation.

85 howed a significantly increased hypothalamic-pituitary-adrenal axis stress response and impaired sens

86 sympathetic nervous system and hypothalamic-pituitary-adrenal axis) transcription factor activation.

87 oes not promote arousal via the hypothalamic-pituitary-adrenal axis, but rather probably acts via bra

88 rine stress hormone system, the hypothalamic-pituitary-adrenal axis, contributes to variability in st

89 a indicate abnormalities in the hypothalamic-pituitary-adrenal axis, including signaling by corticotr

90 cortex (PFC), the amygdala, and hypothalamic-pituitary-adrenal axis, the precise genetic and experien

91 luding on the regulation of the hypothalamic-pituitary-adrenal axis, thereby affecting an individual'

92 growth, and inactivation of the hypothalamic-pituitary-adrenal axis, without affecting energy expendi

93 ocampus-a region modulating the hypothalamic-pituitary-adrenal axis-and somatosensory, viscerosensory

94 ctivity of the amygdala and the hypothalamic-pituitary-adrenal axis.

95 ic conditions, by affecting the hypothalamic-pituitary-adrenal axis.

96 RH) is a major regulator of the hypothalamic-pituitary-adrenal axis.

97 natal growth, cardiovascular development and pituitary-adrenal function of isolated chronic developme

98 gnaling reduces the activity of hypothalamic-pituitary-adrenal pathways via actions in specific brain

99 urotrophic factors, normalizing hypothalamic-pituitary-adrenal reactivity, and the reduction of neuro

100 aint abolished their heightened hypothalamic-pituitary-adrenal responsivity and reduced stress-induce

101 embryonic HSPC numbers via the hypothalamic-pituitary-adrenal/interrenal (HPA/I) stress response axi

102 the expression of genes in the hypothalamic-pituitary-adrenal/stress system (e.g., Crhr1) is one of

103 The hypothalamo-pituitary-adrenocortical (HPA) axis regulates stress phy

104 ts hypothesis and indicate that hypothalamic-pituitary-adrenocortical (HPA) axis regulation is mediat

105 nalog, buserelin using a label-free assay in pituitary alphaT3-1 cells with endogenous GnRH receptor

106 the Galphaq-coupling of the GnRH receptor in pituitary alphaT3-1 cells.

107 optic area that project directly towards the pituitary and form an interface with the pituitary vascu

108 both in thyrotropes and gonadotropes of the pituitary and in Leydig and germ cells in the testes, bu

109 is was conducted in the cerebellum, thalamus-pituitary and liver of tilapia treated with equimolar do

110 < 0.05) in the tilapia cerebellum, thalamus-pituitary and liver, respectively.

111 lls co-localized with dendritic cells in the pituitary and produced increased levels of interferon-ga

112 er sex-biased differential expression in the pituitary as compared to the hypothalamus, with multiple

113 /CORT and obesity in the control of relevant pituitary-axes and whole-body metabolism.

114 He was offered screening for hypothalamic-pituitary axis (HPA) dysfunction because of his suprasel

115 Salivary cortisol (hypothalamic pituitary axis), heart rate variability (sympathetic adr

116 ess are largely mediated by the hypothalamic-pituitary axis, a highly conserved neurohormonal cascade

117 le genes more highly expressed in the female pituitary being related to reproduction, growth, and dev

118 iple genes more highly expressed in the male pituitary being related to secretory function, and multi

119 In tumoral pituitaries, BIM-23A760 also inhibited Ca(2+) concentrat

120 In the adult pituitary, BMPs participate in the control of hormone se

121 ne (GHRH) regulates the release of GH by the pituitary but also exerts separate actions on peripheral

122 xamined their distributions in the brain and pituitary by immunohistochemistry.

123 express high levels of CTLA-4 antigen in the pituitary can cause an aggressive (necrotizing) form of

124 se RP progenitors and for differentiation of pituitary cell types.

125 Here, we used primary pituitary cell-cultures from two normal nonhuman-primate

126 regulatory circuit that relevantly modulate pituitary cell-function.

127 factors involved in the functioning of these pituitary cell-types (e.g. GHRH/ghrelin/somatostatin/ins

128 dipokines on the functioning of all anterior-pituitary cell-types.

129 and LPXRFa-R signaling acts directly on the pituitary cells independent from GnRH or kisspeptin and

130 sive element, we sought to determine whether pituitary cells secrete BMPs or BMP antagonists.

131 In this study, we found that, in pituitary cells, all components of paraspeckles includin

132 Interestingly, we found that pituitary-conditioned medium contains a factor that inhi

133 ar whether other factors besides an enlarged pituitary contribute to the hypersecretion.

134 This high pituitary CTLA-4 expression was associated with T-cell i

135 Pituitary CTLA-4 expression was confirmed in a validatio

136 n a cohort of patients with forebrain and/or pituitary defects.

137 R-7 family, miR-7a2, is essential for normal pituitary development and hypothalamic-pituitary-gonadal

138 e show that ZBTB20 is essential for anterior pituitary development and lactotrope specification in mi

139 o ZBTB20 as a critical regulator of anterior pituitary development and lactotrope specification.

140 disease of numerous organs, its role during pituitary development and tumourigenesis remains largely

141 stained proliferative capacity and disrupted pituitary differentiation.

142 h an increasing number of suspected cases of pituitary diseases, there has been a paradigm shift in t

143 The risk of hypothalamic-pituitary dysfunction (n = 138) was associated with radi

144 guidance for the screening and management of pituitary dysfunction in adult patients with TBI.

145 Pituitary dysfunction is a recognised, but potentially u

146 It commonly leads to pituitary dysfunction.

147 t endocrine-metabolic alterations, including pituitary dysregulations.

148 of POGZ, particularly in the cerebellum and pituitary, early in fetal brain development.

149 CTLA-4 antigen was expressed by pituitary endocrine cells in all patients but at differe

150 Moreover, pituitary enlargement may lead to compressive symptoms,

151 meningeal enhancement, sagging of the brain, pituitary enlargement, and subdural fluid collections.

152 at expression of integrin beta1 in embryonic pituitary epithelial cells is required for angiogenesis

153 Within pituitary epithelial cells, integrin beta1 directs a lar

154 Deletion of pituitary epithelial integrin beta1 before the onset of

155 Primate and human normal pituitaries exhibited similar sst2/sst5/D2 expression pa

156 Finally, we found that primate pituitaries expressed leptin/adiponectin/resistin.

157 and estrogen concentrations via hypothalamic-pituitary feedback regulation and prolonged ligand half-

158 g for ACTH may be necessary to distinguish a pituitary from an ectopic source.

159 Repeated pituitary function assessment at regular intervals is ne

160 e a mechanism by which melatonin can control pituitary function in a seasonal manner.

161 While ovarian feedback on hypothalamo-pituitary function is a well-established concept, the pr

162 no recurrence and partial restoration of the pituitary function was seen.

163 it that contribute to the fine-regulation of pituitary functions.

164 protein and localized a specific area of the pituitary gland (i.e., adenohypophysis) known to secrete

165 well as real data obtained from mouse liver, pituitary gland and data from NIH3T3, U2OS cell lines.

166 (AH) is a rare inflammatory condition of the pituitary gland and usually affects women of childbearin

167 ith a complete absence of vasculature in the pituitary gland at birth.

168 ne with mild lymphocytic infiltration in the pituitary gland but no clinical signs of hypophysitis, a

169 Resection of the pituitary gland causes iatrogenic hypopituitarism which

170 art because no pathologic examination of the pituitary gland has been reported to date.

171 yonic development of the hypothalamus and/or pituitary gland in humans results in congenital hypopitu

172 ocin (OT) originates from secretion from the pituitary gland into the circulation and from absorption

173 of the vertebrate neuroendocrine system, the pituitary gland relies on the progressive and coordinate

174 Patients with craniopharyngeoma or a pituitary gland tumor were excluded.

175 des, significant radioactivity uptake in the pituitary gland was observed (SUV of 0.7 at 30 min pi).

176 neuropeptides from tissue in situ (i.e., rat pituitary gland).

177 e regulators of prolactin secretion from the pituitary gland, also release GABA within the hypothalam

178 1)C-dihydroergotamine in the choroid plexus, pituitary gland, and venous sinuses as expected from the

179 ated by the pars tuberalis (PT) of the fetal pituitary gland, before the fetal circadian system and a

180 is expressed in the developing forebrain and pituitary gland, but its role during hypothalamo-pituita

181 in effective imaging of the adrenal glands, pituitary gland, lymph nodes, pancreas, and thyroid and

182 gulator of developmental angiogenesis in the pituitary gland, thus providing insight into the long-st

183 ly innervating different brain areas and the pituitary gland, which could represent an important fact

184 ther T lymphocytes undergo activation in the pituitary gland.

185 y infiltration of T and B lymphocytes in the pituitary gland.

186 stic mass involving the anterior lobe of the pituitary gland.

187 sequenced from the posterior lobe of the rat pituitary gland.

188 red for angiogenesis in the developing mouse pituitary gland.

189 ion of autoreactive T and B cells within the pituitary gland.

190 d by a 50% threshold for all lesions and the pituitary gland; and for (18)F-FDG (C)-RD of SUVs of the

191 We analyzed at autopsy the pituitary glands of six cancer patients treated with CTL

192 37 surgical pituitary adenomas and 11 normal pituitary glands.

193 mic-pituitary-adrenal (HPA) and hypothalamic-pituitary-gonadal (HPG) axes in underlying some of these

194 The hypothalamic-pituitary-gonadal (HPG) axis is a key biological system

195 ormal pituitary development and hypothalamic-pituitary-gonadal (HPG) function in adulthood.

196 main regulators of the pituitary-thyroid and pituitary-gonadal axes.

197 is of genes associated with the hypothalamic-pituitary-gonadal axis (HPG axis) in fish had become a c

198 The hypothalamic-pituitary-gonadal axis controls puberty and reproduction

199 of metabolic information to the hypothalamo-pituitary-gonadal axis is mediated by leptin receptors o

200 neuroendocrine system along the hypothalamus-pituitary-gonadal axis; however, most studies address ei

201 by affective comorbidities and hypothalamic-pituitary-gonadal dysregulation.

202 and transcriptional analysis of hypothalamic-pituitary-gonadal-liver axis revealed negligible effects

203 lated the Fshb expression levels obtained in pituitary gonadotrope cells perifused with varying GnRH

204 Pituitary gonadotropin hormones are regulated by gonadot

205 GnRH regulates the pituitary gonadotropin's follicle-stimulating hormone (F

206 Sex-dependent pituitary growth hormone (GH) secretory profiles-pulsati

207 The anterior pituitary harbours five distinct hormone-producing cell

208 Pituitary homeobox 1 (PITX1) functions as a tumor suppre

209 Sox2 mutations are associated with pituitary hormone deficiencies and the protein is requir

210 growth pathogenesis associated with combined pituitary hormone deficiency.

211 ntibody that targets the beta-subunit of the pituitary hormone follicle-stimulating hormone (Fsh) inc

212 The anterior pituitary hormone prolactin exerts important physiologic

213 median eminence (ME) to control long-lasting pituitary hormone rhythms essential for homeostasis.

214 amp analyses and are associated with reduced pituitary hormone secretion from AtT-20 cells.

215 idism, and the role of IGSF1 as regulator of pituitary hormone secretion.

216 ction (including serum concentrations of the pituitary hormone thyrotropin and the thyroid hormones t

217 nhibited the expression/secretion of several pituitary hormones (specially GH/PRL), which was accompa

218 aracterized by the deficiency of one or more pituitary hormones and can present alone or in associati

219 bernation, and metabolism, are controlled by pituitary hormones released in response to annual enviro

220 cterized by an increased release of anterior pituitary hormones, whereas altered target-organ sensiti

221 mice, we assayed for changes in thyroid and pituitary hormones.

222 itary gland, but its role during hypothalamo-pituitary (HP) axis formation or involvement in human CH

223 Disruption of Zbtb20 leads to anterior pituitary hypoplasia, hypopituitary dwarfism and a compl

224 t was confirmed in the hGH1 transgenic mouse pituitary in situ Occupancy was reduced in mice fed a hi

225 In conclusion, we show that pituitary-infiltrating lymphocytes proliferate in situ d

226 nhibit FSH secretion, indicating a status of pituitary inhibin B resistance.

227 sbLPXRFa cells might represent the source of pituitary innervation.

228 g., heterotopia, Dandy-Walker malformation), pituitary insufficiency, and/or synpolydactyly.

229 ehaviour and functioning of the hypothalamic-pituitary-interrenal (HPI) axis, the fish equivalent of

230 tment, mitotane therapy within 6 months, and pituitary irradiation within 10 years.

231 as its receptor (LPXRFa-R) in the brain and pituitary is important for understanding their multiple

232 In the pituitary, LPXRFa fibers are closely associated with gon

233 In the assessment of a pituitary mass, objective visual field testing represent

234 n the differential diagnosis of solid-cystic pituitary masses along with clinical correlation, which

235 ormones are in the pars distalis (PD) of the pituitary, melatonin receptors are localized in the pars

236 efore, SOX2 has two independent roles during pituitary morphogenesis; firstly, promotion of progenito

237 th the responses of normal primate and human pituitaries (n = 3-5).

238 file were favouring the diagnosis of AH over pituitary neoplasm.

239 enes are selectively activated in either the pituitary or the placenta by distinct components of a re

240 rgans, including the uterus and the anterior pituitary, or the proliferation of MCF-7a breast cancer

241 onate to adulthood, we investigated a common pituitary origin for hypothyroidism and macroorchidism,

242 menstrual patterns by altering hypothalamic-pituitary-ovarian axis function.

243 ough its central control of the hypothalamic-pituitary-ovarian axis, but also likely through peripher

244 cussion and the function of the hypothalamic-pituitary-ovarian axis.

245 hematical model of the hormonal hypothalamus-pituitary-ovarian control of ovulation in women.

246 o participation of cells in the hypothalamus-pituitary-ovary feedback control loop.

247 del of the female rainbow trout hypothalamus-pituitary-ovary-liver axis to use as a tool to help unde

248 f Foxa2 mRNA in the developing hypothalamus, pituitary, pancreas, lungs and oesophagus of mouse embry

249 d B cells underwent proliferation within the pituitary parenchyma.

250 alterations within the hypothalamic-anterior pituitary-peripheral hormonal axes that are proportionat

251 n, and their fibers innervated the brain and pituitary profusely.

252 deficiencies and the protein is required for pituitary progenitor proliferation, but its function has

253 tect the body from osmotic stress, posterior pituitary-projecting, vasopressin-secreting neurons (VPp

254 apted to the dynamic dopaminergic control of pituitary prolactin secretion, a key reproductive hormon

255 amine neurons are involved in the control of pituitary prolactin secretion, and the GABAergic subpopu

256 hesis and secretion through their effects on pituitary prostaglandin and BMP4 signaling.

257 effects of BIM-23A760 in normal and tumoral pituitaries remains incomplete.

258 higher expression of the Fshb subunit in the pituitary, resulting in elevated serum estrogen and high

259 afV600E and KrasG12D in the developing mouse pituitary, results in severe hyperplasia and abnormal mo

260 ys local-global integration to connect brain-pituitary rhythms and pace hormone secretion.

261 Physiologic distribution was seen in the pituitary, salivary glands, thyroid, and spleen, with lo

262 s expressed in hypothalamic GHRH neurons and pituitary somatotropes.

263 nocorticotropic hormone concentration, and a pituitary source of Cushing's syndrome, from 57 sites ac

264 GH-deficient prophet of pituitary-specific positive transcription factor 1 (Prop

265 POU1F1, a pituitary-specific POU-homeo domain transcription factor

266 oncogenic beta-catenin in Sox2+ young adult pituitary stem cells leads to formation of clusters of s

267 signs of hypophysitis, and four with normal pituitary structure and function.

268 nd regulated in other tissues, including the pituitary, suggesting that locally- and AT-produced adip

269 Patients cured by pituitary surgery alone had long-term survival similar t

270 Pituitary surgery alone is the preferred treatment to se

271 od is a pituitary adenoma, or treatment with pituitary surgery or radiotherapy.

272 Applied to tissue sections of rat pituitary, the platform demonstrated improved spatial re

273 In the pituitary, this innervation was observed close to follic

274 rmones (GPHs) are the main regulators of the pituitary-thyroid and pituitary-gonadal axes.

275 othalamic-pituitary-adrenal and hypothalamic-pituitary-thyroid axes, as well as a rise in systolic bl

276 inant-related activation of the hypothalamus-pituitary-thyroid axis in the kestrels increased elimina

277 he feedback loops involving the hypothalamus-pituitary-thyroid axis is disrupted by these stimulating

278 trophic feedback control of the hypothalamus-pituitary-thyroid axis to euthyroid hormone levels.

279 beta leads to disruption of the hypothalamic-pituitary-thyroid axis with resistance to TH, while muta

280 our findings do not indicate effects on the pituitary-thyroid axis.

281 9.0 days post coitum (dpc) and total loss of pituitary tissue by 12.5 dpc.

282 In adult pituitary tissue, direct cell contacts involving gap jun

283 expression to track transcription in living pituitary tissue.

284 umorigenesis but essential for Rb1-deficient pituitary tumorigenesis.

285 Rb1(+/-) mice incur "two-hit" pituitary tumorigenesis; Skp2(-/-);Rb1(+/-) mice do not.

286 liomas (n = 158), meningiomas (n = 111), and pituitary tumors (n = 154) from 58 patients.

287 Pituitary tumors are frequently associated with mutation

288 ter, white matter, gliomas, meningiomas, and pituitary tumors, allowing their ready discrimination by

289 itary adenomas (NFPAs) are the most frequent pituitary tumors.

290 lary craniopharyngioma (PCP), a benign human pituitary tumour harbouring BRAF p.V600E also contains S

291 In adult cohorts separately, rs1344110 in pituitary tumour-transforming 1 interacting protein (PTT

292 TTG1IP showed significant co-expression with pituitary tumour-transforming 1, the binding factor of P

293 the pituitary and form an interface with the pituitary vasculature, suggesting that preoptic AgRP2 ne

294 cular mechanisms that drive formation of the pituitary vasculature, which is necessary for regulated

295 ngth ELA enabled a distinctive regulation of pituitary vasopressin release.

296 MPs, either produced locally or reaching the pituitary via blood circulation.

297 T cell proliferation in the pituitary was confirmed in patients affected by autoimmu

298 ound that miR-7a2 is highly expressed in the pituitary, where it suppresses golgi glycoprotein 1 (GLG

299 r tumor types (i.e., glioma, meningioma, and pituitary), which were discriminated with an overall sen

300 In anterior pituitary, ZBTB20 is highly expressed by all the mature