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1 tructures or the inappropriate expression of pituitary hormones.
2 denomas that stained positively for anterior pituitary hormones.
3 tructures or the inappropriate expression of pituitary hormones.
4 mice, we assayed for changes in thyroid and pituitary hormones.
5 CART peptide on food intake and circulating pituitary hormones.
6 in binding sulfated glycoproteins including pituitary hormones.
7 R) binds to sulfated glycoproteins including pituitary hormones.
8 ) and one or more of the other five anterior pituitary hormones.
9 mpromised production of one or more anterior pituitary hormones.
10 nous ligands alters plasma levels of several pituitary hormones.
13 nt studies, MIF has been found to be a novel pituitary hormone and the first protein identified to be
14 The capacity of these proteins to activate pituitary hormone and transcription factor gene promoter
16 aracterized by the deficiency of one or more pituitary hormones and can present alone or in associati
17 t of ICV injection of CART peptide on plasma pituitary hormones and finally to examine the effect of
18 nduced changes in activity, vital signs, and pituitary hormones are modulated by the orexinergic syst
20 This process is tightly controlled by the pituitary hormone arginine vasopressin and defective tra
21 , interferon gamma, calcium, phosphorus, and pituitary hormones as well as the secosteroid hormone 1,
22 the cDNA coding for eel somatolactin (SL), a pituitary hormone belonging to the growth hormone (GH)/p
23 , suggesting that the cells not only control pituitary hormones but also may modulate nearby neurons.
24 GSU-Cre, loxP mice had normal levels of most pituitary hormones, but had markedly decreased expressio
25 ACAP) stimulates release of several anterior pituitary hormones by interacting with PACAP receptors o
26 Hypophysectomy and the attendent loss of pituitary hormones, by contrast, decreased MIF protein c
28 ave insufficient levels of multiple anterior pituitary hormones causing short stature, metabolic dise
32 ne deficiency, either alone or with multiple pituitary hormone deficiencies, as identified by clinica
35 2 regions have been associated with combined pituitary hormone deficiency (CPHD) diseases, suggesting
38 n patients with retarded growth and combined pituitary hormone deficiency and also abnormal neck and
40 complex diseases featuring combined anterior pituitary hormone deficiency and, in specific cases, los
41 x transcription factors LHX4 and PROP1 cause pituitary hormone deficiency in both humans and mice.
42 ons in the gene are associated with combined pituitary hormone deficiency in human patients and anima
43 e show here that this novel form of combined pituitary hormone deficiency is characterized by the per
45 isolated growth hormone deficiency, combined pituitary hormone deficiency, and syndromes such as sept
46 ation in human PIT1(R271W), causing combined pituitary hormone deficiency, results in loss of Pit1 as
47 th fronto-temporal lobe hypoplasia, multiple pituitary hormone deficiency, seizures, severe visual im
51 dwarf (dw/dw) mice are deficient in anterior pituitary hormones due to a mutation in the gene encodin
52 ntibody that targets the beta-subunit of the pituitary hormone follicle-stimulating hormone (Fsh) inc
53 statin on pituitary hormone secretion and on pituitary hormone (follicle-stimulating hormone and prol
54 chorionic gonadotropin (CG) and the anterior pituitary hormones follitropin, lutropin, and thyrotropi
59 adenosine controls the secretion of anterior pituitary hormones in vitro, adenosine was incubated wit
60 reover, the pituitary content of a subset of pituitary hormones, including growth hormone, prolactin
62 ed that incomplete glycosylation of anterior pituitary hormones leads to the creation of hormone anta
64 a1,4GlcNAcbeta1,2Manalpha are present on the pituitary hormones lutropin (LH), thyrotropin, and pro-o
65 the pituitary, with consequent reduction in pituitary hormone output and alterations in sexual and o
66 ediates signaling induced by the polypeptide pituitary hormone prolactin (PRL) has been shown to lead
70 issue in the lactotrophic axis, in which the pituitary hormone prolactin is tonically inhibited by tu
71 udy extended these findings by examining the pituitary hormone prolactin, and its response to stress,
73 ry glands, bone, brain, fat differentiation, pituitary hormone regulation, and metabolic effects in m
74 role of the gp130-related cytokines in human pituitary hormone regulation, we tested expression of gp
75 lement-derived cytokine, stimulates anterior pituitary hormone release and activates the hypothalamic
77 anges in hypothalamic regulation of anterior pituitary hormone release, including the decline in medi
80 bernation, and metabolism, are controlled by pituitary hormones released in response to annual enviro
81 median eminence (ME) to control long-lasting pituitary hormone rhythms essential for homeostasis.
82 We show that C3a receptors are expressed in pituitary hormone secreting and non-hormone secreting (f
88 projection to the median eminence to control pituitary hormone secretion possesses a spike initiation
99 nhibited the expression/secretion of several pituitary hormones (specially GH/PRL), which was accompa
101 n the mammalian ovary occur independently of pituitary hormone support, the factors controlling these
103 reased levels of luteinizing hormone (LH), a pituitary hormone that regulates sex steroid synthesis i
105 ction (including serum concentrations of the pituitary hormone thyrotropin and the thyroid hormones t
106 in adenohypophyseal cells and LIF regulates pituitary hormone transcription and cell replication in
108 ted (15-31%, P < 0.05), while other anterior pituitary hormones were not altered by these cytokines.
109 cterized by an increased release of anterior pituitary hormones, whereas altered target-organ sensiti
110 hesis is activated in steroidogenic cells by pituitary hormones, which concomitantly induce transcrip
111 In primates, placental lactogen (PL) is a pituitary hormone with fundamental roles during pregnanc
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