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1 contains glycyrrhizin (a potent inhibitor of placental 11beta-hydroxysteroid dehydrogenase type 2, th
3 hat maternal SERT function impacts offspring placental 5-HT levels, forebrain 5-HT levels, and neurod
4 reterm labor, anemia complicating pregnancy, placental abnormalities, infection during labor, materna
5 ), infection (aRR, 1.85; 95% CI, 1.43-2.29), placental abruption (aRR, 1.68; 95% CI, 1.18-2.38), indu
9 nant IL-10(-/-) mice with A-1254 resulted in placental activation of the Notch/Delta-like ligand (Dll
13 mice limited ZIKV vertical transmission and placental and fetal damage and overall improved placenta
14 function of CD8(+) dT that are permissive of placental and fetal development, and reversal of this dy
18 sitivity and accuracy of PAI for analysis of placental and fetal oxygen saturation (sO2) in mice.
19 In summary, PAI enables the detection of placental and fetal oxygenation during normal and pathol
22 proved for use in pregnant women, attenuated placental and fetal ZIKV infection and ameliorated adver
23 nt transport display convergent co-option by placental and mammary gland cell types to optimize offsp
27 diminished levels of viral RNA in maternal, placental, and fetal tissues, which resulted in protecti
29 uss the potential contributions of maternal, placental, and fetal viral infection to pregnancy outcom
33 ded evidence for pregnancy complications and placental anti-angiogenesis in response to Aroclor 1254
36 Nile virus (WNV), can efficiently infect key placental barrier cells that directly contact the fetal
38 ssociated with congenital disease breach the placental barrier to transit vertically during human pre
39 ZIKV causes microcephaly by crossing the placental barrier, however, the mechanism of trans-place
43 ent.The numerous associations of many of the placental biomarkers of vitamin D metabolism with circul
44 nancy loss, and increased IL-1beta levels in placental blood were associated with pregnancy loss and
45 gestational week 30-32, and delivery, and in placental blood, of 638 women during a longitudinal coho
46 may form a complex to functionally regulate placental cell function through modulation of target gen
49 asmodium falciparum-infected erythrocytes to placental chondroitin sulfate A (CSA) through the PfEMP1
51 Oxidative stress results in reduction of placental CSE activity, decreased hydrogen sulfide produ
54 s markedly enhanced angiogenic responses and placental development in DC expanded IL-10(-/-) dams.
56 egulation of a network of genes required for placental development, suggesting a central role for the
66 lation at E10 causes placental inflammation, placental dysfunction and reduces neonatal brain cortica
68 rameters have the potential to help identify placental dysfunction associated with FGR and may have c
70 hat maternal ageing is associated with utero-placental dysfunction, predisposing to adverse fetal out
73 ption is known to disrupt the ability of the placental enzyme 11beta-hydroxysteroid dehydrogenase typ
75 e loci of genome-wide significance, 835 were placental eSNPs (enrichment fold = 1.68, P = 7.41e-42).
80 ween rodent and human cardiac physiology and placental-fetal development indicate a need for models i
81 g a significant driver of clinical symptoms, placental Flt1 mRNA levels strongly correlate with mater
83 androgen excess in obese dams on metabolism, placental function and fetal growth, female C57Bl6J mice
85 peroxia to quantitatively assess mismatch in placental function in seven monozygotic twin pairs natur
86 We evaluated the effects of phthalates on placental function in vitro by measuring relevant candid
88 ify reduced placental efficiency and altered placental function with AMA in women, with evidence of p
89 olecular basis by which phthalates may alter placental function, and they provide a preliminary mecha
91 associations between maternal phthalates and placental gene expression were reproduced experimentally
92 ta are under tight genetic control, and that placental gene networks may influence postnatal risk of
93 py multiple sites in epigenetically inactive placental genes and in OCT4 Functional manipulation of G
98 etabolism of the mother, in relation to feto-placental glucose utilization and growth, are unknown.
99 f preneoplastic foci in the liver (increased placental glutathione S-transferase and cytokeratin 8-18
101 nsitivity and signalling in relation to feto-placental growth and glucose utilization are unknown.
102 ution of isoforms of VEGF and of the related placental growth factor (PlGF) in the body and resulting
104 r; platelet-derived growth factor AA and BB; placental growth factor; vascular endothelial growth fac
106 a cohort of 90 preterm infants with detailed placental histology and neonatal brain magnetic resonanc
109 y reduced the preterm birth rate and altered placental immune profile with decreased CD8(+) T-cell in
111 ling in WT pregnant mice enhanced ZIKV trans-placental infection although it did not result in fetal
112 ew, we first consider the pathophysiology of placental infection and transplacental transmission of t
113 ns against parasitological endpoints such as placental infection at delivery and health outcomes incl
114 ystals to rats during late gestation induced placental inflammation and was associated with fetal gro
118 er, these data demonstrate the novel role of placental InsRs in sex-specific neurodevelopment and rev
119 off the risk of prolonged fetal exposure to placental insufficiency against the risks of preterm del
120 nfection at embryonic day 6 (E6) resulted in placental insufficiency and fetal demise, infections at
122 l of these observations in sheep models with placental insufficiency are consistent with cases of hum
125 s to reduced oxygen and nutrient supply with placental insufficiency that develop to slow hindlimb gr
130 role of fetal hepcidin in the regulation of placental iron transfer still remains to be characterize
131 kinase 1, providing biological support, as a placental isoform of this protein (sFlt-1) is implicated
132 duced BCRP mRNA up to 10-fold in human model placental JEG3 and BeWo cells and in primary human villo
134 suggest that the proper organization of the placental labyrinth depends on coordinated inter-endothe
136 ular mechanism underlying development of the placental labyrinth, particularly in terms of its endoth
138 in the embryonic vasculature and heart, the placental labyrinths of these embryos exhibited aberrant
140 ogenic activity ex vivo, is expressed at the placental level as revealed by in situ hybridization and
141 This maternal intervention prevented excess placental lipid deposition and hypoxia (independent of s
144 gestational days 85 and 135, they underwent placental magnetic resonance imaging after intravenous g
149 observed that children born to mothers with placental malaria, but not those born to mothers with pe
151 in the risk of histopathologically detected placental malarial infection between the daily TMP-SMX p
152 mary outcome was detection of active or past placental malarial infection by histopathologic analysis
154 teleosts has evolved in a divergent manner: placental mammals have lost the monoaminergic CSF-c cell
157 w that syncytin capture is not restricted to placental mammals, but can also take place in the rare n
158 e C2H2-ZF domain in the common progenitor of placental mammals, but that extant C2H2-ZF domains typic
159 e transcriptomic study of the cervix of four placental mammals, mouse, guinea pig, rabbit and armadil
160 t, compared with the Leu(8)OXT found in most placental mammals, the Cebidae Pro(8)OXT and Saguinus Va
161 ly to have played a role in the emergence of placental mammals, with evidence for multiple, reiterate
167 he patterns of gene expression in eutherian (placental) mammals are consistent with the notion that a
168 igen was seen in the syncytiotrophoblast and placental maternal mononuclear cells by immunohistochemi
169 n (0, 300 or 1000 microg/day; GD 9 - 18) for placental measurements or perinatally (0, 100, 300 or 10
170 lacental and blood vitamin D metabolites and placental messenger RNA (mRNA) abundance of vitamin D me
172 as been presumed to be the primary driver of placental metabolism, and the underlying progenitor cyto
173 th 24,25-dihydroxyvitamin D3 Moreover, these placental metabolites were strongly correlated (r </= 0.
174 -specificity to the placental microbiota and placental microbiome studies should consider regional di
175 ggest that there is niche-specificity to the placental microbiota and placental microbiome studies sh
178 f the IGFs and their signalling machinery on placental morphogenesis, substrate transport and hormone
179 hypoxia (10% inspired O2) from D14 to D19 on placental morphology, transport capacity and fetal growt
180 ons (P </= 0.045) were also observed between placental mRNA abundance of vitamin D metabolic componen
183 ed maternal psychosocial stress with reduced placental mtDNAcn and add to literature documenting raci
184 raumatic-stress-disorder symptom scores with placental mtDNAcn in a racially/ethnically diverse sampl
185 o acid transport and mitochondrial function, placental mTOR folate sensing may constitute the mechani
186 and that maternal folate deficiency inhibits placental mTOR signaling and amino acid transporter acti
187 ty and fetal growth, involving regulation of placental mTOR signaling by folate, resulting in changes
189 nal serum folate is positively correlated to placental mTORC1 and mTORC2 signalling activity in human
192 folate sensing in trophoblast cells matches placental nutrient transport, and therefore fetal growth
194 s of delta(44/42)Ca record a transition from placental nutrition to an adult-like diet and that Ca is
195 to daily TMP-SMX did not reduce the risk of placental or maternal malaria or improve birth outcomes.
196 strong immunological stimulus from prolonged placental or transplacental ZIKV shedding and potential
202 insight into etiological factors underlying placental pathologies associated with intrauterine growt
205 tal outcomes, we explored the association of placental pathology with household air pollution in preg
214 ficantly decreased and regional variation of placental perfusion significantly increased with advanci
221 position versus supine, and in the posterior placental position versus anterior placental position.
226 role of IGFs during pregnancy in regulating placental resource allocation to fetal growth is importa
227 in-like growth factors (IGFs) in controlling placental resource allocation to fetal growth, particula
228 estimates), as did P falciparum detected in placental samples (OR 1.95 [1.48-2.57]; I(2)=33.6%; 31 e
230 ith peripheral infection without evidence of placental sequestration, had increased risk of malaria d
231 ring pregnancy has pointed to alterations in placental signaling, including changes in inflammatory,
234 us retroelements, as a direct repressor of a placental-specific Igf2 transcript (designated Igf2-P0)
236 y within the uterus, amniotic fluid, and the placental structure reveals that the developing fetus is
239 poietic cells, and in the case of pregnancy, placental syncytiotrophoblast cells) and several forms o
240 clampsia (PE), there is increased release of placental syncytiotrophoblast extracellular vesicles (ST
242 distributed lag models, both cord blood and placental telomere length were associated with average w
246 ipants with full data on both cord blood and placental telomere lengths were included, resulting in a
247 ure in vimentin knockout (VimKO) embryos and placental tissue is underdeveloped with reduced branchin
250 along with the regulation of target genes.In placental tissue, 25-hydroxyvitamin D3 [25(OH)D3] was st
255 ncy can compromise the iron availability for placental transfer and impair the efficacy of iron suppl
258 etween prenatal manganese concentrations and placental transfer of manganese with neurodevelopment in
261 itional mother-specific factors, such as the placental transmission of antibodies, cannot be fully ru
265 e studied the cell-cell interactome of fetal placental trophoblast cells and maternal endometrial str
268 two models to study susceptibility of human placental trophoblast to ZIKV: cytotrophoblast and syncy
269 dult offspring were evaluated for effects of placental trophoblast-specific InsR deficiency on stress
273 In this study, we show that primary human placental trophoblasts from non-exposed donors (n = 20)
274 that ZIKV-FLR strain can replicate in human placental trophoblasts without host cell destruction, th
280 these data reveal that Egfl7 is crucial for placental vascularization and embryonic growth, and may
282 tility of the technique in the comparison of placental vessel networks in normal and fetal growth res
283 ique for three-dimensional analyses of human placental vessels; (ii) demonstrate the utility of the t
284 hoblast and syncytiotrophoblast derived from placental villi at term and colonies of trophoblast diff
285 ver established physically in the human, the placental villi, the exocoelomic cavity, and the seconda
286 rnal uterine, endothelial, and immune cells; placental villi, which are bathed in maternal blood, and
287 ng pathologic entities representing abnormal placental villous tissue with unique genetic profiles an
288 hasizes the immunological challenge to clear placental viral infections within the immune-privileged
290 ught to advance the current understanding of placental vitamin D metabolism and its role in modulatin
292 ns, 221 g [95% CI, 6-436]; P = .044) and the placental weight (difference between means, 84 g [95% CI
294 etal deaths, reduced fetal weight, increased placental weight and reduced fetal:placental weight rati
296 increased placental weight and reduced fetal:placental weight ratio compared to 8-12 week controls.
297 t by approximately 10% compared with FA, and placental weight was reduced ( approximately 8%) on GD17
298 roup was decreased (-17%, p < 0.05), whereas placental weight, litter size and crown rump length were
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