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1 station and low birthweight, an indicator of placental insufficiency.
2 d and neonatal anthropomorphic indicators of placental insufficiency.
3 e in litter size was observed, supportive of placental insufficiency.
4  the fetal growth restriction and attenuated placental insufficiency.
5 cts, some of which are indirectly related to placental insufficiency.
6 is hypothesis using a model system mimicking placental insufficiency.
7 may represent an early biomarker of critical placental insufficiency.
8 veral Nsdhl alleles die in midgestation with placental insufficiency.
9 Blimp1 mutant embryos arrest at E10.5 due to placental insufficiency.
10 at at least some of these defects are due to placental insufficiency.
11 cently, Pkd1-/- lethality has been linked to placental insufficiency.
12 on independent of fetal genotype, indicating placental insufficiency.
13 triction, and preterm birth, which stem from placental insufficiency.
14 ortant for correct differential diagnosis of placental insufficiency.
15 n availability (p < 0.05), all indicators of placental insufficiency.
16 f key myocardial enzymes under conditions of placental insufficiency.
17 striction (IUGR), as well as the severity of placental insufficiency.
18 K4 kinase activity causes FGR due in part to placental insufficiency.
19  cases of intrauterine growth restriction or placental insufficiency (5 women had used LMWH).
20  off the risk of prolonged fetal exposure to placental insufficiency against the risks of preterm del
21 t activation in vivo in utero and predicting placental insufficiency and abnormal foetal neurodevelop
22 ay also be important for the pathogenesis of placental insufficiency and cardiac malformations.
23 mall for GA at birth and more likely to have placental insufficiency and exposure to maternal preecla
24 nfection at embryonic day 6 (E6) resulted in placental insufficiency and fetal demise, infections at
25 with MAP3K4 kinase inactivation resulting in placental insufficiency and fetal growth restriction.
26 oncentrations are elevated in the setting of placental insufficiency and fetal stress.
27 une system activation in the pathogenesis of placental insufficiency and identify novel methods for t
28 ion have altered phenotypes, possibly due to placental insufficiency and impaired fetal growth.
29 s underlying fetal growth restriction due to placental insufficiency and in utero hypoxia are not wel
30                                              Placental insufficiency and intrauterine growth restrict
31 r mechanisms involved in the pathogenesis of placental insufficiency and IUGR are largely unknown.
32                       Using a sheep model of placental insufficiency and IUGR, we have previously dem
33 ulin clamp periods in a fetal sheep model of placental insufficiency and IUGR.
34        Low circulating SPINT1 is a marker of placental insufficiency and may identify pregnancies wit
35                                              Placental insufficiency and other conditions affecting f
36 sed intrauterine environment, as in cases of placental insufficiency and perinatal acidaemia.
37 rkers of vascular endothelial activation and placental insufficiency and the occurrence of pre-eclamp
38 totoxicity to neural progenitor cells (NPC), placental insufficiency, and immune responses, remain in
39         Hypoxia resulted in asymmetric IUGR, placental insufficiency, and reduced placental PPAR-gamm
40                                              Placental insufficiency appeared responsible for the inc
41 nic maternal gestational hypoxia, as well as placental insufficiency are associated with increased FG
42 l of these observations in sheep models with placental insufficiency are consistent with cases of hum
43                                              Placental insufficiency can cause fetal growth restricti
44                                   In humans, placental insufficiency can result in intrauterine growt
45                                              Placental insufficiency causes intrauterine growth restr
46 lacenta in the APS model was associated with placental insufficiency characterised by increased oxida
47                                              Placental insufficiency during early-mid gestation may h
48 editing, LNPs have not been investigated for placental insufficiency during pregnancy.
49    There are no reliable screening tests for placental insufficiency, especially near-term gestation
50 is growth factor family in an ovine model of placental insufficiency-FGR, in relationship to uteropla
51 NDINGS: We recruited three women with severe placental insufficiency/FGR and three matched controls.
52 oof-of-concept study suggests that in severe placental insufficiency/FGR, the observed 60-fold reduct
53 neonatal death; birth before 36 weeks due to placental insufficiency, hypertension, or preeclampsia;
54 oncentrations are elevated in the setting of placental insufficiency, hypoxia and elevated stress hor
55 , and more precisely, assess the severity of placental insufficiency in IUGR foetuses.
56  mitochondrial respiration and contribute to placental insufficiency in IUGR pregnancies.
57 sociation between low circulating SPINT1 and placental insufficiency in two other cohorts.
58         We report a rabbit model of in utero placental insufficiency, in which hypertonia is accompan
59 f this study was to determine the effects of placental insufficiency, induced by UPE, on cardiomyocyt
60                                              Placental insufficiency-induced intrauterine growth rest
61                                              Placental insufficiency is a major cause of antepartum s
62 gs also provide a solid basis to explain why placental insufficiency is associated with disorders of
63 ling of the fetal circulation resulting from placental insufficiency is associated with more favourab
64  the absence of fetal arterial hypertension, placental insufficiency is associated with substantially
65                                              Placental insufficiency is caused by inadequate blood fl
66                                              Placental insufficiency is often associated with IUGR; h
67                                           If placental insufficiency is present, the physician must t
68        Defective placentation and subsequent placental insufficiency lead to maternal and fetal adver
69 trauterine growth restriction, brought on by placental insufficiency, likely due to abnormal developm
70 o pregnancy or extra monitoring for possible placental insufficiency may be advisable.
71 etal death in the absence of preeclampsia or placental insufficiency may not meet current classificat
72            When fetal growth is compromised, placental insufficiency must be distinguished from modes
73  has been used to investigate the effects of placental insufficiency on fetal development.
74 ing pregnancy complications characterized by placental insufficiency or fetal hypoxia.
75 ntal (seven patients) abnormalities (but not placental insufficiency or intrauterine growth retardati
76 ular disorders of pregnancy characterized by placental insufficiency, restricted fetal growth and pre
77 fathers trigger an elevated risk of in utero placental insufficiency, revealing a placental origin of
78                                              Placental insufficiency, such as occurs in the complete
79  growth, and may aid our understanding human placental insufficiency syndromes.
80 s to reduced oxygen and nutrient supply with placental insufficiency that develop to slow hindlimb gr
81  mouse model of fetal-growth restriction and placental insufficiency that is induced by a midgestatio
82 on in placentae during pregnancy to identify placental insufficiency that may be indicative of local
83  Fetal growth restriction (FGR) results from placental insufficiency to adequately supply the fetus.
84                    In the univariable model, placental insufficiency was associated with VCDR (B = 0.
85                             Preeclampsia and placental insufficiency were self-reported and abstracte
86 deficit in humans, and is commonly caused by placental insufficiency, which results in fetal hypoxia.
87 EC)-specific deletion of ILK in mice confers placental insufficiency with decreased labyrinthine vasc
88                             To determine how placental insufficiency with reduced oxygen and nutrient