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1 is hypothesis using a model system mimicking placental insufficiency.
2 may represent an early biomarker of critical placental insufficiency.
3 veral Nsdhl alleles die in midgestation with placental insufficiency.
4 Blimp1 mutant embryos arrest at E10.5 due to placental insufficiency.
5 at at least some of these defects are due to placental insufficiency.
6 cently, Pkd1-/- lethality has been linked to placental insufficiency.
7 on independent of fetal genotype, indicating placental insufficiency.
8 cts, some of which are indirectly related to placental insufficiency.
10 off the risk of prolonged fetal exposure to placental insufficiency against the risks of preterm del
11 t activation in vivo in utero and predicting placental insufficiency and abnormal foetal neurodevelop
13 nfection at embryonic day 6 (E6) resulted in placental insufficiency and fetal demise, infections at
14 une system activation in the pathogenesis of placental insufficiency and identify novel methods for t
15 s underlying fetal growth restriction due to placental insufficiency and in utero hypoxia are not wel
17 r mechanisms involved in the pathogenesis of placental insufficiency and IUGR are largely unknown.
20 rkers of vascular endothelial activation and placental insufficiency and the occurrence of pre-eclamp
22 l of these observations in sheep models with placental insufficiency are consistent with cases of hum
24 lacenta in the APS model was associated with placental insufficiency characterised by increased oxida
25 is growth factor family in an ovine model of placental insufficiency-FGR, in relationship to uteropla
26 NDINGS: We recruited three women with severe placental insufficiency/FGR and three matched controls.
27 oof-of-concept study suggests that in severe placental insufficiency/FGR, the observed 60-fold reduct
28 neonatal death; birth before 36 weeks due to placental insufficiency, hypertension, or preeclampsia;
30 f this study was to determine the effects of placental insufficiency, induced by UPE, on cardiomyocyt
33 the absence of fetal arterial hypertension, placental insufficiency is associated with substantially
40 ntal (seven patients) abnormalities (but not placental insufficiency or intrauterine growth retardati
43 s to reduced oxygen and nutrient supply with placental insufficiency that develop to slow hindlimb gr
44 mouse model of fetal-growth restriction and placental insufficiency that is induced by a midgestatio
45 Fetal growth restriction (FGR) results from placental insufficiency to adequately supply the fetus.
47 EC)-specific deletion of ILK in mice confers placental insufficiency with decreased labyrinthine vasc
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