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1 ack caveolae, have increased permeability to plasma albumin.
2 ng to HUVECs 100% and 50%, respectively, and plasma albumin and other proteins prevent a sufficient l
3 ased plasma albumin and the relation between plasma albumin and the edema of protein-energy malnutrit
4 he kinetic changes responsible for decreased plasma albumin and the relation between plasma albumin a
5  that at physiological NO(.) concentrations, plasma albumin becomes saturated with NO(.) and accelera
6 enhances the endocytosis and transcytosis of plasma albumin by podocytes, which may eventually impair
7                                    In HUVEC, plasma albumin caused a sustained decrease in [Ca2+]i fr
8 ood-to-tissue albumin transport, measured as plasma albumin clearance corrected for intravascular vol
9 ally thought to stem from the prevailing low plasma albumin concentration and the decreased transcapi
10 oncentration, which represents the fact that plasma albumin concentration does not reflect its functi
11 ortened in FcRn-deficient mice, and that the plasma albumin concentration of FcRn-deficient mice is l
12 etabolite were associated with drug dose and plasma albumin concentration, and were lower among men a
13             Albumin administration increased plasma albumin concentrations (p <.05 compared with plac
14 y 1 there were no significant differences in plasma albumin concentrations in nonedematous and edemat
15 PN, for which Hpn knockout mice manifest low plasma albumin concentrations.
16 e septic rats and three controls after their plasma albumin had been labeled with Evans blue dye.
17            The decrease in [Ca2+]i caused by plasma albumin is due to an uptake into intracellular st
18 lucocorticoid doses, and in those with lower plasma albumin level.
19                     The effects of serum and plasma albumin on [Ca2+]i in human endothelial cells wer
20                      Because modification of plasma albumin on tyrosine residues generates nitrated a
21                                              Plasma albumin permeation into the joint cavity was also
22 004), protein (r = 0.59; P < .0001), and CSF/plasma albumin ratio (r = 0.60; P < .0001).
23 t is mediated by covalent binding of iRGD to plasma albumin through a disulfide bond.
24                                 No effect of plasma albumin was observed in ECV304 cells.
25                 The results show that normal plasma albumin, which carries few lipids, lowers [Ca2+]i

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