ライフサイエンスコーパス: plasma catecholamine

1 y Score, prehospital Glasgow Coma Scale, and plasma catecholamines.

2 potension was associated with an increase in plasma catecholamines.

3 Elevated levels of plasma catecholamines accompany ischemic AKI, possibly c

4 in the chromaffin granule; and (g) increased plasma catecholamine and Npy levels.

5 first 2 hrs after traumatic brain injury and plasma catecholamine and serum fentanyl concentrations m

6 bradycardic response was prolonged, and the plasma catecholamine and vasopressin responses were dimi

7 sone treatment, femoral vasoconstriction and plasma catecholamine and vasopressin responses were enha

8 lar resistances, ECG, serum cardiac enzymes, plasma catecholamines and atrial natriuretic factor, and

9 ng traumatic brain injury, and elevations in plasma catecholamines and cortisol portend a poor outcom

10 flex impairment, and sympathetic activation (plasma catecholamines and heart rate variability indexes

11 acute hypoxaemia-induced elevations in fetal plasma catecholamines and suppression of glucose-stimula

12 evaluated the acute drug-induced changes in plasma catecholamines and used these results as a basis

13 KO mice displayed hypertension, higher plasma catecholamine, and adipokine levels and lower IL-

14 easured postural blood pressure, heart rate, plasma catecholamines, and systemic norepinephrine spill

15 Indirect indices such as plasma catecholamines are transiently elevated after unc

16 rences in baseline hemodynamic parameters or plasma catecholamines between the 2 groups.

17 eart rate, systemic arterial blood pressure, plasma catecholamine, clonidine, interleukin-6 concentra

18 We manipulated plasma catecholamines (combined adrenaline and noradrena

19 ons in BP during GB were related to baseline plasma catecholamine concentrations (r=-0.31 to -0.41, P

20 of this study is that increases in MSNA and plasma catecholamine concentrations did not differ betwe

21 muscle sympathetic nerve activity (MSNA) and plasma catecholamine concentrations in healthy young and

22 Plasma catecholamine concentrations increased 3-fold dur

23 Plasma catecholamine concentrations increased in both gr

24 Manipulations of plasma catecholamine concentrations influence outcome fr

25 Thus, heart rate power spectral analysis and plasma catecholamine concentrations may prove to be usef

26 Women had lower baseline SBP and plasma catecholamine concentrations than men (P<0.05).

27 Plasma catecholamine concentrations were determined by h

28 w blood oxygen concentrations increase fetal plasma catecholamine concentrations, which lower fetal i

29 ry bypass (CPB) is associated with increased plasma catecholamine concentrations, which might worsen

30 fetuses have chronic hypoxaemia and elevated plasma catecholamine concentrations.

31 ensitization despite comparable elevation of plasma catecholamines during the development of heart fa

32 uscle sympathetic nerve activity (MSNA), and plasma catecholamines evoked by upright tilt in recurren

33 Plasma catecholamine, glucose, and insulin levels were m

34 onary thrombosis suggests that elevations of plasma catecholamines, high shear forces acting on the p

35 ed with the postural heart rate and abnormal plasma catecholamine homeostasis.

36 Plasma catecholamines in NET(-/-) and NET(+/+) mice were

37 Plasma catecholamines in newborn rats (0-2 hr old) were

38 All plasma catecholamines increased after ephedrine administ

39 Plasma catecholamines increased fourfold in the CHF grou

40 ration of recombinant human renalase reduced plasma catecholamine levels and ameliorated ischemic AKI

41 diac hypertrophy is associated with elevated plasma catecholamine levels and an increase in cardiac m

42 Gallein also reduced circulating plasma catecholamine levels and catecholamine production

43 ocorticoid-induced fetal hypertension, fetal plasma catecholamine levels and changes in fetal femoral

44 Plasma catecholamine levels at presentation were markedl

45 In conclusion, large increases in plasma catecholamine levels cause hyperthermia during ex

46 Plasma catecholamine levels in 13 patients with stress-r

47 During SI, plasma catecholamine levels increased progressively and

48 key contributors to the chronically elevated plasma catecholamine levels observed in HF, where adrena

49 In +/+ but not in fa/fa rats, plasma catecholamine levels rose, and both P-STAT3 and P

50 tained plasma glucose levels above 11 mM and plasma catecholamine levels were 5.0-5.5 pmol ml-1 lower

51 infused (AI) at 0.1 microgram kg-1 min-1 and plasma catecholamine levels were elevated 6 pmol ml-1 ab

52 necrosis, and apoptosis were more severe and plasma catecholamine levels were higher in renalase-defi

53 Plasma catecholamine levels were measured at baseline an

54 Plasma catecholamine levels within and between groups di

55 indicated by elevated CB neural activity and plasma catecholamine levels, and elevated reactive oxyge

56 e, respiratory rate, mean arterial pressure, plasma catecholamine levels, and heart rate power spectr

57 ockade (with trimethaphan) on supine SBP and plasma catecholamine levels, and the effect of alpha(1)-

58 ation in heart failure, resulting in lowered plasma catecholamine levels, improved cardiac betaAR sig

59 Based on MDMA-induced changes in plasma catecholamine levels, rats were subjected to the

60 ot accompanied by corresponding increases in plasma catecholamine levels.

61 ardiomyopathy," is associated with increased plasma catecholamine levels.

62 Plasma catecholamines levels 30 mins after MDMA (40 mg/k

63 diphenhydramine on depression, anxiety, and plasma catecholamine metabolites were assessed.

64 of depression, plasma tryptophan levels, and plasma catecholamine metabolites.

65 iovascular disease associated with increased plasma catecholamines, overactivation of the sympathetic

66 late device specifically designed to extract plasma catecholamines prior to their quantification by a

67 Plasma catecholamines provide a reliable biomarker of sy

68 iated with a more than four-fold increase in plasma catecholamines, renin activity, and endothelin co

69 to others factors besides attenuation of the plasma catecholamine response to exercise.

70 , reduced baroreflex sensitivity, diminished plasma catecholamine responses to acute stress, and incr

71 nhanced baroreflex sensitivity and augmented plasma catecholamine responses to acute stress.

72 nts the effect of hypoglycemia to reduce the plasma catecholamine responses to subsequent hypoglycemi

73 e hypoxia (both P < 0.05), without affecting plasma catecholamine responses.

74 occur in the context of increased levels of plasma catecholamines, some critical mechanisms that gov

75 A plasma catecholamine surge was observed in every animal

76 resting and mental stress-induced levels of plasma catecholamines, tissue plasminogen activator anti

77 Plasma catecholamines (unchanged during euglycemia) rose

78 lasma cortisol levels exceeded 17.5 ng ml-1, plasma catecholamines were a major influence on fetal gl

79 Arterial plasma catecholamines were assessed at basal (-30 and 0

80 Arterial plasma catecholamines were assessed at basal (-30 and 0

81 ance, muscle sympathetic nerve activity, and plasma catecholamines were measured at baseline and at 3

82 Changes in plasma catecholamines were not different between the CRT

83 al hormonal investigations include urine and plasma catecholamines with their metabolites, plasma cor