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1 ypoglycemia produced a threefold increase in plasma glucagon.
2 ively, P < 0.05 for both) and no increase in plasma glucagon.
3 increased ( approximately 10-fold) levels of plasma glucagon.
4 ever, produced a more pronounced blunting of plasma glucagon, ACTH, and hepatic glucose production co
6 ng antibody to healthy individuals increased plasma glucagon and amino acid levels, but did not chang
7 sociated with decreased food intake, reduced plasma glucagon and corticosterone concentrations, and d
8 only slightly, it fully normalized elevated plasma glucagon and corticosterone levels and reversed t
9 ose, plasma lipids, liver triglycerides, and plasma glucagon and enhanced pancreatic insulin content,
12 -/-) mice have elevated ( approximately 60%) plasma glucagon and reduced ( approximately 20%) plasma
13 lucose and was accompanied by lowered HbA1c, plasma glucagon, and triglyceride concentrations and exp
14 fatty acid was accompanied by a fall in the plasma glucagon concentration from 122 to 85 pmol/L (P =
16 results in higher plasma glucose and higher plasma glucagon concentrations after a mixed meal and af
17 sfunction, characterized by elevated fasting plasma glucagon concentrations and inadequate postprandi
18 In humans, postprandial lipemia increased plasma glucagon concentrations and led to an inadequate
22 hours and were independent of any changes in plasma glucagon concentrations; these effects were abrog
24 nd the hepatic portal-arterial difference in plasma glucagon decreased slightly from 41 +/- 7 and 4 +
25 whether macronutrient infusion can suppress plasma glucagon during critical illness and study the ro
27 In patients with diabetes, elevations in plasma glucagon, epinephrine, and norepinephrine were bl
28 /- 6 pM), suppression of glucagon secretion (plasma glucagon, I:31 +/- 4, II: 63 +/- 8 pg/ml) doubled
29 nd the hepatic portal-arterial difference in plasma glucagon increased from 43 +/- 5 and 4 +/- 2 to 5
32 tor monoclonal antibody undergo elevation of plasma glucagon levels and alpha-cell expansion similar
33 effect associated with normalization of both plasma glucagon levels and hepatic expression of glucone
35 plus xenin-25 transiently increased ISR and plasma glucagon levels in subjects with NGT and IGT but
38 were associated with protein breakdown, and plasma glucagon levels were inversely correlated with pr
45 sulin secretion (P < .01), and 3-fold larger plasma glucagon-like peptide-1(7-36) (GLP-1(7-36)) excur
46 nd the hepatic portal-arterial difference in plasma glucagon (ng/l) did not change significantly (43
47 nd the hepatic portal-arterial difference in plasma glucagon (ng/l) did not rise significantly (40 +/
48 nd the hepatic portal-arterial difference in plasma glucagon (ng/l) rose from 43 +/- 5 and 5 +/- 2 to
50 tradiol also attenuated hypoglycemia-induced plasma glucagon, pituitary proopiomelanocortin (POMC), a
51 he great potential that normalization of the plasma glucagon profile may have for the treatment of T2
56 ticosteroid levels were altered as expected, plasma glucagon was reduced markedly in the mutant anima
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