ライフサイエンスコーパス: plasma leakage

1 and tight and adherens junction markers with plasma leakage.

2 074) had an adjusted association with severe plasma leakage.

3 laudin-5 are strongly associated with severe plasma leakage.

4 for exacerbated dengue pathogenesis, notably plasma leakage.

5 yte-associated FITC-gelatin fluorescence and plasma leakage.

6 re significantly associated with significant plasma leakage.

7 openings in the endothelium without causing plasma leakage.

8 rrhagic manifestations, and 23% had signs of plasma leakage.

9 icoagulant suppression, vascular injury, and plasma leakage.

10 on status, and disease severity, measured by plasma leakage.

11 es, specifically mast cell degranulation and plasma leakage.

12 f the VE biomarkers with the severity of the plasma leakage.

13 vessels and protects them from VEGF-induced plasma leakage.

14 total VEGF-A in DHF patients at the time of plasma leakage.

15 ilder form of dengue virus infection without plasma leakage.

16 acterized by a transient period of localized plasma leakage.

17 ere correlated with the degree of subsequent plasma leakage.

18 u and Monastral blue as a tracer to quantify plasma leakage.

19 engue hemorrhagic fever, is characterized by plasma leakage and derangements in hemostasis.

20 Plasma leakage and keratinocyte chemoattractant producti

21 that salmeterol pretreatment can reduce the plasma leakage and leukocyte adhesion in early- and late

22 ergic receptor agonist, on ovalbumin-induced plasma leakage and leukocyte adhesion in tracheal blood

23 s vascular remodeling in inflammation, where plasma leakage and leukocyte influx are prominent featur

24 ire the phenotype of venules specialized for plasma leakage and leukocyte recruitment.

25 uctural protein 1 (NS1; anti-NS1 Igs) induce plasma leakage and mortality in mice with warfarinized a

26 viremia levels correlated with the degree of plasma leakage and thrombocytopenia.

27 rations occur in dengue, are associated with plasma leakage, and are correlate with molecules of endo

28 droitin sulfate are strongly associated with plasma leakage, and elevated levels of syndecan-1 and cl

29 ts resulted in both early and late phases of plasma leakage as measured with Evans blue.

30 dly reduced neutrophil (PMN) recruitment and plasma leakage at dermal sites with LTB(4).

31 and a potent inducer of inflammation causing plasma leakage at the site of infection.

32 duced airway hyperresponsiveness and mucosal plasma leakage but had no effect on inflammatory cells o

33 as also able to inhibit neutrophil-dependent plasma leakage, but had no effect on the response induce

34 eans, binds to endothelial cells at sites of plasma leakage, but little is known about the amount and

35 el structure and blood perfusion and blocked plasma leakage by enhanced endothelial barrier function

36 Treatments with the anti-NS1-DR4 Ig led to plasma leakage, coagulopathy, and morality in mice with

37 significantly lower for patients with severe plasma leakage compared to those with no leakage (1.46 v

38 sistance to injury, characterized by reduced plasma leakage, decreased leucocyte adhesion and amelior

39 the relationship between pericytes and blood plasma leakage during photothrombotic occlusion of corti

40 eased vascular permeability and pathological plasma leakage during viral hemorrhagic fevers are large

41 other inflammatory diseases when the rate of plasma leakage from blood vessels exceeds the drainage t

42 y; this has been proposed as a mechanism for plasma leakage in dengue hemorrhagic fever.

43 expression may be an important mechanism of plasma leakage in DHF.

44 and has been hypothesized to play a role in plasma leakage in diabetic retinopathy.

45 r the bradykinin B2 receptor (B2R) inhibited plasma leakage in hamster cheek pouch topically exposed

46 The severity of plasma leakage in patients inversely correlated with pla

47 ough the infection had no effect on baseline plasma leakage, in both strains it potentiated the leaka

48 contribute to both early and late phases of plasma leakage induced by antigen, but most leakage occu

49 ful vascular protective effects: suppressing plasma leakage, inhibiting vascular inflammation, and pr

50 ial (VE) components in dengue infection with plasma leakage is unknown.

51 dengue virus (DV) infection characterized by plasma leakage, is more common in secondary DV infection

52 by early and late phases of vasodilatation, plasma leakage, leukocyte influx, and bronchoconstrictio

53 x events such as bronchoconstriction, airway plasma leakage, mucus secretion and cough.

54 essation of capillary flow, we observed that plasma leakage occurred with three times greater frequen

55 of several viral hemorrhagic fevers, severe plasma leakage occurs at the time of viral clearance and

56 /NO bioavailability is associated with worse plasma leakage, occurs early in dengue illness and corre

57 ed in arterioles also occurs in venules, (2) plasma leakage persists well beyond red cell escape and

58 Plasma leakage results from gaps between endothelial cel

59 y may play a role in the pathogenesis of the plasma leakage seen in dengue hemorrhagic fever and deng

60 The main outcome of interest was plasma leakage severity.

61 se its presentation can resemble more common plasma leakage syndromes, including angioedema or system

62 athophysiological hallmark of DHF and DSS is plasma leakage that leads to enhanced vascular permeabil

63 , particularly the mechanisms underlying the plasma leakage that results in hypovolaemic shock in a s

64 ion with a dengue virus, is characterized by plasma leakage, thrombocytopenia, and hepatic inflammati

65 neutrophil-induced endothelial pores prevent plasma leakage through actomyosin-based pore confinement

66 Plasma leakage was preceded by rapid activation of matri

67 .025-1.307) had an adjusted association with plasma leakage, whereas levels of syndecan-1 (OR = 1.004

68 The ability of Ang1 to block plasma leakage without producing angiogenesis may be the