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1 d-type (WT) mice while maintaining a similar plasma osmolality.
2 ta mRNA correlated inversely with changes in plasma osmolality.
3 onse to angiotensin II; and (c) reduction in plasma osmolality.
4 ipheral circulation in response to increased plasma osmolality.
5 ality failed to decrease at the same rate as plasma osmolality.
6 ncomitant with increases in fluid intake and plasma osmolality.
7 travenous infusion of HYPER saline increased plasma osmolality (294 +/- 3 to 316 +/- 5 mOsm kg(-1) H2
8 induced drinking, vasopressin (AVP) release, plasma osmolality and c-fos expression in the brain of c
9 Loads of 900 and 1200 mOsm/kg both increased plasma osmolality and sodium concentration by 15 min aft
10 smotic stimulus, determined by assessment of plasma osmolality and sodium content, SON neurons exhibi
11       Except for the expected differences in plasma osmolality and sodium, basal measures were simila
12                            Hydration status (plasma osmolality and urine specific gravity) and body c
13 ration, which in turn influence the osmotic (plasma osmolality) and blood volume-dependent compensato
14 atocrit, plasma protein concentration, total plasma osmolality, and plasma COP.
15 ydration status, including hematocrit, total plasma osmolality, and plasma protein concentration, fai
16 s generally accepted that dialysis may lower plasma osmolality at a faster rate than changes in ocula
17 rea bath decreased plasma urea by 21 mM, and plasma osmolality by 22 mosmol/kg H2O, and increased bra
18                              An elevation in plasma osmolality elicits a complex neurohumoral respons
19  decrease in urinary osmolality, and rise in plasma osmolality in the OPC 31260-treated CHF rats as c
20                Plasma volume is expanded and plasma osmolality is decreased, yet vasopressin secretio
21 dration-induced anorexia in which increasing plasma osmolality leads to a centrally generated reducti
22                                The decreased plasma osmolality of P14 rats was not modified by OPC-31
23  (P < .02), despite their consistently lower plasma osmolality (P < .007).
24                                However, only plasma osmolality (P(osm)) showed statistical promise fo
25 ting these results with ongoing behavior and plasma osmolality points to the existence of brain netwo
26 l plasma sodium (WD(5)), the substitution of plasma osmolality (Posm) for sodium (WD(6)), and actual
27 hat individuals classified as DE have higher plasma osmolality (Posm), indicating suboptimal hydratio
28 w; however, plasma sodium concentrations and plasma osmolality remained low.
29                                    Increased plasma osmolality results in increased central as well a
30 , which produced a physiological increase in plasma osmolality to 299 +/- 1 mosmol (kg water)(-1), el
31 g neurones are activated during increases in plasma osmolality to elicit sympathoexcitation.
32                                              Plasma osmolality was consistently reduced by the chroni
33                                              Plasma osmolalities were significantly lower in pregnant
34                      Cardiac output (CO) and plasma osmolality were significantly decreased and plasm
35 ared different equations used for predicting plasma osmolality when its direct measurement was not pr
36  to the LHA responded to a rapid increase in plasma osmolality with increased c-fos mRNA levels.

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