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1 ncreased plasma corticosterone and decreased plasma prolactin.
2 m of effects that includes: (1) elevation of plasma prolactin and corticosterone, (2) short- and long
3                                          The plasma prolactin concentration was higher in the diet an
4 tput; maternal weight, body composition, and plasma prolactin concentration; and infant weight were m
5 ol study to examine the relationship between plasma prolactin concentrations and breast cancer risk i
6  examine, in detail, the association between plasma prolactin concentrations and postmenopausal breas
7 locked haloperidol-induced increases in both plasma prolactin concentrations and the percentage of TH
8            Our prospective data suggest that plasma prolactin concentrations are associated with an i
9 nistration of 200 microl of PRL-AB decreased plasma prolactin concentrations below detectable levels
10 en the Tg mice were fed chow containing IC3, plasma prolactin concentrations increased threefold, BP
11  caused a sustained (up to 12 h) increase in plasma prolactin concentrations that was accompanied by
12  When the Tg mice were fed normal chow (NC), plasma prolactin concentrations were comparable to those
13  s.c.) caused an prompt (by 1 h) increase in plasma prolactin concentrations which was maintained for
14                Thus, a threefold increase in plasma prolactin is sufficient to increase BP significan
15 of an atypical APD, the lack of elevation in plasma prolactin levels (with risperidone and 9-hydroxyr
16 cetic acid (5-HIAA) and fenfluramine-induced plasma prolactin levels are markedly diminished in the T
17 tion, and induces molting, with no change in plasma prolactin levels.
18 ose which significantly reduces food intake, plasma prolactin (PRL), growth hormone (GH) and adrenoco
19 that low CSF 5-HIAA and fenfluramine-induced plasma prolactin reflects chronic, endogenous central ne
20 ddition, clozapine produced fewer effects on plasma prolactin than risperidone or fluphenazine.
21            IBO produced a rapid elevation in plasma prolactin that declined to control levels by 60 m
22                     The effects of increased plasma prolactin were abolished by a genetic absence of

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