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1 sulted in expression of a hybrid CFHR2-CFHR5 plasma protein.
2 d-glycoprotein (AGP) as a chiral acute phase plasma protein.
3 y hemopexin (Hx), an endogenous heme-binding plasma protein.
4 on of selenium-sulfur bond between p-XSC and plasma protein.
5 to identification of a large number of novel plasma proteins.
6 active fragment production in the absence of plasma proteins.
7 d this might be involved in the retention of plasma proteins.
8 n and degradation along with accumulation of plasma proteins.
9 etition of bile acids with FL for binding to plasma proteins.
10 ng the abundance of 44 out of 45 major human plasma proteins.
11 n blood and are largely free from binding to plasma proteins.
12 strated capillaries, causing a major leak of plasma proteins.
13  premature aquation and binding to essential plasma proteins.
14 mplex mixture obtained by digestion of human plasma proteins.
15 cts with a select group of drusen-associated plasma proteins.
16 ssociated functionally with glycosylation of plasma proteins.
17 d thrombosis through its binding of adhesive plasma proteins.
18  acid-lysine-alanine, which binds to clotted plasma proteins.
19 condition of changing radioligand binding to plasma proteins.
20 ts identified a large number of differential plasma proteins.
21  become sensitive to neutralization by mouse plasma proteins.
22 s, however, increased expression of genes of plasma proteins.
23 ermitting routine quantification of multiple plasma proteins.
24 to cause enhanced glomerular permeability to plasma proteins.
25      However, bumetanide is heavily bound to plasma proteins (~98%) and highly ionized at physiologic
26                         Pregnancy-associated plasma protein A (PAPPA) is a metalloproteinase that con
27     Trophoblast-derived pregnancy-associated plasma protein A (PAPPA) is specifically elevated in pre
28                         Pregnancy-associated plasma protein-A (PAPP-A) is a large metalloproteinase s
29  to investigate whether pregnancy-associated plasma protein-A (PAPP-A) is useful for risk assessment
30 ncin metalloproteinase, pregnancy-associated plasma protein-A (PAPP-A), which modulates insulin-like
31 ting metalloproteinase, pregnancy-associated plasma protein-A (PAPP-A).
32 ertebrate-specific gene pregnancy-associated plasma protein-aa (pappaa).
33                              Combinations of plasma proteins accurately identified children with a re
34 distribution, charge, coating molecules, and plasma protein adsorption) that can be effectively tuned
35 o investigate how a systemic absence of this plasma protein affects leukocyte recruitment in alveolit
36 NP) to increase vascular permeability to the plasma protein albumin after an acute plasma volume expa
37 ides and tryptic peptides derived from human plasma proteins, allowing precursor ion selection and CI
38 h by mass spectrometry was identified as the plasma protein alpha1-microglobulin, an established mega
39 ly, we quantified urinary excretion of blood plasma proteins alpha1-microglobulin, albumin, and IgG.
40                        We now show that this plasma protein also is highly susceptible to degradation
41 ecule (Evans blue dye) that is known to bind plasma proteins, an exogenous protein (tetanus toxin fra
42                       SeP is a liver-derived plasma protein and a major supplier of selenium, which i
43                                              Plasma protein and brain tissue binding are very importa
44 ins are representatives of the main types of plasma protein and have different shapes and sizes.
45                               Properdin is a plasma protein and is also released from neutrophil gran
46                                     Among 92 plasma proteins and 135 lipid subspecies quantified with
47             FeCl3 induces aggregation of all plasma proteins and blood cells, independent of endothel
48  1-2% (v/v) 1-butanol mobile phase to remove plasma proteins and concentrate the analytes at the colu
49 gulate the extravasation of small molecules, plasma proteins and inflammatory cells.
50 ng to oedema, excitotoxicity, and ingress of plasma proteins and inflammatory cells.
51  The liver is essential for the synthesis of plasma proteins and integration of lipid metabolism.
52 l tool for the unbiased in-depth analysis of plasma proteins and may advance biomarker discovery, as
53                               After removing plasma proteins and microparticles, 1048 proteins were i
54 stitial space limits the space available for plasma proteins and other macromolecules.
55                                 Transport of plasma proteins and solutes across the endothelium invol
56  altered fucosylation of N-linked glycans on plasma proteins and that glycan biomarkers could improve
57 activated microglia/macrophages, astrocytes, plasma proteins, and blood vessels.
58            Compound 3 was 75% bound to human plasma proteins, and its metabolic stability was much gr
59  platelets with cellular adhesion molecules, plasma proteins, and lipids.
60       We used an aptamer-based assay of 1129 plasma proteins, and patient clinical details were conce
61                               High-abundance plasma proteins are involved in disease-associated pathw
62                                Since certain plasma proteins are regulated through extracellular phos
63  with many human diseases, only a handful of plasma proteins are routinely used in clinical tests.
64 we sought to identify coordinated changes in plasma proteins associated with breast cancer based on l
65                      Our aim was to discover plasma proteins associated with early AD pathology by in
66 tibody prevents mural cell loss and modifies plasma proteins associated with Notch3 activity, includi
67 light mass spectrometry was used to identify plasma proteins associated with renal allograft rejectio
68 ibodies are shown to be directed against the plasma protein beta(2)-glycoprotein I and not against ph
69 oth values were significantly lower than the plasma protein binding (71% +/- 5%) and red cell uptake
70 ere significantly lower (P < 0.001) than the plasma protein binding (75% +/- 3%) and red cell uptake
71 of (99m)Tc-PSMA-I&S was observed due to high plasma protein binding (94%) of the tracer.
72                                          The plasma protein binding (PPB%) was calculated and found t
73            This peptide showed relative high plasma protein binding abilities and a human plasma half
74 n log D, solubility, in vitro clearance, and plasma protein binding also hold in transformation space
75 ng oligonucleotides (MTOs) provide increased plasma protein binding and biodistribution to liver, and
76 ctive starting point, (b) the probability of plasma protein binding and cytotoxicity are often increa
77                                          The plasma protein binding and erythrocyte uptake were deter
78                                          The plasma protein binding and red cell uptake of (99m)Tc(CO
79                                          The plasma protein binding and red cell uptake of (99m)Tc(CO
80 vations are presented to illustrate that low plasma protein binding does not necessarily lead to high
81 boxamide moiety at C-3 significantly reduced plasma protein binding effects in vitro.
82 o metabolic stability, plasma stability, and plasma protein binding for a representative set of compo
83                                              Plasma protein binding of (18)F-FDS was low (<0.1%), and
84 nges for drug development have been the high plasma protein binding of lead structures, interspecies
85                                              Plasma protein binding of Na(2)[(99m)Tc(CO)(3)(NTA)] ave
86 icantly changed the plasma concentration and plasma protein binding of radiotracer.
87 4, aqueous kinetic solubility, and estimated plasma protein binding values in ranges predictive of go
88 and hepatocyte stability, MDCK permeability, plasma protein binding).
89 ro studies show that 2d, 2h, and 2j have low plasma protein binding, a necessary condition for renal
90 ed high kidney extraction and excretion, low plasma protein binding, and high metabolic stability as
91 hat aqueous solubility, Caco-2 permeability, plasma protein binding, human ether-a-go-go-related pota
92                                              Plasma protein binding, red cell uptake, and percentage
93                                              Plasma protein binding, red cell uptake, and percentage
94 ompound 17 demonstrate low clearance and low plasma protein binding.
95  satisfactory liver microsomes stability and plasma protein binding.
96 monkeys, after correcting for differences in plasma protein binding.
97 alian cell lines was reduced, solubility and plasma-protein binding were improved while retaining pot
98 , low intrinsic microsome clearance, and low plasma-protein binding.
99 rafiltration assay was used to determine the plasma protein-binding properties of (177)Lu-cm09.
100 stments, and efforts, the ongoing search for plasma protein biomarkers for disease so far has come up
101 appointing state of affairs may suggest that plasma protein biomarkers have little more to offer for
102 rs, including hepatic transcription factors, plasma proteins, biotransformation enzymes, and metaboli
103                                    Serum and plasma proteins bound to the nanoworms are mostly interc
104                         OT binds strongly to plasma proteins, but a reduction/alkylation (R/A) proced
105 ntify and quantify AGE modification sites in plasma proteins by reversed phase HPLC mass spectrometry
106  peripheral nervous system are shielded from plasma proteins by the blood-brain barrier and blood-ner
107  shock and is characterized by a decrease in plasma protein C (PC) levels.
108 performed a genome-wide association scan for plasma protein C antigen concentration with approximatel
109 n's substrate specificity toward cleavage of plasma protein C into activated protein C (APC), which o
110 ctivity, reduced EPCR shedding, and restored plasma protein C level.
111                                          The plasma protein C3 is a central element in the activation
112 ace were significantly associated with lower plasma protein carbonyl levels in unaffected sisters.
113  in humans was quantified by measuring blood plasma protein carbonyls using the two commercially avai
114                                              Plasma protein carbonyls were associated with an increas
115 lore factors that contribute to variation in plasma protein carbonyls, and to determine whether this
116 ients with organic acidemias showed elevated plasma protein carbonyls, while plasma samples from all
117 e, it engages a tightly regulated network of plasma proteins, cell surface receptors, and regulators.
118 ted with early AD pathology by investigating plasma protein changes at the asymptomatic and symptomat
119 uppress irreversible interactions with blood-plasma protein cocktails.
120 ogen and also vitronectin, and the two human plasma proteins compete for PE binding.
121 he major negative regulator of the AP is the plasma protein complement factor H (FH).
122             This study demonstrated that C3b:plasma protein complexes form in the fluid-phase during
123        The nonspecific binding of heparin to plasma proteins compromises its anticoagulant activity b
124   There was a significant reduction in total plasma protein concentration after plasmapheresis (p < .
125                          We found that total plasma protein concentration increased during hibernatio
126 ontaneous breathing trial-induced changes in plasma protein concentration, 0.96 (0.90-1.01) for chang
127                                              Plasma protein concentration, hemoglobin concentration,
128 s has been shown to be more dependent on the plasma protein concentrations and the type of zeolites t
129 tor, assessed with the median cutoff method, plasma protein concentrations were also not associated w
130                               Measurement of plasma protein concentrations with ELISA revealed a redu
131 ar lung water indexed for ideal body weight, plasma protein concentrations, hemoglobin concentration,
132 e natriuretic peptide level, hemoglobin, and plasma protein concentrations.
133       Histidine-rich glycoprotein (HRG) is a plasma protein consisting of 6 distinct functional domai
134 How the kidney prevents urinary excretion of plasma proteins continues to be debated.
135 ies showed that the complexes consisted of a plasma protein covalently bound to C3b in a 1:1 molar ra
136                We find internal cleavages in plasma proteins created by endo- and exopeptidases, prov
137 ght to be the transcellular pathway by which plasma proteins cross normal capillary endothelium, but,
138 ur mouse brain vasculome with representative plasma protein databases demonstrated significant overla
139 though the concentrations of most individual plasma proteins decreased, as did the white blood cell t
140                   Among the 190 differential plasma proteins detected by iTRAQ, carbamoyl-phosphate s
141 wledge, this marks the lowest LOD for a real plasma protein detection based on label-free LSPR shift
142 tivity and ruggedness, compared to the whole plasma protein digestion approach alone.
143                            Several different plasma proteins displayed a discrete high molecular SDS-
144  interactions contribute to the accretion of plasma proteins during drusen formation.
145                   Kallistatin, an endogenous plasma protein, exhibits pleiotropic properties in inhib
146 M detection of seven out of eight endogenous plasma proteins expressed at ng/mL or subng/mL levels in
147 me or anticipate clinical trajectories using plasma protein expression would allow personalization of
148 ood-brain barrier breakdown characterized by plasma protein extravasation following fibrinogen and Ig
149  One of its most important regulators is the plasma protein factor H (FH).
150 ascade is triggered by the activation of the plasma protein factor XII (FXII) and leads to kallikrein
151 ficial surfaces and biologic substances, the plasma proteins factor XII (FXII) and prekallikrein unde
152 n of P. aeruginosa that binds the four human plasma proteins, Factor H, Factor H-like protein-1 (FHL-
153 s thrombin-catalyzed conversion of a soluble plasma protein, fibrinogen, into a polymeric fibrin clot
154 lin loss or paralysis onset and that, of the plasma proteins, fibrinogen specifically induces rapid a
155 erived hepatocytes produced and secreted the plasma proteins, fibrinogen, fibronectin, transthyretin,
156 or its interaction with collagen type IV and plasma proteins fibronectin and plasminogen.
157 ureus had sufficiently high solubility, high plasma protein free fraction, and favorable pharmacokine
158 gram increased the fraction unbound to human plasma protein from below minimum detection levels, i.e.
159                                              Plasma proteins from blood samples altered the ability o
160 paration and rapid measurement of a panel of plasma proteins from quantities of whole blood as small
161                             We show that the plasma protein haptoglobin prevents the fatty acid-promo
162 Treatment with the naturally occurring human plasma proteins haptoglobin or hemopexin may have benefi
163 face area and high pore volume towards human plasma proteins has been investigated.
164                                           As plasma proteins have become a major thrust in the field
165                  Recently, the liver-derived plasma protein, histidine-rich glycoprotein (HRG), was d
166 ycoprotein (HRG) is a 75-kDa heparin-binding plasma protein implicated in the regulation of tumor gro
167 of a comparison of the quantification of 100 plasma proteins in >1500 LC-MS runs, the SD range of the
168          We aimed to reveal the differential plasma proteins in 272 CHD patients with or without PAH.
169    We performed multianalyte profiling of 50 plasma proteins in 28 patients with persistent HCV infec
170 iologic hemostasis upon injury involves many plasma proteins in a well-regulated cascade of proteolyt
171                 We analyzed a broad range of plasma proteins in children with well-characterized asth
172 h by serving as a comprehensive reference of plasma proteins in humans and accelerate biomarker disco
173                                    Two major plasma proteins in humans are primarily responsible for
174 ized that formation of a fibrin mesh retains plasma proteins in NPs.
175                                              Plasma proteins in patients who responded to standard tr
176                         It bound strongly to plasma proteins in rats (97%), and its labeled metabolit
177                      Here, turnover rates of plasma proteins in rats were measured in vivo using a pu
178 ndergo changes in morphology and internalize plasma proteins in response to this disorder.
179 re the roles of clumping factor A (ClfA) and plasma proteins in supporting adhesion.
180                                    The major plasma proteins in the animal plasmas were identified us
181 r, the mechanisms underlying NP retention of plasma proteins in the submucosa remain unclear.
182 r, the mechanisms underlying NP retention of plasma proteins in their submucosa remain unclear.
183 ding to fibrin and fibrin-associated clotted plasma proteins in tumor vessels.
184 45.3% of 18F-PFH was found to associate with plasma proteins in vivo in normal rats.
185                   We detected changes to 418 plasma proteins in vivo, and detected changes to 262 pro
186 lized platelets in the presence of exogenous plasma proteins, in comparison with suspension-grown cel
187 luenced by interaction of the particles with plasma proteins, including coagulation factor X (FX), wh
188 mass spectrometry (MS) targeted detection of plasma proteins, including post-translational modificati
189 he circulating thyroid hormones are bound to plasma proteins, including three major thyroid hormone-b
190 h intravascular substances as hemoglobin and plasma proteins increases.
191 on on AP53 cells, further showing that these plasma protein inhibitors are active on GAS cells.
192 stinal digestion, intestinal permeation, and plasma protein interaction of polyphenols from a single
193  vitro bioaccessibility, bioavailability and plasma protein interaction of polyphenols from Annurca a
194                               High abundance plasma proteins interfere with detection of potential pr
195 arrier function that restricts flow of large plasma proteins into tissues while allowing small molecu
196                  Fetuin-A is a liver-derived plasma protein involved in the regulation of calcified m
197                 Albumin is the most abundant plasma protein involved in the transport of many compoun
198 glass-based microarray, able to measure >600 plasma proteins involved in cell-to-cell communication,
199 dema or formation of a pseudocyst containing plasma proteins is a prominent characteristic of nasal p
200 igation of chiral interactions of drugs with plasma proteins is of fundamental importance for drug ef
201     C-reactive protein (CRP), an acute-phase plasma protein, is a major component of inflammatory rea
202       Serum AA protein (SAA), an acute phase plasma protein, is deposited extracellularly as insolubl
203                    However, VA1 induced less plasma protein leakage and myeloid inflammatory cell rec
204                                Microvascular plasma protein leakage is an essential component of the
205 cle or dorsal skin, but neutrophil-dependent plasma protein leakage was abolished.
206  loci explained up to 66% of interindividual plasma protein-level variation and, on average, accounte
207  with cell-type-specific gene expression and plasma protein levels sheds light on potential disease m
208 ithin a gene were analyzed for corresponding plasma protein levels using genome-wide association stud
209                                              Plasma protein levels were measured via enzyme-linked im
210 as performed, to evaluate the association of plasma protein levels with dopaminergic system integrity
211 ciation between ARDS-associated genotype and plasma protein levels.
212 ript by >70% and significantly reduced PCSK9 plasma protein levels.
213 ation defined by tumour mutational status or plasma protein levels.
214 ecovery of body weight, reduced glycosylated plasma proteins levels, and long-term survival without s
215 dema or formation of pseudocysts filled with plasma proteins, mainly albumin.
216 fic for C5a, suggesting that this ubiquitous plasma protein may have a more significant role in neutr
217 ween platelet glycoprotein (Gp) IIb/IIIa and plasma proteins mediate platelet cross-linking in arteri
218 ed studies of 3 include its binding to human plasma proteins, metabolic stability using human liver m
219                                              Plasma proteins (n = 163) were profiled in 44 patients w
220                            Recently, a novel plasma protein named MBL/ficolin-associated protein-1 (M
221          Protein S (PS) is a multifunctional plasma protein of the hemostatic and inflammatory pathwa
222 io (a test showing relative amounts of major plasma proteins) of 0.92 (reference range, 0.8-2.0), a u
223 in a mean 70% reduction in circulating PCSK9 plasma protein (p<0.0001) and a mean 40% reduction in LD
224 ysiologic conditions, significant amounts of plasma protein pass the renal filter and are reabsorbed
225  the present study, we report that the inert plasma protein plasminogen (plg) acts as a key regulator
226        Pathogenic microbes acquire the human plasma protein plasminogen to their surface.
227 ain of Epf (EpfN) and increased by the human plasma protein plasminogen.
228 lbumin, but facilitates the loss of IgG from plasma protein pools.
229 ) hydrogel particles for removal of abundant plasma proteins, prior to proteome analysis by MS.
230 tion of genetic variants affecting the human plasma protein profile by combining high-throughput and
231 l-to-cell communication, was used to measure plasma protein profiles in 96 HIV-infected, treatment-na
232 isease (CHD-PAH) has serious consequence and plasma protein profiles in CHD-PAH are unknown.
233                      These data suggest that plasma protein profiles might improve the prediction of
234                 However, although changes of plasma protein profiles reflect physiological or patholo
235                                  We compared plasma protein profiles with ex vivo islet secretome and
236                        In the present study, plasma protein profiling was compared among NEC, sepsis
237 -feet from cerebral microvessels, leakage of plasma proteins, reduction in expression of endothelial
238   However, the foetal blood-brain barrier to plasma proteins remained intact.
239 owever, the exact physiological role of this plasma protein remains enigmatic.
240 Western ligand blot analyses of vitreous and plasma proteins separated by two-dimensional gel electro
241                                          The plasma protein Serum Amyloid P (SAP) inhibits fibrocyte
242                                          The plasma protein serum amyloid P (SAP) reduces neutrophil
243 differentiation is strongly inhibited by the plasma protein serum amyloid P (SAP), and healthy tissue
244 dosis always contain the nonfibrillar normal plasma protein, serum amyloid P component (SAP).
245                     Our results suggest that plasma proteins should be evaluated as a tool for progno
246 aminase-specific labeling to identify FXIIIa plasma protein substrates and their reactive residues.
247 h as scavenger receptors on macrophages, and plasma proteins, such as C-reactive protein (CRP).
248 d protein-1 (MAP-1) is a recently discovered plasma protein that acts as an upstream inhibitor of the
249 ding protein (C4BP), a complement regulatory plasma protein that attenuates the classical and lectin
250                             Haptoglobin is a plasma protein that binds free hemoglobin, thereby inhib
251                             Kallistatin is a plasma protein that exhibits pleiotropic effects in vaso
252      Serum amyloid A (SAA) is an acute-phase plasma protein that functions in innate immunity and lip
253                 Adiponectin is a fat-derived plasma protein that has cardioprotective roles in obesit
254  von Willebrand factor (VWF) is a multimeric plasma protein that mediates platelet adhesion to sites
255 on of iron from transferrin, the circulating plasma protein that normally delivers iron to cells by r
256                Complement factor H (fH) is a plasma protein that regulates activation of the alternat
257 a receptor for vitronectin (Vn), an abundant plasma protein that regulates the terminal pathway of th
258 ase-3 (GPx-3) is a selenocysteine-containing plasma protein that scavenges reactive oxygen species in
259            In univariable analyses, the only plasma protein that was associated with overall survival
260        We also present an 'adsorbome' of 125 plasma proteins that are known to associate with nanomat
261  means of identifying preclinical changes in plasma proteins that are potentially relevant to the ear
262 m of this study was to identify and validate plasma proteins that are predictive of outcome in IPF.
263 ue to the blocking of radioligand binding to plasma proteins that elevated the free fractions of the
264 includes a predominant contribution of blood plasma proteins that is conserved with human SF.
265 cquisition, assimilation and/or recycling of plasma proteins that predicted overwinter survival.
266                    Further, we identified 98 plasma proteins that still foul current "protein-resista
267  was a significant increase in the number of plasma proteins that were related to endocrine system di
268 0183) site-specific changes in the number of plasma proteins that were related to tumor formation, re
269 profiling of a larger portion of circulating plasma proteins (the plasma proteome) will provide oppor
270 o bind, absorb, and load their granules with plasma proteins, this report is one of the first to expl
271 How fibronectin (FN) converts from a compact plasma protein to a fibrillar component of extracellular
272 a list of proteins or peptides against known plasma proteins to assess novelty of their data set.
273 ier changes that allow a different subset of plasma proteins to enter vitreous fluids.
274 oss-talk couples transendothelial leakage of plasma proteins to the cytokine circuitry that coordinat
275 ssed mature liver genes such as cytochromes, plasma proteins, transporters, and other hepatic enzymes
276                                          The plasma protein transthyretin (TTR) is linked to human am
277 studies, creating a reference dataset of 496 plasma protein turnover rates from 4 healthy adults.
278    This study is the first to measure global plasma protein turnover rates in rats in vivo, measure v
279 le is known about how concentrations of many plasma proteins vary between individuals from different
280 es the blood flow through its binding to the plasma protein von Willebrand factor (VWF) and transmits
281                                          The plasma protein von Willebrand factor (VWF) is essential
282                                              Plasma protein von Willebrand factor (VWF) mediates adhe
283                       A core signature of 46 plasma proteins was commonly modulated in all three infe
284                        Normal sialylation of plasma proteins was observed in spite of NANS deficiency
285 ween groups, and biochemical profiling of 40 plasma proteins was performed to identify proteins that
286  the MudPIT-SRM approach to profile abundant plasma proteins, we demonstrated mean increases in peak
287 ng the corresponding effects on inflammatory plasma proteins, we identified cathepsin S as a lead ind
288               It showed low binding to human plasma proteins, weakly inhibited cytochrome P450 isofor
289  more inflammatory than those to which other plasma proteins were adsorbed.
290                                 Differential plasma proteins were first detected by iTRAQ proteomic t
291 tabolic enzymes, some myofibrillar and blood plasma proteins were identified, which were characterise
292          Concentrations of 54 high-abundance plasma proteins were measured simultaneously by liquid c
293                                              Plasma proteins were sampled from male and female fish f
294 shing the binding affinity between drugs and plasma proteins, which is a critical factor to develop e
295 ted to improve the fraction unbound to human plasma protein while retaining biochemical potency.
296        Human alpha-1-antitrypsin (A1PI) is a plasma protein with the function of protecting lung tiss
297                                    Hx is the plasma protein with the highest binding affinity to heme
298 , and turnover rates were quantified for 157 plasma proteins with half-lives ranging 0.3-103 days.
299 poproteins, 135 lipid species, and 211 other plasma proteins with incident CVD (91 events), defined a
300                           The interaction of plasma proteins with metal oxide nanoparticles (NPs) is

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