ライフサイエンスコーパス: plasma renin activity

1 r PA using the ratio of serum aldosterone to plasma renin activity.

2 ifferences between aldosterone excretion and plasma renin activity.

3 rkedly elevated with a relatively suppressed plasma renin activity.

4 d on day 3 despite significant reductions in plasma renin activity.

5 nsin I and II despite continuously increased plasma renin activity.

6 supplemental use of the in vitro stimulated plasma renin activity.

7 TS, there was not a compensatory increase in plasma renin activity (0.79+/-0.58 versus 0.79+/-0.74 ng

8 Postinfusion, the decrease in plasma renin activity (-0.9+/-0.2 versus -0.6+/-0.2 ng/m

9 values <15 ng/dL) but contrasting values of plasma renin activity (15.00 versus 0.56 ng/mL/h; P<0.00

10 Ang 1-7 treatment were associated with lower plasma renin activity (-40%) and serum aldosterone (-48%

11 32 +/- 24 versus 24 +/- 15 mg/dl, P = 0.06), plasma renin activity (7.1 +/- 9.9 versus 3.4 +/- 5.6 ng

12 lability to produce sustained suppression of plasma renin activity after oral administration.

13 Plasma renin activity, aldosterone and arginine vasopres

14 e mean arterial pressure, renal plasma flow, plasma renin activity, aldosterone, urine sodium, and ba

15 Plasma renin activity also increased appropriately when

16 Losartan, EXP3174 and captopril elevated plasma renin activities and comparably and significantly

17 early-morning ratio of plasma aldosterone to plasma renin activity and 24-hour urinary aldosterone an

18 ith ACEI+D, whereas OMA+D resulted in higher plasma renin activity and a delayed increase in aldoster

19 station fetal sheep have minor influences on plasma renin activity and ACTH in normovolaemic fetuses,

20 cardiac nerves and a sustained rise in fetal plasma renin activity and ACTH.

21 The LS diet increased plasma renin activity and aldosterone concentration, whe

22 onism; controlled posture studies to measure plasma renin activity and aldosterone concentrations, fo

23 Plasma renin activity and aldosterone immediately increa

24 th healthy controls and explored the role of plasma renin activity and aldosterone in the regulation

25 fore and after dexamethasone administration, plasma renin activity and aldosterone level.

26 OMA alone did not increase plasma renin activity and aldosterone, but resulted in a

27 Plasma renin activity and angiotensin levels were reduce

28 activity, height, weight, sodium excretion, plasma renin activity and heart rate were examined.

29 Plasma renin activity and insulin concentrations were bo

30 ients with POTS have paradoxically unchanged plasma renin activity and low aldosterone given their ma

31 -four hour urinary sodium was increased, and plasma renin activity and plasma aldosterone levels were

32 At 2 wk, plasma renin activity and plasma aldosterone levels were

33 Plasma renin activity and plasma angiotensin II levels w

34 Furthermore, both plasma renin activity and plasma ET-1 increased with ET-

35 Despite greater intra-group improvements in plasma renin activity and serum aldosterone levels in th

36 al models characterized by various levels of plasma renin activity and significantly potentiated urin

37 Heart rate (HR), blood pressure (BP), plasma renin activity, and aldosterone were measured wit

38 ratio of plasma aldosterone concentration to plasma renin activity, and higher urine aldosterone leve

39 osteronism may cause hypokalemia, suppressed plasma renin activity, and hypertension.

40 l, pro B-type natriuretic peptide, increased plasma renin activity, and increased blood urea nitrogen

41 drome, with normal blood pressure, increased plasma renin activity, and reduced NCC expression and ph

42 Aldosterone excretion rate, plasma renin activity, and size and location of adenomas

43 atremia, hyperkalemia, hypovolemia, elevated plasma renin activity, and sometimes shock and death.

44 Weekly blood pressure, plasma renin activity, Ang II, ET, and catecholamines we

45 at achieved by CA alone, while also reducing plasma renin activity, angiotensin II, aldosterone and v

46 radiol, PAI-1, tissue plasminogen activator, plasma renin activity, angiotensin II, and aldosterone w

47 Plasma renin activity, angiotensin II, and aldosterone w

48 erone levels, and also significantly reduced plasma renin activity, angiotensin II, and vasopressin c

49 There were no changes in plasma renin activity, angiotensin II, or aldosterone.

50 chloremia, metabolic acidosis and suppressed plasma renin activity are variable associated findings.

51 rcalciuria, increased serum aldosterone, and plasma renin activity, are the two major diseases linked

52 independent loci displayed associations with plasma renin activity at genome-wide significance (P<5x1

53 hich is associated with a markedly depressed plasma renin activity because of sodium retention.

54 ane anaesthesia and surgery caused a rise in plasma renin activity but was associated with a suppress

55 tamin D levels and the blood pressure and/or plasma renin activity, but the mechanism is not understo

56 Neither urinary cGMP nor plasma renin activity changed.

57 Plasma renin activity decreased from 80 +/- 88 ng/dL at

58 days of treatment with NTG patches increased plasma renin activity for the entire treatment period.

59 ion rates appear to be accompanied by higher plasma renin activities in mice, compared with rats, rab

60 ects of ACE inhibition on blood pressure and plasma renin activity in both normotensive and hypertens

61 nal kallikrein and renal renin activity, and plasma renin activity in control and diabetic rats and d

62 Hemorrhage increased heart rate and plasma renin activity in intact sheep, but these respons

63 , but it significantly altered the change in plasma renin activity in response to ACE inhibition (-0.

64 Concomitantly, plasma renin activity increased from 3.08 +/- 0.68 (mean

65 Plasma renin activity increased significantly in the cap

66 plemental measurement of in vitro stimulated plasma renin activity insignificantly (p > 0.10) and imp

67 tension with hypokalaemia and suppression of plasma renin activity is known as mineralocorticoid hype

68 nt BP reduction and prolonged suppression of plasma renin activity is observed.

69 ta-analyzed genome-wide association data for plasma renin activity (n=5275), plasma renin concentrati

70 seline cycle (P = 0.001), and an increase in plasma renin activity of 0.14 +/- 0.08 ng/(L . s) from a

71 a combination of the two, compared with the plasma renin activity of the controls.

72 We find no difference in plasma renin activity or plasma aldosterone concentratio

73 multivariate analysis, LVMI correlated with plasma renin activity (p < 0.001) and plasma norepinephr

74 Insulin infusion increased plasma renin activity (P < 0.01) and renal plasma flow (

75 ration produced a dose-dependent decrease in plasma renin activity (P=0.004), with similar trends obs

76 The reduction of plasma renin activity, plasma aldosterone, and arginine

77 Plasma renin activity, plasma aldosterone, plasma and 24

78 Preoperative plasma renin activity, plasma and urinary aldosterone co

79 ours after hospitalization adrenal function, plasma renin activity, plasma noradrenaline and vasopres

80 Ly markedly raised plasma renin activity (PRA) and aldosterone, and exerted

81 weight loss on Iso-induced water intake and plasma renin activity (PRA) and found that weight loss d

82 A BEARS-based method was developed for plasma renin activity (PRA) and was evaluated on fifty-t

83 this study, it was hypothesized that the low plasma renin activity (PRA) is misleading, masking and p

84 nt BP reduction and prolonged suppression of plasma renin activity (PRA) is observed after aliskiren

85 Endothelin, catecholamine, and plasma renin activity (PRA) responses to 24-hour sodium

86 Plasma renin activity (PRA) was similar in young versus

87 els, 24-h urinary aldosterone excretion, and plasma renin activity (PRA) were determined in all group

88 ion of sodium, potassium, and creatinine and plasma renin activity (PRA) were measured in 2937 mildly

89 Supine blood pressure and plasma renin activity (PRA) were measured, and daily sod

90 The plasma hormones, AVP, ANP, plasma renin activity (PRA), angiotensin II, and aldoste

91 this site relate to salt sensitivity and low plasma renin activity (PRA).

92 ) with measurements of serum aldosterone and plasma renin activity (PRA).

93 rats in the highest quartiles of both BP and plasma renin activity (PRA).

94 Plasma renin activity (PRA; >4 ng/mL/hour) was used as a

95 <0.0001), and ratio of plasma aldosterone to plasma renin activity (r=-0.43, P<0.0001) but was indepe

96 tassium and plasma aldosterone concentration-plasma renin activity ratio for patients with hypertensi

97 evel and plasma aldosterone concentration to plasma renin activity ratio.

98 tly alter the magnitude of the ACTH, AVP, or plasma renin activity response to haemorrhage.

99 tients' work-up included plasma aldosterone, plasma renin activity, serum cortisol, and estimation of

100 Salt depletion for 1 wk increased plasma renin activity seven-fold in wild-type mice but o

101 iotensin II receptors with losartan elevated plasma renin activity some 29-fold (P < 0.001) and cause

102 mia also completely eliminated the increased plasma renin activity that accompanied restraint in cont

103 Plasma renin activity was assessed by radioimmunoassay o

104 llowing reduction of RPP to 60 mmHg for 3 h, plasma renin activity was increased more than 7-fold (P

105 Plasma renin activity was markedly suppressed (0.2 +/- 0

106 ary cGMP was 40% greater than in N rats, but plasma renin activity was not significantly greater in C

107 Plasma aldosterone but not plasma renin activity was significantly elevated after 2

108 Plasma renin activity was significantly elevated by losa

109 Plasma renin activity was significantly increased during

110 Plasma renin activity was significantly lower in 13 hype

111 rrhage (20% loss of blood volume), including plasma renin activity, was assessed at 2 and 5 months po

112 Renal tissue kallikrein levels and plasma renin activity were decreased, whereas renal reni

113 aldosterone, atrial natriuretic peptide, and plasma renin activity were drawn at baseline and 2 hours

114 sodium levels, urinary sodium excretion, and plasma renin activity were measured for five time period

115 Renal plasma flow and plasma renin activity were measured serially.

116 Plasma Ang II, ET, catecholamines, and plasma renin activity were unchanged during the progress

117 xcretion, urinary aldosterone excretion, and plasma renin activity were unchanged.