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1 gregation, red blood cell deformability, and plasma viscosity.
2 and included triacylglycerols (2.02 mmol/L), plasma viscosity (-0.10 mPa.s), and serum viscosity (-0.
4 ensity lipoprotein cholesterol, reduction in plasma viscosity and fibrinogen concentration, increase
6 at-rich test meal on postprandial changes in plasma viscosity and serum viscosity was assessed in 12
7 imer, von Willebrand factor (vWF), blood and plasma viscosities, and hematocrit as part of its baseli
8 edimentation rate]), serum fibrinogen level, plasma viscosity, and leukocyte rigidity (assessed in vi
10 <0.01), as well as von Willebrand factor and plasma viscosity (both P<0.05), had significant hazard r
12 bnormalities of hemorheological (fibrinogen, plasma viscosity), endothelial (von Willebrand factor [v
13 ty (P< or =.001) and its major determinants, plasma viscosity, fibrinogen (both P< or =.01), and hema
15 VII, von Willebrand factor, fibrinogen, and plasma viscosity in subjects of the Framingham Offspring
19 443), whereas log fibrinogen (p < 0.001) and plasma viscosity (p = 0.04) were independent associates
21 viscosity, fibrinogen (both P< or =.01), and plasma viscosity (P< or =.05) remained significantly ass
24 fibrinogen), and indexes of blood rheology (plasma viscosity, plasma fibrinogen, and hematocrit) in
29 associated with lower levels of fibrinogen, plasma viscosity, von Willebrand factor, and factor VII.
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