ライフサイエンスコーパス: plasma volume

1 nd hypovolemia led to a similar reduction in plasma volume.

2 rats, a model of normal renin with a raised plasma volume.

3 Alb data provided an independent estimate of plasma volume.

4 uantify the BBB leakage rate and local blood plasma volume.

5 on Rayleigh scattering of microwaves on the plasma volume.

6 ANP), which acts with the kidney to regulate plasma volume.

7 bility may be part of a strategy to maintain plasma volume.

8 n arterial pressure in the face of decreased plasma volume.

9 lung weights, weight gain, hemodilution, and plasma volume.

10 body clearance required for ANP to regulate plasma volume.

11 ndocrine actions of ANP in the regulation of plasma volume.

12 ctivity and aldosterone in the regulation of plasma volume.

13 aldosterone given their marked reduction in plasma volume.

14 may be an indicator of an expanded maternal plasma volume.

15 calculated by subtracting true from apparent plasma volume.

16 nd the ingestion of sodium may help maintain plasma volume.

17 tagged albumin to measure red blood cell and plasma volume.

18 s associated with a significant expansion of plasma volume.

19 ttributed solely to the estimated changes in plasma volume.

20 This method permits analysis with lower plasma volumes (100 microL) and greater sensitivity (to

21 Bed rest led to a reduction in plasma volume (17%), baseline PCWP (18%), SV (12%), LVED

22 Patients with POTS had a greater deficit in plasma volume (334+/-187 versus 10+/-250 mL, P<0.001), r

23 ubjects with obese HFpEF displayed increased plasma volume (3907 mL [3563-4333 mL] versus 2772 mL [25

24 ntional therapy, appear to have a contracted plasma volume, a concept that is in contrast to the wide

25 0 min) more than 95% of the peak increase in plasma volume after volume expansion (4.5% bovine serum

26 % saline was available in addition to water, plasma volumes after 24 h HU were not different from rat

27 essin occurred earlier than the reduction of plasma volume and atrial natriuretic peptide after Heart

28 thelial sodium channel antagonist normalized plasma volume and blood pressure, but only partially cor

29 triuretic peptide (ANP) in the regulation of plasma volume and blood pressure.

30 ure ASGP-R concentration, as well as hepatic plasma volume and flow.

31 Plasma volume and fluid regulatory hormones were measure

32 RBCV, plasma volume and Hb(mass) all increased (P < 0.05) afte

33 We measured plasma volume and plasma levels of atrial natriuretic pe

34 olume can help identify the heterogeneity in plasma volume and red blood cell mass that are features

35 Similar increases of plasma volume and supranormal cardiac index were observe

36 tion because pregnancy produces increases in plasma volume and the hemoglobin concentration decreases

37 H/+ mice have decreased plasma volumes and significantly heavy stiff hearts.

38 ar end-diastolic volume, 2) exhibit a higher plasma volume, and 3) limit kidney injury and free-water

39 r and malaise, 'capillary leak' with loss of plasma volume, and coagulation defects which can lead to

40 Body mass and composition, plasma volume, and fasting concentrations of metabolic c

41 I and mean adjusted PSA concentrations, mean plasma volume, and mean adjusted PSA mass (total circula

42 n brown adipose tissue, minimal increases in plasma volume, and no increases in extracellular fluid v

43 del of high-renin hypertension with a normal plasma volume, and one-kidney, one clip (1K-1C) rats, a

44 of degradation of marker in the myocardium, plasma volume, and relative rate of loss of marker from

45 ance of myometrial quiescence, regulation of plasma volume, and release of neuropeptides, such as oxy

46 in the kidney, but did increase body weight, plasma volume, and the fluid content of abdominal fat pa

47 protein, resulting in chronically increased plasma volume, arterial hypertension, and cardiac hypert

48 outcome variable and the input total RNA or plasma volume as an exposure variable, which is equivale

49 alculated as PSA concentration multiplied by plasma volume), assessed by determining P values for tre

50 Obtained data were adjusted for different plasma volume at the two occasions.

51 protein-bound solutes were greater than the plasma volume, averaging 15 +/- 7 L for PCS and 14 +/- 3

52 decreasing the incidence of TRALI from high plasma volume blood products.

53 iuretic peptide (ANP) acts acutely to reduce plasma volume by at least 3 mechanisms: increased renal

54 Preferential expansion of the plasma volume by infusion of salt-poor hyperoncotic albu

55 tal muscle injury attenuated the increase in plasma volume, cardiac index, or the repayment of system

56 The absence of plasma volume change would suggest that the mechanism po

57 number of transfusions but who received high plasma volume components from female donors.

58 dy, we identified patients who received high plasma volume components from male-only donors and compa

59 f strategies to minimize transfusion of high plasma volume components, fresh frozen plasma and aphere

60 In critically ill recipients of high plasma volume components, gas exchange worsened signific

61 natriuresis and osmotic diuresis, leading to plasma volume contraction and reduced preload, and decre

62 etic and natriuretic effects contributing to plasma volume contraction, and decreases in systolic and

63 With exercise, plasma volume-corrected levels of triglycerides decrease

64 ide most likely results in extracellular and plasma volume depletion and reduced systemic oxygen tran

65 lidation of clinically applicable methods of plasma volume determination as well as enhanced methodol

66 ERPF calculated by the product of fERPF and plasma volume, determined from patient weight, was compa

67 Plasma volume did not change.

68 Plasma volume disturbances have been implicated in some

69 obesity in older adults results in increased plasma volume, eccentric LV hypertrophy, and systolic an

70 ed blood parameters (hemoglobin, hematocrit, plasma volume), exercise parameters (peak oxygen consump

71 ar mass of any of the current macromolecular plasma volume expanders, we found that it filtered readi

72 hemoglobin is more likely to reflect greater plasma volume expansion (and thus better maternal and of

73 normocythaemia (control), anaemia, anaemia + plasma volume expansion (PVX), anaemia + PVX + hypoxia,

74 mized sheep when compared with the resultant plasma volume expansion after a 50 mL/min of 0.9% infusi

75 Fractional plasma volume expansion after rapid infusion of saline o

76 compared with lean subjects, have decreased plasma volume expansion along with impaired iron homeost

77 Current estimates of plasma volume expansion are outdated and do not necessar

78 r study is needed to characterize diminished plasma volume expansion during pregnancy and to understa

79 - (RGZ) induced increases in body weight and plasma volume expansion found in control mice expressing

80 The failure of maternal plasma volume expansion has been implicated in adverse o

81 severe acute hypoproteinemia does not reduce plasma volume expansion in response to 50 mL/min 0.9% sa

82 Plasma volume expansion is an important component of a s

83 ividual patients who also have a compromised plasma volume expansion or pathologic homeostasis.

84 No difference was found in plasma volume expansion produced by a bolus of 50 mL/min

85 The plasma volume expansion reached approximately 20% at the

86 ogesterone exposure are also associated with plasma volume expansion, and a leftward shift in the osm

87 is also accompanied by sodium retention and plasma volume expansion, and pregnant rats are resistant

88 The most frequent effects were due to plasma volume expansion, including conjunctival redness

89 ognition of runner's anemia, which is due to plasma volume expansion, with hemolysis from the poundin

90 these effects may be mediated by inadequate plasma volume expansion.

91 to the plasma protein albumin after an acute plasma volume expansion.

92 ng metabolic dysfunction, such as failure of plasma volume expansion.

93 gnancy (eg, treatment for mild hypertension, plasma-volume expansion, and corticosteroid use) have a

94 g, closed loop: 4.2 +/- 2 mL/kg; p < 0.001), plasma volume, extravascular volume (bolus resuscitation

95 Plasma volume fell, and plasma atrial natriuretic peptid

96 defined as a conjugate dose of 125 microg/mL plasma volume followed at 48 h by a clearing agent in a

97 vascular permeability (K(PS)) and fractional plasma volume (fPV) for each contrast medium.

98 othelial permeability (K(PS)) and fractional plasma volume (fPV) for each tumor.

99 (b) dissociate preferential expansion of the plasma volume from decreases in sodium reabsorption by t

100 low hematocrit may result from an increased plasma volume (hemodilution) or from reduced red blood c

101 ctomy, higher BMI was associated with higher plasma volume; hemodilution may therefore be responsible

102 Further studies evaluating plasma volume in HF may help to improve our understandin

103 d-to-tissue albumin clearance and changes in plasma volume in isoflurane-anaesthetized mice (C57BL/6J

104 e pressure was correlated with body mass and plasma volume in obese HFpEF (r=0.22 and 0.27, both P<0.

105 Despite the lower plasma volume in patients with POTS, there was not a com

106 hormonal systems, few studies have evaluated plasma volume in this condition under treatment.

107 therapy reduced the tumor vessel density and plasma volume in tumors to a greater extent than did the

108 calculation of intravascular volumes (RBCV, plasma volume) in patients with HF according to the Inte

109 patients with decompensated HF, in whom the plasma volume is contracted despite an increase in total

110 Plasma volume is expanded and plasma osmolality is decre

111 onsible for the lack of adequate decrease of plasma volume; its reduction can be taken as a marker of

112 dy mass index (BMI) have greater circulating plasma volumes, lower PSA concentrations among obese men

113 echnique in parallel with [14C]-sucrose as a plasma volume marker.

114 Clinical assessment of plasma volume may be of particular value during treatmen

115 It is likely that significant changes in plasma volume occur during intensification of medical th

116 his acidotic change; however, no increase in plasma volume occurred.

117 y (CH) has been attributed to an increase in plasma volume or a change in cardiac nutrient preference

118 BMI was significantly associated with higher plasma volume (P < .001 for trend) and lower PSA concent

119 n by an effect unrelated to expansion of the plasma volume, perhaps due to an effect of parathyroid h

120 ment over both the Schlegel and renal uptake plasma volume product estimates (p < 0.05).

121 od samples and the Schlegel and renal uptake plasma volume product scintigraphic techniques.

122 Plasma volume (PV) and blood volume (BV) were measured i

123 vial and subsynovial tissues of the TMJ, and plasma volume (PV) and permeability surface area product

124 We sought to test the hypothesis that plasma volume (PV) expansion in heart transplant recipie

125 Plasma volumes ranged from 0.05 to 0.5 ml, and viral loa

126 hanges in plasma aldosterone levels, whereas plasma volumes ranged from 50% to 150% normal accompanie

127 of 6% hydroxyethyl starch (potato-derived) [Plasma Volume Redibag (PVR); Baxter Healthcare, Thetford

128 , including increased urine output, enhanced plasma volume, reduced weight loss, and substantially im

129 We conclude that releasing plasma volume reduces adverse effects of TZD-induced vol

130 iates the early plasma volume reduction; (c) plasma volume reduction as well as ANP release depend on

131 and a PAF-ANP interaction mediates the early plasma volume reduction; (c) plasma volume reduction as

132 ar beds (such as gastro-intestinal tract) to plasma volume regulation.

133 In healthy persons, plasma volume remains relatively constant as a result of

134 nificantly shortened (p = .03), although the plasma volume required to achieve a durable remission wa

135 ing statistics and the overall laser-induced plasma volume suggests that the primary mechanism of par

136 Plasma volume, systemic hemodynamics, and oxygen transpo

137 ingle-copy assay [iSCA]), and increasing the plasma volume tested (Mega-iSCA).

138 Plasma volume, the intravascular portion of the extracel

139 rowth restriction), but so is failure of the plasma volume to expand.

140 cardiomyopathy, hypertension, and increased plasma volumes, together with increased ventricular supe

141 Kb, hepatic plasma volume, Vh, extrahepatic plasma volume, Ve and hepatic plasma flow, F.

142 ed forward-binding rate constant Kb, hepatic plasma volume, Vh, extrahepatic plasma volume, Ve and he

143 plaques were used to estimate the fractional plasma volume (vp) and transfer constant (Ktrans) of con

144 S) product after correction for the residual plasma volume (Vp) occupied by leptin in the vessel bed

145 extracellular space volume (Ve), fractional plasma volume (Vp)] were calculated, and their relations

146 Baseline plasma volume was 123 +/- 20% of normal; it was 122 +/-

147 nfusion, the estimated (89)Zr-trastuzumab in plasma volume was a median 102% (range, 78%-113%) of the

148 Blood plasma volume was highly variable and the only parameter

149 Plasma volume was markedly expanded following albumin in

150 Plasma volume was measured both pre- and postoperatively

151 e plasmapheresis procedures during which one plasma volume was removed and replaced with fresh frozen

152 thelial transfer coefficient, and fractional plasma volume were calculated for each tumor and each CM

153 anges, capillary filtration coefficient, and plasma volume were measured before and during the colloi

154 Plasma volumes were reduced during HU compared to rats i

155 We also measured plasma volume (with Evans blue dye) and maximal orthosta

156 online analysis compatibility, and a higher plasma volume yield.