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1 improves zinc absorption but does not alter plasma zinc.
2 luence on VSMC gene expression is blocked by plasma zinc.
3 o distinguish direct and indirect effects of plasma zinc.
4 patients had low plasma zinc (hypozincemic) (plasma zinc, 45.4 +/- 1.8 microg/dL), whereas 18 had nor
6 g/d as amino acid chelate for 3 wk) elevated plasma zinc and activities of 5'-nucleotidase, a zinc-de
9 , blood was immediately collected to measure plasma zinc and cytokine concentrations and zinc transpo
10 of placebo, increased the concentrations of plasma zinc and decreased the concentrations of plasma h
12 after zinc supplementation were as follows: plasma zinc and free thyroxine concentrations and mononu
13 significantly by iron-zinc deprivation, but plasma zinc and iron were somewhat lower in the deprived
16 pendent association between dietary zinc and plasma zinc and the association of each with bone minera
17 metric variables, breastfeeding, hemoglobin, plasma zinc, and common bacteria/viral respiratory patho
21 rns have been raised about the usefulness of plasma zinc concentration as an indicator of zinc status
28 or of a population's zinc status because the plasma zinc concentration seems to predict whether growt
31 ange that accounted for the observed fall in plasma zinc concentration was a 60% reduction in the rat
32 different doses of supplemental zinc on the plasma zinc concentration, morbidity, and growth of youn
33 iation between the presence of infection and plasma zinc concentration, possibly because the severity
34 uced weight gain and linear growth and lower plasma zinc concentrations (11.8 +/- 0.6 and 9.2 +/- 0.8
36 model suggests that the profound decrease in plasma zinc concentrations after 5 wk of severe zinc dep
38 Z increased with increased dietary zinc, but plasma zinc concentrations and EZP size were unchanged.
39 ecifically, sepsis was associated with lower plasma zinc concentrations and higher SLC39A8 mRNA expre
40 ation with a dose as low as 3 mg/d increased plasma zinc concentrations and reduced diarrhea incidenc
41 sion was highest in patients with the lowest plasma zinc concentrations and the highest severity of i
49 rately high-phytate diet had low TDZ and low plasma zinc concentrations in the third trimester of pre
56 a standard food-frequency questionnaire, and plasma zinc concentrations were measured by using induct
60 patients) of routine micronutrient screens (plasma zinc, copper, selenium, and vitamins A, B-6, C, a
64 e the older subjects had significantly lower plasma zinc, higher ex vivo generation of inflammatory c
65 the time of transplant, 17 patients had low plasma zinc (hypozincemic) (plasma zinc, 45.4 +/- 1.8 mi
71 umans and rodents has little effect on blood plasma zinc levels, and yet cellular consequences of zin
72 only if the C-reactive protein is <20 mg/L (plasma zinc), <10 mg/L (plasma selenium and vitamins A a
74 hropometric, high-precision knee-height, and plasma zinc measurements were made at baseline and at 3,
79 oglobin, hematocrit, transferrin saturation, plasma zinc, or erythrocyte riboflavin values between 6
80 ere significantly lower in men in the lowest plasma zinc quartile (<11.3 micromol/L) than in men with
85 mg/d, phytate intake was 1033 +/- 843 mg/d, plasma zinc was 44.1 +/- 6.0 microg/dL, and the exchange
88 dence of infections was significantly lower, plasma zinc was significantly higher, and generation of
89 sis showed that changes in concentrations of plasma zinc were inversely associated with changes in co
90 thalassemia major (Thal) frequently have low plasma zinc, which has been associated with low bone mas
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