ライフサイエンスコーパス: plasmacytoid

1 Both plasmacytoid and IgE(+)FcepsilonRI(+) myeloid dendritic

2 dent cross-talk between the main DC subsets, plasmacytoid and myeloid DCs (mDCs) was necessary for in

3 o in vivo expansion of IRF8-dependent CD8a+, plasmacytoid, and CD103+ CD11b2 DCs.

4 Macrophages, plasmacytoid, and conventional dendritic cells were each

5 Plasmacytoid bladder cancer is an aggressive histologic

6 alterations in the CDH1 gene occur in 84% of plasmacytoid carcinomas and are specific to this histolo

7 ent with the aggressive clinical behavior of plasmacytoid carcinomas, which frequently recur locally,

8 ction; however, its regulatory mechanisms in plasmacytoid cells (pDCs) still remain unclear.

9 um AFP, we observed a lower ratio of myeloid/plasmacytoid circulating DCs compared with patients with

10 Depletion of either plasmacytoid DC (pDC) alone or the lymphoid-resident DC

11 l fate decision between interferon-producing plasmacytoid DC (pDC) and antigen-presenting classical D

12 and activity in freshly isolated monocytes, plasmacytoid DC (pDC) and in vitro-generated Langerhans

13 ith monocyte mobilization and recruitment of plasmacytoid DC (pDC) and macrophages to lymph nodes, wh

14 Pre-DC share surface markers with plasmacytoid DC (pDC) but have distinct functional prope

15 In normal individuals, plasmacytoid DC (pDC) expressed high levels of CCR5, whe

16 esulted in increased IFN-alpha production by plasmacytoid DC (pDC) from skin/tumor draining lymph nod

17 endritic cells (mDC) and macrophages but not plasmacytoid DC (pDC) had suppressed capacity to stimula

18 (+) DC, CD1c(+) DC, and, to a lesser extent, plasmacytoid DC (pDC) in the blood, spleen, and bone mar

19 and is physiologically important because in plasmacytoid DC (pDC) stimulated with Toll-like receptor

20 examined the number and activation of blood plasmacytoid DC (pDC), CD141(+), and CD1c(+) myeloid DC

21 two distinct subsets, myeloid DC (mDCs) and plasmacytoid DC (pDCs).

22 CD11c(high)CD103(high)) DC and plasmacytoid (plasmacytoid DC Ag-1(high)B220(+)CD11c(int)) DC (pDC) po

23 Irf8 expression is essential for plasmacytoid DC and CD8alpha(+) DC development, and this

24 HC acquisition by thymic CD8alpha(+) cDC and plasmacytoid DC but not SIRPalpha(+) cDC.

25 NSG mice) develop all human conventional and plasmacytoid DC compartments in lymphoid organs.

26 g plays a pivotal role whereby it suppresses plasmacytoid DC development while enhancing that of CD8a

27 eq studies, we show that Irf8 functions as a plasmacytoid DC epigenetic and fate-determining TF, regu

28 set of DCs; the relationship between blastic plasmacytoid DC neoplasia cells and healthy DCs; and cir

29 cluding the myeloid DC subset (mDCs) and the plasmacytoid DC subset (pDCs).

30 ss I and II from thymic epithelial cells but plasmacytoid DC were less efficient.

31 either CD11c as conventional DC or CD123 as plasmacytoid DC).

32 C-like cells, with a concomitant increase in plasmacytoid DC- and CD8alpha(+) DC-specific gene transc

33 culating myeloid DC, and at a lower level in plasmacytoid DC.

34 the numbers of vaccine-resident CD8(+) DCs, plasmacytoid DCs (pDC), along with local interleukin (IL

35 tory DCs, steady-state conventional DCs, and plasmacytoid DCs (pDC), using mouse lacking either AF-1

36 We found that myeloid DCs (mDCs) and plasmacytoid DCs (pDCs) accumulate in the bone marrow du

37 ajor subsets, the interferon (IFN)-producing plasmacytoid DCs (pDCs) and antigen-presenting classical

38 rol of cell survival of two main DC subsets: plasmacytoid DCs (pDCs) and conventional DCs (cDCs) and

39 2 (BDCA2) is expressed exclusively on human plasmacytoid DCs (pDCs) and plays a role in Ag capture,

40 Although plasmacytoid DCs (pDCs) are a primary source of IFN, the

41 Plasmacytoid DCs (pDCs) are predominant cells secreting

42 both conventional dendritic cells (cDCs) and plasmacytoid DCs (pDCs) are required for the crossprimin

43 Although plasmacytoid DCs (pDCs) are the main source of IFNalpha

44 tial dysregulation of myeloid DCs (mDCs) and plasmacytoid DCs (pDCs) at various stages of HIV-1 infec

45 a new DC subset that shares properties with plasmacytoid DCs (pDCs) but potently activates T cells,

46 isingly, IFN expression was not dependent on plasmacytoid DCs (pDCs) but rather was dependent on mDCs

47 Plasmacytoid DCs (PDCs) exposed to PSA do not produce pr

48 ytokine production by myeloid DCs (mDCs) and plasmacytoid DCs (pDCs) in healthy subjects and MS patie

49 n were used to delineate the role of primary plasmacytoid DCs (pDCs) in initiating B cell responses.

50 conventional DCs (cDC) 1 and cDC2 as well as plasmacytoid DCs (pDCs) in the peripheral blood of pigs.

51 we studied monocyte-derived DCs (moDCs) and plasmacytoid DCs (pDCs) in two HPS2 siblings.

52 Plasmacytoid DCs (pDCs) support antiviral immunity by li

53 rentially captured and internalized by human plasmacytoid DCs (pDCs) that express the TIM1 phosphatid

54 1betaHSD1-H6PDH increases the sensitivity of plasmacytoid DCs (pDCs) to GC-induced apoptosis and rest

55 dian frequency of myeloid DCs (mDCs) but not plasmacytoid DCs (pDCs) was observed in the blood of SR

56 d DC1s (mDC1), BDCA3+(high) mDC2s and BDCA2+ plasmacytoid DCs (pDCs) were identified and expressions

57 of inflammatory cells, such as macrophages, plasmacytoid DCs (pDCs), and neutrophils, which were obs

58 e lacking CD11c(hi) lung DCs, but containing plasmacytoid DCs (pDCs), fail tolerization with inhaled

59 istinct subsets: conventional DCs (cDCs) and plasmacytoid DCs (pDCs).

60 HC complexes, along with increased PD-L1, on plasmacytoid DCs (pDCs).

61 RNA for markers of myeloid DCs (mDCs; CD1c), plasmacytoid DCs (pDCs; CD303) and Langerhans cells (LCs

62 Expression of Irf8 in DC9 cells led to plasmacytoid DCs and CD8alpha(+) DC-like cells, with a c

63 -lambda increased production of IFN-alpha by plasmacytoid DCs and IFN-alpha significantly increased p

64 , whereas IDO2 is expressed in both mDCs and plasmacytoid DCs and is not modulated by PGE2.

65 tions to distinct DC lineages, consisting of plasmacytoid DCs and several subsets of classical DCs th

66 hey block viral cell to cell transmission to plasmacytoid DCs and thereby interfere with type-I IFN p

67 , increasing the number of blood myeloid and plasmacytoid DCs by 16- and 60-fold, respectively.

68 Plasmacytoid DCs formed the most discrete cluster, but e

69 th types of CRSwNP, but only myeloid DCs and plasmacytoid DCs from eosinophilic CRSwNP demonstrated a

70 At the functional level, both mDCs and plasmacytoid DCs generate T regulatory cells through an

71 Supernatant from plasmacytoid DCs harvested postinfection with BVDV or re

72 y expressed on activated primary myeloid and plasmacytoid DCs in human and mouse.

73 synthetic double-stranded RNA compared with plasmacytoid DCs in response to a single-stranded RNA eq

74 ations of monocytes, myeloid DCs (MDCs), and plasmacytoid DCs in the lung mucosa.

75 ytes, monocyte-derived DCs, macrophages, and plasmacytoid DCs in the skin, we addressed their dynamic

76 Aggregation of plasmacytoid DCs in the splenic perifollicular region, f

77 On the other hand, recognition of viruses by plasmacytoid DCs inhibits IL-1beta and IL-23 release, in

78 spond more effectively to TLR ligation, with plasmacytoid DCs making more IFN-alpha and both CD8alpha

79 FN production by DCs that lack TLR3, such as plasmacytoid DCs or CD8(-) DCs.

80 Plasmacytoid DCs produced IFN-alpha, whereas natural kil

81 ha(+) conventional dendritic cells (DCs) and plasmacytoid DCs upon TLR-mediated activation and detect

82 Plasmacytoid DCs were not detectable during the whole co

83 d DCs to acute IM plasma resulted in loss of plasmacytoid DCs, and further studies with individual cy

84 onal dendritic cells (DCs) and CD11c1/CD1231 plasmacytoid DCs, and striking granulocytic hyperplasia.

85 Increased numbers of myeloid DCs, plasmacytoid DCs, and their activated subsets were found

86 (MOs), classical dendritic cells (DCs), and plasmacytoid DCs, but whether these possess the attribut

87 r CD8 CTLs, and skewing of DC subsets toward plasmacytoid DCs, coupled with greater CD4 T follicular

88 to bind ICs compared with other myeloid DCs, plasmacytoid DCs, or monocytes.

89 cal infiltrates composed of conventional and plasmacytoid DCs, with the subsequent induction of poten

90 ucing DCs compared with conventional DCs and plasmacytoid DCs.

91 ted an expansion of classical DCs (cDCs) and plasmacytoid DCs.

92 s, including a series of 45 cases of blastic plasmacytoid dendritic cell (BPDC) neoplasms and their p

93 We identified that transient plasmacytoid dendritic cell (pDC) depletion during prima

94 nscription factor is required for B cell and plasmacytoid dendritic cell (pDC) development, but its m

95 HIV-2 favored plasmacytoid dendritic cell (pDC) differentiation into c

96 The plasmacytoid dendritic cell (pDC) is vital to the coordi

97 expression of the monocyte marker, CD14; the plasmacytoid dendritic cell (pDC) marker, BDCA4, identif

98 Blastic plasmacytoid dendritic cell (PDC) neoplasm (BPDCN) is an

99 induced limited type I interferon (IFN) and plasmacytoid dendritic cell (pDC) responses.

100 ell contact, and induction was suppressed by plasmacytoid dendritic cell depletion.

101 eage specification and induced monocytic and plasmacytoid dendritic cell differentiation instead.

102 require specific TLR stimulation, T-cell and plasmacytoid dendritic cell help for distinct activation

103 IFN Regulatory Factor 5 (IRF5) in the human plasmacytoid dendritic cell line Gen2.2 prevented IFNbet

104 dent oncogenic regulatory network in blastic plasmacytoid dendritic cell neoplasm (BPDCN) and demonst

105 Blastic plasmacytoid dendritic cell neoplasm (BPDCN) is a rare m

106 Blastic plasmacytoid dendritic cell neoplasm (BPDCN) is an aggre

107 et al describe a novel treatment of blastic plasmacytoid dendritic cell neoplasm (BPDCN) using an en

108 study of treatment of patients with blastic plasmacytoid dendritic cell neoplasm (BPDCN), an aggress

109 s of CD4 and memory B lymphocytes, decreased plasmacytoid dendritic cell numbers, and increased repre

110 0.0001) and increased regulatory T-cell and plasmacytoid dendritic cell percentages.

111 enotype and gene expression revealed a novel plasmacytoid dendritic cell precursor preferentially mob

112 ed endothelial cells can promote myeloid and plasmacytoid dendritic cell transmigration across endoth

113 We characterized plasmacytoid dendritic cell, natural killer (NK), and T-

114 Thus, upon VSV infection, plasmacytoid dendritic cell-derived IFN-I primarily prot

115 ontrast, the absence of Ly49Q did not affect plasmacytoid dendritic cell-triggering receptor expresse

116 the developmental origin of conventional and plasmacytoid dendritic cells (cDCs and pDCs, respectivel

117 Plasmacytoid dendritic cells (DCs [pDCs]) develop from p

118 significant loss of circulating myeloid and plasmacytoid dendritic cells (DCs) during acute IM, a lo

119 that a highly enriched population of bovine plasmacytoid dendritic cells (DCs) produced IFN in respo

120 factors as well as an increased frequency of plasmacytoid dendritic cells (DCs) that corresponded wit

121 on of the gut-homing receptor alpha4beta7 on plasmacytoid dendritic cells (p < 0.01) and the magnitud

122 of CD8alpha(+)beta(+) and CD8alpha(+)beta(-) plasmacytoid dendritic cells (pDC) and increased recruit

123 Plasmacytoid dendritic cells (pDC) are key regulators of

124 Plasmacytoid dendritic cells (pDC) are specialized in se

125 Plasmacytoid dendritic cells (pDC) are the major produce

126 vesicular stomatitis virus (VSV) particles, plasmacytoid dendritic cells (pDC) are triggered to moun

127 Plasmacytoid dendritic cells (pDC) are type I interferon

128 Plasmacytoid dendritic cells (pDC) constitute the body's

129 Here, we report that AGM plasmacytoid dendritic cells (pDC) express extremely low

130 Plasmacytoid dendritic cells (pDC) have been regarded as

131 edge, nothing is known about the survival of plasmacytoid dendritic cells (pDC) in such situation.

132 We show that plasmacytoid dendritic cells (pDC) of natural hosts disp

133 Following IAV infection, plasmacytoid dendritic cells (pDC) or CD8alpha(+) DC reg

134 Plasmacytoid dendritic cells (pDC) produce IFN-I in resp

135 se to the Toll-like receptor-9 agonist CpGA, plasmacytoid dendritic cells (pDC) produced type 1 IFNs,

136 Plasmacytoid dendritic cells (pDC) rapidly and massively

137 8(+) Tregs, total and IFN-alpha synthesizing plasmacytoid dendritic cells (pDC) relative to plasma vi

138 Enzymatic IDO1 activity, TGF-beta, and plasmacytoid dendritic cells (pDC) were neutralized by 1

139 7 triggers rapid sensing of nucleic acids by plasmacytoid dendritic cells (pDC).

140 ion against cancer using naturally occurring plasmacytoid dendritic cells (pDC).

141 "auto"-regulatory feedback mechanism between plasmacytoid dendritic cells (pDCs) and Breg cells.

142 dendritic cells (DCs) can be distinguished, plasmacytoid dendritic cells (pDCs) and CD16(+), CD1c(+)

143 In this study we determined whether plasmacytoid dendritic cells (pDCs) and CD8 T cells from

144 We show that chronic activation of plasmacytoid dendritic cells (pDCs) and elevated type-I

145 hypothesized that cell-cell contact between plasmacytoid dendritic cells (pDCs) and HCV-infected cel

146 The numbers of plasmacytoid dendritic cells (pDCs) and naive T cells (T

147 1 infection induces persistent activation of plasmacytoid dendritic cells (pDCs) and production of ty

148 Plasmacytoid dendritic cells (pDCs) and type I IFN drive

149 Plasmacytoid dendritic cells (pDCs) are a dendritic cell

150 During microbial infections, plasmacytoid dendritic cells (pDCs) are a main source of

151 Plasmacytoid dendritic cells (pDCs) are considered to be

152 Plasmacytoid dendritic cells (pDCs) are important early

153 Plasmacytoid dendritic cells (pDCs) are important in ant

154 In vitro evidence suggests that plasmacytoid dendritic cells (pDCs) are intimately invol

155 Plasmacytoid dendritic cells (pDCs) are key components o

156 Plasmacytoid dendritic cells (pDCs) are key players in t

157 Plasmacytoid dendritic cells (pDCs) are major type-I int

158 Plasmacytoid dendritic cells (pDCs) are primary producer

159 Airway myeloid dendritic cells (mDCs) and plasmacytoid dendritic cells (pDCs) are professional ant

160 Plasmacytoid dendritic cells (pDCs) are professional typ

161 In chronic diseases, such as HIV infection, plasmacytoid dendritic cells (pDCs) are rendered dysfunc

162 Plasmacytoid dendritic cells (pDCs) are the major produc

163 Plasmacytoid dendritic cells (pDCs) are the major source

164 Plasmacytoid dendritic cells (pDCs) are the major type I

165 During infections and inflammation, plasmacytoid dendritic cells (pDCs) are the most potent

166 Plasmacytoid dendritic cells (pDCs) are the most powerfu

167 Plasmacytoid dendritic cells (pDCs) are vital to antivir

168 of antigen-presenting cells, in particular, plasmacytoid dendritic cells (pDCs) around each MTZ form

169 Plasmacytoid dendritic cells (pDCs) bridge innate and ad

170 the production of type I interferon (IFN) in plasmacytoid dendritic cells (pDCs) by binding to endoso

171 Plasmacytoid dendritic cells (pDCs) efficiently produce

172 Plasmacytoid dendritic cells (pDCs) from females produce

173 LR7 and TLR9 agonists was greatly delayed in plasmacytoid dendritic cells (pDCs) from IRAK1[D359A] mi

174 The developmental origin of IFN-producing plasmacytoid dendritic cells (pDCs) has been uncertain.

175 Plasmacytoid dendritic cells (pDCs) have been proposed a

176 Plasmacytoid dendritic cells (pDCs) have long been impli

177 Plasmacytoid dendritic cells (pDCs) highly populate lung

178 bsets, we found that BCMA was transcribed in plasmacytoid dendritic cells (pDCs) in both blood and ly

179 The potential contribution of plasmacytoid dendritic cells (pDCs) in the presentation

180 of metaflammation by recruiting circulating plasmacytoid dendritic cells (pDCs) into VAT through che

181 ryptococcus neoformans; however, the role of plasmacytoid dendritic cells (pDCs) is unknown.

182 cluding IFN-alpha, and sortilin depletion in plasmacytoid dendritic cells (pDCs) led to a reduction o

183 myeloid dendritic cells (MDDCs/mDCs), and by plasmacytoid dendritic cells (pDCs) of human and murine

184 Plasmacytoid dendritic cells (pDCs) play a crucial role

185 Plasmacytoid dendritic cells (pDCs) play a crucial role

186 Human plasmacytoid dendritic cells (pDCs) play a major role in

187 type I IFN response by a small percentage of plasmacytoid dendritic cells (pDCs) present in the monoc

188 Plasmacytoid dendritic cells (pDCs) produce large amount

189 Plasmacytoid dendritic cells (pDCs) produced FasL in res

190 Plasmacytoid dendritic cells (pDCs) rapidly produce larg

191 g protective antiviral immune responses, and plasmacytoid dendritic cells (pDCs) represent a major so

192 ell TLR7 expression or temporal depletion of plasmacytoid dendritic cells (pDCs) slow progression; ho

193 stem due to unceasing IFN-alpha release from plasmacytoid dendritic cells (pDCs) stimulated by nuclei

194 Ab response against HIV-1 p24 that activates plasmacytoid dendritic cells (pDCs) through FcgammaRIIa

195 terized the immune response of monocytes and plasmacytoid dendritic cells (pDCs) to influenza A virus

196 ntaining nucleoprotein autoantigens activate plasmacytoid dendritic cells (PDCs) to produce type I in

197 RNA from human Huh-7 hepatoma cells to human plasmacytoid dendritic cells (pDCs) triggers pDC alpha/b

198 gp96 preferentially engages conventional and plasmacytoid dendritic cells (pDCs) under low and high d

199 denitis tissues but localization in CD123(+) plasmacytoid dendritic cells (pDCs) was found only in ly

200 Plasmacytoid dendritic cells (pDCs) were considered to b

201 Plasmacytoid dendritic cells (pDCs) were identified as t

202 Plasmacytoid dendritic cells (pDCs) were isolated from w

203 Aberrant activation of plasmacytoid dendritic cells (pDCs) with excessive produ

204 Plasmacytoid dendritic cells (pDCs), a primary source of

205 ic for double-stranded DNA (dsDNA) activated plasmacytoid dendritic cells (pDCs), a type of cell of t

206 BM demonstrated increased CD9(-)Siglec H(hi) plasmacytoid dendritic cells (pDCs), and depletion of pD

207 N-alpha production was primarily mediated by plasmacytoid dendritic cells (pDCs), and was significant

208 Plasmacytoid dendritic cells (pDCs), B cells and NK cell

209 both conventional dendritic cells (cDCs) and plasmacytoid dendritic cells (pDCs), but whereas cDCs pr

210 HIV-1-dependent reduction of ILC3s required plasmacytoid dendritic cells (pDCs), IFN-I, and the CD95

211 , particularly type I interferons (IFNs) and plasmacytoid dendritic cells (pDCs), in the pathogenesis

212 Plasmacytoid dendritic cells (pDCs), prominent cells of

213 This was associated with an influx of plasmacytoid dendritic cells (pDCs), regulatory T cells,

214 ice enhances Fas ligand (FasL) expression on plasmacytoid dendritic cells (pDCs), resulting in apopto

215 oduction upon incubation in vitro with human plasmacytoid dendritic cells (pDCs), whereas lentivirus

216 2 was sufficient for recognition of TgPRF by plasmacytoid dendritic cells (pDCs), whereas TLR11 and T

217 ritical virus-induced IFN-alpha responses of plasmacytoid dendritic cells (pDCs), which can be defici

218 This occurs via selective expansion of plasmacytoid dendritic cells (pDCs), which further augme

219 responses; therefore, we questioned whether plasmacytoid dendritic cells (pDCs), which produce IFN w

220 ompanied by increases in CD80(+) and CD86(+) plasmacytoid dendritic cells (pDCs).

221 A major source of IFN-alphabeta is plasmacytoid dendritic cells (pDCs).

222 miR-126 had high and specific expression by plasmacytoid dendritic cells (pDCs).

223 T cells (CD8+CD40Ig Tregs) interacting with plasmacytoid dendritic cells (pDCs).

224 face molecule selectively expressed by human plasmacytoid dendritic cells (pDCs).

225 ive immunity and are massively produced from plasmacytoid dendritic cells (pDCs).

226 Type I IFN is mainly produced by plasmacytoid dendritic cells (pDCs).

227 able hematologic malignancy originating from plasmacytoid dendritic cells (pDCs).

228 S-1/MAVS signaling and induced type I IFN in plasmacytoid dendritic cells and macrophages, with the l

229 neutrophils promoted cleavage of FcgRIIA on plasmacytoid dendritic cells and monocytes, resulting in

230 ces of type I IFNs during IOE infection were plasmacytoid dendritic cells and monocytes.

231 by inflammatory monocytes, dendritic cells, plasmacytoid dendritic cells and NK cells in all tissues

232 ion by monocytes required TLR7 activation of plasmacytoid dendritic cells and secretion of type I IFN

233 extracellular trap (NET) formation activates plasmacytoid dendritic cells and serves as a source of a

234 ne caused by TLR7 induction of type I IFN by plasmacytoid dendritic cells and subsequent IFN stimulat

235 Upon adoptive transfer of wild-type plasmacytoid dendritic cells and subsequent VSV infectio

236 03 treatment led to pronounced activation of plasmacytoid dendritic cells and substantial increases i

237 Because Tregs and plasmacytoid dendritic cells are known to modulate T-eff

238 We demonstrated that myeloid and plasmacytoid dendritic cells are recruited during the es

239 ent type I interferons (IFN-alpha/beta) from plasmacytoid dendritic cells as well as the production o

240 1 receptor-associated kinase 1 expression in plasmacytoid dendritic cells both in vivo and in vitro,

241 We isolated plasmacytoid dendritic cells from healthy persons and fr

242 Plasmacytoid dendritic cells have been implicated in the

243 cted a higher frequency and proliferation of plasmacytoid dendritic cells in BAL fluid at 3 days post

244 assical dendritic cells and CD11c+ low)B220+ plasmacytoid dendritic cells in both the heart and splee

245 n, and increased classic dendritic cells and plasmacytoid dendritic cells in peripheral blood and bon

246 to induce interferon-alpha in primary human plasmacytoid dendritic cells in response to hepatitis C

247 CXCL4 is the predominant protein secreted by plasmacytoid dendritic cells in systemic sclerosis, both

248 w that type I interferons (IFNs) produced by plasmacytoid dendritic cells inhibit the clearance of ap

249 nfluenza virus, the patient's leukocytes and plasmacytoid dendritic cells produced very little type I

250 It is known that plasmacytoid dendritic cells sense herpesvirus DNA in en

251 that rCl-13(GPC/VGKS) infected fewer splenic plasmacytoid dendritic cells than rCl-13, yet the two vi

252 gressive hematologic malignancy derived from plasmacytoid dendritic cells that typically involves the

253 munity, in which the differential ability of plasmacytoid dendritic cells to produce interferon alpha

254 cells, monocyte-derived dendritic cells, and plasmacytoid dendritic cells to vIL-10 suppresses multip

255 gered the production of type I interferon by plasmacytoid dendritic cells via activation of Toll-like

256 term IDO expression in both conventional and plasmacytoid dendritic cells via autocrine or paracrine

257 une lupus patients can stimulate B cells and plasmacytoid dendritic cells via Toll-like receptors 7 a

258 TLR7.1 plasmacytoid dendritic cells were cell-intrinsically act

259 T cells, B cells, type I IFN, IFN-gamma, and plasmacytoid dendritic cells were contributed to efficie

260 cells (MDDCs), myeloid dendritic cells, and plasmacytoid dendritic cells were examined.

261 ses, frequencies of tolerogenic CD103(+) and plasmacytoid dendritic cells were increased.

262 In addition, infiltrated plasmacytoid dendritic cells were the primary IL-10-secr

263 primary human epithelial cells, B cells, and plasmacytoid dendritic cells with dsDNA viruses induces

264 Depletion of plasmacytoid dendritic cells, a major cellular source of

265 mmune cells such as myeloid dendritic cells, plasmacytoid dendritic cells, and B cells in vitro.

266 al infections, especially by memory T cells, plasmacytoid dendritic cells, and CD56(bright) NK cells.

267 expose immunostimulatory molecules, activate plasmacytoid dendritic cells, and may participate in org

268 mmune factors, such as natural killer cells, plasmacytoid dendritic cells, and the expression pattern

269 onventional, monocyte-derived, lymphoid, and plasmacytoid dendritic cells, as well as activated T-cel

270 Plasmacytoid dendritic cells, B lymphocytes, and eosinop

271 We reveal that some cells, including plasmacytoid dendritic cells, can express both CCR2 and

272 Activation of plasmacytoid dendritic cells, modulation of B-cell respo

273 e, memory and activated subsets, myeloid and plasmacytoid dendritic cells, monocytes, B lymphocytes,

274 and IFN-alpha production from monocytes and plasmacytoid dendritic cells, respectively, retaining th

275 n of pro-inflammatory immune cells including plasmacytoid dendritic cells, T helper cells and plasma

276 ment were also related to the proportions of plasmacytoid dendritic cells, to distinct expression pat

277 e, Ly49Q, the single known MHC-I receptor on plasmacytoid dendritic cells, was shown to bind H-2D(b)

278 ted Treg cell boost involved TNF, OX40L, and plasmacytoid dendritic cells, whereas in a condition of

279 Moreover, CCL22 induces an influx of plasmacytoid dendritic cells, which correlates with high

280 interplay of Tregs, invariant NKT cells, and plasmacytoid dendritic cells, which results in suppressi

281 ed CLEC4C or CD303) is uniquely expressed on plasmacytoid dendritic cells.

282 s again dependent on type I IFNs produced by plasmacytoid dendritic cells.

283 monocytes, conventional dendritic cells, and plasmacytoid dendritic cells.

284 s, with lower expression on conventional and plasmacytoid dendritic cells.

285 nocytes of substances inhibitory or toxic to plasmacytoid dendritic cells.

286 of endothelial and myeloid cells, as well as plasmacytoid dendritic cells.

287 y in mature B cells, T-cell progenitors, and plasmacytoid dendritic cells.

288 Ly49Q functions as a pan-MHC-Ia receptor on plasmacytoid dendritic cells.

289 , in promoting IFN-alpha production by human plasmacytoid dendritic cells.

290 s TLR7 can stimulate them in the presence of plasmacytoid dendritic cells.

291 nduction of type 1 interferon responses from plasmacytoid dendritic cells.

292 e expression, IFNalpha levels, and activated plasmacytoid dendritic cells; 2) higher proinflammatory

293 ways induce IFN-I production in CMV-infected plasmacytoid dendritic cells; however, the initial burst

294 Cs were CD11c(+)B220(+)PDCA-1(+), resembling plasmacytoid dentritic cells (pDCs) of naive mice.

295 Bcl6 and upregulation of genes marking early plasmacytoid differentiation, notably IRF4 and BLIMP1.

296 pure squamous, adenocarcinoma, sarcomatoid, plasmacytoid, or micropapillary disease.

297 Plasmacytoid (p) dendritic cells (DC) are a specialized

298 ral killer (NK) cells and myeloid (mDCs) and plasmacytoid (pDCs) dendritic cells on control of virus

299 conventional (CD11c(high)CD103(high)) DC and plasmacytoid (plasmacytoid DC Ag-1(high)B220(+)CD11c(int

300 breast tumor cells induced the activation of plasmacytoid predendritic cells (pDC), a cell type criti