ライフサイエンスコーパス: plasmacytoid dendritic cell

1 monocytes, conventional dendritic cells, and plasmacytoid dendritic cells.

2 s, with lower expression on conventional and plasmacytoid dendritic cells.

3 nocytes of substances inhibitory or toxic to plasmacytoid dendritic cells.

4 of endothelial and myeloid cells, as well as plasmacytoid dendritic cells.

5 y in mature B cells, T-cell progenitors, and plasmacytoid dendritic cells.

6 Ly49Q functions as a pan-MHC-Ia receptor on plasmacytoid dendritic cells.

7 , in promoting IFN-alpha production by human plasmacytoid dendritic cells.

8 s TLR7 can stimulate them in the presence of plasmacytoid dendritic cells.

9 ith the growing arsenal of markers for human plasmacytoid dendritic cells.

10 ived dendritic cells, or in blood CD1c(+) or plasmacytoid dendritic cells.

11 mice showed reduced type I IFN production by plasmacytoid dendritic cells.

12 that BST2 inhibits type I IFN production by plasmacytoid dendritic cells.

13 R7 ligation is a feature shared by mammalian plasmacytoid dendritic cells.

14 is controlled by the type I IFN secreted by plasmacytoid dendritic cells.

15 pressor cells and putative immune inhibitory plasmacytoid dendritic cells.

16 ling and was not enhanced by the presence of plasmacytoid dendritic cells.

17 odulate inflammatory responses by activating plasmacytoid dendritic cells.

18 lear cells in a manner dependent on TLR7 and plasmacytoid dendritic cells.

19 vated T lymphocytes or on splenic myeloid or plasmacytoid dendritic cells.

20 ells and to stimulate IFN-alpha synthesis by plasmacytoid dendritic cells.

21 nduction of type 1 interferon responses from plasmacytoid dendritic cells.

22 s again dependent on type I IFNs produced by plasmacytoid dendritic cells.

23 ed CLEC4C or CD303) is uniquely expressed on plasmacytoid dendritic cells.

24 e expression, IFNalpha levels, and activated plasmacytoid dendritic cells; 2) higher proinflammatory

25 Depletion of plasmacytoid dendritic cells, a major cellular source of

26 of DOCK2, but not IRF-5, rescued defects in plasmacytoid dendritic cell and B-cell development, and

27 cking the mutation in Dock2 exhibited normal plasmacytoid dendritic cell and B-cell development, larg

28 Irregular cytokine production in plasmacytoid dendritic cells and CD69 CD4 T cells after

29 S-1/MAVS signaling and induced type I IFN in plasmacytoid dendritic cells and macrophages, with the l

30 neutrophils promoted cleavage of FcgRIIA on plasmacytoid dendritic cells and monocytes, resulting in

31 ces of type I IFNs during IOE infection were plasmacytoid dendritic cells and monocytes.

32 by inflammatory monocytes, dendritic cells, plasmacytoid dendritic cells and NK cells in all tissues

33 ion by monocytes required TLR7 activation of plasmacytoid dendritic cells and secretion of type I IFN

34 extracellular trap (NET) formation activates plasmacytoid dendritic cells and serves as a source of a

35 ne caused by TLR7 induction of type I IFN by plasmacytoid dendritic cells and subsequent IFN stimulat

36 Upon adoptive transfer of wild-type plasmacytoid dendritic cells and subsequent VSV infectio

37 03 treatment led to pronounced activation of plasmacytoid dendritic cells and substantial increases i

38 ous levels, including marginal zone B cells, plasmacytoid dendritic cells and T cell populations, and

39 mmune cells such as myeloid dendritic cells, plasmacytoid dendritic cells, and B cells in vitro.

40 script origin analysis identified monocytes, plasmacytoid dendritic cells, and B lymphocytes as prima

41 al infections, especially by memory T cells, plasmacytoid dendritic cells, and CD56(bright) NK cells.

42 modulate the activation of conventional and plasmacytoid dendritic cells, and contribute to the regu

43 including CD14(+) and/or CD16(+) monocytes, plasmacytoid dendritic cells, and in vitro derived monoc

44 expose immunostimulatory molecules, activate plasmacytoid dendritic cells, and may participate in org

45 mmune factors, such as natural killer cells, plasmacytoid dendritic cells, and the expression pattern

46 re a potent stimulus for IFNalpha release by plasmacytoid dendritic cells, and, as such, may play an

47 Because Tregs and plasmacytoid dendritic cells are known to modulate T-eff

48 We demonstrated that myeloid and plasmacytoid dendritic cells are recruited during the es

49 ent type I interferons (IFN-alpha/beta) from plasmacytoid dendritic cells as well as the production o

50 onventional, monocyte-derived, lymphoid, and plasmacytoid dendritic cells, as well as activated T-cel

51 endent mechanisms through TLR7 activation of plasmacytoid dendritic cells, B cells, and monocytes.

52 Plasmacytoid dendritic cells, B lymphocytes, and eosinop

53 1 receptor-associated kinase 1 expression in plasmacytoid dendritic cells both in vivo and in vitro,

54 s, including a series of 45 cases of blastic plasmacytoid dendritic cell (BPDC) neoplasms and their p

55 culating monocytoid dendritic cell (mDC) and plasmacytoid dendritic cell by monoclonal antibody stain

56 We reveal that some cells, including plasmacytoid dendritic cells, can express both CCR2 and

57 e observations demonstrate the importance of plasmacytoid dendritic cells, CD14(dim) monocytes, and C

58 tion status, the numbers of conventional and plasmacytoid dendritic cells (cDCs and pDCs) were higher

59 the developmental origin of conventional and plasmacytoid dendritic cells (cDCs and pDCs, respectivel

60 Plasmacytoid dendritic cells (DCs [pDCs]) develop from p

61 significant loss of circulating myeloid and plasmacytoid dendritic cells (DCs) during acute IM, a lo

62 that a highly enriched population of bovine plasmacytoid dendritic cells (DCs) produced IFN in respo

63 factors as well as an increased frequency of plasmacytoid dendritic cells (DCs) that corresponded wit

64 highly suppressive activity on mouse splenic plasmacytoid dendritic cells, demonstrable in vivo in a

65 ell contact, and induction was suppressed by plasmacytoid dendritic cell depletion.

66 Thus, upon VSV infection, plasmacytoid dendritic cell-derived IFN-I primarily prot

67 eage specification and induced monocytic and plasmacytoid dendritic cell differentiation instead.

68 We isolated plasmacytoid dendritic cells from healthy persons and fr

69 ess IFN-beta and CXCL10, and macrophages and plasmacytoid dendritic cells have a deficiency in activa

70 Plasmacytoid dendritic cells have been implicated in the

71 require specific TLR stimulation, T-cell and plasmacytoid dendritic cell help for distinct activation

72 ways induce IFN-I production in CMV-infected plasmacytoid dendritic cells; however, the initial burst

73 F-alpha secretion and suppressed CpG-induced plasmacytoid dendritic cell IFN-alpha gene expression.

74 receptor 9-mediated recognition observed in plasmacytoid dendritic cells, immune recognition of rAAV

75 cted a higher frequency and proliferation of plasmacytoid dendritic cells in BAL fluid at 3 days post

76 assical dendritic cells and CD11c+ low)B220+ plasmacytoid dendritic cells in both the heart and splee

77 numbers of tolerogenic CD103(+) and PDCA1(+) plasmacytoid dendritic cells in GITR(-/-) mice.

78 n, and increased classic dendritic cells and plasmacytoid dendritic cells in peripheral blood and bon

79 to induce interferon-alpha in primary human plasmacytoid dendritic cells in response to hepatitis C

80 CXCL4 is the predominant protein secreted by plasmacytoid dendritic cells in systemic sclerosis, both

81 w that type I interferons (IFNs) produced by plasmacytoid dendritic cells inhibit the clearance of ap

82 -dependent recruitment and transformation of plasmacytoid dendritic cells into killer cells; this occ

83 Whereas primary plasmacytoid dendritic cells isolated from a heterozygou

84 s transported into endosomal compartments of plasmacytoid dendritic cells, leading to activation of T

85 IFN Regulatory Factor 5 (IRF5) in the human plasmacytoid dendritic cell line Gen2.2 prevented IFNbet

86 ypes of conventional dendritic cells (cDCs), plasmacytoid dendritic cells, macrophages (MPhis), B lym

87 Additionally, plasmacytoid dendritic cells maintained higher levels of

88 Activation of plasmacytoid dendritic cells, modulation of B-cell respo

89 MCs from patients with XLA, freshly isolated plasmacytoid dendritic cells, monocytes, and monocytoid

90 ness-associated genes derived primarily from plasmacytoid dendritic cells, monocytes, and, to a lesse

91 e, memory and activated subsets, myeloid and plasmacytoid dendritic cells, monocytes, B lymphocytes,

92 Next, we showed that, in contrast to plasmacytoid dendritic cells, myeloid dendritic cells, c

93 We characterized plasmacytoid dendritic cell, natural killer (NK), and T-

94 dent oncogenic regulatory network in blastic plasmacytoid dendritic cell neoplasm (BPDCN) and demonst

95 Patients with blastic plasmacytoid dendritic cell neoplasm (BPDCN) have a poor

96 Blastic plasmacytoid dendritic cell neoplasm (BPDCN) is a rare m

97 Blastic plasmacytoid dendritic cell neoplasm (BPDCN) is an aggre

98 et al describe a novel treatment of blastic plasmacytoid dendritic cell neoplasm (BPDCN) using an en

99 study of treatment of patients with blastic plasmacytoid dendritic cell neoplasm (BPDCN), an aggress

100 s of CD4 and memory B lymphocytes, decreased plasmacytoid dendritic cell numbers, and increased repre

101 on of the gut-homing receptor alpha4beta7 on plasmacytoid dendritic cells (p < 0.01) and the magnitud

102 HIV modulates plasmacytoid dendritic cell (pDC) activation via Toll-li

103 We identified that transient plasmacytoid dendritic cell (pDC) depletion during prima

104 nscription factor is required for B cell and plasmacytoid dendritic cell (pDC) development, but its m

105 HIV-2 favored plasmacytoid dendritic cell (pDC) differentiation into c

106 The plasmacytoid dendritic cell (pDC) is vital to the coordi

107 expression of the monocyte marker, CD14; the plasmacytoid dendritic cell (pDC) marker, BDCA4, identif

108 Blastic plasmacytoid dendritic cell (PDC) neoplasm (BPDCN) is an

109 The human plasmacytoid dendritic cell (pDC) receptor BDCA2 forms a

110 induced limited type I interferon (IFN) and plasmacytoid dendritic cell (pDC) responses.

111 of CD8alpha(+)beta(+) and CD8alpha(+)beta(-) plasmacytoid dendritic cells (pDC) and increased recruit

112 Human plasmacytoid dendritic cells (pDC) are important modulat

113 Plasmacytoid dendritic cells (pDC) are key regulators of

114 Plasmacytoid dendritic cells (pDC) are potent APCs known

115 Plasmacytoid dendritic cells (pDC) are rare cells found

116 Plasmacytoid dendritic cells (pDC) are sentinels prepare

117 Plasmacytoid dendritic cells (pDC) are specialized in se

118 Plasmacytoid dendritic cells (pDC) are the major produce

119 Plasmacytoid dendritic cells (pDC) are the producers of

120 vesicular stomatitis virus (VSV) particles, plasmacytoid dendritic cells (pDC) are triggered to moun

121 Plasmacytoid dendritic cells (pDC) are type I interferon

122 Plasmacytoid dendritic cells (pDC) compose one of the ma

123 Plasmacytoid dendritic cells (pDC) constitute the body's

124 Here, we report that AGM plasmacytoid dendritic cells (pDC) express extremely low

125 Plasmacytoid dendritic cells (pDC) have been regarded as

126 Elevated levels of plasmacytoid dendritic cells (pDC) have been reported in

127 by activating toll-like receptors (TLRs) in plasmacytoid dendritic cells (pDC) in SLE.

128 edge, nothing is known about the survival of plasmacytoid dendritic cells (pDC) in such situation.

129 We show that plasmacytoid dendritic cells (pDC) of natural hosts disp

130 ns, focusing on effects of IFN-alphabeta and plasmacytoid dendritic cells (pDC) on Th2 immune respons

131 Following IAV infection, plasmacytoid dendritic cells (pDC) or CD8alpha(+) DC reg

132 Plasmacytoid dendritic cells (pDC) produce IFN-I in resp

133 se to the Toll-like receptor-9 agonist CpGA, plasmacytoid dendritic cells (pDC) produced type 1 IFNs,

134 Plasmacytoid dendritic cells (pDC) rapidly and massively

135 8(+) Tregs, total and IFN-alpha synthesizing plasmacytoid dendritic cells (pDC) relative to plasma vi

136 ing to monocytes, they preferentially engage plasmacytoid dendritic cells (pDC) so generating interfe

137 sed numbers of regulatory T cells (Treg) and plasmacytoid dendritic cells (pDC) that facilitate immun

138 Enzymatic IDO1 activity, TGF-beta, and plasmacytoid dendritic cells (pDC) were neutralized by 1

139 Unexpectedly, plasmacytoid dendritic cells (pDC) were not recruited to

140 We previously reported that plasmacytoid dendritic cells (pDC) within primary breast

141 7 triggers rapid sensing of nucleic acids by plasmacytoid dendritic cells (pDC).

142 ion against cancer using naturally occurring plasmacytoid dendritic cells (pDC).

143 on concomitantly trigger activation of human plasmacytoid dendritic cells (pDC).

144 ports suggest involvement of CD4 T cells and plasmacytoid dendritic cells (pDC).

145 nt on ICOS-L costimulation provided by tumor plasmacytoid dendritic cells (pDC).

146 "auto"-regulatory feedback mechanism between plasmacytoid dendritic cells (pDCs) and Breg cells.

147 dendritic cells (DCs) can be distinguished, plasmacytoid dendritic cells (pDCs) and CD16(+), CD1c(+)

148 In this study we determined whether plasmacytoid dendritic cells (pDCs) and CD8 T cells from

149 Steady-state development of plasmacytoid dendritic cells (pDCs) and conventional den

150 We show that chronic activation of plasmacytoid dendritic cells (pDCs) and elevated type-I

151 hypothesized that cell-cell contact between plasmacytoid dendritic cells (pDCs) and HCV-infected cel

152 factor alpha (TNF-alpha) production by human plasmacytoid dendritic cells (pDCs) and monocytes, and i

153 The numbers of plasmacytoid dendritic cells (pDCs) and naive T cells (T

154 1 infection induces persistent activation of plasmacytoid dendritic cells (pDCs) and production of ty

155 Plasmacytoid dendritic cells (pDCs) and type I IFN drive

156 Plasmacytoid dendritic cells (pDCs) are a dendritic cell

157 During microbial infections, plasmacytoid dendritic cells (pDCs) are a main source of

158 Plasmacytoid dendritic cells (pDCs) are characterized by

159 Plasmacytoid dendritic cells (pDCs) are considered poten

160 Plasmacytoid dendritic cells (pDCs) are considered to be

161 Multiple studies suggest that plasmacytoid dendritic cells (pDCs) are depleted and dys

162 Plasmacytoid dendritic cells (pDCs) are highly specializ

163 Plasmacytoid dendritic cells (pDCs) are important early

164 Plasmacytoid dendritic cells (pDCs) are important in ant

165 Plasmacytoid dendritic cells (pDCs) are innate immune ce

166 In vitro evidence suggests that plasmacytoid dendritic cells (pDCs) are intimately invol

167 Plasmacytoid dendritic cells (pDCs) are key components o

168 Plasmacytoid dendritic cells (pDCs) are key players in t

169 Plasmacytoid dendritic cells (pDCs) are major type-I int

170 Plasmacytoid dendritic cells (pDCs) are potent type I in

171 Plasmacytoid dendritic cells (pDCs) are primary producer

172 Airway myeloid dendritic cells (mDCs) and plasmacytoid dendritic cells (pDCs) are professional ant

173 Plasmacytoid dendritic cells (pDCs) are professional typ

174 In chronic diseases, such as HIV infection, plasmacytoid dendritic cells (pDCs) are rendered dysfunc

175 Plasmacytoid dendritic cells (pDCs) are specialized to p

176 Plasmacytoid dendritic cells (pDCs) are the major produc

177 Plasmacytoid dendritic cells (pDCs) are the major source

178 Plasmacytoid dendritic cells (pDCs) are the major type I

179 During infections and inflammation, plasmacytoid dendritic cells (pDCs) are the most potent

180 Plasmacytoid dendritic cells (pDCs) are the most powerfu

181 Plasmacytoid dendritic cells (pDCs) are vital to antivir

182 of antigen-presenting cells, in particular, plasmacytoid dendritic cells (pDCs) around each MTZ form

183 Plasmacytoid dendritic cells (pDCs) bridge innate and ad

184 the production of type I interferon (IFN) in plasmacytoid dendritic cells (pDCs) by binding to endoso

185 Plasmacytoid dendritic cells (pDCs) constitute a unique

186 rythematosus, type I IFN (IFN-I) produced by plasmacytoid dendritic cells (pDCs) critically promotes

187 r interferon alpha (IFN-alpha) production by plasmacytoid dendritic cells (pDCs) during human immunod

188 Plasmacytoid dendritic cells (pDCs) efficiently produce

189 Plasmacytoid dendritic cells (pDCs) from females produce

190 LR7 and TLR9 agonists was greatly delayed in plasmacytoid dendritic cells (pDCs) from IRAK1[D359A] mi

191 The developmental origin of IFN-producing plasmacytoid dendritic cells (pDCs) has been uncertain.

192 As plasmacytoid dendritic cells (pDCs) have a crucial role

193 Plasmacytoid dendritic cells (pDCs) have been proposed a

194 Plasmacytoid dendritic cells (pDCs) have long been impli

195 Plasmacytoid dendritic cells (pDCs) highly populate lung

196 bsets, we found that BCMA was transcribed in plasmacytoid dendritic cells (pDCs) in both blood and ly

197 The physiologic role played by plasmacytoid dendritic cells (pDCs) in the induction of

198 The potential contribution of plasmacytoid dendritic cells (pDCs) in the presentation

199 Plasmacytoid dendritic cells (pDCs) initiate antiviral i

200 of metaflammation by recruiting circulating plasmacytoid dendritic cells (pDCs) into VAT through che

201 ryptococcus neoformans; however, the role of plasmacytoid dendritic cells (pDCs) is unknown.

202 cluding IFN-alpha, and sortilin depletion in plasmacytoid dendritic cells (pDCs) led to a reduction o

203 myeloid dendritic cells (MDDCs/mDCs), and by plasmacytoid dendritic cells (pDCs) of human and murine

204 Plasmacytoid dendritic cells (pDCs) play a central role

205 Plasmacytoid dendritic cells (pDCs) play a crucial role

206 Plasmacytoid dendritic cells (pDCs) play a crucial role

207 Plasmacytoid dendritic cells (pDCs) play a crucial role

208 Human plasmacytoid dendritic cells (pDCs) play a major role in

209 type I IFN response by a small percentage of plasmacytoid dendritic cells (pDCs) present in the monoc

210 After HIV exposure, plasmacytoid dendritic cells (pDCs) produce high levels

211 While plasmacytoid dendritic cells (pDCs) produce IFN-I upon T

212 Plasmacytoid dendritic cells (pDCs) produce large amount

213 Plasmacytoid dendritic cells (pDCs) produce large amount

214 Plasmacytoid dendritic cells (pDCs) produced FasL in res

215 Plasmacytoid dendritic cells (pDCs) rapidly produce larg

216 Plasmacytoid dendritic cells (pDCs) rapidly produce type

217 Human plasmacytoid dendritic cells (pDCs) represent a highly s

218 g protective antiviral immune responses, and plasmacytoid dendritic cells (pDCs) represent a major so

219 Plasmacytoid dendritic cells (pDCs) selectively express

220 ell TLR7 expression or temporal depletion of plasmacytoid dendritic cells (pDCs) slow progression; ho

221 Plasmacytoid dendritic cells (pDCs) specialize in the se

222 stem due to unceasing IFN-alpha release from plasmacytoid dendritic cells (pDCs) stimulated by nuclei

223 Ab response against HIV-1 p24 that activates plasmacytoid dendritic cells (pDCs) through FcgammaRIIa

224 terized the immune response of monocytes and plasmacytoid dendritic cells (pDCs) to influenza A virus

225 ntaining nucleoprotein autoantigens activate plasmacytoid dendritic cells (PDCs) to produce type I in

226 ion of lymphocytes in the CNS, but access of plasmacytoid dendritic cells (pDCs) to the CNS was unimp

227 RNA from human Huh-7 hepatoma cells to human plasmacytoid dendritic cells (pDCs) triggers pDC alpha/b

228 gp96 preferentially engages conventional and plasmacytoid dendritic cells (pDCs) under low and high d

229 denitis tissues but localization in CD123(+) plasmacytoid dendritic cells (pDCs) was found only in ly

230 Plasmacytoid dendritic cells (pDCs) were considered to b

231 Plasmacytoid dendritic cells (pDCs) were identified as t

232 Plasmacytoid dendritic cells (pDCs) were isolated from w

233 Aberrant activation of plasmacytoid dendritic cells (pDCs) with excessive produ

234 Plasmacytoid dendritic cells (pDCs), a primary source of

235 ic for double-stranded DNA (dsDNA) activated plasmacytoid dendritic cells (pDCs), a type of cell of t

236 BM demonstrated increased CD9(-)Siglec H(hi) plasmacytoid dendritic cells (pDCs), and depletion of pD

237 ilities triggered by activated APCs, such as plasmacytoid dendritic cells (pDCs), and leading to the

238 specific FOXP3(+) regulatory T (Treg) cells, plasmacytoid dendritic cells (pDCs), and myeloid dendrit

239 N-alpha production was primarily mediated by plasmacytoid dendritic cells (pDCs), and was significant

240 Plasmacytoid dendritic cells (pDCs), B cells and NK cell

241 both conventional dendritic cells (cDCs) and plasmacytoid dendritic cells (pDCs), but whereas cDCs pr

242 HIV-1-dependent reduction of ILC3s required plasmacytoid dendritic cells (pDCs), IFN-I, and the CD95

243 , particularly type I interferons (IFNs) and plasmacytoid dendritic cells (pDCs), in the pathogenesis

244 ts by treatment with IFN-alpha suggests that plasmacytoid dendritic cells (pDCs), major producers of

245 en, and surface expression of FcepsilonRI on plasmacytoid dendritic cells (pDCs), myeloid dendritic c

246 Plasmacytoid dendritic cells (pDCs), prominent cells of

247 This was associated with an influx of plasmacytoid dendritic cells (pDCs), regulatory T cells,

248 ice enhances Fas ligand (FasL) expression on plasmacytoid dendritic cells (pDCs), resulting in apopto

249 oduction upon incubation in vitro with human plasmacytoid dendritic cells (pDCs), whereas lentivirus

250 2 was sufficient for recognition of TgPRF by plasmacytoid dendritic cells (pDCs), whereas TLR11 and T

251 ritical virus-induced IFN-alpha responses of plasmacytoid dendritic cells (pDCs), which can be defici

252 This occurs via selective expansion of plasmacytoid dendritic cells (pDCs), which further augme

253 responses; therefore, we questioned whether plasmacytoid dendritic cells (pDCs), which produce IFN w

254 A major source of IFN-alphabeta is plasmacytoid dendritic cells (pDCs).

255 miR-126 had high and specific expression by plasmacytoid dendritic cells (pDCs).

256 T cells (CD8+CD40Ig Tregs) interacting with plasmacytoid dendritic cells (pDCs).

257 face molecule selectively expressed by human plasmacytoid dendritic cells (pDCs).

258 (dsDNA) viruses in the endosome to stimulate plasmacytoid dendritic cells (pDCs).

259 reased the steady state number of intestinal plasmacytoid dendritic cells (pDCs).

260 secretion of type I interferons (IFNs) from plasmacytoid dendritic cells (pDCs).

261 -1 limits differentiation of precursors into plasmacytoid dendritic cells (pDCs).

262 aracterized role in regulating activation of plasmacytoid dendritic cells (pDCs).

263 nsatory interferon response in nonpermissive plasmacytoid dendritic cells (pDCs).

264 a virus to elicit an antiviral response from plasmacytoid dendritic cells (pDCs).

265 ive immunity and are massively produced from plasmacytoid dendritic cells (pDCs).

266 Type I IFN is mainly produced by plasmacytoid dendritic cells (pDCs).

267 able hematologic malignancy originating from plasmacytoid dendritic cells (pDCs).

268 ompanied by increases in CD80(+) and CD86(+) plasmacytoid dendritic cells (pDCs).

269 0.0001) and increased regulatory T-cell and plasmacytoid dendritic cell percentages.

270 viously that donor bone marrow precursors of plasmacytoid dendritic cells (pre-pDCs) can activate don

271 enotype and gene expression revealed a novel plasmacytoid dendritic cell precursor preferentially mob

272 TLR-7/8-activated plasmacytoid dendritic cells produced normal amounts of

273 nfluenza virus, the patient's leukocytes and plasmacytoid dendritic cells produced very little type I

274 and IFN-alpha production from monocytes and plasmacytoid dendritic cells, respectively, retaining th

275 nd type I interferons from human B cells and plasmacytoid dendritic cells, respectively.

276 It is known that plasmacytoid dendritic cells sense herpesvirus DNA in en

277 nfection decreased the number of circulating plasmacytoid dendritic cells, suppressed T cell activati

278 n of pro-inflammatory immune cells including plasmacytoid dendritic cells, T helper cells and plasma

279 that rCl-13(GPC/VGKS) infected fewer splenic plasmacytoid dendritic cells than rCl-13, yet the two vi

280 gressive hematologic malignancy derived from plasmacytoid dendritic cells that typically involves the

281 munity, in which the differential ability of plasmacytoid dendritic cells to produce interferon alpha

282 cells, monocyte-derived dendritic cells, and plasmacytoid dendritic cells to vIL-10 suppresses multip

283 ment were also related to the proportions of plasmacytoid dendritic cells, to distinct expression pat

284 ed endothelial cells can promote myeloid and plasmacytoid dendritic cell transmigration across endoth

285 ontrast, the absence of Ly49Q did not affect plasmacytoid dendritic cell-triggering receptor expresse

286 human and murine models between neutrophils, plasmacytoid dendritic cells, type I IFNs, and endotheli

287 gered the production of type I interferon by plasmacytoid dendritic cells via activation of Toll-like

288 term IDO expression in both conventional and plasmacytoid dendritic cells via autocrine or paracrine

289 une lupus patients can stimulate B cells and plasmacytoid dendritic cells via Toll-like receptors 7 a

290 e, Ly49Q, the single known MHC-I receptor on plasmacytoid dendritic cells, was shown to bind H-2D(b)

291 uction of B cells, natural killer cells, and plasmacytoid dendritic cells were blocked.

292 TLR7.1 plasmacytoid dendritic cells were cell-intrinsically act

293 T cells, B cells, type I IFN, IFN-gamma, and plasmacytoid dendritic cells were contributed to efficie

294 cells (MDDCs), myeloid dendritic cells, and plasmacytoid dendritic cells were examined.

295 ses, frequencies of tolerogenic CD103(+) and plasmacytoid dendritic cells were increased.

296 In addition, infiltrated plasmacytoid dendritic cells were the primary IL-10-secr

297 ted Treg cell boost involved TNF, OX40L, and plasmacytoid dendritic cells, whereas in a condition of

298 Moreover, CCL22 induces an influx of plasmacytoid dendritic cells, which correlates with high

299 interplay of Tregs, invariant NKT cells, and plasmacytoid dendritic cells, which results in suppressi

300 primary human epithelial cells, B cells, and plasmacytoid dendritic cells with dsDNA viruses induces