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1 and fission of caveolae with the endothelial plasmalemma.
2 cally regulated transport of ions across the plasmalemma.
3 dria, the Golgi apparatus, and the dendritic plasmalemma.
4 und vesicles often in close proximity to the plasmalemma.
5  the site of known efflux across the lateral plasmalemma.
6 ociated with microfilaments distant from the plasmalemma.
7 ters adjacent to endoplasmic reticula and/or plasmalemma.
8 ffiliated with endoplasmic reticulum and the plasmalemma.
9 intracellular vesicles into the bone apposed plasmalemma.
10 protein kinase C-epsilon are targeted to the plasmalemma.
11 emmal holes or a complete transection of the plasmalemma.
12 he cell, with no immunoreactivity evident in plasmalemma.
13 m large clusters anchored to the presynaptic plasmalemma.
14  as a result of selective retrieval from the plasmalemma.
15 ers the fusion of synaptic vesicles with the plasmalemma.
16 as well as in astrocytes and skeletal muscle plasmalemma.
17 ates the Na(+) and K(+) gradients across the plasmalemma.
18 ween the forming basal membrane and the yolk plasmalemma.
19 most exclusively to the inner leaflet of the plasmalemma.
20  ROS production, and a partially depolarized plasmalemma.
21  and the displacement of active Rac from the plasmalemma.
22 uated: the core axial array of disks and the plasmalemma.
23 r Ca2+ and create transient nanopores in the plasmalemma.
24 mpartments that influences expression in the plasmalemma.
25 vealing caveolin-1 and Slo1 proximity at the plasmalemma.
26  occurs when synaptic vesicles fuse with the plasmalemma.
27 f VEGFR2 in the plus-end direction to the EC plasmalemma.
28 ng localization to the cytosolic side of the plasmalemma.
29 d with caveolae after their fission from the plasmalemma.
30 abeled axons were localized primarily to the plasmalemma.
31 orylation of Kir1.1 drives expression on the plasmalemma.
32 y was expressed as a membrane protein in the plasmalemma.
33 n link cytoskeletal F-actin filaments to the plasmalemma.
34     There was no inhibition of efflux at the plasmalemma.
35  ionophore that promotes Ca(2+) entry at the plasmalemma.
36 ith (1) dendritic spines (both cytoplasm and plasmalemma), (2) spine apparati located within 0.1 micr
37 P-RhoA to anti-PECAM/NC binding sites at the plasmalemma, actin polymerization into phalloidin-positi
38 retrieval of the vesicular membrane from the plasmalemma after exocytosis.
39 f secretory vesicles from the cytosol to the plasmalemma, an effect not requiring cytokine priming.
40 he fusion of the vesicular membrane with the plasmalemma and by the successive elimination of the lay
41 ized in different domains of the endothelial plasmalemma and differentially distributed throughout th
42  a metastable state and transfer between the plasmalemma and disk region occurs, which is followed by
43 her atrial myocyte ANP-RB colocalizes at the plasmalemma and elsewhere in the cell with the muscle-sp
44 ted budding takes place in parallel from the plasmalemma and from these internal membranes.
45  fusion of channel-bearing vesicles with the plasmalemma and myosin light chain kinase to regulate ce
46 m the COS's distributed bilayer phase, i.e., plasmalemma and older lamellae (membrane recycling).
47  vesicles, whereas D2-LI was found along the plasmalemma and over nearby small clear vesicles.
48 OCE via assembly of STIM1 and Orai1, and the plasmalemma and sarco/endoplasmic reticulum Ca(2+)-ATPas
49 hat provides a structural bridge between the plasmalemma and sarcoplasmic reticulum, is essential for
50 ed the delivery of Golgi-resident as well as plasmalemma and secreted proteins to their normal locati
51 wise required for trafficking of eNOS to the plasmalemma and subsequent activation.
52 ed from microdomains of high Ca(2+) near the plasmalemma and that this aequorin moved, either by move
53 tellite cells lying between the muscle fiber plasmalemma and the basement membrane.
54 at the transport of lignin precursors across plasmalemma and their sequestration into vacuoles are AT
55 confirmed to enhance water transport through plasmalemma and to cluster into orthogonal arrays of par
56  is the major permeating species across both plasmalemma and tonoplast of root cells under toxicity c
57 ence of net efflux of potassium salt at both plasmalemma and tonoplast.
58 d acridine orange (AO) were localized to the plasmalemma and vacuole, respectively.
59 ions examined, often delineated the neuronal plasmalemma, and labeled axons in white matter tracts of
60 tory steps proximal to granule fusion at the plasmalemma, and may facilitate protracted secretion thr
61 lly associated with TRPM2 in the endothelial plasmalemma, and this interaction functioned to suppress
62 enerally accepted model that vesicles at the plasmalemma are guided by cytoskeletal tracks to specifi
63 -alpha1S and GFP-alpha1C were present in the plasmalemma as small punctate foci along much of the lon
64 t there is a membrane network just below the plasmalemma at the cell borders that is connected at int
65 n-2, serine 23 and 36, reduces the number of plasmalemma-attached caveolae and increases the accumula
66           These findings (i) suggest a major plasmalemma-based mechanism of strength loss underlying
67 he phosphoinositide, which is present in the plasmalemma before engagement of the target particle, is
68 tated receptors that were transported to the plasmalemma bound f-Nle-Leu-Phe-Nle-Tyr-Lys-fluorescein
69 p junctions, were present on portions of the plasmalemma, bridging the cytoplasm of neurons and glia
70 th DOR-LI and MOR-LI were detected along the plasmalemma, but only DOR-LI was associated with large d
71 begins to accumulate as invaginations of the plasmalemma, but pinch-off is blocked.
72       Immunocytochemical localization of the plasmalemma Ca(2+)-H(+)-ATPase (PMCA pump) revealed inte
73 into cells with subsequent activation of the plasmalemma Ca(2+)-H(+)-ATPase.
74 nd may derive from a Ca2+ influx mediated by plasmalemma Ca2+ channels, and (b) cold up-regulation of
75 h Ca2+ release from intracellular stores and plasmalemma Ca2+ influx.
76 ls, eNOS in cardiac myocytes is localized to plasmalemma caveolae, specialized lipid microdomains tha
77                 Because eNOS is localized in plasmalemma caveolae, we examined if tyrosine phosphoryl
78 ween Kir6.2 and TMD0, but does not alter the plasmalemma channel density.
79                                              Plasmalemma clathrin-coated structures range from unitar
80 aptic vesicle docking and/or fusion with the plasmalemma correlate with the release of their membrane
81 leted primarily in the hemisphere facing the plasmalemma, creating a gradient of vesicles on its surf
82 volves fusion of secretory granules with the plasmalemma, culminating in the release of the digestive
83 1) protein-coupled receptors, located in the plasmalemma, cytoplasm, and nucleus.
84                     In the second stage, the plasmalemma density reaches a metastable state and trans
85                          In the first stage, plasmalemma diffusion is dominant.
86 ussed in terms of the exosite model, and the plasmalemma diffusion microkinetic hypothesis, for the c
87  inhibited by disruption of cholesterol-rich plasmalemma domains and deletion of PECAM-1 cytosolic ta
88 cission of clathrin-coated vesicles from the plasmalemma during endocytosis.
89 chitin deacetylase has been localized to the plasmalemma-endospore interface.
90 hosphorylation of Kir1.1a serine 44 controls plasmalemma expression.
91 oteins to their surface, thus protecting the plasmalemma from acid-induced hydrolysis.
92 idine receptor (DHPR) in the skeletal muscle plasmalemma functions as both voltage-gated Ca(2+) chann
93 t synaptic and extrasynaptic portions of the plasmalemma in dendrites and somata, many of which also
94 at the native protein is associated with the plasmalemma in intact cells.
95 P inhibitor, NIM811, significantly increases plasmalemma in normally growing cells.
96 calcium release and calcium entry across the plasmalemma in response to intracellular application of
97 d to the TGN by selective retrieval from the plasmalemma in response to signaling sequences in its en
98 erin is deposited immediately outside of the plasmalemma in the cell wall of certain tissues such as
99 ecruitment of Na+,K+-ATPase molecules to the plasmalemma, in a process mediated by protein kinase Cbe
100 ever, distributed uniformly over presynaptic plasmalemma; instead, fluorescence images show "hot spot
101 ocytochemistry confirmed a decrease in hSlo1 plasmalemma localization by myristic acid.
102  CB1 immunoparticles appeared at presynaptic plasmalemma, making asymmetric and symmetric synapses.
103 ell death, the vacuole either fuses with the plasmalemma membrane or disintegrates.
104 ependent mitochondrial Ca(2+) fluxes and the plasmalemma Na(+)/K(+) pump determines the magnitude of
105  targets, such as L-type Ca(2)+ channels and plasmalemma Na+/Ca(2)+ exchanger.
106 ts 3 Na(+) in exchange for 2 K(+) across the plasmalemma of animal cells.
107 4.2 and PPTX colocalize in the region of the plasmalemma of Chinese hamster ovary cells; however, bot
108 a fraction of cellular ANP colocalize at the plasmalemma of cultured atrial myocytes and of freshly d
109 e cytoplasm of dendritic shafts, and (4) the plasmalemma of dendritic shafts.
110        Caveolae open to the apical and basal plasmalemma of endothelial cells increased 2-4-fold with
111                   The ectoenzyme CD39 on the plasmalemma of endothelial cells metabolizes ADP to supp
112  fatty acid (FA) transport across the apical plasmalemma of enterocytes have remained largely unclear
113 G-linked receptor, is present throughout the plasmalemma of isolated cells; its activation also induc
114 drolase-1 (CD39), an enzyme expressed on the plasmalemma of leukocytes and endothelial cells, suppres
115  glycoprotein (MAG), located in the adaxonal plasmalemma of myelin-producing cells, is known to signa
116 rated that dysferlin is not expressed at the plasmalemma of myotubes but mostly localizes to the T-tu
117  for two extracellular K(+) ions through the plasmalemma of nearly all animal cells.
118 Ralpha-I was affiliated with the cytoplasmic plasmalemma of select interneurons and with endosomes of
119 erents converging on similar portions of the plasmalemma of target neurons.
120  within the subsynaptic membrane beneath the plasmalemma of the electrocytes, and talin and acetylcho
121 ated sprouting only when mislocalized to the plasmalemma of the rod cell body.
122 ated co-localization in the Golgi region and plasmalemma of transfected cells and native endothelial
123 analiculi that collected near or streamed to plasmalemma, often next to a synapse.
124     The fluorescence was associated with the plasmalemma only when the beta2 subunit was co-injected
125 asing Ca(2+) from an internal store near the plasmalemma, possibly from synaptic vesicles in the rele
126 in kinase c-src] are concentrated in the NB (plasmalemma proper-enriched) subfraction rather than in
127  from other membrane sources, especially the plasmalemma proper.
128 n (NB) enriched in vesicles derived from the plasmalemma proper.
129 ployed kinetics describing IP3-receptor, DTS-plasmalemma puncta formation, SOCE via assembly of STIM1
130 dosomal recycling of the uPA receptor to the plasmalemma, remained abnormally associated with PAI-1 i
131                    Lysosomal fusion with the plasmalemma results in acidification of the resorptive m
132 Caveolae are specialized microdomains of the plasmalemma rich in signaling molecules and supported by
133 e complexes and increased variability in the plasmalemma-sarcoplasmic reticulum distance.
134                     CAV1 is localized in the plasmalemma, secretory vesicles, Golgi, mitochondria, an
135  Blockade of the synthetase uncovers cryptic plasmalemma sites that bind 5-oxoETE with exquisite spec
136  in the electrophysiological function of the plasmalemma specifically impaired action potential devel
137 on the structure of junctions between SR and plasmalemma suggest that individual release sparks resul
138 tion of numerous ingrowths that increase the plasmalemma surface area for solute uptake.
139 av-1) in 293 cells keeps TFPI exposed on the plasmalemma surface, decreases the membrane lateral mobi
140 tral role of Ca(2+) and K(+) channels in the plasmalemma that regulate Ca(2+) influx and have identif
141 oclast vacuolar proton pump to the polarized plasmalemma, the H+-ATPase decorates microtubules in a m
142 tion of both proteins to raft domains of the plasmalemma, the physical interaction of eNOS with cav-1
143 ther, pre-existing vesicles, and/or with the plasmalemma to form an electrical and a physical barrier
144  the ability of the tibialis anterior muscle plasmalemma to generate and conduct action potentials.
145 and/or with intact or damaged regions of the plasmalemma to repair small (1-30 microm) plasmalemmal h
146 ated propagation of the Ca2+ signal from the plasmalemma to the nucleus.
147 bute to constitutive as well as pathological plasmalemma turnover in dependence on mitochondrial stre
148 ease is indirect and possibly related to the plasmalemma unitary Ca2+ current magnitude.
149          Inhibition of Na+ influx across the plasmalemma using lidocaine, low extracellular Na+, or t
150 ions, and IL-6, tight junction proteins, and plasmalemma vesicle protein (PLVAP) were detected by Wes
151    Previously, we found that the presence of plasmalemma vesicle-associated protein (PLVAP) in retina
152                                              Plasmalemma vesicle-associated protein (Plvap) is an end
153 rget of this antibody has been claimed to be plasmalemma vesicle-associated protein-1 (PV-1) and neur
154 he caveolin-1-positive, albumin-transporting plasmalemma vesicles.
155 d anti-gp60 antibody colocalized in the same plasmalemma vesicles.
156 cial fluorescent dye RH 414, which marks for plasmalemma vesicles.
157 though glucose transporter 1, claudin-3, and plasmalemma vesicular-associated protein have been ident
158 strated that SAGA-1 is anchored in the sperm plasmalemma via a GPI-lipid linkage.
159 anged on Western blot, but the amount in the plasmalemma was increased.
160 r of hemidesmosomes per microm of epithelial plasmalemma was subnormal.
161 to bind to GM1 ganglioside on the macrophage plasmalemma, we demonstrate that these glycosphingolipid
162  connections between their membranes and the plasmalemma were sometimes visible by serial sectioning.
163 strated preferential TRPC7 expression in the plasmalemma, whereas TRPC3 was distributed throughout th
164 as indicated by the marker LAMP2 near/at the plasmalemma), which can explain the advantage of junctio
165 sertion of H/K-ATPase into the parietal cell plasmalemma, while its cessation is associated with rein
166 ated either with select vesicles or with the plasmalemma within preterminal axons and axon terminals.

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