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1 Plasmin activity was blocked using alpha(2)-plasmin inhibitor.
2 plasmin(ogen) and is only a kinetically slow plasmin inhibitor.
3 lycan mimetics (NSGMs), as direct allosteric plasmin inhibitors.
4 up is a beneficial element for the design of plasmin inhibitors.
6 Covalent incorporation (cross-linking) of plasmin inhibitor alpha(2)-antiplasmin (alpha(2)-AP) int
7 d human dermal fibroblasts plus the specific plasmin inhibitor alpha(2)-antiplasmin or dermal fibrobl
8 d into the vitreous humor with KA, whereas a plasmin inhibitor, alpha-2-antiplasmin, failed to attenu
11 was fused to a sequence derived from alpha2-plasmin inhibitor (alpha2-PI1-8) that is a substrate for
13 Neuritogenesis is also impaired by alpha(2)-plasmin inhibitor, antibodies directed against t-PA and
15 ardiocytes with anti-uPAR or anti-uPA Abs or plasmin inhibitor aprotinin prior to coculturing with he
16 ith GAS were simultaneously treated with the plasmin inhibitor aprotinin, a significant reduction in
17 e of the thrombin inhibitor, hirudin, or the plasmin inhibitor, aprotinin, consistent with the interp
20 ed by either tPA-deficiency or infusion of a plasmin inhibitor, both of which also block neuronal deg
22 nd by microsomes was inhibited by bdellin, a plasmin inhibitor from the medicinal leech Hirudo medici
23 e macrophage surface is protected from serum plasmin inhibitors, interaction of AgLDL with macrophage
24 a2-antiplasmin (alpha2AP, also called alpha2-plasmin inhibitor) is the main physiological inhibitor o
25 antiplasmin (alpha2AP), also known as alpha2-plasmin inhibitor, is the major inhibitor of the proteol
26 iplasmin (alpha(2)AP), a major physiological plasmin inhibitor, or anti-annexin II IgG, blocked EMT b
27 asminogen participates in neuritogenesis, as plasmin inhibitors reduced both neurite outgrowth and ne
28 vage pathway in the setting of urokinase and plasmin inhibitors to promote clearance of the provision
32 of our recently described substrate-analogue plasmin inhibitors, which were cyclized between their P3
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