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1 describe the molecular characterization of a plasmodesmal-associated protein kinase (PAPK).
2                                              Plasmodesmal-associated protein kinases could play a cen
3 teins have a high affinity for the mesophyll plasmodesmal binding site(s).
4 ate movement of viral DNA across nuclear and plasmodesmal boundaries, respectively.
5                                   Either the plasmodesmal channels involved in SE/CC unloading are ex
6 vum L.) grains to evaluate the dimensions of plasmodesmal channels involved in sieve element/companio
7 show that regulating callose accumulation at plasmodesmal channels is a common strategy to alter plas
8  interaction between such proteins/RNPCs and plasmodesmal chaperones/receptors.
9 specially the way in which calcium regulates plasmodesmal closure.
10 ts with callose synthesis inhibitors suggest plasmodesmal connectivity as a potential mechanism for t
11 microchannels, and provide direct proof that plasmodesmal dilation is a prerequisite for the cell-to-
12                                              Plasmodesmal enriched cell fractions and the contents of
13 le of interacting with proteins present in a plasmodesmal-enriched cell wall fraction.
14 e performed on a tobacco (Nicotiana tabacum) plasmodesmal-enriched cell wall protein preparation usin
15                                              Plasmodesmal flux is regulated by a variety of environme
16 o lines previously reported to have impaired plasmodesmal function as well as in wild-type seedlings
17 pathway in conjunction with the regulator of plasmodesmal gating Plasmodesmata-located protein5.
18            A Cm-PP16 interaction partner, Nt-PLASMODESMAL GERMIN-LIKE PROTEIN1 (Nt-PDGLP1) was identi
19                                          Two plasmodesmal-localized beta-1,3 glucanases (PdBGs) were
20 lin, but not fungal chitin, is mediated by a plasmodesmal-localized Ca(2+) -binding protein Calmoduli
21 feedback circuit that regulates the level of plasmodesmal-localized callose in order to locally downr
22 ation of, and protein translocation through, plasmodesmal microchannels, and provide direct proof tha
23 act with a motif involved in the dilation of plasmodesmal microchannels.
24  allows these Hsc70 chaperones to engage the plasmodesmal non-cell-autonomous translocation machinery
25 n-accumulating regions of sxd1 leaves due to plasmodesmal occlusion at the bundle sheath-vascular par
26 xpression of GAT1 in mature leaves increased plasmodesmal permeability and led to a delay in senescen
27 ich different environmental stressors affect plasmodesmal permeability are not well understood.
28                                    Moreover, plasmodesmal permeability is strongly altered by applied
29 esmal channels is a common strategy to alter plasmodesmal permeability under both pathogen infection
30 e regulatory effect of MP phosphorylation on plasmodesmal permeability was host dependent, occurring
31 r exchange between plant cells determined by plasmodesmal permeability.
32 ion protein was located in the centre of the plasmodesmal pore, between paired callose platelets.
33             A 34-kD protein, isolated from a plasmodesmal preparation, exhibits calcium-independent k
34                   The mutations in genes for plasmodesmal proteins have provided valuable genetic too
35 opsis line (pdko3) mutated in genes encoding plasmodesmal proteins is defective in some, but not all,
36 ction of flowering may represent a change in plasmodesmal selectivity at this time or that a period o
37                                              Plasmodesmal size exclusion limit increased to greater t
38                This system likely involves a plasmodesmal switch that would prevent the dissipation o
39 els of calreticulin severely interfered with plasmodesmal targeting of TMV MP, which, instead, was re
40 t involves supracellular control achieved by plasmodesmal trafficking of informational molecules, her
41                 Callose deposition modulates plasmodesmal transport in vivo, but little is known abou
42                   As the threshold value for plasmodesmal transport of phloem sap proteins falls with
43 netic approach to identify mutants affecting plasmodesmal transport.

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