ライフサイエンスコーパス: plateau phase

1 , inflammation in control mice remained at a plateau phase.

2 on titre, followed by a distinct switch to a plateau phase.

3 s relative to the original template into PCR plateau phase.

4 owing a rapid burst phase followed by a slow plateau phase.

5 during the cardiac action potential waveform plateau phase.

6 burst phase and 1.0178 +/- 0.0003 during the plateau phase.

7 he burst phase and 1.0022 +/- 0.0003 for the plateau phase.

8 3-mo expansion phase followed by a long-term plateau phase.

9 entified performance deterioration after the plateau phase.

10 g two distinct time intervals separated by a plateau phase.

11 ells, but only for fractionated radiation in plateau phase.

12 of stimulation but not during the subsequent plateau phase.

13 ill present with reduced efficacy during the plateau phase.

14 hyperpolarizing for pulses during the early plateau phase.

15 nts' growth rates either slowed or reached a plateau phase.

16 cer cells growing either exponentially or in plateau phase.

17 ) and [Ca(2+)](i) remained higher during the plateau phase.

18 diabetic corneas lost the normal rising and plateau phases.

19 fibrils with characteristic lag, growth, and plateau phases.

20 respective of PrP(C) expression level, and a plateau (phase 2), which continues until clinical onset

21 resurgence of outward current terminates the plateau phase and is thus a key regulator of action pote

22 -furanosyl) uracil (FMAU) were determined in plateau-phase and exponentially growing cultures of 3 hu

23 of initial peak and the remaining 40% of the plateau phase, as administration of TEA in combination w

24 RTTs also displayed the initial increase and plateau phase, but the third region was virtually absent

25 ment to decrease the telomerase activity (in plateau phase cells of RKO, HeLa; and growing cells of R

26 a mitotic shake-off technique as well as in plateau-phase cells arrested by growing to confluency.

27 ould, likewise, induce mutations specific to plateau-phase cells, aprt mutations induced by amsacrine

28 phase cells, but were much less prevalent in plateau-phase cells.

29 of 74 aprt mutations induced by treatment of plateau phase Chinese hamster ovary CHO cells with the r

30 s induced by amsacrine in both log-phase and plateau-phase CHO cells were analyzed.

31 ) after treatment with 1 microM amsacrine in plateau phase, compared with a spontaneous frequency of

32 reased in all cell lines as cells moved from plateau-phase conditions to exponential growth.

33 TK(1) activities and S-phase fractions under plateau-phase conditions, consistent with a loss of norm

34 ter was controllable in both exponential and plateau phase cultures and support the plausibility of u

35 not be induced until 48 h after infection of plateau phase cultures but could still be induced 180 h

36 n daily fractions of 2.0 Gy were examined in plateau phase cultures of human tumor cells of varying i

37 Using plateau-phase cultures, we investigated the response of

38 ase from intracellular stores, followed by a plateau phase due to Ca2+ influx.

39 he timing of NCX current reversal toward the plateau phase earlier in the action potential.

40 ression profiling analyses were conducted on plateau phase human lung cancer (A549) cell cultures tre

41 h and hasten involution in proliferative and plateau phase IH.

42 currents with a pronounced nondesensitizing plateau phase in cells that co-expressed both subunits.

43 end of an exponential increase followed by a plateau phase in successive cycles.

44 ted a transient [Ca(2+)](i) signal without a plateau phase in the presence of extracellular Ca(2+) an

45 ch apoptosis rarely occurred, followed by a "plateau" phase in which cell loss by apoptosis was compe

46 e action potential shape during the peak and plateau phases is determined primarily by transient outw

47 initial peak observed within minutes, and a plateau phase lasting up to 48 h.

48 However, the plateau phase measurement of the Halimeter was 25% great

49 Data from 57 patients with plateau phase MM and a marrow PCLI of more than 1.0% wer

50 igh PCLI in patients with apparently stable, plateau phase MM is an adverse parameter that may predic

51 lly patients have been observed with stable, plateau phase MM with minimal numbers of residual light-

52 We report here that G1-phase arrest in plateau-phase monolayer cultures of both M1 and MR1 cell

53 t 5% of the log-phase mutants and 16% of the plateau-phase mutants were +1 frameshifts, and all but o

54 All 3 tracers concentrated in the plateau phase, nonproliferating cells at levels many-fol

55 The plateau phase of a human ventricular myocyte is analysed

56 ibrillation shocks and shocks applied at the plateau phase of a normal action potential produced by v

57 (EAD), or abnormal depolarization during the plateau phase of action potentials, is a hallmark of lon

58 ll single Ca(2+) channel currents during the plateau phase of broad action potentials raise local Ca(

59 nitial mobilization peak had dissipated, the plateau phase of calcium entry was unchanged (92 +/- 9 n

60 current (ICaL) is a major determinant of the plateau phase of cardiac action potential and has a crit

61 ectively, the rising phase and the prolonged plateau phase of cerebellar Purkinje cell (PC) action po

62 L cells in tissue culture in logarithmic and plateau phase of growth have been made.

63 ere in the exponential phase compared to the plateau phase of growth.

64 wild type, and the mutant never attained the plateau phase of infection in mouse lungs.

65 monstrate that the establishment of the high plateau phase of infection is dependent on non-antibody-

66 In populations of quiescent myocytes, the plateau phase of the [Ca2+]i signal evoked by 2-M-S-ATP

67 l number of LCCs gating in mode 2 during the plateau phase of the action potential (AP) can trigger E

68 ch induces additional Ca2+ influx during the plateau phase of the action potential, further slowing t

69 by reducing depolarizing current during the plateau phase of the action potential.

70 and a large increase in the duration of the plateau phase of the action potential.

71 The plateau phase of thermal hyperaemia was significantly at

72 ere consistent with a model wherein a normal plateau phase of variable length was followed by initiat

73 ally to the initial peak and modestly to the plateau phases of thermal hyperaemia.

74 Several studies noted a plateau phase or maturation in the surgical learning cur

75 responses to stretch: a rise phase (R) and a plateau phase (P) and an exponential phase (E).

76 To the degree that these factors diminished plateau phase pulmonary function, they may be important

77 progressively suppressed the voltage of the plateau phase (r=-0.90) with increasing Ito1 density.

78 The stress-induced growth inhibition of plateau-phase spheroids was accompanied, at the cellular

79 and neocarzinostatin were more mutagenic in plateau-phase than in log-phase cells.

80 loads >500 nM, recovery showed an additional plateau phase that was abolished i nm-chlorophenylhydraz

81 oads > 500 nM, recovery showed an additional plateau phase that was abolished in m-chlorophenylhydraz

82 emoved during the stable, CCh-induced Ca(2+) plateau phase, the decline of cytosolic Ca(2+) is much f

83 ivity changed little as cells moved from the plateau phase to exponential growth, suggesting that in

84 idine-resistant Ca(2+) channels, whereas the plateau phase was attributed to Ca(2+) release from inos

85 n, rapid Ca2+ fluxing was enhanced while the plateau phase was diminished in lupus T cells.

86 by ATP and/or thapsigargin was lower but the plateau phase was similar in the PMCA1a expressing cells

87 ase (median duration of the installation and plateau phases was 6 [IQR 4-9] and 4 days [3-10], respec

88 [Ca(2+)](i) levels attained during the plateau phase were similar to or higher than those requi

89 Two studies noted a plateau phase, where increases in years of experience we

90 f an initial peak followed by a steady state plateau phase which was observed for stimulation duratio

91 ng upstroke phase that initiated a sustained plateau phase, which was associated with Ca(2+) spikes i

92 burst phase and 1.0019 +/- 0.0007 during the plateau phase, while those for the hydroxylamine leaving