ライフサイエンスコーパス: plateau potential

1 ially inactivated during a slow up-ramp to a plateau potential.

2 y probenecid promoted the sADP to generate a plateau potential.

3 cation conductance ((CaN)) in generating the plateau potential.

4 ated depolarizing pulses summed to promote a plateau potential.

5 ation current (ICAN) capable of generating a plateau potential.

6 persists after the collapse of the dendritic plateau potential.

7 ight temporal correlation with the dendritic plateau potential.

8 time-locked with the regenerative dendritic plateau potential.

9 R), and no neurons were found that displayed plateau potentials.

10 aptic inputs for the generation of dendritic plateau potentials.

11 ursting, mixed mode oscillations, and pseudo-plateau potentials.

12 (CaN) by flufenamate abolished both sADP and plateau potentials.

13 urst firing to boost NMDA currents and allow plateau potentials.

14 by OT stimulation lead to the activation of plateau potentials.

15 ing, high-amplitude depolarizations known as plateau potentials.

16 uration, and spatial compartmentalization of plateau potentials.

17 Plateau potentials after the antidromic spikes or local

18 l glutamatergic network, which together with plateau potentials, allow amplification of hyperdirect c

19 ure, WT CA1 neurons developed a spike-evoked plateau potential and an increased spike-evoked dendriti

20 t I(to1) plays a crucial role in setting the plateau potential and overall APD, supporting a causativ

21 cally altered, with a significantly elevated plateau potential and prolonged AP duration.

22 63 is required for B50 to elicit the B31/B32 plateau potential and the motor program.

23 rofoundly reducing extra spikes, eliminating plateau potentials and allowing temporally stationary, s

24 ium channel mediated regulation of dendritic plateau potentials and dendritic excitability underlies

25 cantly prolonged the duration of evoked Ca2+ plateau potentials and increased the whole-cell I(Ca) in

26 Additionally, we show that generation of plateau potentials and LTP induction in dorsal CA1 neuro

27 Conversely, plateau potentials and self-sustained discharges are ver

28 ated by 5-HT and NA synapses and have robust plateau potentials and self-sustained discharges.

29 uted distant to the channels responsible for plateau potentials and self-sustained discharges.

30 inal motoneurons is essential for generating plateau potentials and self-sustained discharges.

31 ting conductance and threshold for eliciting plateau potentials and thus increased the occurrences of

32 arized voltage range of the action potential plateau potential, and prolonged action potential durati

33 iable, contained long-duration supratheshold plateau potentials, and high spike probability, suggesti

34 These data suggest that nigral plateau potentials are mediated by a calcium-activated n

35 switched from phasic to continuous firing as plateau potentials became non-inactivating.

36 The soma was driven into a plateau potential by each of these inputs, during which

37 4-aminopyridine, increased the amplitude of plateau potentials by allowing them to recruit neighbori

38 , calcium transients outlast local dendritic plateau potentials by severalfold.

39 Plateau potentials can be elicited in nigral GABAergic n

40 g of glutamate and GABA, we demonstrate that plateau potentials can broaden the spatiotemporal window

41 ut triggered in many cases a long excitatory plateau potential capable of triggering repetitive actio

42 These plateau potentials coexist in single subthalamic neurons

43 The incidence of plateau potentials decreased to 39% of neurons by P10-P1

44 ately mirrors the glutamate-evoked dendritic plateau potential (dendritic UP state).

45 This subtle inhibitory control of plateau potential depends on the location and kinetics o

46 c of nigral dopamine cells, are converted to plateau potentials following application of apamin, a po

47 ifedipine, in a concentration known to block plateau potential generation, also affects bursting acti

48 e antagonist of L-type Ca2+ channels, blocks plateau potential generation; however, its effects on fi

49 muscarinic agonist oxotremorine restores the plateau potential in B31/B32 and eliminates the necessit

50 Together, our analysis shows that the plateau potential in B31/B32 is not endogenous but condi

51 e blocks the B50-elicited motor program, the plateau potential in B31/B32, and, notably, a slow compo

52 ensity of I(to1) progressively depressed the plateau potential in Kv4.3-infected guinea pig myocytes

53 intrinsic membrane conductance mediating the plateau potential in lateral septal neurons, possibly as

54 Epileptiform bursts with an underlying plateau potential in neurons are a cellular correlate of

55 The I(CAN)-mediated plateau potential in nigral GABAergic neurons likely aff

56 se in input efficacy, all induced by a large plateau potential in the distal dendrites of CA1 pyramid

57 o reduced the duration and amplitude of Ca2+ plateau potentials in both saline- and cocaine-withdrawn

58 pathways results in the generation of large plateau potentials in distal dendrites of CA1 pyramidal

59 olarization, persistent firing, or prolonged plateau potentials in interneurons and evokes sustained

60 gh activity-dependent feedback inhibition of plateau potentials in magnocellular neurosecretory cells

61 tes phasic bursts by autocrine inhibition of plateau potentials in magnocellular neurosecretory cells

62 interacts with both the graded expression of plateau potentials in motor neurons to generate spasms,

63 molecular layer that generate long-duration plateau potentials in response to excitatory synaptic in

64 ventral CA1 dendrites, however, can generate plateau potentials in response to temporally dispersed e

65 nd nickel all abolished both stimulus-evoked plateau potentials in SGCs and synaptic barrages in down

66 inputs at the distal dendrites could trigger plateau potentials in SPNs.

67 ged glutamate triggered abnormally prolonged plateau potentials in the deafferented neurons when stro

68 Here, we report silent plateau potentials in these cells.

69 However, how plateau potentials interact with subsequent excitatory a

70 pecial class of regenerative events known as plateau potentials introduces the possibility of digital

71 Emerging evidence suggests that the plateau potential is mediated by neuronal canonical tran

72 ce associated with preceding activation of a plateau potential is referred to as 'warm up'.

73 Furthermore, activation of a plateau potential is thought to manifest itself as a dec

74 This phenomenon, referred to as a plateau potential, is due to the activation of monoamine

75 ere we report the presence of a postsynaptic plateau potential mediated by L-type Ca2+ channels using

76 kers nimodipine and nifedipine abolished the plateau potential observed under control conditions but

77 644 or activation of NMDA receptors enhanced plateau potentials observed under control conditions and

78 Plateau potentials observed under control conditions as

79 optically driven spike trains can result in plateau potentials of 10 mV or more, causing incidental

80 ence of its frequency and by the presence of plateau potentials on the falling phase of low threshold

81 Prolongation of plateau potentials or block of Kv4.2 channels at branch

82 plifying synaptic currents without incurring plateau potentials or self-sustained discharges.

83 These dendritic plateau potentials produce widespread Ca2+ influx, large

84 ld presynaptic activity and calcium (Ca(2+)) plateau potentials produced a large potentiation with an

85 iated by a fraction of interneurons in which plateau potentials produced large and widespread calcium

86 Accordingly, activation of a plateau potential should result in a decrease in the exc

87 s and in particular, the ability to generate plateau potentials, similar to that of STN neurons witho

88 y distinct in its preferential access to the plateau potential that represents the committed step tow

89 Here we report that the large depolarizing plateau potential that underlies the epileptiform burst

90 ger spike trains, summing IPSPs decayed to a plateau potential that was relatively independent of fir

91 rs voltage-dependent Ca(2+) channel-mediated plateau potentials that are confined to the stimulated d

92 ursts, action potentials are superimposed on plateau potentials that are generated by summation of de

93 neurons, correlated inputs trigger dendritic plateau potentials that drive neuronal plasticity and fi

94 Stronger electrical stimulation evoked small plateau potentials that had significant longer-lasting l

95 suggest astrocytes as a source of dendritic plateau potentials that have been implicated in localize

96 al calcium signals are produced by dendritic plateau potentials that require both vibrissal sensory i

97 reveal characteristic electrical transients (plateau potentials) that trigger and shape dendritic cal

98 itude of spontaneously occurring depolarized plateau potentials (up events).

99 The plateau potential was abolished in calcium-free buffer,

100 f VGKCs in regulating the generation of Ca2+ plateau potential was also studied in mPFC neurons.

101 These results suggest either that plateau potentials were not engaged (or were rapidly ext

102 For phrenic motoneurones, no plateau potentials were observed in either state (except

103 In expiratory motoneurones, plateau potentials were observed in the decerebrates, bu

104 In approximately one-third of these neurons, plateau potentials were observed under control condition

105 We found that dendritic plateau potentials were produced by an interaction betwe

106 Plateau potentials were readily evoked in non-phrenic ce

107 ls thus underlie repolarization of dendritic plateau potentials, whereas Kv4.2 channels confine these

108 stunned myocytes demonstrated a decrease in plateau potential without a change in resting membrane p

109 acellular Ca(2+) abolished both sADP and the plateau potential without affecting Ca(2+) spikes.

110 nd BAPTA dialysis, increased the duration of plateau potentials without affecting their amplitude or