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1 were independently associated with elevated plateau pressure.
2 each independently associated with elevated plateau pressure.
3 lness may decrease the incidence of elevated plateau pressure.
4 ressure was related to barotrauma, including plateau pressure.
5 e reached after 10 s of decay was termed the plateau pressure.
6 H2O in supine obese patients; p < 0.001) and plateau pressure (15.6 [14-17] vs 22 [18-24] cm H2O in s
7 al volume (1A) and limitation of inspiratory plateau pressure (1B) for acute respiratory distress syn
8 t all the obese patients, without increasing plateau pressure (24 [19-25] vs 22 [18-24] cm H2O at zer
9 /- 1.1 vs. 9.8 +/- 1.5 mL/kg; p < .0001) and plateau pressure (27.5 +/- 6.4 vs. 33.8 +/- 8.9 cm H2O;
10 e eucapnic group showed significantly higher plateau pressures (27.0 +/- 2.5 versus 20.9 +/- 3.0; p =
11 (76 +/- 7 to 53 +/- 6 cm H2O; p<.001) and in plateau pressure (28 +/- 2 to 18 +/- 3 cm H2O; p<.001),
12 6) were mechanically ventilated to the same plateau pressure (30-32 cm H2O) with high-strain (VT = 1
13 .3 mL/kg, respectively (p < .001), with mean plateau pressure = 30.6 and 24.9 cm H2O (3.3 kPa), respe
14 positive end-expiratory pressure, 10 cm H2O; plateau pressure, 30 cm H2O) while receiving intravenous
17 low auto-PEEP and a large difference between plateau pressure and auto-PEEP was only seen after expir
18 verity of lung injury, this group had higher plateau pressure and more excessive spontaneous breathin
19 nd either driving pressure, tidal volume, or plateau pressure and positive end-expiratory pressure, V
20 ressure, which is the difference between the plateau pressure and the level of positive end-expirator
22 01), even in patients receiving "protective" plateau pressures and VT (relative risk, 1.36; 95% CI, 1
24 al volume, positive end-expiratory pressure, plateau pressure, and driving pressure evaluated at 24 h
25 ume, respiratory rate, mean airway pressure, plateau pressure, and hemodynamic variables were recorde
27 based on patient's age, PaO2/FIO2 ratio, and plateau pressure at 24 hours after acute respiratory dis
28 Assessment of respiratory mechanics included plateau pressure, auto-positive end-expiratory pressure,
29 (6 versus 12 ml/kg), baseline PEEP, baseline plateau pressure, baseline tidal volume, Acute Physiolog
30 ad lower Pa(o(2))/Fi(o(2)) ratio, had higher plateau pressure, but also had a lower ICU mortality rat
31 d on the values of age, PaO2/FIO2 ratio, and plateau pressure calculated at 24 hours on protective ve
34 cable pulmonary mechanical concepts, such as plateau pressures, driving pressure, transpulmonary pres
35 tory pressure levels and significantly lower plateau pressures during extracorporeal membrane oxygena
36 (2))/FI(O(2)), and decreased end-inspiratory plateau pressure from 16.6 +/- 1.0 to 11.9 +/- 0.5 cm H(
37 on from 7.8 +/- 1.5 to 5.2 +/- 1.1 L/min and plateau pressure from 25 +/- 4 to 21 +/- 3 cm H2O and ra
38 racorporeal membrane oxygenation initiation, plateau pressure greater than 30 cm H2O before extracorp
39 % of patients with FIO2 greater than 40% and plateau pressure greater than 30 cm H2O received low tid
40 ients with plateau pressure less than 30 and plateau pressure greater than or equal to 30 with those
41 tic regression model for predicting elevated plateau pressure had an area under the receiving operato
43 entilation with lower tidal volume and lower plateau pressure improves mortality in patients with acu
44 ought to determine the incidence of elevated plateau pressure in acute lung injury /acute respiratory
47 entilation and limitation of end-inspiratory plateau pressure is important in the management of ARDS
48 rotocol recommends limiting tidal volume and plateau pressure; it also recommends increasing respirat
51 predicted body weight and an end-inspiratory plateau-pressure limit of 30 cm of water, clinical outco
53 ody weight) and lower inspiratory pressures (plateau pressure < 30 cm H2O) (moderate confidence in ef
54 volumes > or =10 mL/kg body weight; 2) keep plateau pressure < or =30 cm H2O, arterial pH at 7.30 to
55 sitive end-expiratory and/or end-inspiratory plateau pressure may be appropriate for one patient but
56 raprotective ventilation strategy minimizing plateau pressure may be required to improve outcome.
59 ight (based on patient gender and height), a plateau pressure of >30 cm H2O, and a peak airway pressu
62 Individual sighs (2 x 10 s at inspiratory plateau pressure of 30 cm H2O) largely restored normal a
63 e end-expiratory pressure was increased to a plateau pressure of 30 cm H2O, and end-expiratory occlus
64 ited to less than 5-7 cc/kg per breath and a plateau pressure of 30 cm of water or less provides the
67 15 cm H2O [interquartile range, 13-15]), the plateau pressure - positive end-expiratory pressure diff
68 nformation on airway resistance (Raw), final plateau pressure (Pp), and peripheral lung compliance (C
69 usted to maintain a constant end-inspiratory plateau pressure (Pplat) of about 25 cm H2O in both grou
70 riving pressure greater than or equal to 19, plateau pressure provided a slightly better prediction o
71 based on mechanical parameters, such as the plateau pressure, respiratory system compliance, or tran
72 al volume (1B) and limitation of inspiratory plateau pressure strategy (1C) for acute lung injury (AL
73 rotocol, noncompliance with tidal volume and plateau pressure targets was associated with significant
74 lower tidal volume ventilation often have a plateau pressure that exceeds Acute Respiratory Distress
75 etween mechanical ventilation settings (i.e. plateau pressure, tidal volume, and positive end-expirat
76 ysis, increasing values of age, lactate, and plateau pressure under ECMO were associated with death.
78 kg +/- 1.8 mL/kg predicted body weight, mean plateau pressure was 27 cm H2O +/- 6 cm H2O, and mean po
82 cts; b) the differences in tidal volumes and plateau pressures were modest; or c) reduced tidal volum
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