ライフサイエンスコーパス: platelet dysfunction

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1 unction, culminating in thrombocytopenia and platelet dysfunction.

2 NS1 Ab-mediated endothelial cell damage and platelet dysfunction.

3 ions as an important mechanism for inherited platelet dysfunction.

4 ve been associated with thrombocytopenia and platelet dysfunction.

5 n the presence of thrombocytopenia or severe platelet dysfunction.

6 une system, as well as thrombocytopenia with platelet dysfunction.

7 gical postcardiopulmonary bypass bleeding is platelet dysfunction.

8 ing with thrombocytopenia and/or evidence of platelet dysfunction.

9 The existence of animal models of platelet dysfunction and currently used antiplatelet the

10 s, hemodialysis is associated with transient platelet dysfunction and decreased membrane expression o

11 ited in the perioperative setting because of platelet dysfunction and gastrointestinal toxicity.

12 urring TxA(2)R variant to be associated with platelet dysfunction and the first in which loss of rece

13 is characterized by oculocutaneous albinism, platelet dysfunction, and in some patients, pulmonary fi

14 s associated with familial thrombocytopenia, platelet dysfunction, and predisposition to acute leukem

15 bg mutation is characterized by NK cell and platelet dysfunction, and systemic treatment of WT mice

16 , MLC phosphorylation, thrombocytopenia, and platelet dysfunction associated with RUNX1 mutations.

17 tic peptide, in the mouse model of inherited platelet dysfunction because of mutation of the myosin 9

18 N substitution causes clinically significant platelet dysfunction by reducing ligand binding establis

19 tion in Glanzmann's thrombasthenia patients, platelet dysfunction can be a result of genetic variabil

20 proposed mechanisms that induce TIC, such as platelet dysfunction, endogenous anticoagulation, endoth

21 understanding of TIC, including the role of platelet dysfunction, endothelial activation, and fibrin

22 GRP2 that account for bleeding diathesis and platelet dysfunction in 2 unrelated families.

23 ilar to that in Glanzmann thrombasthenia and platelet dysfunction in addition to impaired leukocyte a

24 Platelet dysfunction in CF could contribute to pulmonary

25 Platelet dysfunction in DKO mice is revealed by prolonge

26 However, the impact of insulin therapy on platelet dysfunction in patients treated with P2Y12 anta

27 cular explanation for the universally severe platelet dysfunction in this disease, and the cumulative

28 Platelet dysfunction is a common cause of bleeding after

29 Platelet dysfunction is a major cause of excessive micro

30 In this study, we uncovered evidence that platelet dysfunction is a third possible mechanism for b

31 Platelet dysfunction is common.

32 present study indicate that diabetes-induced platelet dysfunction is mediated largely by calpain acti

33 Whereas platelet dysfunction is severe and consistent, immune de

34 by virus-induced IFN-alpha/beta that causes platelet dysfunction, mucocutaneous blood loss and suppr

35 However, the molecular mechanism for platelet dysfunction observed in patients with diabetes

36 ide generation may play an important role in platelet dysfunction observed in patients with diabetes.

37 ), carriage of haemophilia-A gene (one), and platelet dysfunction (one).

38 ndrome is an autosomal recessive disorder of platelet dysfunction presenting with mild thrombocytopen

39 disorder characterized by thrombocytopenia, platelet dysfunction, splenomegaly, reticulocytosis, and

40 Although the transient platelet dysfunction that occurs after platelet contact

41 Platelet dysfunction was apparent after completion of th

42 Platelet dysfunction was independent of glycemic control

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