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1 holine residues on pneumococcal cell wall to platelet-activating factor receptor.
2 lin receptor, or cell wall phosphorylcholine/platelet-activating factor receptor.
3 signaling can be augmented via the epidermal platelet-activating factor receptor.
4 or-negative epithelial cell line KB with the platelet-activating factor receptor.
5 nthesized platelet-activating factor and the platelet-activating factor receptor agonist 1-palmitoyl-
6 oupled receptors and excluded key candidates platelet-activating factor receptor and CCR5.
7 experiments revealed that L5 signals through platelet-activating factor receptor and lectin-like oxid
8 s demonstrate the differential expression of platelet-activating factor receptor and TLR-4 in uterine
9 B versus 1 microM in KB cells expressing the platelet-activating factor receptors) and increased the
10 d receptor-1, protease activated receptor-3, platelet activating factor receptor, and factor V.
11 ine, an H1R antagonist, blocked EAE, and the platelet activating factor receptor antagonist CV6209 re
12 ventricular injection with the intracellular platelet-activating factor receptor antagonist BN 50730
13 rom the international phase III trial of the platelet-activating factor receptor-antagonist Lexipafan
14 nti-eotaxin and anti-RANTES mAbs, but not by platelet-activating factor receptor antagonists (CV6209,
15 ted by pretreatment with the two competitive platelet-activating factor receptor antagonists CV-6209
16 s NK(2), calcitonin gene-related peptide, or platelet-activating factor receptor antagonists had no e
17             Pretreatment of HaCaT cells with platelet-activating factor receptor antagonists, or over
18                                              Platelet-activating factor receptor blockade protected s
19 tion was assessed, and whether the epidermal platelet-activating factor receptor could augment toxin-
20 f oxidative stress-induced signaling seen in platelet-activating factor receptor-expressing cells was
21 ctive effect resulted in part from decreased platelet-activating factor receptor expression on endoth
22 antibody or therapeutic agents that modulate platelet-activating factor receptor expression, may conf
23 D synthase, histamine receptor type 1 (H1R), platelet activating factor receptor, Ig Fc epsilon recep
24 stry studies demonstrate the presence of the platelet-activating factor receptor in both endometrial
25                            Expression of the platelet-activating factor receptor in KB cells lowered
26 studies suggest involvement of the epidermal platelet-activating factor receptors in oxidant-mediated
27 milar involvement for an unrelated GPCR (the platelet-activating factor receptor), indicating that it
28 nally, retroviral-mediated expression of the platelet-activating factor receptor into the platelet-ac
29 let-activating factor and express functional platelet-activating factor receptors linked to calcium m
30 platelet-activating factor receptor into the platelet-activating factor receptor-negative epithelial
31 d by retroviral-mediated transduction of the platelet-activating factor receptor-negative epithelial
32 creased adherence mediated by binding to the platelet activating factor receptor on epithelial cells.
33                 Anaphylaxis was inhibited by platelet-activating factor receptor or histamine recepto
34 eir ability to generate oxidized lipids with platelet-activating factor receptor (PAF-R) agonist acti
35 by generating oxidized lipid agonists of the platelet-activating factor receptor (PAF-R).
36 regation was blocked using a monoclonal anti-platelet activating factor receptor (PafR) antibody and
37 er of melanoma metastasis, via activation of platelet-activating factor receptor (PAFR) and cAMP-resp
38 dditional recognition system mediated by the platelet-activating factor receptor (PAFr) and directed
39 e hypothesis that influenza up-regulates the platelet-activating factor receptor (PAFr) and thereby p
40                                Inhibitors of platelet-activating factor receptor (PAFR) blocked LTA-
41            We have previously shown that the Platelet-Activating Factor Receptor (PAFR) engagement in
42                                          The platelet-activating factor receptor (PAFr) has been sugg
43 hibitors identified a prominent role for the platelet-activating factor receptor (PAFr) in hypoxia-as
44                                              Platelet-activating factor receptor (PAFR) is a G protei
45  Chinese hamster ovary cells, the M1AChR and platelet-activating factor receptor (PAFR) mediate MAPK
46                                              Platelet-activating factor receptor (PAFr) mediates pneu
47 f synaptic activity in a manner dependent on platelet-activating factor receptor (PAFR) signaling.
48 polymeric immunoglobulin receptor (pIgR) and platelet-activating factor receptor (PAFr) to attach and
49 iciency in interleukin 1alpha (IL-1alpha) or platelet-activating factor receptor (PAFR), an important
50  of A. baumannii binds to A549 cells through platelet-activating factor receptor (PAFR), resulting in
51                      Moreover, expression of platelet-activating factor receptor (PAFR), which is kno
52 nvolved in immune responses include CD36 and platelet-activating factor receptor (PAFR).
53 r airway cells is mediated by host-expressed platelet-activating factor receptor (PAFR).
54 ent studies used model systems consisting of platelet-activating factor receptor-positive and -negati
55 3T cDNA exhibited a pronounced impairment in platelet-activating factor receptor-stimulated inositol
56                 The ability of the epidermal platelet-activating factor receptors to modulate oxidant
57    Bacterial lipoteichoic acid activates the platelet-activating factor receptor, which is G protein-

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