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1 donor's blood samples (both whole blood and platelet-rich plasma).
2 imilar to that observed in vitro using mouse platelet rich plasma.
3 bility to inhibit ADP-induced aggregation in platelet-rich plasma.
4 counts for approximately 20% of total VWF in platelet-rich plasma.
5 DP-induced aggregation of human platelets in platelet-rich plasma.
6 nic acid-induced platelet aggregation in rat platelet-rich plasma.
7 ntent of anandamide in either macrophages or platelet-rich plasma.
8 ergizes with soluble collagen in aggregating platelet-rich plasma.
10 RPAI-1 inhibits in vitro the ADP-dependent platelet-rich plasma aggregation by collagen (COLL), TRA
12 ibition of aggregation of human platelets in platelet rich plasma, an IC50 of 6.8 nM for the inhibiti
13 orn trypsin inhibitor-treated whole blood or platelet rich plasma and subsequent array scanning via a
14 togenous bone and anorganic bovine bone with platelet-rich plasma and a bioabsorbable collagen membra
15 telet aggregation in response to collagen in platelet-rich plasma and firm adhesion on VWF under arte
18 arkedly enhanced thrombin generation in both platelet-rich plasma and platelet-poor plasma, indicatin
20 ibition of platelet aggregation in the human platelet rich plasma assay with IC(5)(0) values below 50
21 hat fails to inhibit platelet aggregation in platelet-rich plasma, blocked the inhibitory effect of t
23 llograft (ADM) to that of a CPT plus ADM and platelet-rich plasma (CPT/PRP) 4 months post-surgically.
26 groups: demineralized freeze dried bone with platelet-rich plasma (DFDB + PRP), DFDB alone, and no tr
27 se of autologous fibrin membrane and the eye platelet-rich plasma (E-PRP) clot could be considered as
28 ncoated stents were then immersed in porcine platelet-rich plasma for two min and the platelet cyclic
32 ated primary glomerular endothelial cells to platelet-rich plasma from patients, or patient platelet-
33 hibited when compared to the wild-type using platelet-rich plasma from the principal donor, but adhes
36 of blood microparticles (MPs) obtained from platelet-rich plasma of healthy individuals was characte
37 anufacture of whole-blood-derived platelets (platelet-rich plasma or buffy coat intermediate steps) r
38 Shear-induced GPVI shedding also occurred in platelet-rich plasma or washed platelets isolated from a
41 nts or sample volumes can be used, either of platelet-rich plasma or whole blood from human subjects
43 ormed in platelets, platelet microparticles, platelet-rich plasma, platelet-poor plasma, and serum.
45 th PMN and PMN-RBC combinations suspended in platelet rich plasma (PRP) than in platelet poor plasma
46 played inhibition of platelet aggregation in platelet rich plasma (PRP) with an IC(50) value of 53 nM
50 as to evaluate the regenerative influence of platelet-rich plasma (PRP) added to xenogenic bone graft
51 ion of CD62P by wild-type mouse platelets in platelet-rich plasma (PRP) and caused their secretion of
53 lyze the content and specific effect of both platelet-rich plasma (PRP) and platelet-poor plasma (PPP
57 nhanced by 127% (P < 0.001) in study patient platelet-rich plasma (PRP) compared to control PRP and c
60 lcium sulfate hemihydrate (MGCSH) mixed with platelet-rich plasma (PRP) for extraction socket preserv
64 e mineral (NBM) with and without addition of platelet-rich plasma (PRP) has been shown to result in s
65 ch surrounding intra-articular injections of platelet-rich plasma (PRP) has not produced clear eviden
66 hput microfluidic removal of leukocytes from platelet-rich plasma (PRP) in a continuous flow regime.
67 of autologous platelet-rich fibrin (PRF) and platelet-rich plasma (PRP) in the treatment of intrabony
68 al benefits provided by the incorporation of platelet-rich plasma (PRP) into a regenerative protocol
74 of recalcified platelet-free plasma (PFP) or platelet-rich plasma (PRP) supplemented with corn trypsi
75 al intrabony defects, the benefits of adding platelet-rich plasma (PRP) to a bone replacement graftin
76 plored through a spectrophotometric assay on platelet-rich plasma (PRP) treated with the thromboxane
80 aggregation tests (PATs) were performed with platelet-rich plasma (PRP), a shorter lag time was measu
84 tomorphometrically analyzes the influence of platelet-rich plasma (PRP), low-level laser therapy (LLL
85 ractionated to yield red blood cells (RBCs), platelet-rich plasma (PRP), platelet-poor plasma (PPP),
88 ng of recombinant human TPO (rhTPO) to human platelet-rich plasma (PRP), washed platelets (WP), and c
93 specifically in VAT, in addition to elevated platelet-rich-plasma (PRP) NPY, compared to control fema
94 12-hydroxyeicosatetraenoic acid to P-12LO-/- platelet-rich plasma rescues the hyperresponsive phenoty
95 Platelet aggregation assays with citrated platelet-rich plasma reveal that the primary and seconda
96 d at baseline in increasing proportions with platelet-rich plasma sampled 4 hours after loading dose.
98 ing, hydrogel-coated PVC membranes soaked in platelet-rich plasma showed less adhesion and activation
100 ctor Xa-inhibited (250 nM apixaban), diluted platelet rich plasma that had been loaded with the calci
101 ld be prevented by simply reducing the pH of platelet-rich plasma to about 6.5 prior to centrifugatio
102 Using gel-filtered platelets derived from platelet-rich plasma treated with alpha-tocopherol (500
103 r, second-wave aggregation induced by MDC in platelet-rich plasma was inhibited by aspirin, ADP scave
106 the coagulation cascade in platelet poor or platelet-rich plasma, when activation was initiated by t
108 roparticles in megakaryocyte cultures and in platelet-rich plasma, which are predominantly derived fr
109 atelets derived from apheresis compared with platelet-rich plasma whole-blood-derived platelets.
110 ma cofactor, because a 35-hour incubation of platelet-rich plasma with 125I-factor V showed no specif
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